paeds general Flashcards

1
Q

septic screen in kids

A
F
Urine output
Culture bloods
Culture urine 
LP
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2
Q

child with sepsis - how would you recognise it..?

A

Recognition of a child at risk:
If a child with suspected or proven infection AND has at least 2 of the following:
• Core temperature < 36°C or > 38.5°C
• Inappropriate tachycardia (Refer to local criteria / APLS Guidance)
• Altered mental state (including: sleepiness / irritability / lethargy / floppiness)
• Reduced peripheral perfusion / prolonged capillary refill

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3
Q

define JIA

A

Joint inflammation
Idiopathic (not caused -
Age under 16

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4
Q

what are the criteria for kawasakis

A
four of:
Conjunctivitis
Rash
Adeopathy
Strawberry tongue
hands
and 
BURN  - fever for 5 days or more
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5
Q

management of heart disease in kids

A

high energy regular feeds
Furosemide
ACEi
Digoxin

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6
Q

causes of failure to thrive in children:

A

Inadequate caloric intake/retention

Inadequate nutrition (breastmilk, formula and/or food)
Breast feeding difficulties
Restricted diet (e.g. low fat, vegan)
Structural causes of poor feeding eg. cleft palate
Persistent vomiting
Anorexia of chronic disease
Error in infant formula dilution
Early (before 4 months) or delayed introduction of solids

Psychosocial:
Parental depression, anxiety or other mood disorders
Substance abuse of one or both parents
Attachment difficulties
Disability or chronic illness of one or both parents
Coercive feeding (including feeding child whilst asleep)
Difficulties at meal times
Poverty
Behavioural disorders
Poor social support
Poor carer understanding
Exposure to traumatic incident/family violence
Neglect of this infant or siblings
Current or past Child Protection involvement

Inadequate absorption:
Coeliac disease
Chronic liver disease
Pancreatic insufficiency eg. Cystic fibrosis
Chronic diarrhoea
Cow milk protein intolerance
XS calorie utilisation
Chronic illness
Urinary tract infection
Chronic Respiratory disease eg. Cystic Fibrosis
Congenital heart disease
Diabetes Mellitus
Hyperthyroidism
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7
Q

management of failure to thrive

A

half the size, twice as often

Careful growth charts

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8
Q

treating constipation in children

A
  1. movicol

2. stimulant eg. Senna

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9
Q

HSP - complications

A

Intussuception

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10
Q

causes of eneuresis

treatment of idiopathic

A

gu malf
dm
utis

tx if over 5:
sublingual desmo
1 wk break every 5 months

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11
Q

causes of eneuresis

treatment of idiopathic

A

gu malf
dm
utis

tx if over 5:
sublingual desmo
1 wk break every 5 months

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12
Q

what types of cerebral palsy are there?

?causes
?botox

A
  1. spastic
  2. ataxic
  3. athetoid
  4. mixed

botox :

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13
Q

ddx’s for vacant episodes

A
hearing 
epilepsy
visual problems
behavioural - adhd / autism 
learning disability 
brain tumour 
iatrogenic - medications
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14
Q

how might you investigate her symptoms?

A

history - social interactions / regression / reccurent ear infections

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15
Q

what is the pathognomic finding of a petit mal seizure on EEG?

A

3hz spike and wave

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16
Q

treatment for absence seizures? (petit mal)

A

ethosuxamide

valproate

17
Q

investigation of squint

A
  1. gross
  2. corneal light reflex
  3. cover test
18
Q

causes of squint?

A

short / long sightedness

atigmatism

19
Q

serious neonatal stuff:

A

issues of prematurity

nec
gastro
neonatal jaundice
resp: bronchopulmonary dysplasia

20
Q

what is the triad of haemolytic uraemic syndrome

A

thrombocytopenia
microangiopathic haemolytic anaemia (Coombs test negative)
AKI

21
Q

what is the common infection causing HUS?

how does this infection cause HUS

A

E. coli O157
circulating toxins produced by this (verotoxin / shigella) bind to endothelial receptors - causing deposition of fibrin and thrombin in the microvasculature
erythrocytes are damaged as they pass through the microvasculature - get caught and damaged the partially occluded small vessels

platelets sequestered but without the cascade of clotting factors

22
Q

when can children with HUS go back to school??

A

after 2 negative stools

23
Q

presentation of HUS?

A

The classical presenting feature is profuse diarrhoea that turns bloody 1 to 3 days later

It is rare for the diarrhoea to have been bloody from the outset

24
Q

management of HUS?

A

General management includes appropriate fluid and electrolyte management, antihypertensive therapy and dialysis where required.

Circulating volume must be kept up to protect the kidneys; simply replacing losses with crystalloid and keeping up with faecal loss is inadequate, as circulating volume will be lost by vascular leakage.

Where kidney failure occurs, indications for dialysis are as for any other cause of acute kidney failure.