Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

Peds > Peds ID uworld > Flashcards

Peds ID uworld Flashcards

1
Q

causes of meningitis in children

A

v3 mos

  • GBS
  • e coli and other gram negatives
  • listeria
  • herpes simplex virus

3 mos to 10 yrs

  • s pneumo
  • neisseria meningitidis

^11 yrs
-neisseria meningiditis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

tf

low wbc can be a sign of sepsis in an infant

A

t

espec w left shift (bands)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

define neonatal sepsis

A

systemic bacterial infection v28 days of life

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

define early vs late-onset neonatal sepsis and what bug is the most common cause of each

A

early v7 days of life

late 7-28 days of life

GBS is the most common cause of both

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

most common cause of meningitis in peds v3 mos old

A

GBS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

how have early onset vs late onset neonatal sepsis incidence changed relatively to each other and how is this accomplished

A

early onset, v7 days vertical transmission from mom during birth, has been much reduced thanks to Universal Screening and intrapartum abx

late onset 7=28 days horizontal transmission (person to person with unwashed hands) has not changed in incidence

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

signs of neonatal jaundice

A

non-specific

poor po, irritable, hyperthermic or hypothermic, respiratory distress, vomiting, jaundice…

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

symptoms of neonatal meningitis

A

hypotonia, full fontanelles, apnea, seizures

plus nonspecific sepsis symptoms
poor po, irritable, hyperthermic or hypothermic, respiratory distress, vomiting, jaundice.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

TF
all neonates with suspected sepsisshould get bcx ucx csfcx and empiric abx

why or why not?

A

T
high incidence in newborns
non-specific presentation
high morbidity and mortality

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

TF
h flu b is a common cause of newborn sepsis

why or why not?

A

F

  • low prevalence (herd immunity from older age vaccinations)
  • protective maternal antibodies
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

when to think Listeria vs GBS as cause of newborn sepsis

A

usually GBS in kid v3mos, just the most common

think more Listeria if seems pregnant mom had flu-like symptoms after unpasteurized dairy, canned meats

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

chorioretinitis, hydrocephalus, intracranial calcifications in newborn think…

A

congenital toxoplasmosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

duration and symptoms of catarrhal phase of pertussis

A

1-2 weeks

mild cough, rhinitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

duration and symptoms of paroxysmal phase of pertussis

A

2-6 weeks cough with inspiratory whoop, posttussive emesis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

duration and symptoms of convalescent phase of pertussis

A

weeks to months

gradual resolution of cough, whoop, posttussive emesis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

diagnose pertussis

A

culture or pcr of nasopharynx
(or dx clinically if classic symptoms)

lymphocyte predominant leukocytosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

treat pertussis

wait to confirm diagnosis or treat based on clinical suspision?

A

macrolide (azythromycin, clarithromycin, erythromycin)

treat based on clinical suspicion, do not wait to confirm with nasopharyngeal pcr or cx or lymphocytosis on labs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

paroxysmal cough, posttussive emesis, subconjunctival hemorrhages, lymphocytosis
think…

A

pertussis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

tf

prior pertussiss infection and/or immunization with acellular pertussis vaccine provide lifelong immunity

A

F
immunity to pertussis wanes
that is why we give 5 DTaP doses during infancy
boost with Tdap age 11-18 and each pregnancy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

heterophile antibody test for…

A

mono

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

tf
paroxysmal cough, posttussive emesis, subconjunctival hemorrhages, lymphocytosis
in unvaccinated patient
think flu

A

F
flu sx fever, cough, myalgias

this patient likely has pertussis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Congenital Rubella Syndrome
clinical triad
diagnosis
prevention

A

cataracts/glaucoma
sensorineural hearing loss
congenital heart disease (PDA)

Rubella IgM
PCR

maternal immunization with live attenuated rubella prior to conception

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

newborn
SGA small for gestational age
cataracts/glaucoma
PDA

think…

A

congenital rubella syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

prenatal maternal infection with rubella looks like…

A

asymptomatic
vs
prodrome fever cough conjunctivitis followed by diffuse maculopapular rash

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

when does maternal infection with rubella transmit greatest risk to fetus

A

1st trimester

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

common presentation of congenital toxoplasmosis

A

maternal exposure to cat litter

macrosephaly, chorioretinitis, diffuse intracranial calcifications

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

infectious disease reasons to perform c-section

combine c-section with for even better results

A

maternal HIV with high viral load
active genital herpes lesions

combo with prenatal antiviral therapy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

tf

congenital rubella syndrome is preventable with c-section and antiviral therapy

A

f
only preventable with vaccination.. no antivirals and transmission through placental blood flow in first trimester usually, well before birth

HIV and HSV preventable with c-section and antivirals

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

neonatal group B strep disease manifests as…

A

sepsis, pneumonia, meningitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

name a group A strep

A

strep pyogenes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q 
Acute Rheumatic Fever
epi
clinical features
late sequela
prevention
A

peak incidence 5-15yo
2x more common in girls

major JONES criteria Joints migratory arthritis, Carditis, Nodules subq, Erythema marginatum, Sydenham chrea

minor fever, arthralgias, CRP ESR, prolonged P-R interval

mitral regurgitation/stenosis late

treat GAS pharyngitis with Penicillin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
32
Q 
child with
friction rub, diffuse ST elevations
continuous irregular and rapid irregular jerks
subcutaneous nodules
elevated ESR

suspect…

A

Acute Rheumatic Fever
history of untreated sore throat and fever

(pericarditis, chorea)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
33
Q

tf

penicillin for strep pyogenes pharyngitis because does not self-resolve

A

F
does self-resolve

but give 10 days oral penicillin to prevent acute rheumatic fever

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
34
Q

treat acute rheumatic fever

why

A

long-acting intramuscular benzathine penicillin G until adulthood - to eradicate bacterial carriage to prevent recurrent ARF

corticosteroids for severe chorea

NSAIDS for pericarditis and arthritis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
35
Q

most common organism responsible for pericarditis and myocarditis in peds

also causes pharyngitis (herpangina) in young children

A

cocksackie virus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
36
Q

tf

strep pneumo is group A strep

A

F
strep pyogenes is group A strep

strep pneumo is “ungrouped”… because lacks lancfield carbohydrate antigen (low yield)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
37
Q

TF

There is a lyme vaccine

A

Fish

Not available ANYMORE… poor demand, side effect concerns, never a recommended immunization

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
38
Q 
Cat scratch disease 
Etiology
Gradual or Rapid onset
Clinical features 
Dx
Tx
A

Bartonella henslae from cat scratch bite or flea rarely

Gradual onset

Papule at scratch site
Regional tender lymphadenopathy
W/wo Fever of unknown origin ^14 days

Clinical dx w/wo serologies

Tx with Azythromycin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
39
Q

How long can LAN from cat scratch take to resolve

A

Can take 1-2mos

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
40
Q

TF

Cat scratch disease typically resolved spontaneously

A

T…

But still treat with Azyhromycin…

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
41
Q

How long can LAN from cat scratch take to resolve

A

Can take 1-2mos

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
42
Q

Where is lymphadenopathy from mycobacterium tuberculosis most common and is it tender?

A

Cervical lymphadenopathy

Strikingly NON-tender

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
43
Q

Pasturella multocida infection
Vector
Time to symptoms
Symptoms

A

Dog or Cat bite

1-2 days to symptomatic cellulitis or other soft tissue infection

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
44
Q

Salmonella enteritidis infection
Vector
Symptoms

A

Reptiles including Turtles

Mesenteric adenitis and enteritis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
45
Q

Impetigo
Microbiology
Clinical features
Treatment

A

Staph aureus, GAS pyogenes
-painful pruritic pustules, honey-crusted lesions

Staph aure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
46
Q

5 Factors predisposing to IMPETIGO

A

Colonization with staph aureus or gas pyogenes

Hot humid weather

Crowding

Poor personal hygiene

Preexisting lesion like eczema or big bite

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
47
Q

TF

Lymphadenopathy and fever w impetigo

A

T local lymphadenopathy common

F fever unusual

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
48
Q

TF

Impetigo is contagious

A

T

So counsel hand washing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
49
Q

TF

Impetigo is contagious

A

T

So counsel hand washing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
50
Q

Why treat Impetigo

How treat impetigo

A

Speed recovery and limit contagious transmissiom

Topical mupiricin if limited skin infection, to avoid side-effects and resistance of systemic abx

Systemic cephalexin dicloxacillin or clindamycin if extensive skin infection

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
51
Q

TF

Consider getting antibody titers, antistreptolysin O, and skin lesion swab and culture to dx impetigo

A

F mostly

antibody titers take a long time to get back and not really useful

52
Q

TF

Consider getting antibody titers, antistreptolysin O, and skin lesion swab and culture to dx impetigo

A

F mostly

antibody titers take a long time to get back and not really useful

53
Q

Rubeola aka

A

aka measles

54
Q 
Measles
Transmissiom
Presentation
Prevention
Treatment
A

Airborn transmission

Prodrome

55
Q

When is measles patient contagious

How long are infectious measles airborn particles capable of lingering in the air in a contained space like airplane or clinic waiting room

What kind of isolation / precautions

A

Most contagious during prodrome

56
Q

Child traveled to endemic country and got measles 6 weeks later, where did he get the measles?

A 
Probably domestically (thanks antivaccers)
Symptoms manifest 1-3 weeks after exposure
57
Q

What kind of precautions for MRSA and Rotavirus

A

Contact precautions

Gown, gloves

58
Q

What kind of precautions for flu and rsv

A

Droplet precautions - surgical mask

59
Q

What kind of precautions for measles and varicella

A

Airborne precautions - negative pressure, n95 mask

60
Q

Why the difference between droplet and airborne precautions

A

Droplets larger, shorter range - surgical mask appropriate

Airborne particles smaller, travel farther linger longer - need negative pressure and N95 masks

61
Q

What is the orbital septum

A

Fibrous portion of the eyelids extending from the orbital rim

62
Q

4 distinguishing features of orbital cellulitis vs periorbital

A

Pain with extraocular movement
Opthalmoplegia
Proptisis
Vision impairment

63
Q

Define hypothermia quantitatively

A

v36C 96.8F

64
Q

Empiric abx choice for neonatal sepsis

A

Amp and Gent

65
Q

TF

CT before LP in neonate suspected of meningitis

A

F

Open fontanelle relieves pressure so can LP without risk of hernia

66
Q

TF

Neonates do not experience cns herniation after lumbar puncture

A

T
F
Open fontanelle relieves pressure so can LP without risk of hernia

67
Q

Galactosemia
Pres
First step in management

A

Infant with lethargy vomiting jaundice hepatomegaly

Stop breast feeding

68
Q

Clinical presentstion of rubella infection in

Neonate

Peds

Adult

A

Neonate congenital sensorineural hearing loss, pda, cataracts, often intrauterine growth restriction / SGA

Peds fever and cephalocaudal spreading maculopapular rash

Adults fever and cephalocaudal maculopapular rash PLUS arthritis and arthralgias
OR asymptomatic

69
Q

Confirm congenital rubella syndrome suspected clinically

A

Infant serum IgM

70
Q

Congenital varicella syndrome

Clinical triad

A

Limb hypoplasia
Cataracts
Skin lesions (scarring)

71
Q

Congenital syphilis symptoms

A

Hepatomegaly
Nasal congestion/snuffles
Osteoarticular destruction
Maculopapular rash

Sensorineural hearing loss late if goes untreated

72
Q

Most common pathogens of orbital cellulitis

A

Staph aureus
Strep pneumo
Other strep

73
Q

Laterality of eye swelling in cavernous sinus thrombosis

A

Often starts unilaterally but quickly progresses to bilateral (24-48 hours)

74
Q

Cranial nerves passing through cavernous sinus, can be affected by cavernous sinus thrombosis

A

III IV V VI

75
Q

Inflammatory demyelinating condition causing acute vision loss and pupillary defects

A

Optic neuritis

76
Q

Most common causes of osteomyelitis in infants and children

A

Infants
STAPH AUREUS
GBS
Ecoli

Children
STAPH AUREUS
Strep pyogenes

77
Q

When to suspect
Staph epidermidis
Pseudomonas
Klebsiella

As causes of osteomyelitis

A

Staph epidermidis - prosthesis

Pseudomonas - uti or urinary instrumentation hx

Klebsiella - uti hx or urinary instrumentation hx

78
Q

Cat bite
Microbiology
Management

A

Pateurella multocida
Gram negatives

Irrigate/clean

Amoxicillin/clavalunate (Augmentin) ppx
-amox gets pasteurella, added clav grants coverage to oral anaerobes

Td booster

Do not suture closed

79
Q

TF

Obs and close follow-up for cat bite not located on hands feet or genitalia in immunocompetent patient

A

F
Can obs in this situation if dog or human bite

But cat bites need augmentin (amox/clavalunate) because risk of infection is much higher

80
Q

TF

Azithromycin pox for cat scratch

A

F

Azithromycin does get bartonella but no need for ppx for scratch – ppx with augmentin is for cat BITES

81
Q

Clindamycin coverage

A

Gram positive skin flora
(Staph aureas, strep pyogenes

Oral anaerobes

82
Q

lyme endemic areas in US

A

a little Virginia and north of there va md de pa ny new england

also minnesota and wisconsin… west great lakes`

83
Q

erythema migrans

A

spreading annular rash with central clearing

e.g. lyme

84
Q

lyme causative organism

A

borrelia burgdorferi

ixodes scapularis vs anaplasmosis vs babesiosis (tick)

85
Q

first sign of lyme disease usually

how long after tick bite

A

erythema migrans (spreading annular rash with central clearing

1-2 weeks after bite

maybe flu-like fatigue headache myalgias arthralgias
and regional lymphadenopathy

86
Q

what percentage of lyme patients recall tick bite

A

25%

87
Q

prevent lyme

A

avoidance and rapid recognition and removal of ticks

  • repellants DEET (NN diethyl metatoluamide), Permethrin
  • long clothing
  • tick checks and bathing (wash away unattached ticks)
88
Q

lyme prophylaxis

A

doxycycline reserved for pts with ixodes scapularis (tick) attachment for ^36 hours

89
Q

swimming in brackish water off the new england coast may expose to what infectious disease

A

vibrio vulnificus

  • cellulitis
  • sepsis with hypotension and bullous skin lesions
90
Q

most common viral meningitis pathogens

A

90% are non-polio enteroviruses, such as echovirus and coxsackie virus

91
Q

age preference of viral meningitis

A

infants

and decreases with age

92
Q

viral meningitis
csf analysis
treatment
time to resolution

A

pleocytosis with lymphocyte predominance (may be neutrophil predominance early)

protein normal to slightly elevated

glucose normal

supportive treatment
7-10 days to resolution in most patients

93
Q

bacterial meningitis

csf analysis

A

pleocytosis with neutrophil predominance
increased protein
decreased glucose
bacteria on gram stain

94
Q

tuberculous meningitis
csf analysis
acuteness of presentation

A

lymphocyte predominant pleocytosis (like virus, not bacteria)

very high protein, low glucose (like bacterial)

subacute presentation

95
Q

which is the more common viral meningitis, ebv or enteroviruses?

A

non-polio enteroviruses (coxsackie, echo) 90% of cases

96
Q

when to suspect the following as causes of acute cervical adenitis

staph aureus
strep pyogenes
anaerobic bacteria (prevotella buccae)
bartonella henslae
mycobacterium avium
adenovirus
abv/cmv
A

staph aureus - pronounced erythema, tenderness

strep pyogenes - pronounced erythema, tenderness

anaerobic bacteria (prevotella buccae) - dental caries, peridontal disease

bartonella henslae - papular nodular site of cat scratch or bite

mycobacterium avium - gradual onset, nontender

adenovirus - bilateral, pharyngoconjunctivitis

ebv/cmv - bilateral, mono

97
Q 
acute unilateral lymphadenitis
most common cause
most common age
most common nodes affected
what happens if left untreated
tx
A

strep pyogenes GAS
staph aureus

v5yo
submandibular nodes
untreated can progress to suppuration and abscess
tx w empiric clindamycin (MRSA and GAS coverage)

98
Q

most common cause of acute bilateral lymphadenitis

A

adenovirus and other URIs

ebv and cmv too

99
Q

most common cause of Subacute unilateral lymphadenitis in peds

A

mycobacterium avium

-very slow onset, non-tender lymph node

100
Q

perianal pruritus, especially at night,in peds
think…
dx
tx

A

pinworm (enterobius vermicularis)
eggs on tape test
albendazole or pyrantel pamoate
-treat patient and all household contacts

101
Q

in addition to nocturnal perianal pruritus, how might pinworm enterobious vermicularis present in a prepubertal female

A

vulvovaginitis

102
Q

life cycle of pinworm enterobious vermicularis

A

adults live in intestines
females migrate distally to lay eggs in perianal skin at night
scratch, mouth, swallow, repeat

103
Q

typical distribution of atopic dermatitis (eczema) in kids

does it typically involve the groin or genitals?

do symptoms change with time of day?

A

popliteal and antecubital fossas (flexor surfaces)

spares groin and genitals typically

no symptom change with hour, present throughout

104
Q

healthy prepubertal female with vaginal pruritus, erthema, discharge, history of recent abx use, think…

A

candidal vulvovaginitis

105
Q

differentiate vulvovaginitis from pinrowrm enterobious vermicularis vs candida

A

pinworm enterobiuos vermicularis will have excoriated and erythematous perianal area as well

106
Q

dry thickened erythematous plaques on popliteal and antecubital flexor surfaces in a school-aged child think..

A

atopic dermatitis (eczema)

107
Q

define lichen sclerosus

A

benign, chronic, progressive dermatologic condition characterized by marked inflammation, epithelial thinning, and distinctive dermal changes accompanied by pruritus and pain. usually occurs in the anogenital region, but can develop on any skin surface and in women, men, and children

108
Q

perianal and vulvar pruritus with vaginal discharge or bleeding, hypopigmentation and ecchymosis of affected area, think…

A

lichen sclerosus

109
Q

classic croup(laryngotracheitis
pathogen
age
symptoms

A

parainfluenza
6mos-3years
barky cough, stridor, hoarse voice

110
Q

classic epiglottitis
pathogen
age
symptoms

A

h flu
unvaccinated children
sore throat, dysphagia, drooling, tripoding

111
Q

classic bronchiolitis
pathogen
age
symptoms

A

RSV
v2yo
wheezing, coughing

112
Q

what does steeple sign on neck xr signify

A

subglottic edema eg in croup

113
Q

treatment
and purpose of treatment
for croup

A

corticosteroids (dexamethasone) for mild cases
racemic epi nebulized for stridor at rest

to reduce subglottic edema

114
Q

laryngomalacia
mechanism
pres
infectious symptoms?

A

collapse of supraglottic structures during inspiration

chronic inspiratory stridor beginning in neonatal period, worse in supine position

no infectious symptoms (no fever, rhinorrhea, congestion, cough)

115
Q

retropharyngeal abscess
age
symptoms

A

v4yo

high fever, muffled voice, limited neck rotation due to pain

116
Q 
malaria
path
clinical features
complications
dx
prevention
protective factors
A

path - plasmodium falciparum vivax ovale malariae parasites by mosquito bite

clinical features - CYCLIC FEVERS q2-3days with OTHER NON-SPECIFIC SYMPTOMS aka periodic febrile paroxysms, nonspecific malaise headache nausea vomiting abdominal pain diarrhea myalgia pallor jaundice petechiae hepatosplenomegaly

complications

  • peds - hypoglycemia metabolic acidosis seizure coma
  • adults - jaundice acute renal failure acute pulmonary edema

dx - thin and thick peripheral blood smears with giemsa-stained parasites visualized microscopically

prevention - antimalarials (atovaquone, doxycycline, mefloquine, chloroquine, hydroxychloroquine), nets, insecticides

protective factors - hemoglobinopathies (HbS HbC thalassemias), partial immunity from prior malaria infection

117
Q

why is malaria particularly scary in infants and young children

A

increased susceptibility to life-threatening complications

-eg CEREBRAL MALARIA seizures delirium coma

118
Q

why is ciprofloxacin kept on hand by travelers

A

to treat traveler’s diarrhea

-e coli, campylobacter, shigella, salmonella

119
Q

malaria should be suspected in…

A

any ill patients who have traveled to an endemic tropical region - symptoms are NON-SPECIFIC malaise headache nausea vomiting abdominal pain diarrhea myalgia pallor jaundice petechiae hepatosplenomegaly

ESPECIALLY if FEBRILE PAROXYSMS aka cyclic fevers q2-3 days

120
Q

fever and parotitis after a non-specific prodrome in an unimmunized child
think…

A

Mumps

121
Q

Mumps presentation
complications
age most common
how do symptoms vary by age

A

fever and parotitis after a non-specific prodrome in an unimmunized child

aseptic meningitis (fever headache nuchal rigidity)
orchitis in postpubtertal males
-infertility possible

most common in school-age children - mild disease or even asymptomatic

more severe and more complications (aseptic meningitis, orchitis) in adolescents and adults

122
Q

primary reason for universal mumps vaccination

A

prevention of orchitis

123
Q

tf

facial nerve palsy and mastoiditis are complications of parotitis from mumps

A

false
mumps - fever and parotitis in unvaccinated… potentially aseptic meningitis and orchitis as compx

facial nerve palsy and mastoiditis can complicate otitis media

124
Q

tf

pneumonia is a complication of primary measles or varicella infections

A

T…!

125
Q

tf

mumps is usually self-limited

A

T
self-limited fever and parotitis

but may be complicated by aseptic meningitis or orchitis/infertility

Peds (12 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions