PBL2 Flashcards

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1
Q

what happens when you bleed

A

HR goes up to compensate for drop in BP in order to maintain cardiac output. CO=HRxSV

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2
Q

how do we know that Cody is not bleeding

A
  • we know he is not bleeding because his heart rate decreases from 82 to 64
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3
Q

why is Cody still breathing but has shallow breathing

A
  • he has a C5/C6 fracture, but c3 c4 c5 phrenic nerve innervation keeps the diaphragm alive, therefore he has breathing but restricted breathing
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4
Q

what is the babinski sign a sign of

A

Positive babinski sign is a sign of upper motor neurone damage in the corticospinal tract

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5
Q

what is spinal cord shock

A

A state of temporary loss of function in the spinal cord

All functions below lesion are depressed or absent due to removal of all descending motor pathways

Gives impression of flaccid paralysis and loss of sensation

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6
Q

how long does spinal cord shock last for

A
  • often only lasts 1 day

- can last months

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7
Q

what happens after spinal cord shock

A

Neurones gradually regain their excitability and the flaccid paralysis gives way to spastic paralysis

Oedema and inflammation reduced

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8
Q

what happens when you have upper motor neurone lesions

A

Damage to the CNS

Spastic paresis

Hypertonia

Clonus

  • Distributed to groups of muscles, never individual muscles. Stretch reflexes hyperactive
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9
Q

what happens when you have lower motor neurone lesions

A

Damage to PNS

Flaccid paralysis

Limited to muscles innervated by damaged motor neurones or axons

Pronounced atrophy

hypoactive stretch reflexes

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10
Q

where does the corticospinal tract decussates

A

Corticospinal tract – decussates at the medulla, damage leads to ipsilateral motor deficits

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11
Q

where does the spinothalmic tract decussate

A

Spinothalamic tract – decussates in spinal cord – lesions lead to a contralateral deficit at the level of lesion

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12
Q

where does the DCML decussate

A

DCML – gracile and cuneate fasciculi. Decussates at medulla. Damage leads to ipsilateral loss of proprioception

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13
Q

what are the pyramidal tracts

A
  • lateral and anterior corticopsinal tracts
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14
Q

what are the extrapyramidal tracts

A

rubrospinal
reticulospinal
vestibulospainl tract

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15
Q

what is the prognosis of Cody

A

Further functional recovery is poor. CNS regeneration is poor, lost neurons won’t be replaced

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16
Q

why can’t Cody maintain his blood pressure and why does he have warm peripheries

A

In spinal cord injury, lose the inability to vasoconstrict. Vasodilation causes warm peripheries and low BP.

17
Q

why is the heart rate low

A

Also because of the feedback mechanism to the heart, HR does not increase to compensate

18
Q

what is shock

A

Shock is inadequate perfusion to tissue, sufficient to cause end organ damage

19
Q

what are the 2 main sensory tracts in the spinal cord

A

spinothalamic and dorsal columns

20
Q

describe input into spinothalamic tract

A
  • smal diameter sensory neurones which are unmyelianted or thinly myelinated
21
Q

what information does the spinothalmic tract relay

A
  • crude touch, pain, temperature
22
Q

where is the spinothalamic tract in the spinal cord

A

ventrolateral spinal tract

23
Q

what are the inputs into the DCML

A
  • carry information from larger diameter myelinatedneurones
24
Q

What information is in the DCML

A
  • vibration
  • proprioception
  • discriminatory touch
25
Q

what is the babinskin response to

A
  • it is in response to a polysyantpic reflex
  • it is evoked to by stimulation to the sole of the foot
  • this normally produces a flexion withdraws reflex
26
Q

what is the normal response to the babinski when the corticospainl tract is normals

A
  • toes curl down and adduct together
27
Q

what is the abnormal baninskin sign when the corticospainl tract is damaged

A
  • big toe dorsiflexes and the other toes fan out

- this is normal in a baby

28
Q

what does the babinski sign due to

A

upper motor neurone damage

29
Q

Define the term spasticity and state its clinical significance

A

Increased muscle tone and exaggerated tendon reflexes; Sign of upper motoneuron damag

30
Q

State two reasons for weakness in Cody’s breathing.

A

Damage to the motoneurons of C5 innervating the diaphragm.
Complete injury at C5 level results in loss of control on lower motoneurons innervating external intercostal and abdominal muscles