Peds week 2 Flashcards

(76 cards)

1
Q

When do deciduous teeth erupt?

A

At 6 months and begin shedding between 5-8 yrs.

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2
Q

Epiglottis is ______, ______, and at a more _____ angle from the trachea making it more difficult to lift

A

Epiglottis is narrower, longer, and at a more acute angle from the trachea making it more difficult to lift

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3
Q

The narrowest part of the pediatric airway is:

A

The cricoid cartilage. In adults it is at the vocal chords

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4
Q

The pediatric larynx is at:

A

C3-4

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5
Q

The adult larynx is at:

A

C4-5

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6
Q

What shape is the pediatric airway?

A

Funnel shaped

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7
Q

Length of the trachea (vocal cords to carina) in neonates and children up to one year of age is ___cm or ___

A

Length of the trachea (vocal cords to carina) in neonates and children up to one year of age is 5-9 cm or 2-2.5 inches

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8
Q

DO NOT ____ THE ETT once you go through the vocal cords

A

DO NOT BURY THE ETT once you go through the vocal cords

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9
Q

Do NOT overextend…keep the head ______

A

Do NOT overextend…keep the head parallel

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10
Q

The epiglottis is ____ shaped

A

omega

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11
Q

True/False: It is more difficult to lift an infants epiglottis with a laryngoscopic blade

A

True

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12
Q

A small decrease in airway size (edema from inflammation or trauma) results in what?

A

a large increase in resistance to flow

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13
Q

Infants are _____ ______ breathers until 3-5 months of age because?

A

Infants are obligate nasal breathers until 3-5 months of age because the major source of resistance to airflow is the lower airways

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14
Q

What can occlusion of the nares can cause?

A

complete airway obstruction

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15
Q

Overcoming the resistance of the nares accounts for only ___% of the work of breathing for infants as compared to ___% in adults

A

Overcoming the resistance of the nares accounts for only 25% of the work of breathing for infants as compared to 60% in adults

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16
Q

What kind of breathing do children under 6 months of age rely on?

A

diaphragmatic breathing

Thorax is weak and unstable

Diaphragm contains a smaller percentage of Type 1 (slow twitch, fatigue-resistant) muscle fibers

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17
Q

Ribcage/intercostal muscle contribution to ventilation is ___to___%

By 9 months of age, ribcage contribution increases to ___%

A

Ribcage/intercostal muscle contribution to ventilation is 20-40%

By 9 months of age, ribcage contribution increases to 50%

By 12 months of age, chest wall is stable and resists inward recoil of lungs

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18
Q

FRC is ____

A

FRC is small (not functional)

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19
Q

Infant respiratory rate is ____

A

30-50

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20
Q

Infants tidal volume is ___

A

7

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21
Q

Infants dead space is ____

A
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22
Q

Alveolar ventilation is _____

A

100-150

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23
Q

Functional residual capacity is ____

A

27-30

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24
Q

Oxygen consumption is ___

A

7-9

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25
Anesthesia reduces FRC by causing ____ ____ ____ and impaired intercostal and diaphragm activity
peripheral airway collapse
26
ETT Size (mm ID) =
age in years +16 / 4
27
There should be an audible air leak around the tube at a pressure between ___-___ cm H20 (we typically go with 20 cm H2o)
There should be an audible air leak around the tube at a pressure between 15-25 cm H20 (we typically go with 20 cm H2o)
28
What is the biggest factor for post operative croup?
caused by excessive tube size
29
Is cuffed ETT is always a half size smaller?
Yep
30
Is percussion and auscultation is of limited usefulness in small children?
Yep. Limited usefulness
31
URI within the past 2-6 weeks =
significant risk of bronchospasm
32
Canceled Cases:
Wheezing Green nasal drainage Fever
33
Laryngospasm
Stimulation in stage 2 Sustained tight closure of the vocal cords during light planes of anesthesia (creates a state of central disinhibition) Stimulation of the superior laryngeal nerve, not the recurrent laryngeal nerve Contraction of the adductor muscles of the larynx (lateral cricoarytenoids, thyroarytenoids, and cricoarytenoids)
34
Causes of laryngospasm
Inhalation of volatile agents Excessive secretions in the airway Presence of URI (hyper-irritable) Manipulation of the airway (intubation, extubation) Stimulation of the visceral nerve endings in the pelvis, abdomen, and thorax
35
Treatment for laryngospasm
Remove irritating stimulus (suction) Remove debris from airway Deepen anesthesia as appropriate 100% O2 via tight fitting face mask *Sustained positive airway pressure (30-40 cm H2O) Manual forward displacement of the mandible If airway maneuvers fail – atropine, succinylcholine, and consider intubation Succinylcholine 0.4mg/kg IV 4mg/kg IM Atropine 20mcg/kg IM/IV
36
Post Intubation Laryngeal Edema
Potential complication of intubation in all children but incidence is greatest in children ages 1-4
37
Causes of post-intubation laryngeal edema
Mechanical trauma to the airway during intubation Placement of a tube that produces a tight fit (no leak up to 40 cm H2O)
38
Treatment for post intubation laryngeal edema
Humidification of inspired gases Aerosolized racemic epinephrine 0.5ml of 2.25% solution in 2-3 ml saline – vasoconstriction of capillaries in subglottic mucosa Re-intubation Tracheostomy
39
Epiglottitis
Rare cause of infectious upper airway obstruction in children Etiology – haemophilus influenzae type B Occurs in children 3-6 yrs. Pathology – systemic septicemic process with local erythema and edema most marked in the epiglottis, aryepiglottic folds, and supraglottic connective tissue
40
Symptoms of epiglottitis
Rapid clinical progression of symptoms, <24 hrs. Dysphagia, dysphonia, drooling, inspiratory stridor, distress High fever, >39°C
41
Treatment for epiglottitis
O2 Urgent intubation of the trachea under general anesthesia – NEED to be in the OR Antibiotics, antipyretics Fluids
42
Anesthetic Management for epiglottitis
Transfer the child to the OR – do nothing to upset or agitate the child Smooth, controlled inhalation induction with sevoflurane, child in a sitting position, CPAP applied to the circuit Obtain IV access, administer atropine Achieve Stage III, do no precipitate laryngospasm ENT HAS TO BE PRESENT Place the child supine, intubate the trachea with a small ETT a size and ½ smaller MAINTAIN SPONTANEOUS VENTILATION Through direct laryngoscopy by the ENT surgeon to confirm diagnosis
43
Anesthetic implications with epiglottitis
Expect a slow induction due to the partially obstructed airway Inflammation of the airway may enhance irritability and increase the potential for coughing, breathholding, and laryngospam CV depressant effects of inhalation agent magnified r/t hypovolemia Rigid ventilating bronchoscope and surgical airway equipment must be available With severe obstruction and mucosal swelling, identification of supraglottic structures may be difficult May choose to replace oral tube with nasal tube intubated for 48-96 hrs.
44
Recovery of epiglottitis
Extubation considered when temp and white count fall Resolution of the swelling signaled by audible leak around ETT Extubation only after direct laryngoscopy in the OR under GA to confirm that swelling of epiglottis has resolved
45
How can you tell if epiglottitis swelling has resolved?
by audible leak around ETT
46
Laryngotracheobronchitis (croup, subglottic infection)
Accounts for 90% of infectious upper airway obstruction in children Etiology – parainfluenzae virus type 1 and 2, influenzae A, respiratory syncytial virus Occurs in children <2 years of age Pathology – mucosal and submucosal edema within the circoid ring (decreased luminal size)
47
Laryngotracheobronchitis
Gradual onset and progression of symptoms, 24-72 hrs. History of URI progressing to hoarse cry or barking cough Low grade fever <39ºC
48
Treatment for laryngotracheobronchitis
O2 with cool aqueous mist Aerosolized racemic epi (vasoconstriction of capillaries in subglottic mucosa, Beta adrenergic bronchodilatory effect) Albuterol Corticosteroids. Stabilize cell membrane integrity, decrease release of inflammatory mediators Antipyretics Intubation of the trachea is RARE unless exhaustion occurs
49
Foreign Body Aspiration
Most frequent site is right mainstem Cough, wheezing, decreased air entry into affected lung, URI, pneumonia
50
Treatment of Foreign Body Aspiration
Laryngoscopic or endoscopic removal Best to remove object within 24 hrs. Risks of leaving the foreign object: -Migration of the aspirated material -Pneumonia -Residual pulmonary disease
51
Anesthetic Management for airway obstruction
Induction technique will depend on severity of airway obstruction: With airway obstruction, inhalation of volatile agent in O2 maintaining spontaneous ventilation Without airway obstruction, IV induction with standard agents
52
After anesthetic management for airway obstruction continued..
Achieve Stage III of anesthesia, perform direct laryngoscopy for the purpose of anesthetizing the vocal cords to prevent laryngospasm – lidocaine 1% 1-2 mg/kg With airway obstruction, avoid NDNMBs: Positive pressure ventilation may contribute to migration of the aspirated material Narrow bronchoscope creates high resistance to gas flow Typically there is a large gas leak around the bronchoscope
53
Anesthetic Management continued
Maintenance – O2, IH agent Skeletal muscle paralysis may be required for removal if the aspirate object is too large to pass through the moving vocal cords – succs, cisatracurium Emergence – pt. may or may not be intubated after removal of the foreign body Post-op – aerosolized racemic epi, corticosteroids to reduce subglottic edema
54
Foreign body aspiration complications
Airway obstruction Fragmentation of the foreign body Arterial hypoxemia Hypercarbia Subglottic edema from trauma to the tracheobronchial tree – foreign body, instrumentation
55
Tonsillectomy and Adenoidectomy implications
Upper airway obstruction Massive hypertrophy Chronic upper respiratory infections Obstructive sleep apnea
56
Anesthetic management for Tonsillectomy and Adenoidectomy
Pre-medication oral or intranasal midazolam IH induction with sevoflurane Intubation – deep (sevoflurane + propofol 1-2mg/kg) or with short acting NDNMB Analgesia – MSO4 0.1mg/kg or Fentanyl 1-2mcg/kg (Obstructive sleep apnea cut meds to ½ dose) Steroid –dexamethasone 0.3-1mg/kg Emergence – extubate when child fully awake…don’t *need* to
57
Post-tonsillectomy Bleeding
Bleeding that continues or recurs after tonsillectomy and requires surgical intervention (packing or suturing) Early: Incidence 0.2-2% *Within first 24 hrs. 99% within first 6 hrs. Secondary/Delayed Incidence 0.1-3% 24 hrs. up to 2-3 wks. post-op, peak - day 7
58
Post-tonsillectomy Bleeding risk populations
Older pts. Presence of inflammation, infection Pre-op ingestion of ASA, NSAIDS – inhibition of platelet function Coagulopathy Patients that have had strep multiple times
59
Post-tonsillectomy Bleeding prevention
Careful dissection in tonsilar capsule Meticulous hemostasis Avoid surgery during/immediately after acute inflammation, infection Avoid blind vigorous suctioning Avoid use of NSAIDS
60
Post-tonsillectomy bleeding symptoms
Frequent swallowing The kid will throw up blood Tonsil bleed is an RSI Need to know hemoglobin and urine specific gravity (how dehydrated) 1.010-1.015
61
Post-tonsillectomy Bleeding clinical presentation
Hypovolemia Anemia Agitation Shock Stomach full of blood Active bleeding (poor visualization of glottis)
62
Post-tonsillectomy Bleeding preop evaluation
Assessment of volume status: BP (orthostatic changes), HR, urine output, mucus membranes, skin turgor, sensorium Labs, H/H, urine specific gravity/osmolality E stablish IV access-rehydration or transfusion must begin immediately SEND for BLOOD
63
Post-tonsillectomy Bleeding management
2 Suctions AT THE HEAD OF THE BED!!! In case the one clots
64
Choanal Atresia
Occlusion of one or both posterior nares Atresia is partially or totally bony in 90% of cases Freq. association craniosynostosis Since neonates are obligatory nose breathers, bilat. choanal atresia causes suffocation if the mouth is not kept open (oral airway or large rubber nipple secured in the mouth) Unilateral atresia may go undiagnosed for months or years; is eventually diagnosed due to the presence of intractable unilateral nasal drainage Surgical correction or tracheostomy must be performed within the first few days of life
65
Anesthetic Management of choanal atresia
66
Anesthetic Management of choanal atresia
Awake intubation with oral RAE tube Maintenance – O2/N20/IH, NDNMB may be used, opioids for analgesia Emergence – wide awake for extubation Post op – partial or intermittent airway obstruction may persist for some time, so the infant must be observed with appropriate monitoring until airway patency is assured Stents are place – baby will transfer to ICU
66
IV Induction: Advantageous because…
Asleep without going through Stage 2 Risk of Laryngospasm very low
67
Stages chart
68
anatomical differences between pediatric and adult airways
69
Anesthetic Management of choanal atresia
Awake intubation with oral RAE tube Maintenance – O2/N20/IH, NDNMB may be used, opioids for analgesia Emergence – wide awake for extubation Post op – partial or intermittent airway obstruction may persist for some time, so the infant must be observed with appropriate monitoring until airway patency is assured Stents are place – baby will transfer to ICU
70
Anesthetic Management of choanal atresia
Awake intubation with oral RAE tube Maintenance – O2/N20/IH, NDNMB may be used, opioids for analgesia Emergence – wide awake for extubation Post op – partial or intermittent airway obstruction may persist for some time, so the infant must be observed with appropriate monitoring until airway patency is assured Stents are place – baby will transfer to ICU
71
Laryngospasm dose of succs
Succinylcholine 4mg/kg IM 0.4 mg/kg IV Positive pressure 40 mm Hg – This is the reason you bring the mask and oral airway with every child – and why you transport with O2 in the event of spasm the kid has been oxygenated prior to event rather than starting at room air
72
Opioid doses for peds
½ the dose for fatigued children ½ the dose for children with OSA Narcan o.5 mcg/kg
73
Anesthetic Management of choanal atresia
Awake intubation with oral RAE tube Maintenance – O2/N20/IH, NDNMB may be used, opioids for analgesia Emergence – wide awake for extubation Post op – partial or intermittent airway obstruction may persist for some time, so the infant must be observed with appropriate monitoring until airway patency is assured Stents are place – baby will transfer to ICU
73
Nausea/Vomiting
Zofran 0.1-0.5mg/kg
74
Anesthetic Management of choanal atresia
Awake intubation with oral RAE tube Maintenance – O2/N20/IH, NDNMB may be used, opioids for analgesia Emergence – wide awake for extubation Post op – partial or intermittent airway obstruction may persist for some time, so the infant must be observed with appropriate monitoring until airway patency is assured Stents are place – baby will transfer to ICU