periodontial diseases and systemic health Flashcards
modifying factors for periodontal disease
diabetes, pregnancy/puberty/menopause, smokeing
what can smoking, diabetes, or pregnancy influence in the periodontium
susceptibility to gingivitis and periodontitis, plaque growth and composition, clinical presentation, disease progression, and response to periodontal therapy
how can diabetes affect your mouth?
xerostomia, candida, periodontitis, multiple periodontal abscess
t/f. glycemic control is worse after periodontal therapy
false. it gets better because periodontitis can increase insulin resistance
oral bacteria involved in type 1 diabetes
capnocytophaga
oral bacteria involved in type 2 diabetes
p intermedia, c rectus, p gingivalis
diabetes effects on healing and Tx response
decreases collagen sysnth by fibroblasts
increased degradation by collagenase
glycosylation of existing collagen and wound margins
defective remodeling and degradation of new collagen
in what trimester is gingival inflammation the highest
2nd and 3rd
microbial effects during pregnangy
increased p intermedia, increase in spirochetes, napthoquinones from steroids used by p intermedia
host effect during pregnancy
increased vascular permeability = increased gingival exudate
decreased keratinization
decreased PMN chemotaxis and phagocytosis, Ab, Tcell response
describe the relationship between menopause, osteoporosis, and periodontitis
due to decreased absorption and increased elimination of Ca
osteoporosis and post menopausal pts may not cause periodontal disease, but it may affect the severity of the preexisting disease
daily exposure
cigarettes per day
cumulative exposure
pack years (# packs smoked per day x # years smoked)
what qualifies a current vs former vs nonsmoker
current smoked > cigs in lifetime and currently smoke
former smoked >100 in lifetime and do not currently smoke
nonsmoker have not smoked > 100 cigs
t/f. inflammation in response to plaque accumulation is reduced in smokers compared to nonsmokers
true.
smokers are how much more likely to develop periodontitis
4x
t/f. 42% of perio cases attribute to current smoking
true. and 11% are attributable to former smokers
smokers are how much more likely to have aggressive perio
3.8x
t/f. negative effects of smoking on host response are not reversible
false. they are
what happens to the microbial enviornment in smokers with perio
increase in pathogens in shallow perio pockets and increased levels of pathogens in deep pockets
smokers will typically have increased levels of what pathogens?
t. forsythia, Aa, P ging
what happens to the immune-inflammatory response in smokers with perio
increased TNF alpha, PgE2, and neutrophil collagenase and elastase in GCF, increased production of PgE2 by monocytes in response to LPS
what happens to the oral physiology of smokers with perio
decreased blood flow, decreased clinical signs of inflammation, increased recovery time
for implant therapy, smoking increases the risk of failure by how many times
2x, and it is greater in the max than the mand
t/f. peri implant bone loss is increased in non smokers
false
approximately how many poor treatment responders are smokers
90%
t/f smoking is associated with tooth loss
true. everything bad happens to your periodontal health if youre a smoker
t/f. there are 30% more site with probing depth reduction of >2 mm in quitters vs nonquitters
true.
name 3 benefits of smoking cessations
shift toward less pathogenic microbiota, recovery of the gingival micro-circulation, improvements in immune-inflammatory response
