Peds week 3 Flashcards
For deoxygenated blood to exit the fetus, it passes through the __ arteries and then the __ arteries to the placenta
hypogastric arteries
umbilical arteries
for oxygenated blood to reach the fetus, after leaving the placenta it travels through the __ vein, which connects with the inferior vena cava via the ductus __ venosus
umbilical vein
ductus venosus
For the last time, during FETAL circulation, the PVR is __ and the SVR is __
PVR high (b/c lungs are bypassed) SVR low
What four defects define tetralogy of fallot? and what kind of shunt?
pulmonary stenosis (leads to right to left shunt) overriding aorta RV hypertrophy (also right to left shunt) ventricular septal defect
what is the most common CYANOTIC congenital heart disease?
cyanosis = a right to left shunt that bypasses lungs. The most common is Tetralogy of fallot
How to treat a “tet spell” aka acute worsening of tetralogy of fallot
100% fio2, hyperventilation (decreasing etco2 decreases PVR and encourages pumonary circulation, increase preload with IVF, sedate, increase SVR with phenylephrine to reverse shunt, beta blocker to slow HR
when repairing tetralogy of fallot, what side do you put the a-line on?
the opposite side of the clamped subclavian artery, most probably place it on the left side as the right subclavian will probably be clamped
what are teh anesthetic management goals for tetralogy of fallot?
maintain volume, increase SVR with phenylephrine to lessen shunt, decrease PVR to lessen shunt
an infant with tetralogy of fallot will have normal __ and __, but decreased __
normal pH and PaCO2, decreased PaO2
nitrous for a child with tetralogy of fallot?
NO, INCREASES PVR and worsens shunt
list three conditions that increase right to left shunt associated with tetralogy of fallot?
acidosis, hypercarbia, hypotension, volatile anesthetics, anything that causes histamine release
One more time, what four characteristics define tetralogy of fallot?
VSD, right ventricular outflow obstruction (pulmonary stenosis), RV hypertrophy, and overriding aorta
what is the most crucial part of successfully reversing the transposition of the great arteries?
connecting the coronary arteries to the neo-aortic root
what is truncus arteriosus?
a big VSD with one common valve for both ventricles, leads to constant mixing of oxygenated and deoxygenated blood.
what is a double outlet right ventricle?
both the PA and aorta arise from the right ventricle, often with a large VSD
what’s up with hypoplastic left heart syndrome?
very small LV due to mitral and/or aortic valve stenosis or atresia (closure), so blood flows from the Left atrium to the RA/RV via ASD, thus the RV is acting as a single ventricle, and systemic blood has to go from RV to PA and then to aorta via PDA for systemic circulation
what is a norwood stage 1?
creation of neo-aorta and placement of BT shunt for passive pulmonary blood flow
during norwood surgery, should PVR be increased or decreased?
NEITHER, they have to be just right. because if the PVR increased then cyanosis would result, and if PVR decreased then the lungs would flood and systemic circulation would be low
KEEP SPONTANEOUSLY BREATHING WITH FiO2 21% AND PROSTAGLANDIN INFUSION TO KEEP PDA OPEN
oxygen is a drug slide
I have no clue, you’ll have to read it yourself. Slides 69 and 70. Seems contradictory to last weeks info.
If you want to maintain a PDA
give prostaglandin
if you want to close a PDA
give indomethacin or PDA ligation
why must you AVOID the IJ vein for a norwood?
will use it in the future for a glenn shunt
norwood surgical considerations
high dose opioid, venous access via femoral or umbilical vein