periodontial diseases and systemic health Flashcards

1
Q

modifying factors for periodontal disease

A

diabetes, pregnancy/puberty/menopause, smokeing

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2
Q

what can smoking, diabetes, or pregnancy influence in the periodontium

A

susceptibility to gingivitis and periodontitis, plaque growth and composition, clinical presentation, disease progression, and response to periodontal therapy

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3
Q

how can diabetes affect your mouth?

A

xerostomia, candida, periodontitis, multiple periodontal abscess

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4
Q

t/f. glycemic control is worse after periodontal therapy

A

false. it gets better because periodontitis can increase insulin resistance

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5
Q

oral bacteria involved in type 1 diabetes

A

capnocytophaga

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6
Q

oral bacteria involved in type 2 diabetes

A

p intermedia, c rectus, p gingivalis

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7
Q

diabetes effects on healing and Tx response

A

decreases collagen sysnth by fibroblasts
increased degradation by collagenase
glycosylation of existing collagen and wound margins
defective remodeling and degradation of new collagen

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8
Q

in what trimester is gingival inflammation the highest

A

2nd and 3rd

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9
Q

microbial effects during pregnangy

A

increased p intermedia, increase in spirochetes, napthoquinones from steroids used by p intermedia

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10
Q

host effect during pregnancy

A

increased vascular permeability = increased gingival exudate
decreased keratinization
decreased PMN chemotaxis and phagocytosis, Ab, Tcell response

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11
Q

describe the relationship between menopause, osteoporosis, and periodontitis

A

due to decreased absorption and increased elimination of Ca
osteoporosis and post menopausal pts may not cause periodontal disease, but it may affect the severity of the preexisting disease

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12
Q

daily exposure

A

cigarettes per day

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13
Q

cumulative exposure

A

pack years (# packs smoked per day x # years smoked)

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14
Q

what qualifies a current vs former vs nonsmoker

A

current smoked > cigs in lifetime and currently smoke
former smoked >100 in lifetime and do not currently smoke
nonsmoker have not smoked > 100 cigs

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15
Q

t/f. inflammation in response to plaque accumulation is reduced in smokers compared to nonsmokers

A

true.

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16
Q

smokers are how much more likely to develop periodontitis

A

4x

17
Q

t/f. 42% of perio cases attribute to current smoking

A

true. and 11% are attributable to former smokers

18
Q

smokers are how much more likely to have aggressive perio

A

3.8x

19
Q

t/f. negative effects of smoking on host response are not reversible

A

false. they are

20
Q

what happens to the microbial enviornment in smokers with perio

A

increase in pathogens in shallow perio pockets and increased levels of pathogens in deep pockets

21
Q

smokers will typically have increased levels of what pathogens?

A

t. forsythia, Aa, P ging

22
Q

what happens to the immune-inflammatory response in smokers with perio

A

increased TNF alpha, PgE2, and neutrophil collagenase and elastase in GCF, increased production of PgE2 by monocytes in response to LPS

23
Q

what happens to the oral physiology of smokers with perio

A

decreased blood flow, decreased clinical signs of inflammation, increased recovery time

24
Q

for implant therapy, smoking increases the risk of failure by how many times

A

2x, and it is greater in the max than the mand

25
Q

t/f. peri implant bone loss is increased in non smokers

A

false

26
Q

approximately how many poor treatment responders are smokers

A

90%

27
Q

t/f smoking is associated with tooth loss

A

true. everything bad happens to your periodontal health if youre a smoker

28
Q

t/f. there are 30% more site with probing depth reduction of >2 mm in quitters vs nonquitters

A

true.

29
Q

name 3 benefits of smoking cessations

A

shift toward less pathogenic microbiota, recovery of the gingival micro-circulation, improvements in immune-inflammatory response