Periodontitis Flashcards
Types of periodontitis
A). Chronic Periodontitis
B). Aggressive periodontitis
C). Periodontitis as a manifestations of
D). Necrotizing periodontal disease
E). Abscesses of periodontium
F).Periodontal lesions associated with endodontic lesions
G).Developmental or acquired deformities or conditions
Chronic periodontitis
generalized (>30%) localized (30%)
- Porphyromonas gingivalis
- Modified by systemic diseases e.g. diabetes mellitus and HIV infection
- Cigarette smoking and emotional stress
- Subgingival calculus
Severity of disease can be :
i). Slight : CAL of 1-2mm
Ii).Moderate : CAL OF 3-4mm
Iii). Severe: CAL of 5mm or more
Aggressive periodontitis
Actinobacillus Actinomycetemcomitans
Rapid attachment loss, rapid bone destruction in a clinically healthy individual
Familial aggregation
Phagocyte abnormalities and hyper-responsive monocyte/macrophage phenotype
Increased prostaglandin and interleukins
Localized form: Involves first molar or incisor (usually maxillary teeth) with proximal attachment loss (at least two permanent teeth, one of which is a first molar); onset around puberty
Generalized form: Pt is usually under 30 years of age; Generalized proximal attachment loss affecting at least three teeth other than first molars and incisors; poor serum antibody response to infecting agents
Periodontitis as a manifestations of systemic disease
- Acquired neutropenia
- Leukocyte adhesion deficiency syndrome
Necrotizing periodontal disease
- Necrotizing Ulcerative Gingivitis (NUG)
Fusobacterium nucleatum; Spirochetes and Pi - Necrotizing Ulcerative Periodontitis (NUP)
Fusobacterium nucleatum; Spirochetes and Pi
Necrotizing Ulcerative Gingivitis (NUG)
Primary predisposing factor is bacterial plaque
Diseases is complicated by presence of secondary factors such as:
Acute psychological stress, smoking and poor nutrition (all contributing to immunosuppression)
Usually seen in young adults or teens
NUG: Clinical features
Fever; lymphadenopathy (submandibular and cervical lymph nodes); malaise
- Punched out, crater like depression at the crest of the interdental papillae
- Involves marginal gingiva and attached gingiva
- Craters are covered by greyish pseudomembranous slough (yellowish white/grayish)
-Linear erythema demarcates normal and diseased gingiva
Pseudopockets may be present, Gingival haemorrhage, Excessive salivation, Halitosis present, Painful for patient
If not treated, may lead to NOMA
Necrotizing Ulcerative Periodontitis (NUP)
Affects deeper tissues of the periodontium, resulting in loss of connective tissue attachment and alveolar bone
Seen in systemically healthy patients, often patients that had multiple episodes of NUG, or at a site previously affected with periodontitis
Patients are often immune-compromised through systemic conditions , such as HIV
In these patients the prognosis depends on not only reducing local and secondary factors, but also dealing with systemic problem
Species found : Fusobacterium nucleatum; Spirochetes and Pi
NUP: Clinical features
Same signs and symptoms as NUG
Deep interproximal craters
Denudation and sequestration of interdental alveolar bone
Involvement of buccal and lingual alveolar bone
Periodontal attachment loss (at times with no periodontal pocket formation due to necrosis
Abscesses of periodontium
Gingival, Gingival and Gingival abscess
i). Gingival abscess: Usually due to irritation in sulcus region
Ii). Periodontal abscess: Associated with a pre-existing periodontal pocket, caries or both
Swelling located around the involved tooth
Iii). Pericoronal abscess: Usually seen on erupting wisdom tooth(mostly lower 8’s)