Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

parasitology 2 > paramphistomatidosis, dicrocoeliosis, schistomatidosis, alariosis, opisthorchiosis > Flashcards

paramphistomatidosis, dicrocoeliosis, schistomatidosis, alariosis, opisthorchiosis Flashcards

1
Q

what species can u find rumen fluke?

A

in grazing domestic and wild ruminants worldwide

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

veterinary significance rumen fluke

A

high in tropical and subtropical climate areas

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

mention some rumen fluke species

A
  • Paramphistomum ichikawai
  • P. leydeni
  • P. cervi
  • Calicophoron ( syn. Paramphistomum daubney)
  • C. microbothrioides
  • C. microbothrium
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

mention some predominant rumen flike species in Europe

A
  • Paramphistomum ichikawai
  • P. leydeni
  • P. cervi
  • calicophoron daubneyi
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

rumen fluke, adults, site, egg

A

Adults: 6-12 mm long, 2-4 mm wide, pink reddish, conical not flat.

Site: rumen, reticulum

Egg: like F hepatica but colourless, a bit larger and very resistant

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

rumen fluke cycle
( paramphistomosis)

A

life cycle : indirect
intermediate host: freshwater or amphibious snail

MI–>SPO–>RE–>CE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

infection rumen fluke
( paramphistomosis)

A
  • infected per os
  • immatures–> duodenum, abomasum –> rumen, reticulum
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

rumen fluke ( paramphistomosis)
PP

A

3-4 months, in lambs 40- 50 days

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

rumen fluke ( paramphistomosis)

pathogenesis, clinical signs

A

acute or intestinal paramphistomatidosis: tissue destruction, inflammation, local haemorrhages, necrosis, diahrrhoea, weigh loss.

chronic or rumen paramphistomatidosis: no symptoms

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

diagnosis and treatment
rumen fluke ( paramphistomosis)

A

DIAGNOSIS:
acute form:
- no eggs, juvenil flukes in faeces

chronic form:
- faceal egg count, look at the color of egg can be simular to F. hepatica

TREATMENT:
Actue form:
- stop grazing on infected pasture
- niclosamide, albendazole

Chronic form:
- addult flukes are more difficult to remove
- oxyckizanide, closantel

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Dicrocoeliosis
Lanceolate fluke disease
small liver fluke

A
  • worldwide in grazing animals, hare, rarely pig, horse, human
  • veterinarian significance: moderate
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Dicrocoeliosis
Lanceolate fluke disease
small liver fluke

is caused by

A
  • Dicrocoelium dendriticum ( syn D. lanceolatum) lancet or small liver fluke
  • Dicrocoelium hospes in tropical regions
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Dicrocoeliosis
Lanceolate fluke disease
small liver fluke

adult, site, egg

A

adult : 8-12 mm, lancet shape,pied, integumentum without spines, brown uterus and white vitellaria,

site: bile ducts, gall blatter

egg: 40-48 um , oval dark brown, thick shelled with operculum, contains miracidium, inside two germinal cells

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Dicrocoeliosis
Lanceolate fluke disease
small liver fluke

lifecycle, hosts

A

lifecycle: indirect

host: 2 intermediate hosts
- terrestrial snails
- ants

MI –> SPO1 –> SPO2 –> CE
(metacaria in ants)

per os infection, rare in humans but can happen

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Dicrocoeliosis
Lanceolate fluke disease
small liver fluke

PP

A

7-9 weeks

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Dicrocoeliosis
Lanceolate fluke disease
small liver fluke

pathogenesis

clinical signs

A

pathogenesis:
thickening of bile duct wall, oedema, cholangitis, cirrhosis, fibrosis

clinical signs:
rarely anemia, oedema

17
Q

Dicrocoeliosis
Lanceolate fluke disease
small liver fluke

treatment

A

in heavier infections only (benzimidazoles at higher doses)
albendazole, praziquantel

  • they usually dont bother to treat

heavily infected livers are not suitable for human consumptions

18
Q

schistomatidosis ( bilharziosis)
blood fluke disease

distribution
veterinary significance

A

a chronic and lethal tropical disease

distribution: tropical and subtropical zones

veterinary significance: low

19
Q

schistomatidosis ( bilharziosis)
blood fluke disease

caused by

A

-Schistosoma bovis
-Schistosoma mattheei
-Schistosoma japonicum
-Schistosoma mansoni
-Schistosoma haematoboium

20
Q

schistosoma bovis

A

Schistosoma bovis:
- cattle, sheep, pig, goat, camles, horses, monekeys ( man)

  • africa, middle east, asia, south europe
  • portal and mesenteric veins
21
Q

Schistosoma mattheei:

A

Schistosoma mattheei:
- domestic and wild ruminants, zebra, monkeys , man

  • south and central africa, middle east
  • intestinal, hepatic and bladder veins
22
Q

schistosoma japonicum

A
  • most domestic and wild animals, man
  • south and east asia
  • portal and mesenteric vein
23
Q

schistosoma mansoni

A
  • man
  • tropical africa, south america
  • intestinal schistosomosis
24
Q

schistosoma haematobium

A
  • man
  • tropical africa, middle east
  • urinary or bladder schistosomosis

( eggs was found in mumies from 13th centuries B.C)

25
Q

schistomatidosis ( bilharziosis)
blood fluke disease

adult, longevity, eggs

A

adult: 0,5-3 cm long,
- thread like in shape,
-sexual dimorphism,
-male is broad and flat, thin shred like and a bit longer is female.

longevity: many years ( 20-30 in humans)

eggs:
-130- 280 um in size,
- spindle shaped or spiny
- contain miracidium when passed out in faeces or urine

26
Q

schistomatidosis ( bilharziosis)
blood fluke disease

host, lifecycle, pp, infection, infective stage

A

life cycle: indirect
intermediate host: water snails
pp: 30-40 days
infection: percutan or per os
infective stage: furcocercaria

MI–> SPO1–> SPO2–> CE

27
Q

schistomatidosis ( bilharziosis)
blood fluke disease

Pathogenesis

A

pathogenesis:
- eggs enter lumen of excretory organs
50% –> passed out of body
50% –> trapped in tissues, carried away by blood circulation, lymph

28
Q

schistomatidosis ( bilharziosis)
blood fluke disease

clinical signs

A

3 distinct syndromes

Cercarial dematitis:
- rash, schistome or swimmer itch

Acute schistostomiosis:
- katayama fever ( fever, urticaria, malaise, diarrhea)

chronic schistostomiosis:
- caused by eggs travelling in body
- trapped eggs –> granulomatous inflammatory immune response

29
Q

Opisthorchiosis
cat liver fluke

host, caused by

A

hosts: fish consuming carnivores and man

caused by : Opisthorchis felineus, cat liver fluke

30
Q

Opisthorchiosis
cat liver fluke

adult, site, eggs

A

adult: 8-13 mm long, lancet shape
site: bile ducts, gallbladdder ( duct of pancreas)
eggs: oval, 16- 30 um, inside miracidium

31
Q

Opisthorchiosis
cat liver fluke

lifecycle, pp, pathogenesis, treatment

A

life cycle: indirect with water snails
pp: 3-4 weeks
pathogenesis: cholangio- carcinoma
treatment: praziquantel

MI –> SPO –> RE –> CE

32
Q

alariosis
country

A

-europe, asia and america
- temperate climate
- not coommon in dogs and cats, but in wild carnivores

33
Q

alariosis
caused by

A

Alaria alata
alaria mariciane (cats)
alaria canis

34
Q

alariosis

site, egg

A

site: small intestine
egg: 98-134 * 62-68 um, brownish, operculum inside zygote

35
Q

alariosis

life cycle

A

indirect with 2 intermediate hosts
snails- cercaria
tadpoles, frogs - mesocercaria

MI–>SPO–>RE1–>RE2–>CE –> MCE

parasitology 2 (6 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions