Past exam questions Flashcards

1
Q

Describe the paediatric GCS.

A
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2
Q

How do you assess a patient with a potential fracture?
Pain, swelling and limited range of motion.

A
  • Inspect: for open fracture while limiting movement, observe any displacement.
  • Neurovascular examination:
    • Pulses
    • Perfusion: cap refill
    • Neuro function: assess sensory and motor (may need to give analgesia for this)
  • Assess for compartment syndrone
  • Check for associated injuries
  • X-ray
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3
Q

What is the importance of a transient lucid period/ lucid interval after a head trauma?

A

A lucid interval is a temporary improvement in a patient’s condition after a traumatic brain injury, after which the condition deteriorates. A lucid interval is especially indicative of an epidural hematoma. An estimated 20 to 50% of patients with epidural hematoma experience such a lucid interval

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4
Q

Describe the clinical relevant pathophysiology of HSV.

A
  • HSV-1 generally associated with orofacial ulcers
  • HSV-2 generally associated with genital ulcers
  • Family: Herpesviridae
  • Subfamily: Alphaherpesvirinae
  • ds-DNA virus
  • Properties of the virus:
    • Neurovirulence: Invades and replicates in the nervous system
    • Latency: HSV-1 in trigeminal (mostly), HSV-2 sacral nerve root (S2-S5)
    • Reactivity
  • Needs to vector to be transmitted
  • Transmitted by close personal contact: infects mucosa
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5
Q

What are the causes of AF?

A
  • Cardiac:
    • Ischaemic heart disease
    • HTN
    • Vavlvular heart disease (mitral stenosis/ regurgitation)
    • Pericardial disease
    • Cardiomyopathies: dilated, hypertrophic
  • Pulmonary:
    • PE
  • Infections:
    • Acute infections
  • Metabolic:
    • Electrolyte disturbance: hypokalaemia, hypomagnesaemia.
    • Thyrotoxicosis
    • Phaeochromocytoma
  • Drugs:
    • Alcohol
    • Caffeine
      *
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6
Q

What are the proposed mechanisms of AF?

A
  • Focal activation: in which AF originates from an area of focal activity. This activity may be triggered, due to increased automaticity, or from micro re-entry. Often located in the pulmonary veins.
  • Multiple wavelet mechanism: in which multiple small wandering wavelets are formed. The fibrillation is maintained by re-etry circuits formed by some of the wavelets. This process is potentiated in the presence of a dilated LA- the alrger surface area facilitates continuous waveform propagation
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7
Q

What are the ECG changes AF? that are seen in

A
  • Irregularly irregular rhythm
  • No P waves
  • Rate = 120-160/min
  • QRS complex usually <120 ms
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8
Q

What are the symptoms of AF?

A
  • Palpitations
  • Breathlessness
  • Fatigue
  • Syncope
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9
Q

What is the treatment of AF?

A
  • Medical:
    • Rate control: Digoxin, beta-blockers, and rate-limiting calcium antagonists (verapamil, diltiazem)
    • Reduce risk of thromboembolism: warfarin, bridge with LMWH.
    • Treatment of underlying disease i.e. cardiac disease.
  • Surgical:
    • Cardioversion
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10
Q

What is the CHADVASC score?

A
  • CHADVASC score is a way of assessing a patients risk of stroke within the next year and assess their need for anticoagulation when they have AF.
    • CHF = 1
    • HTN = 1
    • Age: 65-74 = 1, >75 = 2
    • Diabetes = 1
    • Sex: female = 1
    • Vascular disease (MI, PVD) = 1
  • Risk of stroke:
    • Score 0 = 5%
    • Score 5-8 = 70%
  • Need for anti-thrombotic therapy:
    • Score 0 = no therapy
    • Score 1 = either anticoagulate or anti-platelet
    • Score >2= anticoagulate
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11
Q
A
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