Physiological Changes and Adaptation in the Older Adult Flashcards
Muscle strength
Peaks at 30
Constant loss after 50 (20-40% by age 65 in the nonexercising adult)
Changes in muscle fiber composition
loss of type II fast twitch and increase in proportion of type I fibers
Collagen changes
Denser, irregular due to cross linkages, loss of water content and elasticity - affects tendons, bone, cartilage
CT becomes
denser and stiffer
leads to inc risk of sprains, strains, tendon tears
Loss ROM
inc risk of adhesions and contractures
Gait changes
Dec amp and speed, slower cadence
Shorter steps, wider stride, inc DS, dec trunk rotation and arm swing
Cartilage changes
Dec water content
becomes stiffer, fragments, erodes
by age 60 more than 60% of adults have degenerative changes and cartilage abnormalities
Bone mass
Peak bone mass at 40
Dec between 45 and 70
Loss of Ca, dec bone marrow RBC production
IVD changes
flatten, less resilient due to loss of water content (30% loss by age 65) and loss of collagen elasticity, trunk length and overall height dec
Postural changes
Forward head
Kyphosis of thoracic
Flattening of lumbar
Hip and knee flexion contractures with prolonged sitting
Clinical implications - Skeletal changes
Maintenance of WB is important
Nerve cells
Atrophy of them in cerebral cortex
overall loss of brain weight 6-11% between ages of 20 and 90
Accelerating loss after age 70
Brain morphology
Gyral atrophy
Ventricular dilation
Loss in cortex (esp frontal and temporal)
Presence of lipfuscins, senile, or neuritis plaques and neurofibrillary tangles (NFT)
More selective loss in BG
Changes in SC and peripheral nerves
Neuronal loss and atrophy - 30-50% loss of anterior horn cells
30% loss of posterior roots/sensory fibers by age 90
Vision changes
Rapid decline between ages 60 and 90
visual loss high as 80% by age 90
Presbyopia
visual loss in middle and older ages characterized by inability to focus properly and blurred images due to loss of accommodation, elasticity of lens
Cataracts
Opacity, clouding of lens due to changes in lens proteins
results in gradual loss of vision - central first, then peripheral