Pediatric Dentistry Flashcards
max. conc. of nitrous should not exceed
flow rate is
50%
6 L/min
earliest symptom of conscious sedation is
light headedness
drug used for pediatric sedation by acting on CNS to induce sleep is
CHLORAL HYDRATE
-kids enter period of excitement and irritability before becoming sedated
short acting barbiturates include
secobarbital (seconal)
pentobarbital (nembutal)
sedatives by oral admin. but aren’t as good, non-analgesic, can cause hyper-excitability
standard prophylaxis for kids is
amox
50 mg/kg 1 hr before
pulpectomy (RCT) is indicated when
- periapical pathology
- canals filled with ZOE paste (min. tissue rxn)
pulpotomy
preserves radicular VITAL pulp when the entire coronal pulp is amputated; allows resorption and exfoliation of the primary tooth, but preserves its role as a natural space maintainer
-coronal pulp shows evidence of inflammation and degen. change
2 pulpotomy techniques
- Calcium hydroxide - tx PERMANENT teeth when there is a pathologic change in pulp at carious exposure site; indicated for peranent teeth with immature root development and healthy pulp tissue in canals
- Formocresol - tx PRIMARY teeth with carious exposure, success depends on VITAL root tip. Formecrosl causes surface fixation of pulp tissue and odontoblast degeneration.
indications and contraindications for Formecresol pulpotomy
indications - tooth sensitive to sweets, pulp exp during caries excavation, xray shows deep caries to pulp
contra - internal resorption on xray, tooth painful with swelling
direct pulp capping is used on
PERMANENT teeth (not primary cause of CaOH’s alkaline pH), tooth is ASYMPTOMATIC, there’s a small exposure
with mild irritation, resolves itself
with severe irritation, internal resorption can occur
contraindications of a direct pulp cap on primary teeth
spontaneous pain
large exposure
excessive bleeding
xray evidence of internal resorption
indirect pulp capping goals
preserve pulp vitality, prevent exposure, save tooth structure, arrest caries, promote reparative dentin formation
indications - permanent teeth with rampant caries, large caries close to pulp
contraindications - don’t use when spontaneous pain, furcation involvement, pulpal involvement, don’t do in KIDS
adolescents with rampant caries may need caries control before final restoration to arrest lesions. this involves __
removing gross caries, placing CaOH and an interim restoration like IRM (reinforced ZOE)
ER tx of fractures of permanent teeth with immature apices
Class I - smooth edges, restore
Class II - CaOH to exposed dentin and restore
Class III - CaOH, place temp, if exposure is big then perform CaOH pulpotomy -> pulpectomy eventually
Class IV - CaOH pulpotomy -> pulpectomy eventually
in a kid with a fully formed apex, if there is a pinpoint exposure and it has been a while (day) since it happened, what is tx?
what if it happened right away?
1 day - conventional RCT
imm - direct pulp cap with CaOH
MOST COMMON craniofacial malformations (50% of all defects)
cleft palate and cleft lip
Cleft lip
during 5th-6th week of embryonic life, from failure of maxillary and frontonasal processes to merge
- more common in males
- more common on left side
Class I - unilateral notch of vermillion NOT into lip
Class II - unilateral notch of vermillion extending into lip but not nasal floor
Class III - unilateral notch of vermillion but into lip and floor of nose
Class IV - bilateral clefting
-occurs during 4-6 wks of pregnancy
Cleft palate
opening in roof of mouth where 2 sides of palate did not unite, occurs in 6th-8th week of embryonic life
- fissure in midline
- isolated clefts more common in females
- impaired speech and swallowing
Class I - only soft palate
Class II - both palates but not alveolar process
Class III - both palates and alveolar process on one side of premaxilla
Class IV - soft palate and continues thru alveolus on both sides of premaxilla
Acute Necrotizing Ulcerative Gingivitis (ANUG, Vincent’s Angina, Trench Mouth)
clinical manifestations
tx
painful hyperemic gingiva, punched out papilla, covered by GRAY pseudomembrane with foul odor
- fusopirochetal infxn caused by FUSIFORM/FUSIBACTERIUM, SPIROCHETES, P. INTERMEDIA
- assoc. with poor OH, common in conditions with crowding and malnutrition, stress and smoking risk factors too
- YOUNG ADULTS 15-35 yrs.
clinical manifestations - inflamed, painful, bleeding gingiva, poor appetite, fever, malaise, odor
tx - debridement, H2O2, abx therapy
Primary (Acute) Herpetic Gingivostomatitis
viral infection
- characterized by: inflamed gingiva, sore throat, fever, malaise, lymphadenopathy, small fluid filled vesicles on mucosa of the lips/tongue/gingiva
- round ulcers with RED AREOLAE on CHEEKS
- self limiting, lasts 7-10 days
- tx: oral fluids
- common in PRESCHOOL kids
Atrophic Gingivitis
gingival recession without bone loss
Acute Lymphocytic/Lymphoblastic Leukemia (ALL)
most common pediatric cancer, form of acute leukemia most responsive to therapy
signs - fatigue, pallor, weight loss, easy bruising -> fever, hemorrhages, weak, bone/joint pain, repeated infxns
oral features - gingival oozing, petechiae, hematoma, ecchymosis, oral ulceration, pharyngitis, gingival infxn unresponse to therapy, submandibular lymphadenopathy
-susceptible to candida infxns (Nystatin)
most common type of leukemia in kids is
lymphoblastic leukemia
Apert Syndrome
cranial-limb anomaly
- malformations of skull, midface, hands, feet (NOT blindness)
- assoc. with supernumerary teeth, crowding, Class III
major features - prematurely fused cranial sutures, retruded midface, fused fingers and toes
Autism
appears in first 3 yrs, 4x more common in boys
- difficult to tx autistic pt cause of impaired communication
- can use sedatives and reduce loud sonds
Attention Deficit Disorder (ADD)
what tx?
short attention span, hyperactivity, impulsive behavior
- cause unknown
- 10x more common in males
Methylphenidate (ritalin) - mild CNS stimulant
Amphetamines (dextroamphetamine)
Achondroplasia
short-limb dwarfism
-most kids die before 1
clinical features - disproportionate short stature, prominent forehead, depressed bridge of nose, small maxilla causing overcrowding of teeth, Class III malocclusion
Gigantism oral features
- enlarged tongue
- teeth tipped buccal or lingual
- mand prognathism
- long roots
Pituitary dwarfism oral features
- delayed eruption and exfoliation
- short clinical crowns, smaller roots
- smaller dental arch -> malocclusion
- mandible underdeveloped
Cellulitis
acute spreading infxn of dermis and subcutaneous tissues, causing pain, erythema, edema, warmth
caused by - Group A streptococci & Staph Aureus
-harder to tx in kid cause dehydration
Ludwig’s Angina
cellulitis that affects submandibular, sublingual, submental spaces
- causes elevation of tongue and mouth -> obstruction of airway
- hospital immediately
Cretinism (child hypothyroidism) is a
deficiency caused by congenital absence of THYROXINE
- severe HYPOthyroidism in a kid characterized by defective mental and physical development
- dwarfed body with curved spine and pendulous abdomen
- distorted limbs
- severe mental retardation
dental findings - underdeveloped mandible, overdeveloped maxilla, enlarged tongue causing malocclusion, delayed tooth eruption, longer retention of deciduous teeth
Cystic Fibrosis
INHERITED disease of exocrine glands
- sticky mucus from faulty transport of Na+ and Cl in cells lining organs
- glands most affected are in pancreas, respiratory system, sweat glands
- inherit defective copy of CF gene
- dx tool is sweat test (elevated Na and Cl)
- dark teeth
combo of steatorrhea, chronic resp. infxn, functional disturbances in secretory mechanisms of various glands
Cleidocranial Dysplasia (dDysostosis)
INHERITED disorder of bony development
- absent or incompletely formed clavicles
- characteristic facial appearance, dental abnormalities
- supernumerary teeth, delayed eruption, peg-shaped teeth, missing teeth
Ectodermal Dysplasia
HEREDITARY condition caused by abnormal development of skin, hair, nails, teeth, sweat glands
- different types but X-linked anhidrotic is most common
- anodontia or oligodontia, conical shaped anteriors
- atrophic skin, defective hair, hypoplastic sweat glands
Anhidrotic ectodermal dysplasia
most common, only affects males, characterized by lack of perspiration
retained primary teeth are an oral manifestation of what 2 conditions
Ectodermal and
Cleidocranial Dysplasia
Diabetes
what is the triad?
oral complications?
body can’t properly use and store glucose
triad - polydipsia (thirst), polyphagia (hunger), polyuria (freq. urination)
Type I Diabetes (insulin dependent, juvenile-onset) - body stops making insulin, prone to ketoacidosis, blindness may develop
oral complications - xerostomia, infection, poor healing, perio disease, burning mouth syndrome
Diphtheria
acute, contagious disease caused by bacterium CORYNEBACTERIUM DIPHTHERIA
-production of a systemic toxin damaging to heart and CNS
Down Syndrome (Trisomy 21)
CONGENITAL, chromosomal abnormality
low caries rate high prevalence perio disease delayed eruption malocclusion enamel dysplasia delayed mental and physical development short, stocky build, broad, flat face, slanty eyes prominent, thick tongue **heart defects common, SBE prophylaxis is required for dental tx** reduced resistance to infxn
Gingivostomatitis
sores on mouth and gingiva caused by HERPES (HSV-1), characterized by inflammation of gingiva and mucosa, common esp. in kids
Acute (Primary) Herpetic Gingivostomatitis
kids <3 (1-5 yrs) with prodromal symptoms (fever, malaise, irritability, headache, dysphagia, vomiting, lymphadenopathy)
- tx by mild topical (ex. Dyclone) for pain
- virus that causes this is closely related to herpes that causes chickenpox (varicella zoster)
common sequelae of Acute Herpetic Gingivostomatitis
- recurrent herpes labialis (cold sores)
- spherical vesicles
- kid will have circulating anti-herpes antibodies (HSV-1 antibodies)
Herpangina
viral infection (strain of coxsackie A)
- young kid
- oral ulcers (white to whitish gray base and red border on roof of mouth and throat)
- fever, sore throat, headache
- goes away in a week, tx is palliative
Hemangioma
most COMMON BENIGN TUMOR of INFANTS
- vascular birthmarks, biologically active
- 5x more common in girls
- common on lips, tongue, buccal mucosa, flat or raised, deep red or blushish
- removed surgically
ex. asymptomatic blue lesion on tongue, minimal increase in size 5 yrs
Lymphangioma
well circumscribed nodule or mass of lymphatic vessels, often in neck and axilla
-compressible and spongy red to blue translucent lesions
tx by excisional biopsy
Neurofibroma
firm, encapsulated tumor by proliferation of SCHWANN CELLS
- on tongue, buccal mucosa, vestibule, palate
- can become malignant (5-15%)
MULT. LESIONS are assoc. with NEUROFIBROMATOSIS (Von Recklinghausen’s Disease)
Nursing Bottle Caries (Baby Bottle Tooth Decay/Bottle Mouth Syndrome)
widespread caries, most commonly affecting MAXILLARY INCISORS
-in combo with Strep mutans
max incisors > max and mand 1st molars > mand. canines
Pierre Robin Syndrome
HEREDITARY disorder
-presents micrognathia, glossoptois (down displacement/retraction of tongue), high arch or cleft palate
Porphyria
3 major findings?
dx?
INHERITED disorders, abnormalities in production of heme pigments, myoglobin, ctyochromas
-can cause discoloration of teeth
3 major findings
- Photodermatitis (light sensitivity causing rash)
- Neuropsychiatric complaints
- Visceral complains (ab pain, cramping)
Dx - red urine, purple brown teeth, sensitive to sunlight, gets blisters and swelling on face/hands in sunlight
Causes of tooth discolration (4 conditions)
- Porphyria
- Cystic Fibrosis - dark
- Erythroblastosis Fetalis - destruction of erythrocytes, blue-green
- Tetracycline therapy - yellow to brown, gray to black
Rieger’s Syndrome
delayed sexual development, hypothyroidism, dental features like hypodontia, underdeveloped premaxilla, cleft palate, protruding lower lip