Path 2 Flashcards

1
Q
A

“bread & butter”, adhesions

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2
Q
A

Fibrinous pericarditis

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3
Q
A

hemorrhagic pericarditis (malignancy, TB)

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4
Q
A

bacteria, fungi

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5
Q
A

dilated cardiomyopathy, most common,

etiology: etoh & drugs, genetics

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6
Q
A
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7
Q
A

fibrous scar formation in cardiomyopathy

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8
Q
A
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9
Q

myocytes arranged haphazardly

A

hypertrophic cardiomyopathy

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10
Q
A

restrictive cardiomyopathy (amyloidosis)

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11
Q
A
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12
Q
A

restrictive cardiomyopathy (hemochromatosis)

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13
Q
A

acute bacterial endocarditis, staph aureus & other virulent bacteria, large friable vegetations on valve cusps

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14
Q
A
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15
Q
A

subacute bacterial endocarditis, strep viridans & other less virulent bacteria

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16
Q
A
17
Q
A

vegetations on valve leaflets (bacterial endocarditis)

18
Q
A

clinical presentation of bacterial endocarditis

19
Q
A

clinical presentation of bacterial endocarditis

20
Q
A

small vegetations on valve cusps in RF

21
Q
A

aschoff bodies in myocardium (Acute RHD)

22
Q
A

aschoff bodies, myocarditis in acute RHD

23
Q
A

endocarditis dominates in chronic RHD

24
Q
A

fishmouth mitral stenosis in chronic RHD

25
Q
A

fishmouth valves, mitral stenosis in chronic RHD

26
Q
A

Non Bacterial Thrombotic Endocarditis

  • d/t hypercoagulable states e.g. cancer, chr illness, trauma from indwelling catheter
  • less # of vegetations, non destructive, but friable
27
Q
A

Non Bacterial Thrombotic Endocarditis

-pink fibrin, no inflammation or bacteria

28
Q
A

Libman-Sacks Endocarditis d/t SLE

-small sterile vegetations, trigger inflammation, can occur anywhere

29
Q
A

Types of vegetations