Pathology Flashcards

1
Q

Acute Cervical Facet

A

Complaint: unilateral neck pain or “locking”

Acute onset - “slept wrong”

Sudden, one-sided, neck pain

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2
Q

Etiology

Acute cervical facet

A

Most common: spondylosis (aging risk factor)

Less common: secondary trauma from sports or MVA

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3
Q

Mechanism

Acute cervical facet

A

Sudden backward, SB or rotation
or
Sustained position

Pathophys: entrapment of small piece of synovial membrane of z-joint

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4
Q

S/S

Acute cervical facet

A

Sx local
Rarely radiates past GH joint, maybe lower neck and upper back

Limited ROM associated with closing or down glide of mid-c/s facet

R-sided involve: no rot or SB to right (decrease ipsi SB, ipsi rot, ext)

No extension

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5
Q

prognosis

acute cervical facet

A

Excellent

Resolves around 1-2 weeks + common recurrence

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6
Q

examination

acute cervical facet

A

(+) painful AROM (3/6 motions)

(+) unilateral PA on involved side/segment

Local mm guarding / possible spasms

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7
Q

treatment

acute cervical facet

A

Control pain and acute sx

Joint mob for flex or ext + rotation w/ traction superimposed

Applied in pain free direction (opening) → direction of pain

Restore full AROM → strengthening

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8
Q

forward head posture

A

Can result in myofascial pain syndrome

Often coexists upper crossed syndrome

Insidious onset

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9
Q

mechanism

forward head posture

A

Aggravating: repeated inefficient muscle use

Upper crossed muscles:
Shortened:
Suboccipitals
Pec major and minor
Subscap
Scaneli
SCM

Lengthened:
Trap
Rhomboids
Deep cervical flexors
Deep cervical extensors

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10
Q

s/s

forward head posture

A

Persistent neck and shoulder girdle ache/muscular tension (not an acute onset)

Sx reproduction with trigger points

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11
Q

examination

forward head posture

A

Exam unremarkable except muscle imbalances noted in flexibility and strength

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12
Q

treatment

forward head posture

A

Interventions to diminish muscular tension
Provide ergonomic cuing
Treat the trigger points (inhibit, elongate, prevent)

Treat the muscle imbalances
Flexibility exercises to sub-occ, pecs, scal, scm, subscap
Strengthen: trap, rhom, DNF, deep neck ext’s

Postural and ergonomic education

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13
Q

cervical muscular headache

A

Progression of myofascial pain syndrome from FHP

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14
Q

mechanism

cervical muscular headache

A

Onset with:
Postural static load
FHP

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15
Q

s/s

cervical muscular headache

A

Major sx: headache

Areas of pain:
upper back and neck
Base of head and ears
Above the ears
Jaw
Above the eyes

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16
Q

examination

cervical muscular headache

A

Neuro screen clear / Imaging clear

Specific findings:
Suboccipital muscular tension, tenderness, tightness, symptoms provocation
Unilateral or bilateral OA flexion limitations

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17
Q

treatment

cervical muscular headache

A

Soft tissue

Joint mobs to craniovertebral region

Postural and ergonomic education/retraining

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18
Q

HNP Protrusion – W/O Spinal Nerve Root Involvement

A

Clinical picture NOT as well defined as with lumbar

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19
Q

Findings/exam

HNP w/o nerve root

A

With mild to mod HNP protrusion
increased pain with sitting and with neck flexion

Often lacks extension
Extension will cause increased centralization of pain, while lessening peripheral pain

Forward head posture

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20
Q

s/s

HNP w/o nerve root

A

Maintaining correct posture to allow disc to heal
“head back, chin in” – progressing to cervical ext ex’s
Neck flexibility and strengthening

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21
Q

HNP Protrusion – With Spinal Nerve Root Involvement

A

Most often caused by DDD

Often need MRI/CT scan for dx

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22
Q

area of most affected

HNP w/ nerve root

A

Most common at C5-6 segment = involves C6 nerve root

23
Q

S/S

HNP w/ nerve root

A

Worsening of sx starting centrally at base of neck → shoulders and arms (conditions get worse)

Referred pain to upper-T/S

Cloward’s areas

24
Q

examination

HNP w/ nerve root

A

Correcting FHP or to perform extension of C/S = increase peripheral S/S
Protrusion is too big to be reduced with maneuvers

25
treatment | HNP w/ nerve root
Reduce protrusion = restore normal posture Manual traction combined with passive axial ext and/or passive backward bending exercises is often effective “play” with angle of pull with manual traction
26
Whiplash
Acceleration Injuries to the Cervical Spine * Anatomic injury to longus-colli and SCM * Anterior column ligamentous tears * Annular tears of anterior discs * Sympathetic nervous system plexus damage * Nerve root trauma * Esophageal damage * Closed head injury
27
mechanism | whiplash
Majority of the cases, no injuries can be identified Symptoms attributes to musculo-ligamentous sprain Transfer of energy into the neck May result from rear-end or side impact collision Can occur with other accidents Impact of injury can occur on soft tissue and bone in the neck Can lead to a variety of clinical presentations known as WAD Whiplash injury feasible at 5 MPH
28
s/s | whiplash
Localized and referred pain, paresthesias Diffuse muscular tension, tenderness, and weakness Movement restrictions in the upper quarter Headache Blurred vision Dizziness Dysphagia
29
examination | whiplash
Neuro screen clear / Imaging clear Specific findings: Suboccipital muscular tension, tenderness, tightness, symptoms provocation Unilateral or bilateral OA flexion limitations NDI = greater than 15/50 VAS = greater than 5/10 Expectations of recovery = poor expectations of recovery
30
prognosis | whiplash
40% of clients will continue to have symptoms at 6 months! 10-25% will continue to have symptoms at 2 years!
31
Quebec task force (WAD) | whiplash
WAD 0 - No complaints or physical signs WAD 1 - Neck complaints only, but no physical signs WAD 2 - Neck complaints and musculoskeletal signs WAD 3 - Neck complaints and neurological signs WAD 4 - Neck complaints and fracture / dislocation
32
treatment | whiplash
Reduce muscle spasm Passive modalities early Soft collar Proper weaning instructions are critical Proper fit is important Progress to active therapies Counseling/CBT PRN patient education and counseling strategies that promote an active lifestyle and address cognitive and affective factors (chronic neck pain with mobility deficits)
33
Rheumatoid Arthritis (RA)
With RA an overactive immune system attacks the joints It usually takes many years for the disease to reach the neck BUT it has been reported that ~1/4 experience neck pain at the time of RA diagnosis Aggravating: morning & inactivity (different pattern than OA neck) **PRECAUTION: High cervical instability!!!!**
34
s/s | RA
Pain, swelling and stiffness Neck pain and cervicogenic headache
35
treatment | RA
Hands off the neck! Modalities, STM to proximal shoulder regions Gentle mid range postural awareness Isometric strengthening Positional support with ADLs
36
Cervical Spondylosis
Risk factor is advancing age Chronic degenerative condition affecting: Contents of spinal canal-nerve roots and/or spinal cord Cervical vertebral bodies (eventually = degeneration facets) Intervertebral discs (IVDs) = disc space narrowing
37
progression of cervical spondylosis
Changes in the osseous and fibroelastic boundaries (DDD & DJD) of the spinal canal and/or lateral recess DJD and/or DDD → normal of spinal canal → myelopathy DJD and/or DDD → narrowing lateral foramen
38
mechanism | cervical spondylosis
Initial clinical presentation hypermobility of segment but progresses to chronic hypomobility * Disc bulges outward * Angle of tension on ligaments changed * Weight bearing develops on uncinate processes anterior-laterally
39
s/s | cervical spondylosis
Slow onset * Can be asymptomatic for a long time * As the space for neural structures becomes smaller, the risk of developing motor and/or sensory disturbances increases Exacerbated by minor trauma Pain and muscle guarding * Localized * Referred Neuro – radiculopathy Diagnostic imaging Long term hypomobility and long term progressive stiffness
40
Examination | cervical spondylosis
Reduced SB initial stages followed by reduced sagittal plane motion in later stages
41
treatment | cervical spondylosis
Address limiting impairments Electrotherapeutic modalities to control pain and increase extensibility of the connective tissue Molded cervical pillows Limited immob of CS with collar if nerve root irritation is significant Manual techniques * Stretch adaptively shortened tissues * ROM exercises as tolerated * Isometrics and cervical stab as motion is restored
42
Mechanical traction-B level evidence in the 2017 Neck Pain Clinical Practice Guidelines | cervical spondylosis - treatment
Chronic neck pain w/radiating symptoms (mechanical I/M) Chronic neck pain w/mobility deficits when combined with TS mob/manip & TE (I/M man or mech txn)
43
cervical stenosis
Associated with spondylosis(degenerative process) Usually in patients over 50 years Chronic and slowly progressive
44
mechanism | cervical stenosis
Narrowing of the spinal cord Central or lateral Disc space narrowing leads to loss of cervical lordosis Loss of disc height also leads to buckling of the ligamentum flavum posteriorly Osteophytes form from the facets, uncovertebral joints, and posterior vertebral margins
45
s/s | cervical stenosis
* Neck pain; not always severe. * Pain, weakness, or numbness in the shoulders, arms, and legs. * Hand clumsiness * Burning sensations, tingling, and pins and needles in the involved extremity
46
examination | cervical stenosis
Symptoms aggravated by extension of the neck: * In extension = narrowing of the spinal canal by 20%
47
treatment | cervical stenosis
Flexion exercises Note: In flexion, widening of spinal canal by 31%
48
cervical myelopathy
A disorder in the cervical region of the spinal cord that disrupts or interrupts the normal transmission of the neural signals
49
risk factors | cervical myelopathy
Present in 90% of individuals by age 70 * Most common spinal cord dysfunction in people over 55 years * Males > Females * Asian Descent
50
prognosis | cervical myelopathy
Prognosis worse the more severe the stage Degenerative (cervical spondylotic myelopathy) Progresses in varying fashions
51
mechanism | cervical myelopathy
Direct compression of the spinal cord by bony or fibrocalcific tissues Ischemia caused by compromise of the vascular supply to the cord Repeated trauma secondary to normal flexion and extension of the neck
52
s/s | cervical myelopathy
* Neck pain, HA, dizziness * Radicular arm pain * May have only bilateral LE sxs (no UE) * Bowel & bladder disturbance * Hyperreflexia (UEs & LEs), clonus * Multisegmental weakness and/or sensory changes * Intrinsic wasting and sensory disturbance of the hands * Hoffman’s & Babinski reflex * Loss of dexterity * Wide-based unsteady gait
53
examination | cervical myelopathy
* Gait Deviation; * (+) Hoffman’s; * Inverted Supinator sign (C6 DTR with Finger flexion); * (+) Babinski; * age > 45 years
54
stages | cervical myelopathy
Mild: involves hand and arm symptoms, but does not prevent performance of normal ADL’s Moderate: considerable difficulty using their arms and legs, which affects performance of ADL’s Severe: require ambulatory aides, and often confined to bed, chair or home