Physiology of Intermittent Sports Flashcards

Not actually needed for exam

1
Q

What tools can be used to assess demands during performance?

A

GPS Monitoring

Prozone (cameras)

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2
Q

What physical demands should be considered during performance?

A
  • Implications of pacing strategies due to ever-changing gameplay
  • Many high-intensity running and sprinting in top players, therefore this declines fast
  • Demands are also position specific
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3
Q

Football specific crap that was on the board

A
  • 10-12km covered during a match
  • fatigue develops in all players
  • Longer high-intensity periods of running make it more likely to win
  • tactics can be developed specific to players
  • different tactics, different demands (4-5-1 vs 4-4-2)
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4
Q

How can metabolic demands be measured during gameplay?

A

Masks obvs cant be used so use Polar HR monitoring, which can be integrated with GPS monitoring

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5
Q

What is the correlation between ATP resythesis and capacity?

A

From PCr to Aerobic, rate is highest in PCr but capacity is highest in aerobic

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6
Q

How can VO2 be monitored during gameplay?

A

During an incremental TTE, record HR during test to estimate VO2 on the field!

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7
Q

What happens to BLa during play?

A

increaes during matchplay (4010 mMoL), indicating reliance on anaerobic glycolysis

Lactate can be oxidised into substrate, allowing it to continue for longer, however this is at a slow rate.

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8
Q

What happens to PCr during high-intensity exercise?

A

Declines and is then resynthesised during recovery
PCr recovery significant is linked to muscle O2 recovery during exercise
Therefore aerobic is important during recovery

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9
Q

What happens during Purine Nucleotide Metabolism (ATP degradation)?

A

ATP is broken down into 2 phosphates to create energy
myokinis attach these together
Ammonia is produced within bloodstream

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10
Q

What happens to Ammonia during a match?

A

Venous blood ammonia production increases for the first half, lowers slightly during half time and then increases again during second half
As ammonia can cross the blood-brain barrier, this is a cause on central fatigue

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11
Q

How can fatigue development be measured?

A

Through GPS monitoring or repeated (30m) sprints

MVC - time taken to strap in etc can make less accurate

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12
Q

What are the sites of fatigue?

A

Central - CNS (brain, spinal cord, motor neurons

Peripheral - peripheral nerves (alpha motor neurons), neuromuscular junctions, muscle fibres

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13
Q

How to measure global and peripheral fatigue?

A

Global - MVC

Peripheral - potentiated twitch

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14
Q

What are the main metabolic changes during high-intensity intermittent exercise?

A
  • Increased oxidative energy metabolism

- Lower aerobic glycolysis

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15
Q

What is the fundamental mechanism of excitation contraction coupling?

A

Depolarization (excitation) of muscle fibre

  • nerve impulse travels down T-tubules, releasing calcium from SR
  • Calcium binds to troponin, exposing ctive sites on actin
  • permits strong binding state between actin and myosin, causing contraction
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16
Q

What are the consequences of increased plasma K+?

A
  • Increased K+ in interstitial fluid surrounding cells
  • Membrane becomes depolarized (effects on action potential)
  • Action potential amplitude is induced
  • inactivation of voltage-gated Na+ channels
17
Q

What are the consequences of Ca2_ release from SR?

A
  • Impaired (slower) Ca2+ reuptake following exercise
  • impaired muscle relaxation
  • release to cause contraction but must be reuptaken by SR or stiffness occurs

consequence - impaired muscle contraction

18
Q

What are the benefits of greater fat oxidation?

A
  • Greater ATP production than glycogen
  • Spares glycogen stores
  • Massive reserves
19
Q

What are the implications of anaerobic production?

A
  • Muscle glycogen and PCr reserves are limited, advantageous to preserve
  • Accumulation of Pi, ADP and H+ in muscles is linked to muscular fatigue
  • Oxidative metabolism is key in recovery
20
Q

Na/K+ pumps can be improved with training, what are the implications of this?

A

Increase K+ in cells may limit K+ in interstitial fluid which might

  • delay progressive membrane depolarization
  • delay lower action potential amplitude
  • delay fatigue
21
Q

What are the benefits of fast twitch fibres during intermittent exercise type 2a

A
  • faster nerve conduction
  • faster muscle contraction
  • greater PCr and glycogen stores (anaerobic energy substrates)
  • more anaerobic enzymes
  • greater SR and Ca2+ stores, faster calcium release
  • larger cross-sectional area - more myofibrils
  • more forceful contractions