Perio Flashcards

1
Q

Factors influencing gingival recession

A
Periodontal phenotype
Presence of periodontal disease and its treatment 
Muscle attachments
Trauma
Tooth position 
Iatrogenic factors
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2
Q

Gingival phenotype

Bone morphology

A

Gingival phenotype = gingival thickness + keratinised tissue width < can be measured by inserting probe
Bone morphology = thickness of bone plate

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3
Q

Types of trauma:

A

Mechanical - TB (evidence inconc.), choice of TB
Physical - piercings, poor dentures, habitual behaviors, foods
Chemical - drugs, irritants, tobacco

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4
Q

Tooth positions:

A

Bucco-lingual position and orientation of tooth can influence thickness of overlying gingival tissues
Mandibular incisors most susceptible to post-ortho recession
Correlation between labial bone dehiscences and recession defects
Deep overbite - trauma to palatal tissues and recession - Akerley classification

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5
Q

Iatrogenic factors

A
Overheated/USS tips
Encroachment on supra-crestal width 
Poorly contoured restorations - plaque retentive 
Surgical procedures
Irritants
Rubber dam clamps
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6
Q

Sequalae of gingival recession

A

Dentine hypersensitivity
Non carious cervical lesions/root caries - pulp necrosis
Impaired aesthetics - open embrasures, exposed crown margins

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7
Q

Classification of gingival recession

A
Millers classification - class I - IV - old
New classification - WWP (2017) based on Cairo et al., 2011
RT1 (full c), RT2 (partial c), RT3 (no c)
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8
Q

Conservative management gingival recession:

A

Reassurance
Baseline records for objective monitoring - clinical photos, dated study models, clinical measurements
Recognition and management of contributing factors - plaque retentive, tooth position, ortho, remove traumatic features, mx of habits
Sensitivity - F- varnish, dentine bonding agents, cervical lesions restored with GIC/composite

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9
Q

Non-surgical management gingival recession:

A

Pink composite
Pink ceramic
Gingival veneers

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10
Q

Surgical management gingival recession:

A
  1. Pedicle flap procedures - coronally advanced flap, rotational flaps
  2. Free soft tissue grafts - free gingival graft, connective tissue graft, processed tissue graft
  3. Regeneration procedures - guided tissue regeneration, enamel matrix derivative, leukocyte and platelet rich fibrin
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11
Q

Indications for gingival recession surgery

A
Progressive breakdown 
Hypersensitivity 
Aesthetics 
Unfavourable contour of gingival margin - limiting effective plaque control 
? prior to ortho if dehiscence likely
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12
Q

Contraindications for gingival recession surgery:

A
Smoking 
Poor OH 
Periodontal pocketing
Uncontrolled aetiological factors e.g. self-inflicted trauma
Severe defects e.g. RT3, wide defects 
Poor donor sites 
Poor access for surgery 
MH considerations
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13
Q

Pedicle flap procedures (2):

A

Coronally advanced flap (CAF) - coronal shift of pedicle flap onto exposed root surface. Ensure at least 3mm keratinised tissue apical to defect. + Reported as most predictable for generalised&localised defects if sufficient and good donor tissue.
+ Can be used in combo with grafts to increase expected RC.

Rotational flap - double papilla flap - 2 papillae slide from adjacent tooth and sutured over to cover exposed root surface. Requires tissue of adequate length thickness and width of KT adjacent to defect.
+ Good for localised defects, esp if narrow defect
- potential bone loss or gingival recession at donor site

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14
Q

Free soft tissue gingival graft procedures:

A

Free gingival graft (FGG): not 1^ aim of root coverage but can be by-product. Done when wanting to ^ width of band of keratinised tissue. Can be to improve local OH, deepen vestibule, prevent fraenal reattachment, pre-ortho/implants if risk of recession high/show through
Donor site - premolar region of palate, avoid palatine vessels. Prepare recipient site - split thickness flap to expose underlying CT, extend 3mm beyond defect, donor graft placed in situ on recipient bed, interrupted sutures to close. Donor ideally 2mm thick and larger than recipient site.

Subepithelial CT graft (CTG): - similar to FGG, but aim of this procedure is full root coverage. Split thickness flap w/2 lateral relieving incisions. Donor tissue harvested from palate using trap door technique - avoiding rugae. Graft placed over recipient site and split thickness flap sutured over top of graft
++ dual blood supply from sub-epithelial tissue base and overlying flap

Processed soft tissue grafts - xenogenic collagen matrices (XCM) or cellular dermal matrix allographs (ADMAs). Porcine bioresorbable product which encourages regeneration of keratinised tissue around teeth/implants w/recession defects.
- Limited evidence but
+ good for pt’s with lack of donor tissue e.g. thin gingival & periodontal phenotypes
Split thickness flap raised, XCM cut to size of defect, moistened, placed in recipient site
Can be combined wirh coronally advanced flaps

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15
Q

Regeneration precedures:

A

Guided tissue regeneration - used for mgt of alveolar bone loss or localised bony defects. Can be used for reconstruction of recession defects.
+ possibility periodontal regeneration
- studies show CTG better

Enamel matrix derivative - porcine product used in combo with CAF for periodontal regeneration of recession defects.
+ similar root coverage achieved compared to CTG, no need for donor site
- systematic review shows CTG + CAF achieves better keratinised tissue thickness than EMD + CAF
- not suitable for all recession defects

Leukocyte and platelet rich fibrin - 2nd gen platelet concentrate used in conjunction with CAF. LRPF source of autogenous growth factors - pt’s blood taken and centrifuged. PRF layer used
- limited evidence as new technique

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16
Q

Systematic review gingival recession procedures:

A

All root coverage procedures can provide significant reduction in recession depth and CAL gain for Miller Class I and II recession-type defects. Subepithelial CT graft-based procedures provided the best outcomes for clinical practice because of their superior percentages of mean and complete root coverage, as well as significant increase of keratinised tissue.
(Chambrone et al, 2015)

17
Q

Limitations of ‘closed’ RSD

A

May not stop progressive and aggressive disease completely
Difficult to visualise morphology of roots
Persistent acute episodes e.g. perio abscess
Deep complex bone defects
Severe hyperplasia or tissue deformity
Pathology e.g. epulides

18
Q

Aims of periodontal therapy

A
Gain access to root surface for effective debridement 
Visualisation of bone defects 
Improvement in tissue contour 
Reduction/elimination of pockets 
Removal of chronically inflamed tissue 
Encourage regeneration 
Removal of hyperplastic gingival tissue 
Crown lengthening
19
Q

Indications for gingivectomy

A

Hyperplasia
False pockets
Need to have adequate attached gingiva

20
Q

Indications for open flap debridement

A

Deep persistent bleeding (suppurating) pockets

21
Q

Indications for apically repositioned flap

+ disadvantages

A

Crown lengthening
Pocket elimination
Unsuccessful gingivally encroaching restorations

Roots exposed - sensitivity, caries
Poor aesthetics - gummy smile

22
Q

Gingivectomy

A
LA
Pocket depth markings - probe depths then pierce from outside depth of pocket (Goldmans technique). External bevel incision 
Incisions
Removal of excised tissue 
Scaling 
Hameostasis 
Dressing
23
Q

Flap procedures - stages:

A
LA
Incisions 
Raise flap with periosteal elevator
Currettage 
RSD
Irrigation 
Sutures 

Incisions - sulcular - maintains ST, hoping for regeneration
resect - resecting epithelium to eliminate pocket. Will cause recession

24
Q

Modified Widman flap

A

Ramjford and Nissle 1974
Incision 1mm from gingival margin preserving interdental papillae
Flap raised only exposing few mm of bone
Intracrevicular/horizontal incisions to release pocket lining
Currettage of bone
Debrdiement root surfaces - USS, hand, irrigation w/saline
Replacement flaps to cover interdental bone and suture

25
Q

Modified Widman flap

A

Ramjford and Nissle 1974
Incision 1mm from gingival margin preserving interdental papillae
Flap raised only exposing few mm of bone
Intracrevicular/horizontal incisions to release pocket lining
Currettage of bone
Debrdiement root surfaces - USS, hand, irrigation w/saline
Replacement flaps to cover interdental bone and suture

26
Q

Post-op perio txt advice

A

Analgesia
Suture removal 7-10 days
CHX 0.2% mouthrinse 2x day for first 2-3 weeks
Not to perform mechanical cleaning in area and chew in this period over txt area
Soft TB
2 weekly dental visit to monitor plaque control

27
Q

Signs of success of flap surgery

A
Decrease in inflammation
Less bleeding on probing 
Decrease in pocket depths 
Increase in attachment 
Elimination of pus
No increase in mobility
Improvement of tissue contour
Stabilisation of bone levels
Regeneration
28
Q

Evidence - perio:

A

Ramjford et al 1987 and Gothenburg group evaluated outcome of surgical vs non-surgical
All surgical resulted in decrease in PD, greater reduction in deeper sites
Surgical showerd greater short term reduction in PDs but long term variation in results between S vs NS
Sites with shallow probing depths - surgical created greater attachment loss than NS, but in deeper sites >7, greater attachment seen w S
In molar sites, surgical outcomes better than NS
Surgery w/o osseous = or greater attachment gain than w/osseous contouring
Deliberate excision of pocket by surrettage did not improve healing result
Pts with good post op plaque control mainted clinical attachment levels and PD more consistently than pt’s with poor OH

29
Q

Guided tissue regeneration - perio:

A

Is used for the prevention of epithelium migration along the cemental wall of the pocket and maintains space for clot stabilisation
GTR - placing membrane barrier over bone, seperating the gingival epithelium from the bone and PDL
This prevents epithelial migration and favours repopulation of area by PL and bone cells
CT has chance to develop further into defect as epithelial tissues develop faster than CT
Most membranes bioresorbable e.g. BioGuide
Non resorbable not really used any more

30
Q

EmgoGain

A

Enamel matrix derivative
Made from enamel matrix proteins from tooth germs of swine and uses propylene glycoralginate as matrix
Differentiates cells at dental follicle into cementoblasts to produce cementum which anchors collagen fibres
Applied as gel to exposed surface during surgery for intra-bony defects
Biological action after apply Emdogain:
1. Attraction
2. Attchment and proliferation
3. Differentiation
4. Alveolar bone

Predictability Emdogain:
Hedbed et al
87% sites >2mm attachment gain
Majority of sites >4mm attachment gain 
Ave. bone infill = 69%
Preditable way to enhance periodontal regeneration outcome
31
Q

Review protocol post regeneration therapy

A

1 week - suture removal from donor sites, removal from other site at 10-12 days, OH support, gentle cleaning
4-6 weeks - suture removal for CTG/FGG and OH support
3/12 - initial probing
6/12 some techniques may require longer before probing
9/12 post op PA to assess bone levels
12/12 - some root coverage may continue to occur by creeping attachment

32
Q

Wound healing

A
  1. Haemostasis - blood clot, matrix for cell migration
  2. Inflammation - growth factors in clot recruit inflammatory cells - hours. Day 3 - macrophages releasing cytokines and growth factors to transition into granulation tissue
  3. Granulation - day 4 - migration and prolif of fibroblasts, endothelial cells and SM cells. 7 days - initial collagen
    Maturation - fibroblasts replace provision fibrin matrix with collagen rich matrix, endothelial cells - angiogenesis

Perio healing differs slightly as one vascular side and one avascular side. Pocket reduction following nonsurgical therapy occurs through production of long junctional epithelium at 1 week, resolution of inflammation at 2 weeks, CT remodelling and collagen fibre maturation at 2-3 months so no reprobing prior to 3 months

33
Q

Factors affecting healing

Local and systemic

A
Local:
Infection
Poor blood supply
Foreign bodies
Movement - graft destabilisation and tooth mobility 
Ionising radiation 
UV light 

Systemic:
Age
Poor nutrition - vit C and D
Hormones - corticosteroids can suppress migration and macrophages. Androgenous corticosteroids from stress can suppress granulation tissue formation
Smoking
Uncontrolled diabetes - more prone to infection
Haematological abnormalities - may impair clot formation

34
Q

Histological patterns of wound healing

A

No repair
Long junctional epithelium attaches to root surface
CT attachment to the root surface
New bone seperated from root surface
New bone with resorption or ankylosis to root surface
New attachment apparatus