papers to quote Flashcards

1
Q

biologic with ppr to quote

A

gargiulo 1961

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2
Q

freq of snacking paper to quote

A

Duggal 2001 implication: children where usage of fluoridated toothpaste is low, frequency of snacking would be
important factor in caries development

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3
Q

VT pulp therapy primary teeth

A

coll et al 2024-clinical decision tree

IPC or calcium silicate cement pulpotomy is likely to increase vital pulp therapy success over other VPTs such as direct pulp capping
and other pulpotomies after 24 months (moderate certainty).

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4
Q

Ecological plaque hypothesis (ppr yr) =disease is the result of

A

Marsh (2003) =disease is the result of an imbalance in the overall microbiome due to ecological factors [ like presence of sugars and lower pH] , resulting in an enrichment of some cariogenic pathogens=> caries development

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5
Q

Ozgur et al 2017

A

Hemostasis could not be achieved after 10 minutes, either the inflamed pulp tissue has not been
completely removed or the radicular pulp has been infected. In the study, the pulp was regarded as healthy if hemostasis was
achieved within five minutes.

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6
Q

Evidence presented in this review of the literature indicates formocresol, when used judiciously, is unlikely to be
genotoxic, immunotoxic, or carcinogenic in children.

A

conference paper by Milnes 2008

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7
Q

Infective endocarditis (IE)

A

AHA Guidelines 2007
infection of the endocardium, usually affecting the heart valves. It can be acute [staph aureus] or subacute[strep. viridans]-typical post dental procedures.

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8
Q

IE prophylaxis dosage-adult

A

No penicillin allergy: amoxicillin 2g 1hr PO once OR cefazolin 1g/ Ampicillin 2 g IM or IV once

Penicillin allergy: a macrolide (e.g., azithromycin 500 mg PO once ) OR doxycycline 100mg PO once

PO- Per os - latin for oral route of adminstration

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9
Q

who received IE prophylaxis - 4 categories

A

Cardiac risk factors requiring IE prophylaxis (for procedures that may cause bacteremia)
1. Presence of prosthetic cardiac valve or material
2. History of endocarditis
3. Certain types of congenital heart disease (CHD), e.g., unrepaired cyanotic CHD-Tetralogy of Fallot in DSynd/ Persistent truncus arteriosus-digeorge syndrome], repaired CHD (within 6 months of repair), iii.repaired CHD with residual post-operative shunt or regurgitation
4.Valvulopathy in cardiac transplant recipients

FYI-acyanotic CHD- PDuctusA; vsd, asd, avsd; coarctation of aorta

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10
Q

which medical condition do NOT need AB

A
  1. pacemakers
    2 complete closure=Septal defect
    3 Peripheral vascular grafts-hemodialysis
    4 Coronary artery stents
    5 Vena cava filters
    6 Pledgets
    7 CNS ventriculoatrial shunts
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11
Q

what procedures need AB prophylaxis?

A
  1. manipulation of gingival tissues [perio/exo] or PERIapical region of teeth [rct]
  2. perforation of oral mucosa
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12
Q

no AB prophylaxis needed?

A
  1. anaesthetic injections of non infected tissues.
  2. xray
  3. Removable prosthesis/ ortho appliance
  4. adjst ortho appliance
  5. placement of brackets
  6. shedding of primary teeth
  7. bleeding from trauma of lips/oral mucosa
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13
Q

dosage for AB prophylaxis for children

A

No penicillin allergy: amoxicillin 50mg/kg 1hr PO once OR cefazolin/ Ampicillin 50mg/kg IM or IV once

Penicillin allergy: a macrolide (e.g., azithromycin 15 mg/kg PO once ) OR doxycycline >45kg -100mg PO once// <45kg 2.2mg/kg

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14
Q

Extraradicular Infection
Characteristics:
occur dt earlier …. infection
tx

A

Extraradicular Infection Overview
Characteristics:
1 Microbial Invasion in the inflamed periapical tissues surrounding the tooth root.
2 Sequel: Almost always follows an intraradicular infection.
3Treatment: (RCT)

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15
Q

Independent of Intraradicular Infection
Example:
Treatment:

A

Independent of Intraradicular Infection
Example: Apical actinomycosis.
Tx:Requires endodontic surgery for resolution.

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16
Q

ASA Physical Status Classification System

A

ASA 1-A normal healthy patient bmi 18.5-24.9kg/m2
ASA ii-A patient with mild systemic disease- without substantive functional limitations 30-40 BMI
ASA iii- severe systemic disease-with functional limitations BMI ≥40
ASA IV-severe systemic disease that is a constant threat to life
ASA V-moribund patient who is not expected to survive without the operation
ASA VI-declared brain-dead patient whose organs are being removed for donor purposes

17
Q

Flemmig et al., 1999; Lindhe et al., 1999 clinical features of C. Periodontitis

A

1.CAL
2. Periodontal pockets
3. Alveolar bone loss
4. Gingival inflammation
5.Gingival recession/enlargement
6. BOP
7. Increased mobility, drifting and/or tooth loss

18
Q

Howeffective are access flaps
compared to repeated
subgingival instrumentation?

A

In the presence of deep residual pockets (PPD ≥ 6 mm) in patients with stage III periodontitis after
the first and second steps of periodontal therapy, we suggest performing access-flap surgery.

In the presence of moderately
deep residual pockets (4–5 mm),
we suggest repeating subgingival instrumentation.
· Supporting literature Sanchez et al. (2020)

19
Q

Fluoride varnish works via 3x
reduce bacterial sp

A

topical mechanisms that include inhibition of demineralization, and enhancement of remineralization, at low levels is bacteriostatic, and at high levels is bactericida-S. mutans

20
Q

evidence-based clinical recommendations for the arrest or reversal of non-cavitated carious lesions on Occlusal surfaces of primary or permanent teeth
include:

A

1 Sealants plus 5% NaF varnish (varnish application every 3-6 months)- not 0.9% difluorsilane
2 Sealants alone
3 1.23% Acidulated phosphate fluoride (APF) gel (application every 3-6 months)
4 Resin infiltration plus 5% NaF varnish
5 0.2% NaF mouthrinse (once per week plus supervised brushing

-2 to 3 times greater chance of being arrested or reversed than results with no treatment and that “the combination of sealants plus 5% NaF varnish was the most effective at arresting or reversing noncavitated occlusal lesions.
only 2.26% fluoride varnish is recommended for children younger than six years of age.

21
Q

Absorption of the topical anaesthetic drug is key to successful desensitization, with attached gingiva being an area of slow absorption. why?

A

Prompt absorption occurs in tissues without keratinization, such as vestibular mucosa, while slow absorption occurs in keratinized tissue, such as attached gingiva and hard palate.

22
Q

Pavicic et al. (1992) highlight that pathogens may evade the effects of mechanical debridement due to….So,…

A

their ability to invade periodontal tissues or reside in anatomical tooth structures inaccessible to periodontal instrumentation. In such cases, adjunctive chemotherapeutic agents can help manage these pathogens.

23
Q

Addy and Renton-Harper (1996) have noted that there is no data to show that susceptible treated periodontal patients derive any …

A

long-term benefits by using chemical therapeutics alone. They also state that there is no basis for continuous long-term use of anti-plaque agents. T

24
Q

Chlorhexidine gluconate are considered the gold standard among antiseptic mouth rinses due to their….3x but can cause…

A

1broad-spectrum antimicrobial activity-by disrupting cytoplasmic membrane,
2 substantivity (the ability to bind to salivary proteins and release slowly over time8-12hrs), and
3 efficacy in reducing plaque and gingivitis.

but causes tooth staining -prolonged use

25
Q

minocycline microspheres (…) and doxycycline gels (….) have shown efficacy in reducing probing depth and improving clinical attachment levels when used adjunctively with scaling and root planing.

A

minocycline microspheres (Arestin)
doxycycline gels (Atridox)

26
Q

Essential Oil Mouthwashes: those containing… demonstrated….

TRICLOSAN,/,COLGATE,TOTAL- have both …effects and reduction of plaque.

A

thymol, eucalyptol, menthol, and methyl salicylate, have demonstrated efficacy in reducing plaque and gingivitis.

antimicrobial and anti-inf

27
Q

, Porphyromonas gingivalis can …. and ….

A

evade host defenses
capnophilic

28
Q

Lindhe 1983Furcation Classification (Grade)
Grade I: Horizontal loss of periodontal support <
Grade II
Grade III

A

Furcation *
Grade I: Horizontal loss of periodontal support < than or equal to one-third of tooth width.
Grade II: Horizontal loss greater than one-third of tooth width, but not encompassing the total width of the furcation area.
Grade III: Through-and-through horizontal loss of periodontal support, encompassing the total width of the furcation area.

29
Q

Guided Tissue Regeneration (GTR)
1 Indications: 4x
2 Contraindications
3 Influencing Factors :

4Healing Mechanism of Action:

A

1 Angular/vertical bony defects, Class II furcation involvements, desire to regenerate periodontal structures.
2 Poor plaque control, poor patient compliance to f/up, systemic conditions affecting wound healing/hopeless prognosis.
3 Defect morphology, systemic health, bacterial infection control, stability of regenerative membrane/absence of mechanical trauma during the healing phase, + patient’s post-op care compliance.

4 Use of a barrier membrane to facilitate selective repopulation by periodontal ligament and bone cells [NOT epit/ging CT], promoting regeneration.

30
Q

Flap Surgery with Osseous Reshaping
1 Indications:
2 Contraindications:
3 healing conditions,
4 Healing Mechanism:

A

1 Deep pockets unresponsive to non-surgical therapy; need for bone recontouring.
2 Poor oral hygiene, uncontrolled systemic conditions like diabetes/ poor healing conditions.
3 Overall health, smoking status, oral hygiene, bony defect morphology, clinician skills.
4 Direct access for debridement and reshaping; healing through reattachment at a new, more apical position.

31
Q

Fluoridation level in SG: sg national water agency = dec caries incidence [new cases] + prevalence [existing cases]
2023 SG average F is 0.??ppm

A

in SG average F is 0.45ppm - 2023