Peds Respiratory Flashcards

1
Q

General concepts

A

Look first; what do you see?
Is the breathing labored? Is there grunting? Are there retractions? Is there wheezing? Is there stridor? Do you have lung sounds bilaterally? What does it mean if you don’t?
Is there oxygen present? Style of device? How are they breathing through nose, mouth, chest, abdominal? Is there a need for assist control by a ventilator? Is there a set rate?
Where do you take a pulse ox? In infants vs adults? What would you do if you had a reading less than 90?
Is there diaphoretic skin?
Is there a need to be upright?
Is there inability to swallow? Is there drooling?
What is the tone of the skin?
What do you hear when you auscultate for lung sounds?
Do the physician orders correlate to the symptoms?
Is there an IV running? What is the rate? How does this relate to lung sounds?
What is the urine output? Is there retention? How do you know that? How does this relate to lung sounds?
Is there swelling ie pitting edema in the extremities? How does this relate to the heart?
If you hear “wet lungs” what is it helpful to do? Would sitting upright help this situation?
Would Albuterol aerosol be helpful?
If there is drooling present what does that mean to you? What should you not do? What action does this imply?

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2
Q

Respiratory disorders general

A

Respiratory disorders are the most common cause of illness and death in infants and children.

Distress is due to
Immature immune systems
Smaller upper and lower airways
Underdeveloped supporting cartilage

Usually self-limiting, but distress due to mucus & edema obstructing the airways

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3
Q

Peds respiratory differences General

A

Smaller upper and lower airways
Underdeveloped supporting cartilage
Less well-developed intercostal muscles
Alveoli have less elasticity
Tongue is large in proportion to oropharynx
Eustachian tubes relatively horizontal
Tonsilar tissue enlarged
Larynx is more anterior, more flexible
Smaller septum; nasal bridge is flat and flexible
Vocal cords located at C3-4 versus C5-6 in adults (Contributes to aspiration if neck is hyperextended)
Smaller airway
Airway narrowest at cricoid ring instead of at vocal cords
Airway diameter is 4 mm (20 mm in adult)

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4
Q

Peds differences - upper

A

Obligatory nose breather (< 4 months)
Tongue is large in proportion to oropharynx
Larynx is more anterior, more flexible
Eustachian tubes shorter and more horizontal
Epiglottis is large and floppy
Tonsillar tissue enlarged
Trachea shorter, bifurcation higher up
Tracheal diameter size of straw, cartilage rings easily compressed

More smooth muscle makes airway more reactive or sensitive to foreign substances

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5
Q

Peds differences - lower and general

A
Lack of or insufficient surfactant   (preemie)
Less well-developed intercostal muscles
Alveoli have less elasticity
Faster respiratory rate
Apnea periods common with newborn

Hyaline membrane disease/BPD Broncho pulmonary dysplasia

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6
Q

Causes of respiratory compromise

A
Upper or lower respiratory infections
Sedative medications
CNS disorders
Musculoskeletal deformities
Congenital anomalies
Aspiration 

CNS disorders such as muscular dystrophy or cerebral palsy
Genetic defects with curvature of the spine
Diaphramatic hernias

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7
Q

Infection Preventive Teaching measures

A

Adequate rest
Good nutrition
Good hygiene
Especially HANDWASHING

Nurses must remember to wash hands and not touch eyes/nose (teach patients also)
Change toothbrush esp with bacterial infections (24 hr after starting antibiotics

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8
Q

History, questions to ask

A

Time of onset of problem ? Rapid or gradual ?
Previous episodes of respiratory distress ?
Presence of a cough ?
Shortness of breath ?
Nasal flaring ?
Nasal congestion ?
Noisy respirations ?
Decreased level of awareness or response ?
Anxiety, confusion, ability to be consoled ?
Difficulty feeding ?
Any episodes of apnea ?

Questions should also include

- medication history
- living environment
- exposure to respiratory infection
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9
Q

Significant past history

A

Low birth weight/SGA
Prematurity/Early and Late
Chronic illnesses, including asthma
Previous hospitalization for respiratory illness

International travel
Asthma stemming from BPD and venting

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10
Q

Physical assessment general

A

Consider the child’s chronological age and developmental level
Be creative in gaining cooperation
Always use appropriate size equipment

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11
Q

Phys assess- inspection

A

Evaluate color, rate, ease of breathing
Children< 6-7 years of age: abdominal breathing
Children> 6-7 years of age: thoracic breathing
Note shape and size of chest
Note nasal flaring, SOB, retractions, grunting, stridor*

Nasal flaring: a serious sign of air hunger, widening of nares for more oxygen
Retractions: abnormal chest wall movement at inspiration; intercostally and substernally
Grunting: noise audible with or without a stethoscope
Stridor: shrill, harsh sound during inspiration or expiration or both. Air flow through a narrowed segment
Coloring: more in trunk and less to extremities

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12
Q

Phys inspection - auscultation

A

Follow a systematic pattern
Check for presence or absence of breath sounds
Check for adventitious breath sounds
Listen for intensity, pitch, and duration

Adventitious breath sounds:
Crackles: heard on inspiration
result as air moves through fluid filled alveoli as in pneumonia
may not change after coughing
sound can be simulated by rolling hair between your fingers
Rhonchi: low pitched sounds heard throughout respiration
air passing through thick secretions
may clear after coughing
Stridor high pitched sound heard on inspiration as in croup
Wheezing: heard throughout respirations high pitched musical sound result of air passing thru constricted bronchioles as in asthma

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13
Q

Upper airway problems

A
Sinusitis
Allergic rhinitis
Epistaxis
Otitis media*
Pharyngitis
Tonsillitis*
Croup*
Epiglottitis*
Laryngitis
Foreign body

Upper respiratory infection – (URI) – common cold

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14
Q

Otitis Media

A

Middle ear infection
Otitis Media Acute
Otitis Media with Effusion

Peak ages between 6 months and 3 years
Two types
May cause hearing loss or eardrum puncture
Often caused by bacteria: Streptococcus pneumonae or haemophilus influenzae
Can be caused by virus: RSV respiratory syncytial virus
Of all children 84% will have at least one episode before age 3

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15
Q

Otitis media causes/factors

A

The Eustachian tube is more horizontal, shorter and wider, so secretions do not drain efficiently.
A negative pressure occurs in the middle space , and fluid creates a medium for bacterial growth.
Babies who drink bottles laying flat in a crib rather than a baby seat

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16
Q

Acute otitis media Sx

A

Febrile High Grade/febrile seizures
Earache, toddlers may pull on ear, infants may rub ear on things
Vomiting, anorexia
Irritability, sleep disturbance, persistent crying
Otoscope findings: tympanic membrane red and bulging.

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17
Q

Otitis media interventions

A
Administer analgesics
Administer antibiotics if ordered*
Promote drainage
Promote comfort
Encourage fluids
Instruct parent about the importance of completing the prescribed medications

30 degree elevation, warm or cold compresses, encouraging fluids to liquify secretions

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18
Q

Otitis media with effusion Sx

A

Tinnitus
Conductive hearing loss may occur
Afebrile
Otoscope findings: tympanic membrane dull gray, orange, immobile

Fluid in the middle ear space without symptoms of an acute ear infection
Children with OME do not act sick*.
Common after acute ear infection
Eustachian tube, which connects the inside of the ear to the back of the throat, becomes blocked
Usually drains secretions from the tube and are swallowed.
When Eustachian tube is partially blocked, fluid accumulates in the middle ear. Bacteria already inside the ear becomes trapped and begins to multiply.

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19
Q

Tonsilitis

A

Frequent colds and throat infections

Irritation in throat w/ pain, radiating to ears, difficulty eating and drinking, occasional fever, inflamed tonsils

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20
Q

All about tonsilitis

A

What are tonsils?
mass of lymphoid tissue encircling nasal and oral pharynx

Purpose of tonsils?
prevent bacteria, virus, allergens, etc attempting to attack the human body. As lymphoid tissues, they produce Lymphocytes. which form an immunological network of self-defense.
Symptoms of tonsillitis?
Sore throat (gradual with viral);
DIFFICULTY SWALLOWING (DYSPHAGIA) from pain or large size due to inflammation
Erythema and inflammation of pharynx and tonsils (vesicles on tonsils with viral)
Fever
Apnea during sleep with obstruction of air passage
Snoring due to obstructed air passage
Malaise, loss of appetite, headache, body ache
Examination:
inflamed, red, congested tonsils.
visible small pus pockets
enlarged and mildly painful neck glands palpable
:
Intervention
Differentiate between viral and bacterial with culture
Comfort with rest, soft/liquid diet, cool mist vaporizer, warm gargles, lozenges, Tylenol (codeine)
Antibiotics if bacterial
Surgery after 3-4 infections/year not responding to medical therapy
Analgesics
No red food or liquida)
Discourage coughing or clearing throat

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21
Q

Croup / Acute Laryngotracheobronchitis LTB

A
Assessment/History
What treatment or meds have been given?
How effective were meds?
Any difficulty swallowing?
Drooling present?
Has the child been ill?
What symptoms noted &amp; what changes?

Differentiate by LOCATION and causative ORGANISM – VIRUS OR BACTERIA
Usually caused by a virus, preceded by a URI
Low grade fever, barking, seal like harsh coughworse at night usually absent by day
Narrowing of the larynx, trachea, bronchi….if severe, stridor may be seen and other signs of resp distress…less likely to cause obstruction than epiglottis,

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22
Q

Croup mgmt at home

A

Place child in bathroom with hot shower running, followed by blast of cool air.
Humidifiers
Fluids
Ibuprofen

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23
Q

Croup management in hospital

A

Assess & monitor ABC’s
High flow humidified O2
Limit exam/handling to avoid agitation
Be prepared for respiratory arrest & CPR
Do not place instruments in mouth or throat
Supportive nursing care; administer steroids as ordered.

Corticosteroids such as Dexamethasone with side effects of hypertension and elevated glucose levels
Beta adrenergics such as epinephrine are rapid acting aerosols which decrease inflamation and mucus production Beta adrenergics can lead to tachycardia, hypertension, headache and anxiety
Milk products increase mucus production….

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24
Q

Epiglottitis

A

An acute inflammation & swelling of the epiglottis and upper respiratory structures caused by bacteria/ H.influenza (or strains of strep & staph)
Secretions pool & child unable to swallow
Usually in children 3-6 years of age
A true medical emergency

Progresses to complete obstruction within a few hours of onset.
Like bacterial meningitis, sicker than viral…
Epiglottis is worse than croup, it’s the location of the swelling as well as the bacteria…
Hib vaccine has decreased illness, but did not eliminate it.
Your book categorizes epiglottitis as a type of croup. Keep them separate in your mind.
Usually caused by Staph or Strep or H influenzae

Upper respiratory viral infection -> bacterial infection
Mostly ages 6 months to 3 years
More prevalent in fall and spring
Edema causes narrowed airway lumen
Severe cases: complete obstruction
25
S/S of epiglottitis
``` May be sitting in Tripod position May be holding mouth open, with tongue protruding Muffled or hoarse cry Inspiratory stridor Tachycardia, tachypnea Pale, mottled, cyanotic skin Anxious, focused on breathing, lethargic Very sore throat Nasal flaring Looks very sick and high fever ```
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Assessment/ Hx for epiglottitis
When did the child become ill? Was it sudden? Is it suddenly worse after a couple of days or hours? Sore throat? Can the child swallow liquids or saliva? Is drooling present? Is there a high fever? Is there difficulty breathing? ``` Abrupt onset, rapid progression Sore throat Appears anxious/agitated* Unable to swallow & drooling* Absence of spontaneous cough* High temperature 102-104°F ``` Tachypneic, nasal flaring, retractions, pallor STRIDOR present “Cherry Red” epiglottis
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DCs of Epiglottitis
``` 4 Ds Drooling Dysphagia Dysphonia Distressed respirations ```
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Nsg Assessment for Epiglottitis
``` Assess for patent airway Assess for presence of inspiratory stridor Measure body temperature Assess RR, HR, color Note presence of drooling Assess pulse oximetry ```
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Signs of impending airway obstruction
Increased pulse and respiratory rate Substernal, suprasternal, intercostal retractions Flaring nares Increased restlessness
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Two most significant nursing dx for epiglottits
Ineffective Airway Clearance R/T edema and placement of artificial airway Impaired Gas Exchange R/T edema of the epiglottis
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Epiglottitis mgmt
Assess & monitor ABC’s, oxygen saturation Have child sit up Maintain patent airway High flow humidified O2; “blow by” if child won’t tolerate mask Limit exam/handling to avoid agitation Be prepared for respiratory arrest, assist ventilations and perform CPR as needed Artificial airway may be necessary Administer antibiotics Epinephrine may be required Administer analgesia as ordered Keep a calm and quiet environment
32
Lower airway problems
``` Bronchitis Bronchiolitis Respiratory syncytial virus (RSV)* Pneumonia Pertussis Tuberculosis Foreign body aspiration Smoke inhalation Asthma* Cystic fibrosis* Sudden Infant Death Syndrome (SIDS)* ```
33
Bronchiolitis
Problem Respiratory infection of the bronchioles Occurs in early childhood (younger than 1 year) Caused by viral infection Assessment/History Length of illness or fever? Recent history of child or family having URI? Taking any medications? How much fluid has the child been drinking? Edema in mucous membrane lining in walls of bronchioles plus inc mucus causing obstruction and inc airway resistance
34
Respiratory Syncytial Virus RSV
Most common cause of bronchiolitis and pneumonia among infants and children under 1 year of age. Illness begins most frequently with fever, runny nose, cough, and sometimes wheezing Most children recover in 8 to 15 days. Inflammation of smaller bronchioles characterized by thick mucus During their first RSV infection, between 25% and 40% of infants and young children have signs or symptoms of bronchiolitis or pneumonia UP to 2% require hospitalization- usually under 6 months of age
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Bronchiolitis RSV Manifestation
``` Acute respiratory distress Tachypnea May have intercostal and suprasternal retractions Nasal flaring, circumoral cyanosis Fever & dry cough think thick mucus May have wheezes - inspiratory & expiratory Confused & anxious mental status Possible anorexia, dehydration Nasal discharge ``` Most clinical laboratories use antigen detection assays to diagnose infection Elisa test: nasopharyngeal washing Chest xray H & H
36
Nsg Dx for RSV /Bronchiolitis
Impaired gas exchange r/t edema and increased secretions Ineffective airway clearance r/t increased secretions Fluid volume deficit r/t decreased food and fluids
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Management for RSV/Bronchiolitis
``` Management Assess & maintain airway Clear nasal passages if necessary IV LR or NS @KVO rate Contact precautions High risk infants may also receive RSV Immunoglobulin (Respigam) or Monoclonal Antibodies (Palivizumab) prophylactic vaccination ``` Intubate if airway management becomes difficult or fails Palivizumab = Synagis prophylactic vaccination Given as an IM injection before the start of the RSV season usually October to May and then every month during RSV season for a total of 5 injections at the cost of about 1000 dollars for the smallest single dose Treatment is recommended for high risk groups: Infants born at 29 weeks to 35 weeks gestation who are 3 months or younger at the start of the RSV season Infants and children under the age of 2 with congenital heart defects Infants born at 35 weeks gestation with chronic lung disease or neuromuscular disorders Symptom RX only (e.g., acetaminophen to reduce fever). Children with severe disease may require oxygen therapy and sometimes mechanical ventilation Ribavirin aerosol may be used in the treatment of some patients with severe disease but not routine due to concerns re side effects. Easily spread from hand to eye, nose or other mucous membrane Separate room or with other RSV patients HANDWASHING Contact precautions: gloves, gowns, mask, goggles; careful nurse assignments
38
Respigam IG considerations
Watch high risk premature infants and children for tachycardia, HTN, vomiting, diarrhea, wheezing, tachypnea, rales
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Sudden infant Death Syndrome SIDS
Sudden and unexplained death of a seemingly healthy baby. Most SIDS deaths usually occur in children who are between 2 months and 4 months Typically, a peacefully sleeping baby simply never wakes up. In most cases, no cause is ever found. multiple factors cause SIDS infant must have some sort of biological vulnerability, such as a heart or brain defect, combined with an environmental stressor, such as stomach sleeping, before SIDS can occur must also occur during a critical developmental period in the baby's life, typically during the first six months. These three factors — vulnerability, critical developmental period and outside stressor — combine to form what's known as the triple-risk model.
40
SIDS research
In 2006, researchers discovered that abnormalities in a part of the brain that helps control breathing and arousal likely play a role in SIDS. Other research has focused on the way babies breathe while they're asleep — especially their response to low blood oxygen levels (hypoxia) — and on heart function Investigations link SIDS and long QT syndrome, an electrical disturbance in the heart that causes sudden, extremely rapid heart rates
41
SIDS Prevention
``` Avoid smoking Encourage supine sleeping position (back to sleep) Avoid soft mattress, blankets, pillows Discourage bed sharing Encourage breast feeding, pacifiers Avoid overheating during sleep Vary infant’s head position ``` Since 1992 death rate decreased by 40% Babies found huddled in corner with blankets over head; frothy blood tinged oral/nose; full diaper
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Chronic Lower Airway Problems
Asthma * Bronchopulmonary dysplasia (BPD) Cystic Fibrosis * Tuberculosis
43
Asthma
Pathophysiology Chronic recurrent lower airway disease with episodic attacks of bronchial constriction Multiple precipitating factors Age of diagnosis The decreased size of child’s airway due to edema & mucus leads to further compromise ``` MOST COMMON CHRONIC DISEASE OF CHILDHOOD: 5 million children suffer/yr; 80% dx before age 5 Precipitating factors: Exercise psychological stress respiratory infections changes in weather & temperature genetics ``` Occurs commonly during preschool years, but also presents as young as 1 year of age Labeled as RAD (Reactive Airway Disease)
44
Asthma Causative factors
``` Dietary habits Time indoors Energy efficient homes Decrease in breastfeeding Survival of low birth weight babies ``` Western dietary habits (which commonly include more fast foods and less fruits, vegetables, fiber, minerals, and other nutrients) Children are spending more time indoors watching television, playing video games, or using the computer and are, therefore, overexposed to indoor allergens making homes more energy-efficient may result in dust mites being trapped inside them Survival rates are now higher in low-birth-weight babies, who may be more susceptible to asthma Declining rates in nursing may be a contributor. Breast milk contains important anti-inflammatory substances, such as omega-3 fatty acids, which might protect against asthma
45
Asthma Assessment
History Physical Exam assessments SOB, shallow, irregular respirations, increased or decreased respiratory rate Pale, mottled, cyanotic, cherry red lips Restless & scared Cough Inspiratory & expiratory wheezing, rhonchi* Tripod position History Time of last attack & severity Presence of fever Medications, treatments administered * Diminished breath sounds could indicate decrease in air passage Higher incidence in September Back to school exposure to cold virus with lots of kids and inside Exercise increase with sports High ragweed allergens
46
Asthma management in hospital
``` Administer bronchodilators (know SE’s) Administer corticosteroids, inhaled and oral/IV as ordered Assess & monitor ABC’s Apply oxygen (Humidified if possible) Provide IV of LR or NS at a KVO rate Prepare for possible vomiting Assess pulse oximetry Intubate if airway management becomes difficult or fails ``` Beta-adrenergic drugs Rinse mouth after inhalers. Bronchodilators 1st, then corticosteroids.
47
Status asthmaticus
``` Sweat profusely Insists on sitting upright Severe respiratory distress Bronchospasm Either suddenly agitated or suddenly quiet Hypoxic ``` Medical emergency that may result in death Symptoms are refractory to initial bronchodilator therapy in the emergency department. Patients report chest tightness, rapidly progressive shortness of breath, dry cough, and wheezing. Therapy directed at improving ventilation Correct dehydration and acidosis with IV fluids; NPO Meds inhaled albuterol Cortisone Subq epinephrine or terbutaline
48
Asthma Prevention of attacks
Teach regarding correct use of peak flow meters, inhalers and nebulizers ``` Allergen control -Drug therapy-Sometimes hyposensitization No smoking near children Allergen control Avoid temperature extremes Meal planning Good hygiene Activity tolerance Moist air Correct use of peak flow meters, inhalers and nebulizers ```
49
Cystic Fibrosis
A multisystem disorder affecting exocrine glands. Transmitted by an autosomal recessive gene that both parents must carry for child to be affected Deletion of chromosome 7 Absence of gene decreases chloride-ion and water transport at cellular level 25% chance that each child will have the disease, 50% chance that each child will be a carrier. A chronic, sometimes fatal disease. Most common inherited disease of children One of the most common causes of childhood death Affects multiple organs so have MSOF NOT the most common chronic resp illness of children – asthma is. 70% of population is diagnosed by age 2 and 55% under age of 18 The mean survival age is mid 30’s CFF 2011 As they age: Infertile Hyponatremic Build up of amylase lipase trypsin Inability to absorb fat protein carbohydrates Chronic lung infections leading to vent dependence
50
Cystic fibrosis problems
Exocrine glands produce tenacious mucous leading to Obstruction of the small passageways of the bronchioles small intestine pancreatic and bile ducts. ``` Complications Malabsorption Syndrome Congestive heart Failure Diabetes Mellitus (IDDM)‏ Pneumonia ```
51
Cystic fibrosis manifestations
``` Recurrent respiratory infections Use of accessory muscles, nasal flaring Productive cough Barrel chest CHF Meconium ileus Steatorrhea FTT (failure to thrive) Deficiencies in the fat soluble vitamins (ADEK)‏ IDDM in older children Salty taste of skin due to high concentrations of sodium and chloride in sweat ```
52
CF tests to Dx and Tx
``` Sweat test-still best way to diagnose CF* Family hx of CF or positive newborn blood screening 72- hour fecal fat Fasting blood sugar Sputum culture and sensitivity PFT’s Prenatal chorionic villi sampling Liver enzymes ``` ``` The sweat test does not hurt and you can stay with your baby throughout the procedure. A special gel is put on your baby’s skin on an arm or leg and small patches with wires cover the gel for about 5 minutes. This causes your baby to sweat. The skin may feel warm and tingly while the patches are in place, but it is not painful. The sweat is then collected on a gauze pad or disk. After about 30 minutes, the gauze or disk is removed and taken to a laboratory to measure the salt. The sweat test takes about one hour from start to finish.Diagnosis: amount of chloride in sweat. It’s positive if greater than 60 meq/liter Family history Altered sweat electrolyte content Absent pancreatic enzymes Chronic pulmonary involvement ```
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More CF assessment
``` Stool specimen for absence of trypsin and fecal fat content Assess respiratory status Measure height and weight Assess for signs of malabsorption Assess for growth and development Serum albumin to assess nutritional status Metabolic panel for hydration status Liver enzymes to assess liver function ``` ``` Stool specimen for absence of trypsin and fecal fat content Assess respiratory status Measure height and weight Assess for signs of malabsorption Assess for growth and development Serum albumin to assess nutritional status Metabolic panel for hydration status Liver enzymes to assess liver function ```
54
Nsg Dx for CF
Ineffective Airway Clearance R/T tenacious and increased pulmonary secretions Impaired Gas Exchange R/T air trapping in alveoli secondary to airways obstructed by thick mucus Altered Nutrition: less than Body Requirements R/T poor nutrient absorption in the intestine
55
CF Interventions
Promote airway clearance and gas exchange Assess respiratory status Assess pulse oximetry Administer bronchodilators, antibiotics (inhaled and systemic) Perform chest PT, postural drainage & teach this Promote cough Position for comfort Administer IV and po fluids as ordered Assess vital signs especially temperature Monitor WBC count Assess color, odor and amount of sputum
56
CF Prevention of complications
Practice good handwashing/isolation from those with resistant bacteria, (Pseudomonas aeruginosa, Burkholderia cepacia ) Keep immunizations up to date Take antibiotics and do pulmonary hygiene as ordered No anti-tussives; cough is critical Promote optimum nutrition; teach re diet and to administer pancreatic enzymes with meals Promote effective individual/family coping
57
CF Nutrition
Promote optimum nutrition Hi calorie, Hi protein with supplemental snacks Administer pancreatic enzymes with meals and snacks Administer vitamin supplements Monitor weight Monitor I & O Note, color, frequency and consistency of stools
58
Newer Research and Tx for CF
Lung Transplant Hypertonic Saline New inhaled antibiotics to target resistant bacteria VX-770, faulty gene targeted therapy in Phase 3 Clinical Trials .Compacted DNA, In Phase 1 Clinical Trials
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Abx for Resp. Disorders
Penicillins- commonly used for ear infections Macrolides- used often in PCN allergy and with lower respiratory infections Erythromycin Cephalosporins-also used in ear infections, often 2nd line (10% cross reactivity/allergic reaction in pts with pcn allergy)