Peds PA exam 2 Flashcards
Physical Assessment of Head
Look for;
torticollis
examine from above, side, with fingertips, for sutures; Face: forehead and upper orbit, orbitonasal zygomatic area, maxillomand. area, posterior cranial vault
Macrocephaly Assessment
serial head measures measure parents head developmental hx transilluminate skull look for skeletal dysplasia listen for bruit look for sunset eyes signs of neurocutaneous disorder
Causes of Microcephaly
Genetic defect Karyotype intrauterine infections antenatal radiation exposure to drugs/chemicals perinatal insult
Scaphocephaly/Dolichocephaly
positional problems in premies
Plagiocephaly
common with back head sleep position
no growth perpendicular to sagittal suture
bitemporal narrowing
cranial elongation in anterior-posterior direction
face will be pushed toward affected side
frontal bossing due to pressure on the occiput
ear on affected side will be forward
parallelogram shape
craniosynostosis
rare-skull sutures close and bony plates of skull become fused too early in ilfe
develops while fetus is still in womb
1/2500 births
Sagittal suture closes prem.
Simple or isolated craniosynostosis or complex/syndromal
Brachycephaly
b/l coronal or lambdoidal early
broad skull with short base and recessed lower forehead
brachy/turi/acrocephaly is associated with: Apert syndrome, Crouzon syndrome, Pfeifffer syndrome
Crouzon’s Syndrome
craniosynostosis most often of coronal and lamboid, sutures
underdeveloped midface w/ receded cheekbones/bulging eyes
Ocular proptosis-which is prominence in eyes due to shallow orbits
Esotropia & wideset eyes
Apert Syndrome
Ø Very high brachycephalic head Ø Severe syndactyly affecting all limbs. Ø Syndactyly involves bony fusion. Ø Apert is the second most common craniofacial syndrome after Crouzon.
Oxycephaly
closure of all sutures except squamosal
Lambdoidal Craniostosis
if there is lambdoidal synostis the head can’t expand in frontal region on affected side of the head, so the growth will be affected on unaffected side
growth restriction on affected side»overgrowth everywhere else
Trapezoid
Alopecia Areata Vs Tinea Capitis
AA:
distrib: patchy, multifocal
onset: abrupt, waxes/wanes
appearance: thin w/ abrupt bare patches, exclamation point hairs
Degree of shed: prominent
age of onset: any, first before 20 usually
other: personal or fam hx of autoimmune, need TSH
Tinea Capitis:
distrib: specific area-focal or multifocal
onset: gradual or abrupt
appearance: none to some inflamm, scaling
age of onset: common in childhood, any
other: contact w/ animal w/ dermatophytes
midface hypoplasia
assoc w sleep apnea & stridor
dental problems
maybe hearing problems
Micrognathia
mandible has not grown asm uch asit should-small mandible and chin
surgical advancement of mandible and chin via intraoral route
prognathic mandible
lower jaw is too prominent
occurs w/ congenital overgrowth of lower jaw
pahologic such as gigantism due to pituitary overgrowth and other tumors
ex) Jay leno
Sturge Weber Syndrome
non elevated purple venous malormation in distribution of trigeminal nerve, opthalmic division
high incidence of dev. delay
ocular complications like glaucoma on affected side
Parotiditis- Types & causes
Wind parotiditis: air forced into parotid duct with glandular swelling- when children learn to lay wind instrument or blow balloons
Viral causes: mumps, parainfluenza types 1 and 3, HIV, CMV, coxackie
Bacterial: staphylococcus aureus, streptococcus species
can have tender and red gland w/ significant systemic symptoms
Differential Diagnosis for Parotiditis
recurrent parotiditis associated w/ pain & fever
abscess of parotid
rare: cat scratch, TB
metabolic: slow, progressive, painless, no inflammation
starvation-hypoproteinemia, anorexia, cushing
Obstructive enlargement-stones
Autoimmune: lupus w/ recurrent swelling as initial sign, sjogen syndrome
Myopia
when anterior-posterior diameter ofe eye is too long relative to refracting power of cornea and lens
hyperopia
eye is too short relative to refracting power of eye. focal point of image occurs posterior to retina and image that forms ont he retina is blurred
Astigmatism
refractive eye error that causes blurred vision
when optical system of the eye ( cornea usually) is not perfectly spherical
strabismus
when eyes do not move in synchronous pattern
Esotropia
type of strabismus-inward deviation
exotropia
outward deviation, type of strabismus
latent strabismus
apparent only on dissociation of vvision of the eyes (covering one eye)
phoria-exophoria, esophoria, hyperphoria
hypertropia
misalignment of eyes (strabismus) whereby visual axis of one eye is higher than the fellow fixating eye
hypotropia
similar condition, focus being on the eye w/ visual axis lower than fellow fixating eye
Esophoria
tendency toward inward deviation of eye, usually b/c of extraocular muscle imbalance
exophoria
form of heterophoria-tendency of eye to deviate outward
comitant strabismus
same deviation in all fields of gaze
incomitant strabismus
limited eye movement and size of deviation is different in different fields of gaze
most commonly where there is paralysis of one or more extraocular muscle
Neurological- lesions of 3rd 4th CN from trauma, tumor, infection, ICP
Muscular- direct involvement of muscles
Myasthenia gravis, congenital problems
Causes of comitant strabismus
hereditary, familial predisposition
sensory deprivation
Accomodative- freqeuent convergence in children who are hypermetropic
Testing for Stabismus
hirschberg/corneal light red reflex/bruckner cover test evaluate extraocular movement binocular status-steropsis tests
Amblyopia
poor vision caused by abnormal dev secondary to abnormal visual stimulation
pressence of associated findings: strabismic amblyopia, anisometropic amblyopia, deprivational amblyopia
Strabismic amblyopia
results from abnormal binocular interaction
causes fovea of 2 eyes to be presented iwth different images
Anisometropic Amblyopia
fovea are also presented with different images r/t unequal refractive error
refractive amblyopia
most commonly in hyperopic patients, may occur in patients w myopia or astigmatism as well though
Deprivation amblyopia**
least common and most serious type of amblyopia***
severe visual deprivation due to occlusion of visual axis/distortion of foveal image
Vision Screening
routine screening at age 18 been changed to a risk assessment
visual acuity screen is recommended at ages 4 and 5 years
instrument based screening to assess risk at ages 12 and 24 months
+ well visits at 3-5 years
0-6month old vision screening
ocular history
vision assessment
external inspection
ocular motility assessment-should fix and follow by 6months
pupil examination
red reflex exam- refer infants with abnormal red reflex or history of retinoblastoma in parent/sibling
6-12 month screening
ocular hx, external inspection, red reflex, pupil exam, ocular motility, visual acuity fixte and follow
wen to refer: infants with strabismus, w/ chronic tearing or discharge
who fail instrument based screen
12mo-3years screening
the usual testing + at age 3, start vision screening using optotypes
Refer; who cannot read at least 20/40
4-5 years old screening
usual tests, instrument based screen plus using optotype
over 5 years old screening
ocular hx, vision assessment, external inspection of eyes/lids, ocular motility assessment, pupil examination, red ref, visual acuity, + opthalmoscopy
Refer: who cannot read at least 20/32 w either eye; must identify majority of optoypes on 20/32 line and those not reading at grade level
Assessing for Ocular motility
3-6 feet in front of child
patient follow your giner w eyes- Double H
-check convergence- go in toward patient’s nose
Hirschberg test
Negative: both reflexes are located .4mm nasal to center of pupil
Positive: if one reflex is located in a position other than .5mm nasal, suspect strabismus
Cover Test
watch for flick movements of eye; small angle strabismus
Bruckner/Red Reflex
darken room
adjust opthalmoscope so light is not TOO bright
dial to zero
use left hand and left eye to examine LEFT eye; right to right
have patient stare at point on wall
identifies strabismus as well as asymmetric refractive errors
Unequal refractive errors: reflexes will differ in brightness
Pupil Assessment
integrity dependes on intact iris, CN II and III, and sympathetic nerves innervating eye
Hippus
Normal pupil is continuous motion dilating in and out by small contractions
more prominent in pediatric patients exposed to bright light
Direct & conensual
direct- ipsilateral constriction
consensual- flash light in one eye and other pupil reacts
Aniscoria
Simple
0.4mm or more difference in size of R and L pupil not related to drugs, ocular injury to inflammation
Relative Afferent Pupillary Defect/Marcus Gunn Pupil
most common pupil problems
occurs w/ significant optic nerve or retinal disease & when there’s a difference in disease process between R and L eyes
In Marcus Gunn pupil, the pupil where the light is shined
will constrict strongly when light is shining into the eye
but as light moves to illuminate the abnormal eye, both pupils dilate (react as though the light was dimmer
l The eye that dilates when the light is shown on it, is the
abnormal or Marcus Gunn Pupil
Iris coloboma
isolated iris coloboma does not interfere w/ vision
Aniridia
absence of iris
1/3 associated w/ Wilm’s tumor
Assessment of Red Eye
External inspection of eyes and lids- suspect orbital injury if eye is red at limbus (where iris meets sclera);
-ocular motility, pupil exam, vision screen
Ciliary Injection
injection at limbus!! Does not spare limbus
lessens as it moves to palpebral conjunctiva
seen in uveitis, keratitis, acute angle glaucoma
Chemosis
swelling of conjunctiva from allergic conjunctivitis and in kids w/ pharyngoconjunctival fever
Subconjunctival Hemorrhage
blood under conjunctiva
causes: trauma, sudden increase of pressure in chest
S&S: assoc w/ history of trauma
-if mild and no other sign of ocular injury- benign will disappear in 1 week, no tx
corneal abrasion
trauma, foreign body/chemical exposure
S&S:
sensitivity to light, extreme pain, trouble opening eye, tearing, foreign body sensation
dx: area of epithelial defect stains w fluorescein dye and lights up w/ blue light
Hyphema
blood between anterior chamber (space between the iris/cornea)
from trauma, blunt or penetrating; can be assoc w other eye injury
Conjunctivitis- bacterial
bacterial- H influ, staph, chlamydia
RED, purulent discharge* THE hallmark, swollen lid, one or both eyes
**antibiotic drops do NOT cure gonococcal conjunctiivitis- suspect gonorrhea and send to ER-need IV antibiotis
Viral Conjunctivitis
Adenovirus, Herpes simplex, zoster, molluscum contagiosum
S&S- irritation, light sensitivity, swollen lids, watery discharge, some mucous, NOT purulent (distinguishes from bacterial)
often enlarged tender lymph node is present in front of ear
Conjunctivitis- Herpes
skin vesicles
corneal infection w “dendrite”- hallmark
Allergic Conjunctivitis
environmental allergen, seasonal, sneezing/congestion
Hallmark: ITCH
Blepharitis
eyelid inflammation/infection
cause- staph infection at lash base
skin oil glands inflamed
*lice can cause this!
Chalazion
obstructed meibomian gland
hyphema
layering of blood in anterior segment
hypopyon
pus in anterior segment
iridocyclitis
inflammation of iris
iridogonesis
quivering of iris when patient moves eye
synechia
adhesion between iris and cornea
conductive hearing loss
impairment of outer and or middle ear conductive mechanism only; sound does not make it past the TM. ex) wax build up, foreign object
Screening; Eustachian tube dysfunction, ear fluid, hole in eardrum, fixed middle ear bone
Sensory Hearing Loss
damage of cochlea (outer hair cells or outer & inner hair cells)
mixed hearing loss
presence of both conductive and sensory impairment
neural hearing loss
damage to auditory neurons (spiral ganglia) and / or auditory branch of eith nerve. ex) auditory neuropathy and dysynchrony
central hearing loss
damage to auditory structures in brainstem, thalamo cortex/cortex.
Early Hearing loss causes
maternal rubella, CMV, genetic disorders causing malformation of cochlea, sepsis, o2 deprivation, hyperbillirubinemia
Medications-lasix, aminoglycosides, vanco
Genetic Syndromes associated with Hearing loss
Waardenburg: white forelock and eye abnormalities
Jerveil syndrome, Lange Nielson: deafness assoc w/ long QT –>sudden death! EKG!
Renal Abnormalities
kidneys and ears form at same time in utero;
Alport’s syndrome- hearing loss + hematuria
sensorineural hearing loss- screening
noise induced hearing loss
presbycusis
meiniere’s (more adults)
tumors of auditory nerve- Acoustic Neuroma causes sudden progressive loss
External Assessment
shape, size, (large: fragile x), symmetry, variations frmo normal
presence of dimples, tags, nodules: remnant of first brachial cleft
position of ear- rotated? should be along straight plan with outer canthus of eye
swelling, drainage, erythema, enlarged pre/post auricular nodes, external otitis w/ cellulitis causes ear to protrude
Brachial Cyst
*pseudomonas smell** distinct odor
Crouzon’s Ears
low, rotated backwards