Phase 4 2015 Flashcards

1
Q

Define compartment syndrome

A

Life and limb threatening event
Occurs when swelling of the tissues in an anatomic compartement occludes the vascular supply leading to hypoxia and eventually necrosis
Susequent rhabdomyolysis can cause renal failure

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2
Q

Treatment of compartment syndrome

A

Urgent fasciotomy

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3
Q

4 treatments for the management of a fracture

A

Resuscitation
Reduction
Restriction
Rehabilitation

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4
Q

Features of a viral conjunctivits

A
Serous discharge 
Lack of itch 
Red eyes 
Gritty eyes 
Morning crusting 
Hx of an URTI
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5
Q

Signs of viral conjunctivitis

A

Preauricular lymph nodes
Recurrence of conjunctivitis
Bilateral eyes involved
Pinpoint conjunctival haemorrhages

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6
Q

Mode of inheritance of Polycystic kidney disease and name the chromosomes

A

Autosommal dominant
PKD1 Chrom 16
PKD2 Chrom 4

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7
Q

Signs observed on examination in appendicitis

A

Rebound tenderness
Guarding
Rovsing’s sign: pain is > in the RIF than the LIF when the LIF is pressed
Psoas sign: pain on extending the hip if retrocaecal appenditis

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8
Q

What is a cohort study

A

Population without the disease is followed over time

Measures incidence

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9
Q

Define attributable risk

A

The rate of disease in the exposed that may be attributed to the exposure, i.e. incidence in exposed minus incidence in unexposed.

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10
Q

Relative risk

A

Ratio of risk of disease in the exposed to the risk in the unexposed, i.e. incidence in exposed divided by incidence in unexposed.
Measures strength of association

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11
Q

Define confounding

A

A situation in which the estimate between an exposure and an outcome is distorted because of the association of the exposure with another factor (confounder) that is also independently associated with the outcome.

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12
Q

Name the blood test you would do for a dementia screen

A
FBC
B12/Folate 
ESR
U&E
LFT
Ca2+
TFTs
Serology for syphilis
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13
Q

Drugs used in dementia

A

Anti-cholinesterase inhibitors : rivastigmine

NMDA receptor antagonist: memantine

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14
Q

Signs of hypovolaemic shock

A
Cool/Clammy 
CRT>2 secs
Low BP
Reduced urine output 
Tachycardia
Reduced GCS
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15
Q

Discuss the mechanism behind a mallory weiss tear

A

Excessive vomiting increases intraabdominal pressure
Tear in the mucosal layer of the oesophagus
Reaches the oesphageal venous or arterial plexus

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16
Q

Most common causing organism of CAP

A

Strep pneumonia
Mycoplasma pneumonia
Haemophilus influenza
Moraxella

OTHERS
Chlamydia
Legionella
Staph aureus

17
Q

Outline the management of a renal laceration

A

Grade 1-4
Can be managed conservatively with strict bed rest until gross haematuria has resolved
Intervention is required if
- Persistent bleeding (ie, enough to necessitate repeated transfusions)
- Expanding perinephric hematoma
- Renal pedicle avulsion or other significant renovascular injuries

18
Q

What are 4 pathological mechanisms of ascites

A

Hypoalbuminia
Portal hypertension
Reduce oncotic pressure

19
Q

Why perform an asictic tap

A

New-onset ascites: To determine aetiology, To differentiate transudate versus exudate, To detect cancerous cells.
Suspected spontaneous or secondary bacterial peritonitis.

20
Q

Describe the formation of oesophageal varices

A

Progressive liver fibrosis + regeneration of the contractile elements in the liver vascular bed
Portal HTN
Splanchnic vasodilation
Increased cardiac output
Salt and water retention
Hyperdynamic circulation
Formation of collateral between lower oesophagus and carida of the stomach

21
Q

Reduce the risk of bleeding in oesophageal varies

A

Non selective beta blocker: Propanalol