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SET 7 (Ortho & Pedo) > Pedo (doc micks) > Flashcards

Pedo (doc micks) Flashcards

1
Q

When does crowns of primary teeth begins to calcify?

A

3.5-6mos. utero
4-6mos. utero
14-24 wks utero
2nd trimester

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2
Q

‼️📌Remember

Enamel of primary central incisor is completed at what age?

A

1 1/2 - 2 1/2 months of age

15month old child except Canine & 2nd molar

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3
Q

6 y/o molar

A

1st molar

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4
Q

12 y/o molar

A

2nd molar

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5
Q

Crown permanent incisor

A

Apical & Lingual

Inclination : Labial Inclination

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6
Q

What do you where in primary tooth

A

Overretained deciduous tooth

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7
Q

📌📌‼️‼️

It takes___ years for most crowns to complete its formatiom , except for 1st molar____ & cuspid ____

A

4-5 yrs
3yrs
6yrs

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8
Q

‼️📌Crowns of permanent central incisors are completely calcified at what age?

A

4-5 yrs

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9
Q

From start of calcification to root completion it would take ____ yrs
except canines ___ yrs

A

10 yrs

13yrs

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10
Q

Permanent teeth erupt when approximately ____ of its root is completed

A

2/3

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11
Q

Apex fully developed _____ years after eruption

A

2-3yrs

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12
Q

Max.

A

61245378

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13
Q

Mand.

A

61234578

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14
Q

present at time of birth
mandibular incisor region
hypocalcified

A

Nata teeth

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15
Q

present within the first 30 days after birth

hypocalcified

A

Neonatal Teeth

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16
Q

Tetracycline staining can affect a child’s teeth until what age?

A

<8 y/o

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17
Q

5 y/o taking tetracycline side effects can be:

A

Canine
PM
2nd molars

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18
Q

Tx of Natal teeth

A

Extraction to prevent aspiration

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19
Q

Natal teeth of mobile

A

Extraction to prevent aspiration to the Righ lung

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20
Q

Natal teeth not mobile

A

No tx

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21
Q

‼️📌📌📌Nolla’s Stage of Tooth Development‼️‼️

A 
Stage 0- Absence of crypt
Stage 1- Presence of crypt
Stage 2- Initial calcification
Stage 3- 1/3 of crown completed
Stage 4 - 2/3 of crown completed
Stage 5- crown almost completed
Stage 6- crown completed, root formation begins
Stage 7- 1/3 of root completed
Stage 8- 2/3 of root completed
Stage 9 - root almost completed ; open apex
Stage 10- root completed ; closed apex
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22
Q

Characteristics of Primary Mand. 1st molar (D)

A 
Doesn't resemble any other teeth
Pot belly appearcance
No central fossa
BIG MB cervical ridge
rounded & short distal surface
Flat & long mesial surface
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23
Q

early extractiom of primary

A

Delayed eruption
drifting
Malocclusion

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24
Q

REMEMBER‼️📌📌

only ant. teeth that have a greater width than height

A

Primary Maxillary Central Incisors

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25
Q

All anterior teeth

A

4 lobes

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26
Q

All PM

A

4 lobes

expcept mand. 2nd PM - 5 lobes

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27
Q

First molars

A

5 lobes

Max. 1st molar - 4 lobes & 5 lobes

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28
Q

2nd molars

A

4 lobes

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29
Q

Most common lesion primary molar

Permanent

A

Bifurcation lesion
Reason: Because many accessory canal pulpal floor
Apical lesion (Periapical)

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30
Q

separation anxiety

mother is cenrer of his world

A

2 yrs old

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31
Q

big talker
Brief attention span
exhibit tantrums

A

4 yrs old

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32
Q

loves to learn new thing
likes to dramatize things
“susceptible to praises”

A

8 yrs old

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33
Q

increase interest in appearance

peer group oriented

A

12 yrs old

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34
Q

📌‼️‼️BE

What id the general characteristics 12 y/o

A

Reject Parental Authority

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35
Q

‼️📌📌First dental visit

A

As soon as first tooth eruptd or within 6mos. of first tooth eruption
no later than first bday

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36
Q

First tooth beush

A

First tooth erupts

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37
Q

when 2 teeth touchong

A

First flossing

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38
Q

‼️📌📌BE

Infant oral care cleaning the gauze bedtime

A

0-6mos.

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39
Q

minimal apprehension

A

Cooperative

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40
Q

deficient in comprehension & or communication skills

A

Lacking cooperative ability

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41
Q

Special needs

A

Autism
Cerebral Palsy
ADHD
Down syndrome

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42
Q

inclined plane / tongue blade

A

Ant. Cross bite

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43
Q

capable of behavimg but disruptive in dental setting
3-5 y/o
6-8 y/o

A

Potentially cooperative

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44
Q

temper tantrums

3-6 y/o

A

Uncontrolled

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45
Q

overprotective parents

I donl’t want to attitude

A

Defiant

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46
Q

shielding or hesitating behavior

3-5 y/o preschool

A

Timid

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47
Q

white knuckler

7 y/o

A

Tense cooperative

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48
Q

absence of tears

A

Whinning

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49
Q

spoiled brat

A

Incorrigible

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50
Q

least ideal

A

Fearful

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51
Q

‼️📌BE

What should the dentisf do when communicating mute child?

A

A. Maintain a natural communication

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52
Q

‼️📌 Frankl Behavioral Rating Scale
Fearful
shows extreme negativism
Definitely negative

A

Frankl I

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53
Q

negative

shows negative attitude but NOT PRONOUNCED

A

Frankl II

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54
Q

Positive

Likes & accepts thr tx but CAUTIOUS

A

Frankl III

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55
Q

Definitely positive

Laughing & enjoying the situation

A

Frankl IV

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56
Q

uses voice to control child , FIRM

establish authority

A

Voice Control

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57
Q

verbal praises & toys

A

Positve Reinforcement

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58
Q

diverts px education short attention span

A

Distraction

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59
Q

posture
body language
patting the shoulder

A

Non- verbal communication

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60
Q

gradual exposure to new stimuli or experience

A

Desensitization

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61
Q

learning by observation

A

Modeling

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62
Q

Aversive Technique or

A

HOM

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63
Q

Used for px who can understand but is DEFIANT

Not for handicapped & very young children

A

Aversive Technique / HOM

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64
Q

uses physical restraining equipments

should not be presented as punishments

A

Protective Stabilization

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65
Q

Protective Stabilization

A

Parent
Assistant
head lock

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66
Q

Passive restraint

A

Papoose

Pedi wrap

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67
Q

Determines a child behavior

A
  1. Age
  2. Mothers’ Anxiety
  3. Past medical history
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68
Q

‼️📌📌‼️

Drugs don’t use for premedicating 8 y/o severe apprehension

A

Naloxone (narcan)

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69
Q

Most common drug oral sedation

A

Chloral Hydrate

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70
Q

Advantages of Sedation of Chloral hydrate

A

Ease of Administration

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71
Q

Disadvantages of Chloral Hydrate

A

Slow onset of action
Child may spit
Nausea & vomiting
Respiratory depression- excessive dose

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72
Q

most common inhalation

A

Nitrous Oxide Oxygen Inhalation

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73
Q

mild odourless & reversible inhalation drug

Produces ONLY ANALGESIA & minimal conscious sedation

A

Nitrous Oxide Oxygen

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74
Q

Most common adverse effect of Nitrous Oxide

A

Nausea

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75
Q

Adminstering concentration of N2O

A

70% N202

30% O2

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76
Q

Maintaining concentration

A

30% N202

70% O2

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77
Q

Properties of NO

A

Anxiolytic
Analgesic
Amnesic

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78
Q

oversedste ma reverse ant effect

A

Flumazenil (IM)

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79
Q

Proper response to N2O2?

A

Feeling of floating & giddiness tingling of digits

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80
Q

Combined volume of gases delivered?

A

3-5 L (min dental decks

4-6 L

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81
Q

What to do during termination of N2O2?

A

100% 02 inhaled not <3-5mins prevent Diffusion Hypoxia

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82
Q

no peer involvement

A

Solitary

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83
Q

observing others play

A

On looking

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84
Q

play activity along side

A

Parallel play

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85
Q

with interaction

A

Associative play

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86
Q

highest form of play

A

Cooperative

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87
Q

📌‼️‼️‼️REMEMBER

1st bacteria to colonize the mouth after birth?

A

Strep. Salivarius

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88
Q

Bacteria associated with PLAQUE?

A

Strep. Sanguis

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89
Q

pH of Saliva

A

6.2- 7.6

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90
Q

Critical enamel ph

A

5.5-5.7

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91
Q

Initial sign of dental caries

A

White spots & Incipient caries / white spot lesions

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92
Q

tooth mineral is affected firtmwhen there is sctivd csries?

A

CRbonate

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93
Q

Incipient proximal decalcification

Proximal lesin primary molar

A

Below the contact / Gingival to the contact area
Radiograph : Children: Bitewing
Proximal Caries

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94
Q

used for prevention & control of caries

Most effective way is systemic

A

Fluoride Therapy

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95
Q

systemic

A

Fluoridation

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96
Q

topical, cavity varnish , toothpaste

A

Fluoridization

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97
Q

📌‼️‼️‼️REMEMBEr

Fluoride can inhibit what enzymes

A

Phosphatase

Enulase

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98
Q

Where is the site of excretion of fluoride

A

Kidney

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99
Q

Optimal Fluoride Concentration for public water

A

0.7-1.2 ppm

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100
Q

What determines the fluiride content in a community water

A

Climate/ Temp.

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101
Q

Rule of 6

A
  • No supplemental systemic fluoride
    fluoride level > 0.6 ppm f
    px is < 6mos.
    px older 16 y/o
102
Q

Demineralization

A

Carbonate
Calcium
Phosphate

103
Q

RemineralizRion

A

Fluoride
Calcium
Phosphate

104
Q

Indication of N2Op

A

Minimal to moderate anxiety

105
Q

Contrainidications N2O

A

Nasal obstruction
Uncooperative
Active pulmonary Infection

106
Q

Topical fluoride will NOT CAUSE FLUOROSIS only

A

Systemic will

107
Q

Advantage

A

Proximal & smooth surfaces benefits the most

108
Q

Fluoride converts HYDROXYAPATITE crystals into

A

FLUOROAPATITE

109
Q

Toothpaste contains

A

1,100 ppm of Fluoride

110
Q

‼️📌📌BE

Types of Fluoride

A
  1. NaF
  2. APF
  3. SNF
111
Q

concentration of NaF

A

2-5%

112
Q

APF concentration

A

1.23%

113
Q

SnF concentration

A

8%

114
Q

Tooth discoloration

A

SnF - Brown
NaF-none
APF- noe

115
Q

pH of NaF

A

pH- 9.2

116
Q

APF

ph

A

pH -+3-3.5

117
Q

SnF

ph

A

ph - 2.1-2.3

118
Q

child know how to spit

A

APF

119
Q

Adult lethal dose

A

4-5g

120
Q

Child Lethal Dose

A

15mg/kg

121
Q

Fluoride Varnish

A

5%
>22,600 ppm
> 22.6 mg f/ml

122
Q

Supplemental fluoride factors to consider

A

Age

Fluoride content commonly water

123
Q

‼️📌📌REMEMBER

Silver Diamine Fluoride

A

Black Staining

124
Q

contains how much ppm F?

A

38% silver ammonia fluoride

125
Q

Side effect

A

Black staining but can stop arrest the active caries 44,800 ppm F

126
Q

Tx of Fluoride Toxicity

A

Syrup of Ipecac

Milk of Magnesia- induce vomiting

127
Q

anomaloes n #

A

Initiation

Proliferation

128
Q

anomLy in enamel structure or dentin

A

Histodiff.
Amelogenesis
Dentinogenesis

129
Q

anomaly size or shape

A

Morphodifferentiation

130
Q

Tissue formation

Hypoplasia Enamel

A

Apposition

131
Q

excessive ingestion of fluoride

A

Calcification

fluorosis/ mottled enamel

132
Q

emerge eruption

A
133
Q

uses weight in lbs / 150 adult dose

A

Clark’s Rule

134
Q

uses age

A

Young’s rule

135
Q

infants

infant dose- age in mos

A

Fried’s rule

136
Q

susceptible to decay
Histodifferentiation
Yellow teeth
Hypersensitivity

A

Amelogenesis Imperfecta

137
Q

Types of AmelogenisismImperfecta

A

Enamel Hypoplasia
Enamel hypocalcification
Enamel hypomaturation

138
Q

Allergic to Penicillin

A

Erythromycin

139
Q

Term for permanent teeth having hypoplastic defect crown resulting a local infection / trauma

A

Turners Tooth

140
Q

‼️📌📌What vitamin & mineral deficiency NOT in enamel hypoplasia

A

Vit. B

141
Q

Enamel Hypoplasia deficiency

A

A, D, C
calcium
phosphorus

142
Q

sign . gray brown teeth, opalescdnt hue, weak enamel

A

DI

143
Q

BE. Radiographic of DI

A

Obliterated Pulp chamber

Extensive deposits of 2ndary Dentin

144
Q

Type of Dentinogenesis Imoerfecta

associated w/ OI

A

Type I

145
Q

Blue sclera
Brittle /Fragile Bone
Hearing Loss

A

Osteogenesis Imperfecta

146
Q

most common type of OI, not associated w/ OI/ Hereditary Opalescent Dentin

A

Type II Dentinogenesis Imperfecta

147
Q

multiple pulpal exposure & PA lesions

Brandywine Type DI/ Teeth w/ shell like appearance

A

Type III Dentinogenesis Imperfecta

148
Q

Type I of Dentin Dysplasia

A
  • Rootless teeth / radicular type
  • short pointed root &pulp
  • absent pulp chamber & pulp canal
  • multiple PA lesion
149
Q

coronal

thistle tube shaped pulp chamber

A

Type II Dentin Dysplasia

150
Q

Rampant caries due to Sleeping with Feeding Bottle

aka. baby bottle tooth decay

A

Early Childhood caries/ Nursing Bottle caries

Severe Early Childhood Caries

151
Q

pattern of Early Childhood Caries

A

Cervical Max. Incisors ➡️ Max. Posterior➡️ Mand. posterior➡️ Mand. Incisor

152
Q

Most common early childhood caries

A

Max.Incisors

153
Q

Least early childhood caries

A

Mand. Incisors - tongue & saliva

154
Q

Only involves Gingiva
Rare: Preschool occurs in 15-30 y/o only GINGIVA
signs & symptoms: Painful hyperemic gingival Punched out erosions covered by GRAY PSEUDOMEMBRANE, FETID ODOR
Microorganisms: FuPS

A

Necrotizing Ulcerative Gingivitis/ Vincent’s Infection, / Vincent Angina/ Trench Mouth
Fusobacterium, Prevotella Intermedia , Spirochetes

155
Q

Tx. NUG

A

Debridement
Antibiotic
Hydrogen Rinses

156
Q 
will develop Class III Max. Deficiency, small maxilla, class 3 malocclusion , delayed eruption &:exfoliation of teeth underdeveloped mandible
short limbs
dwarfism
A

Achondroplasia

157
Q

enlarged tongue & longer root ; Skeletal Class III

A

Gigantism

158
Q

Skeletal Class III after epiphyseal plates closed

A

Acromegaly

159
Q

affect children ⬇️ 3 y/o very contagious

Fever, painful reddish ulcerations in gingiva & mouth

A

Primary Herpetic Gingivostomatitis

160
Q

What is the causative agent of Primary Herpetic Gingivostomatitis

A

HSV-1

161
Q

what do you call the secondary infection?

A

Recurrent Herpes Labialis / Cold sores / Fever Blister

162
Q

painful white / yellow ulcers with bright red that causes apthous ulcers

A

Coxsackie virus

163
Q

Herpangina, Hand-foot- mouth d.

A

Coxsackie A

164
Q

Mouth blisters

A

Herpangina

165
Q

<1cm & last for 2 weeks

A

Minor Apthous Ulcers

166
Q

> 1cm & last for more than 2 weeks w/ scarring

A

Major Apthous Ulcers

167
Q

Clusters of Ulcers

A

Recurrent Herpetiform

168
Q

📌‼️‼️Frequenr recurrences of ulcers should be screened for

A

DM & Behcet’s Syndrome

169
Q

Diabetes Mellitus trias

A

Polydipsia
Polyphagia
Polyuria

170
Q

uveitis - inflammation of uvea

vasculitis- inflammation of BV

A

Behcet’s Syndrome

171
Q

occurs during 6th-8th week in utero

A

Cleft Palate

172
Q

occurs during 5th- 6th week in utero

A

Cleft lip

173
Q

Syndromes associated in cleft lip & palate

A

Stickler Syndrome
Van Der Wonde / Lip Pits - obturator
Di George syndrome

174
Q

📌‼️‼️‼️ 2 Medial Nasal Process failed to fuse to form imtermaxillary segment

A

Median Cleft Lip

175
Q

Correctin of Cleft on Soft palate

A

Staphylorrhapy

176
Q

Hard Palate

A

Uranorrhapy

177
Q

‼️📌📌📌

Most common malocclusion cleft lip & palate px

A

Class 3 Maxillary Retrusion

178
Q

📌‼️‼️BE

Pattern of Caries Development Primary 2nd molar

A

Occlusal
Mesial
Distal

179
Q

DDx, of ANUG

A

Interdental papilla

Necrotic

180
Q

DDx.Primary Herpetic Gingivostomatits

A

Interdental papilla intact

181
Q

‼️📌BE

Most common cause Generalized Acute Gingival Inflammation in preschool/children

A

Acute Herpetic Gingivostomatitis

182
Q

Cleft lip
Class I
Class II

A

Microform / Incomplete cleft lip

183
Q

Uranoschisis

A

cleft on hard palate

184
Q

staphyloschisis

A

soft palate

185
Q

RULE of 10

A

10 wks
10 lbs
10 gm/ dl hemoglobin

186
Q

delayed up to 9-18 months after birth

A

Cleft Palate repair

Palatoplasty

187
Q

micronagthia, airway obstruction
posterior displacement tongue ;glossoptosis
Bird face
Cleft palate

A

Pierre Robin Syndrome

188
Q

Absent Nasal Bone; associated with Supernumerary
Needs affection & love
Less common to dental caries & more prone to PD- weak immune system
Delayed eruption
Heart Defects are common (Tetratology of Fallot) PROV

A

Trisomy 21( Down’s Syndrome)

189
Q 
Fish face
down slanting eyes
eyelids coloboma
underdeveloped cheek bone 
micrognathis
malformed ears
A

Treacher Colling Syndrome / Mandibulofacial Dysostosis

190
Q

📌‼️‼️Most common type gingivitis seen in children

A

Eruption Gingivitis

191
Q

Physical & mental retardation
Associated with Congenital Hypothyroidism
Delayed Eruption

A

Cretinism (hypothyroidism in Children)

192
Q

Delayed ERUPTION

A

Hypothyroidism

Hypopituitarism

193
Q

Absence of Clavicle
Clinical sign few teeth
Radiographic sign: numerous supernumerary teeth

A

Cleidocranial Dysplasia/ Cleidocranial Dysostosis / Marie- Sainton Syndrome

194
Q

‼️📌📌📌Best interest of dentist & unerupted teeth

A

Cleidocranial Dysplasia / Cleidocranial Dysostosis/ Marie - Sainton Syndrome

195
Q

delayed ERUPTION

A

Gingival Fibromatosis

196
Q

bluish purple, elevated area of tissue, appears before eruption of a tooth

A

Eruption Cyst

197
Q

whitish gingival cyst of Newborns, buccal, lingual surface of alveolus

A

Bohn’s Nodules

198
Q

Most common hemophilia in Children
Cause : fx 8 deficiency
more common males

A

Hemophilia A

199
Q

abnormal Ectoderm
affects skin, hair, sweat glands & tooth enamel
Alveolar bone development is Lacking -due to absence of permanent tooth

A

l Ectodermal Dysplasia

200
Q

Median Palatine Raphe

A

Epstein Pearl

201
Q

Crest of alveolar mucosa

A

Dental Lamina Cyst

202
Q

Hemp. C

A

Rosenthal Syndrome XI

203
Q

Contraindicated Hemophilic px

A 
Aspirin
Anticoagulant
Warfarin
Coumadin
Heparin
204
Q

absence of all teeth

A

Anodontia

205
Q

6 or more missing

A

Oligodontia

206
Q

less than 6

A

Hypodontia

207
Q

Vital tooth with provoked pain/ reversible pulpitis
1.8mm of dentin thickness between pulp & carious lesion
root resorbed Not greater than or equal 2/3 (deciduous)

A

Pulpotomy

208
Q

Procedure of Pulpotomy

A 
Removal caries
Access prep
Removal of Coronal pulp
Medicament placement
ZOE
Crown (SSC)
209
Q

hypohidrosis (lack of perspiration)
hypodontia (missing teeth)
hypotrichosis (fine sparse hair)

A

Anhidrotic Ectodermal Dysplasia

210
Q

infected pulp with spontaneous pain/ nocturnal pain
non- vital pulp with periradicular lesion
root resorbed NOT greater than or equal to 2/3 (deciduous)

A

Pulpectomy - Internal resorption/ Pink tooth of Mummery

211
Q

📌Contraindication Pulpectomy

A

Large bifurcatipn lesion
bone loss
mobile
non-restorable tooth

212
Q

📌‼️‼️Buckley’s solution

  1. Formaldehyde
  2. Cresol
  3. Glycerin
  4. Water
A

19%
35%
15%
31%

213
Q

physiologic development of Apex
Seen in young permanent teeth pulpotomy or direct pulp cappimg
Success & complete apical closure : tootg asymptomatic

A

Apexogenesis

214
Q

CaOH➡️ GI➡️ final restoration- small carious / mechanical exposure
MTA

A

Direct pulp capping

215
Q

GI- final restoration - carious lesion result to pulp exposure

A

Indirect pulp capping
infected dentin- remove
Affected dentin- stay
Indirect Pulp Capping

216
Q

MTA- GI- Final restoratiom= CaOH

A

Partial Pulpotomy / Cvek Pulpotomy

217
Q

Non- vital young permanent teeth with open apex

A

Apexification / Root End Closure

218
Q

Procedure apexification

A

Canal filled with CaOH or MTA

After apical closure, proceed to RCT

219
Q

Featheredge finish line

A

Anterior Strip off Crowns

220
Q

incisal Ant. SOC

A

1-1.5mm

221
Q

Labial & Proximal Ant. Strip off Crowns

A

1mm

222
Q

Lingual Reductiof of Ant. Strip off crowns

A

0.5mm lingual

223
Q

try in crown

A

Passive fit SOC

224
Q

cervical 3rd -facial surfsce

A

Undercut

225
Q

lingual surfsce: vents / holes

A

for excess material

226
Q

remove excess & cure

A

GI & Composite

227
Q

1mm subgingival Featheredge finish line
1.5mm overall reduction size
GI cement
snap fit

A

SSC

228
Q

Direction SSC

A

Lingual Buccal
Cement: Type I - GIC
Common errors- Ledges Interproximal
*Surface least amount reduction: Lingual

229
Q

Primary teeth
Vital
Spontaneous pain
Provoked pain

A

Pulpectomy

Pulpotomy

230
Q

Loosening of teeth
Slight-
Moderate

A

Observe only
Passive repositioning
Active repositioning then stabilize

231
Q

Displaced
Intrusion, Lateral Luxation
- Passive repositioning
- Active repositioning then Stabilize

A
232
Q

displace outside of its socket

A

Active repositioning then stabilize

Extrusion

233
Q

Extrusion
Primary
Permanent

A

Primary- Reposition, no tx
Severe - extract
Permanent- Reposition & stabilization

234
Q

completely out of the socket
Do not reimplant- Primary
- Reimplant &:stabilize for 2 wks

A

Avulsion

235
Q

Avulsion

A

Take radiograph to verify
Milk
Saliva
Last option- Water

236
Q

Chief cause failure of Avulsed tooth

A

External resorption

237
Q

Vital spontaneous pain

A

RCT- irreversible pulpitis

238
Q

Avulsed tooth w/ incomplete root formation when RCT

A

only if the tooth exhibit pulpal necrosis

after reimplantation

239
Q

tender to touch
no mobility
no displacement
Tx. Observe (permanent & primary)

A

Concussion

240
Q

mobility no displacement
tender to touch
TxOnserve
Flexible splint 2 wks monitor pulpal condition

A

Subluxation

241
Q

no mobility
displaced labially / palatally
Primart Interference
Severely displaced

A

Lateral subluxation
Selective grinding
Extract

242
Q

Avulsion

A 
Hank's Solution
Milk
Saliva
Reimplant
Stabilize for 2 wks
243
Q 
Ellis Classification of tooth trauma EDENTRDLD
I
II
III
IV
V
VI
VII
VIII
IX
A 
Enamel 
Dentin
Exposed pulp
Non- vital / without loss of crown
Teeth loss- trauma
Root fracture
Displacement
Loss of Crown
Deciduous Teeth
244
Q

📌‼️‼️REMEMBER
Primary ant.teeth
Is not common
more common

A

Fracture

Displacement

245
Q

Permanent ant. teeth
More common
Not common

A

Fracture

Displacement

246
Q

Most common ankylosed tooth

A

Mand.1st Molar

247
Q

2 Terms Ankylosis

A
  1. Submerged tooth

2. Replacement resorption

248
Q

Root fracture least favorable prognosis

Good prognosis

A

Cervical 3rd

Apical 3rd

249
Q

Diagnose Ankylose Radiograph

A

Cessation of eruption

250
Q

Replace the tooth bud

A

Sectioning

SET 7 (Ortho & Pedo) (3 decks)

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