Perio Flashcards

1
Q

extent

A

<30% localised
>30% generalised

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2
Q

stage

A

severity
1 - <2mm or 15% coronal
2 - coronal third
3 - mid third
4 - apical third

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3
Q

grade

A

progression
A - slow - <0.5
B - moderate - 0.5-1.0
C - rapid - >1.0

% bone loss / age

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4
Q

stability

A

stable; BoP <10%, PPD <=4mm

remission; BoP >=10%, PDD <=4mm

unstable; PDD >=5mm or >=4mm + BoP

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5
Q

investigations

A

diet diary
MPBS
6PPC
PA radiographs

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6
Q

further questions for pt

A

pt motivation
OH techniques
smoking
MH

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7
Q

why could non-surgical therapy fail to eliminate bacteria from perio pockets

A
  • deep/inaccessible pockets
  • furcation lesion
  • infra bony defect
  • presence of virulent resistant bacteria
  • poor pt compliance w OH
  • systemic/host factors impairing healing
  • smoking
  • poorly controlled DM
  • medication [phenytoin, ca channel blockers]
  • immunocompromised
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8
Q

problems that limit usefulness of oral abx in the tx of periodontitis

A
  • not bacterial infection but host mediated inflammatory
  • abx resistance
  • limited penetration of sub gingival biofilm
  • disruption of normal microflora
  • side effects
  • need mechanical disruption
    = compliance and OH needed
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9
Q

management of lateral periodontal abscess

A

reassure
incise and drain or through pocket w LA if needed
thorough supra+subgingival debridement, just short of length
occlusal adjustment if needed
analgesia

systemic PenV 2 tabs 4x day 5 days
amoxicillin 500mg tds 5 days

after acute phase - review and assess, continue pd tx

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10
Q

signs of improved health post periodontal abscess

A

reduced pocket depths
resolution of infection
decreased swelling/erythema
improved systemic conditions

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11
Q

bacteria involved in necrotising gingivitis

A

fusiform bacteria
spirochetes

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12
Q

clinical signs and symptoms

A

punched out papillae, ID, ulcers, swelling, pain
pseudomembrane formation, grey/white slough when wiped reveals bleeding underlying connective tissue
tissue destruction
halitosis

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13
Q

necrotising gingivitis risk factors

A

high stress
young
poor OH
smoking
immunocompromised
malnutrition
HIV/AIDs

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14
Q

outline management of necrotising gingivitis

A

reassure pt
superficial debridement w ultrasonic to remove soft and mineral deposits, gentle
increase depth each day 2-4
limit mechanical OH as may impair healing
chx 0.2% 2xday

abx if systemic or failing to resolve
metronidazole 400mg tds 3 days

close follow up, daily if possible
after acute phase - tx of existing condition, address risk factors

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15
Q

info for pt for informed consent

A

nature of procedure
risks vs benefits
alternatives
reason why needed
risks of nothing
costs
aftercare

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16
Q

crushing pain across chest and left arm

  • diagnosis
  • immediate management
A

myocardial infarcation

999
abc
oxygen 15l/min
300mg aspirin to chew
monitor vital signs, prepare for CPR

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17
Q

after open flap curettage

post-op instructions to minimise complications

A

analgesia
avoid brushing directly on area, soft brush, chx
avoid surgical site w tongue/fingers
care with hot as encourage bleeding
avoid spicy, salty, acidic
if bleeding, gentle pressure
ice to swelling
avoid strenuous activity
avoid smoking and alcohol
recognise complications and number to call back

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18
Q

post open flap curettage, you review at one week and all is good

when should you review next and why

A

4-6 weeks
healing at 1-2 weeks but takes weeks for full tissue regeneration and reattachment of PD fibres

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19
Q

clinical signs of improved health following non-surgical therapy

A

<20% plaque, <10% BoP
reduced inflammation, erythema
reduced pocket depths [ideally <=4mm]
epithelium reattachment [decreased probing depths due to long junctional epithelium]

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20
Q

what is SIRS
parameters

A

systemic inflammatory response syndrome

serious, exaggerated defence response form body causing severe inflammation throughout, due to infection/trauma/burns etc

temp <34 >36
pulse >90
resp >20
WCC <4 >12

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21
Q

what is a periodontal abscess

A

abscess from periodontal origin, usually existing pocket
accumulation of pus within gingival walls
destruction of collagen fibre attachment and alveolar bone

cause = disease exacerbation, complex morphology, changes in microbiota, increased bacterial virulence, reduced host response

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22
Q

signs + symptoms of periodontal abscess

A

ovoid elevation in gingiva along lateral root
pain, localised swelling, associated pus
bite may feel high
bleeding, tenderness of probing
increased mobility
TTP
suppuration through root spontaneous or w pressure

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23
Q

differentiate periodontal abscess and periapical abscess

A

sensibility =
PD normal, PA not respond as necrosis

probing =
PD deep pocket, PA normal

radiograph =
PD vertical bone loss, PA radiolucency/widened PDL

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24
Q

management of occlusal trauma in pt w periodontal disease

A

address perio - non-surgical

correct occlusal stability - smooth premature contacts, reduce high cusps, ortho, occlusal adjust to distribute forces more evenly

parafunctional habits - splint, relaxation, CBT etc

splinting - temp stabilise if excessively mobile, or for debridement
doesn’t affect rare of PD destruction, can worsen OH

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25
Q

what factors influence localised mobility

A

periodontal - inflammation, attachment loss, height/width of PDL

teeth - number, shape, length of roots

PD abscess

occlusal - trauma, bruxism, tooth migration

endo - pathology, RR

trauma

iatrogenic - high rests causing excessive forces, ortho forces

medications - bisphosphonates

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26
Q

when may splinting be advised

A

advanced mobility due to loss of attachment - discomfort, difficulty eating
stabilisation for tx
post-trauma

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27
Q

why is there a decrease in mobility post-perio tx

A

reduced inflammation
improved attachment level
reorganisation and reattachment of PDL
improved occlusal stability
bone healing
long junctional epithelium

28
Q

what could you do if PDL is still widened post perio tx
in occlusal trauma pt

A

reassess perio health - plaque, bleeding, probing, occlusal trauma
splinting
management parafunction

29
Q

what bacteria is implicated in periodontitis

A

P.gingivalis
T.denticola
T.forsythia

30
Q

how is angular bone loss caused

A

vertical osseous defect, occurs when bone loss progresses down the root of the tooth
bone destruction occurs at an angle, not uniformly around a tooth
“V” shaped pattern

31
Q

how do you classify angular defects/infrabony

A

based on number of walls remaining
1 wall
2 wall
3 wall

32
Q

what is the limitation of treatment for angular bone loss

A

limited access of non-surgical therapy
root surface debridement may not be successful and effectively access these deep defects

33
Q

alternative tx for angular defects, beside non-surgical

A

periodontal surgery = access surgery/open flap debridement

regenerative periodontal surgery = guided tissue regeneration, enamel matrix derivative

34
Q

what is the difference between horizontal and angular bone loss

A

horizontal =
more generalised, uniform loss of bone height across alveolar ridge, parallel plane

angular =
infrabony, localised bone loss non-uniformly, unevenly, creating a “V” shape

35
Q

define localised and generalised bone loss

A

localised = <30% of teeth
generalised = >30% of teeth

36
Q

define mild, moderate and severe bone loss

A

mild = <15% or 2mm of root
mod = coronal third
severe = mid third
very severe = apical third

37
Q

how does healthy periodontium react to traumatic occlusion

A

PDL widening until forces adequately dissipated
should then stabilise
may cause mobility as a result

RETURN TO NORMAL

38
Q

how does healthy but reduced periodontium react to traumatic occlusion

A

PDL widening until forces adequately dissipated
less resilient
less able to adapt and recover

tooth more vulnerable, increase mobility, further degradation of periodontal health

39
Q

how does periodontitis respond to traumatic occlusion

A

worsening of periodontitis
increased mobility
progressive attachment loss
increased bone loss
exacerbation of inflammation
gingival recession
root resorption

risk tooth loss

40
Q

what is chlorhexidine

A

biguanide gluconate
broad spectrum antimicrobial
topical antiseptic

41
Q

chlorhexidine mode of action

A

membrane disruption =
disrupts cell membrane, binds and causes leakage of cell causing death

bactericidal effect =
bactericidal action against wide range bacteria

inhibition of bacterial adhesion =
prevents bacteria adhering to oral surfaces

42
Q

define substantivity

A

ability of substance to bind to tissues/surfaces and maintain antimicrobial action over time

43
Q

what is chlorhexidine substantivity

A

good
remains active for several hours
up to 12hrs of residual effect

44
Q

give 2 common doses of chlorhexidine

A

0.12% - general oral hygiene, 10-15ml 30secs 2x day

0.2% - antibacterial purpose, tx, 10-15ml 30secs 2x day

45
Q

side effects of chlorhexidine

A

staining
taste alteration
mucosal irritation
xerostomia
sore throat
parotid gland swelling

46
Q

uses of chlorhexidine

A

OH adjunct
post-surgical care [xla, perio surgery]
oral infections - necrotising, RAS
peri-implantitis management
pericoronitis
denture stomatitis
primary herpetic gingivostomatitis
osteonecrosis

47
Q

what is TIPPS

A

behavioural change theory, aim to make pt more confident in ability to perform effective plaque biofilm removal, plan how and when to look after oral

TALK
INSTRUCT
PRACTICE
PLAN
SUPPORT

48
Q

what 7 things are recorded on periodontal pocket chart

A

teeth present/not present
pocket depths
gingival margin
clinical attachment level
furcation
mobility
bleeding

49
Q

disadvantages of pocket charting

A

time consuming
uncomfortable for pt
subjective
based on operator technique
doesn’t give comprehensive view on bone loss
limited predictive value

50
Q

local factors for gingival recession

A

restoration overhangs
poor IP contact points
plaque/biofilm accumulation
toothbrushing trauma
poor OH
traumatic incisor relationship
abrasive TP
frenum
misaligned teeth
ortho tx
piercings

51
Q

how can you measure gingival recession

A

CPITN probe relative to CEJ

cairo classification

52
Q

how is localised recession managed

A

record magnitude clinically/study models to monitor progression
alter habits - TB, TP, single tuft
improve OH
remove plaque + retentive factors
desensitising agents

if bad
gingival veneers
crowns
mucogingival surgery

53
Q

tooth 15 has been root treated, has 9mm pocket and vertical body defect radiographically

give 3 differential diagnosis
what special investigations

A

endo-perio lesion, periodontal abscess, subgingival root fracture

PA radiograph, percussion/palpation test, transillumination, BPE, 6PPC if indicated, sensibility tests

54
Q

pt wants implant
what would you look for?

A

good compliance
good OH
quality of remaining bone
height/width of bone
proximity to roots/anatomcial structures
MH
smoking status
age
gingival biotype

55
Q

2 interventions for inadequate bone level pre-implant

A

autograft - same
allograft - cadaver
xenograft - animal
alloplast - synthetic

56
Q

indications for regenerative periodontal surgery

A

infrabony defects >=3mm
class 2/3 furcation defect
deep pockets >=6mm
no medical contraindications
reasonable tooth prognosis
non-resolved pockets following good quality non-surgical tx

57
Q

if regenerative periodontal surgery fails, how else can this root be managed

A

resective surgery
root resection
root separation
xla

58
Q

why is diabetes a risk factor in periodontal disease

A

impaired/altered immune response
impaired wound healing
overproduction of pro-inflammatory cytokines

hyperglycaemia leads to AGE, which makes connective tissue more susceptible to destruction and damages collagen/connective tissue

59
Q

tests for diabetes

normal vs diabetic values

A

HbA1c =
normal <42mmol/L, <5.7%
diabetic >48mmol/L >5.6%

fasting blood sugar test =
normal <100mg/dL
diabetes >126mg/dL

glucose tolerance test =
normal <139mg/dL
diabetes >200mg/dL

60
Q

how does smoking affect periodontal tissues

A

vasoconstriction
increased keratinisation
impaired antibody production
decreased Th lymphocytes
increased pro-inflammatory cytokines

impaired wound healing

61
Q

what is interleukin-1

A

cytokine involved in regulation of immune and inflammatory responses

pro-inflammatory cytokine mediatior
promotes tissue destruction via MMP
induces bone resorption via activating osteoclasts

62
Q

name 3 oral conditions associated with inflamed gingiva extending beyond mucogingival junction

A

periodontitis
acute necrotising periodontitis
desquamative gingivitis

63
Q

medications associated with gingival hyperplasia

A

phenytoin
cyclosporin
amlodipine

64
Q

how do you manage gingival hyperplasia

A

identify cause
improve OH
surgical management if needed - gingivectomy/gingivoplasty

65
Q

describe BPE values

A

0 - <3.5mm, no calculus/BoP
1 - <3.5mm, BoP
2 - <3.5mm, calculus
3 - 3.5-5mm
4 - >5mm
* - furcation

66
Q

how is mobility graded

A

0 - 0.1-0.2mm
1 - 1mm
2 - >1mm
3 - horizontal + vertical movement

67
Q

how is furcation graded

A

1 - <1/3 tooth width
2 - >1/3 tooth width
3 - through through