Patho Flashcards

1
Q

What is pneumonia

A

An inflammatory condition of the lungs leading to an abnormal alveolar filling with consolidation and exudation

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2
Q

4 stages of pneumonia

A

Congestion-24 hours
Red hepitisation- 2-3 days
Grey hepetisation 4-6 days
Resolution 6 days +

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3
Q

Congestion

A

Vascular engorgement

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4
Q

Red hepetisation

A

RBC, leukocytes fill the alveolar spaces

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5
Q

Grey hepetisation

A

progressive disintegration of RBC

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6
Q

Resolution

A
  • the consolidated exudate within the alveolar spaces undergoes progressive digestion
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7
Q

What is bronchiectasis

A

the permanent abnormal dilation of one or more of the lungs bronchi
Causing sputum to be built leaving patient prone to infection

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8
Q

Clinical features of bronchiectasis

A

75% dyspnoea (laboured breathing) and wheezing
50% chest pains
Coughing and sputum production

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9
Q

What is cystic fibrosis

A

an-inherited autosomal recessive disease
The result of a gene mutation- single defect on chromosome 7

CTFR protein is a channel protein and controls the flow of water and chloride ions between membranes

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10
Q

Symptoms of CF

A

persistent cough
Inflammation in lungs
Cross infection
Impaired diaphragm from enlarged liver

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11
Q

What is COPD

A

COPD is characterised by airflow obstruction which is…
• Progressive in severity
• Not fully reversible
• Does not change markedly over several months
• Umbrella term for chronic bronchitis, emphysema and chronic asthma

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12
Q

What are airways lined by?

A

Cells which produce mucus
- Tiny hairs called cilia which continually beat

• Mucus traps dust particles and bacteria
• The cilia move the mucus along until it reaches the throat and we swallow it or cough
Defence mechanism

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13
Q

What is bronchitis

A

• A chronic disease of the lungs where the bronchi becomes inflamed
The inflammation causes more mucus to be produced, which narrows the airway and makes breathing more difficult

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14
Q

What is emphysema

A

A condition where the alveoli of the lungs become inflamed and lose their natural elasticity
• They over expand and lose their ability to fill up and contact properly
• as air fills up in these sacs, some rupture and become one sac reducing the surface area for the exchange of oxygen and carbon dioxide
• When you breath out, the trapped air cannot be released and breathing becomes more difficult

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15
Q

What is asthma

A

• Episodic increase in airway obstruction caused by various stimuli resulting in increased airway resistance
• Symptoms= breathlessness, wheeze, tight chest

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16
Q

Causes of COPD

A

Ciggerate smoking
• Occupational exposure- coal miners
• Social deprivation

17
Q

Clinical signs of COPD

A

breathlessness on exertion
• Cough
Increased sputum

18
Q

Treatment and medication for COPD

A

• Smoking cessation
• 4 times more likely to quit with help, advice and nicotine replacement
• Stop smoking services
• Stopping smoking will help slow the progression of the disease
• we can refer as physios at anytime

Medication
• Inhalers
• Steroids and antibiotics
• Mucolytics
• Flu and pneumonia vaccines

19
Q

What is pulmonary rehab

A

• exercise
• Education
• Self management
• Diet
• Lifestyle modifications

20
Q

Coronary heart disease

A

sometimes called ischamic heart disease or ccoronary artery disease
• a disease where the coronary arteries are blocked or narrowed
• Included the following diseases:
-Angina
-Myocardial infarction
-Heart failure

21
Q

CHD risk factors and symptoms

A

Age
• Gender
• Social deprivation
• Smoking
diet

Symptoms

• Pain, discomfort, pressure, tightness, angina
• Dizziness
• SOB
• Fatigue
Nausea

22
Q

Angina

A

• a chest pain due to an inadequate supply of oxygen to the heart muscle
Types:
• Stable- happens at predictable times eg stress or exercise
• Unstable- happens when no particular demand is being placed on the heart
• Micro vascular- affects the very smallest of the vessels

23
Q

Myocardial infarction

A

MI is when the blood supplying the oxygen to the heart is severely reduced or cut off. The result is ischemia of the heart muscle and scar formation

Diagnosis MI:
Blood tests:
-Looking for altered cardiac enzymes ( released when heart muscle is damaged)
• Troponin found in the myofilament of the heart muscle and released into the blood stream when the heart muscle is damaged

24
Q

MI consequences

A

Sudden death
• Arrhythmia
• Heart failure
• Cardio genie shock
• Rupture
• Thrombus formation

25
Q

Heart failure

A

The heart is unable to adequately pump blood around the body usually due to heart becoming to stiff or weak

26
Q

Diabetes

A

A metabolic disorder
• It is a absence, deficit or resistance to insulin leading to hyperglycaemia (excess of glucose in blood)

27
Q

What are the two types of diabetes and risk factors for each

A

• Type 1- insulin dependent mellitus
• Type 2- non insulin dependant diabetes mellitus

Risk factors :
• type 1: Family history, viral infection
Type 2- obesity, hypertension, poor lifestyle, diet

28
Q

Physiotherapy involvement for diabetes

A

• secondary to complications
• Awareness of complications eg healing
• Education on life style changes
• Higher musculoskeletal pain
• Falls