Paeds Flashcards

1
Q

What is croup and it’s primary cause?

What are the major age groups affected?

A
  • Acute laryngo/tracho/bronchitis w sub glottic inflamm and oedema
  • 80% cases cause by parainfluenza virus
  • 6 months - 6 years. Most common 1-2 years.
  • Peaks in autumn
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2
Q

What is the typical presentation of croup?

Including the specification for mild, moderate and severe

What are the signs of impending RF?

A
  • 12-48 Hx fever, non specific cough, rhinorrhoea
  • Sudden onset seal like barking cough, hoarseness
  • Symptoms worsen at night

Mild –> barking cough, no stridor, no recession, no agitation or lethargy at rest
Mod –> barking cough w stridor and recession. No agitation or lethargy at rest
Severe –> barking cough w stridor and recession. Agitation and lethargy at rest

Cyanosis, decreasing consciousness, pallor, recession

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3
Q

What are the investigations for croup?

What would an x ray show?

A

Clinical diagnosis

-Antero/post/lateral x ray –> steeple sign –> narrow trachea

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4
Q

What is the management of croup and its differing severity?

A
  • Most cases actually self limiting and resolve w/i 48 hours
  • Single dose dexamethasone 0.15mg per kg

Mild

  • Manage at home
  • Fluids, paracetomal for fever
  • Admit to hospital when –> <3 months, congenital heart defect, inad fluid intake (e.g no wet nappy w/i 12 hours or <75% normal), not coping, live far, immunodeficeincy
  • Advise to seek Tx when at home and –> continous stridor develops, recession, agitation

Mod-severe

  • Admit to hospital
  • 8/10L oxygen by blow by
  • Possible nebulised adrenaline when agitated
  • Too ill to swallow dexa –> consider IM dexa or 2mg nebulised budesonide.
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5
Q

What is pyloric stenosis?

A
  • Pyloric sphincter –> ring of smooth muscle between the stomach and duodenum. When hypertrophies –> narrows lumen.
  • After eating increasing peristalsis to move food from stomach to duodenum, when pyloric stenosis food can’t get through and therefore goes up oesophagus and results in projectile vomiting.
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6
Q

What is the typical presentation of pyloric stenosis?

What would be the finding on examination?

A
  • Typically presents in first few weeks of life
  • Projectile vomiting typically 30mins after feeding
  • Failure to thrive, skinny, pale.

-On examination –> olive like mass in upper abdomen due to pyloric hypertrophy.

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7
Q

What investigations are carried out in suspected pyloric stenosis?

A
  • Blood gas –> hypochloride. Metabolic alkalosis due to vomiting up stomach acid hydrochloric acid.
  • Diagnostic –> Abdo US –> visulise thicken pylorus
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8
Q

What is the management for pyloric stenosis?

A

-Laparoscopic pyloromyotomy –> Ramstedt’s operation. An incision is made in the smooth muscle of the pylorus to widen the canal allowing that food to pass from the stomach to the duodenum as normal.

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9
Q

What is whooping cough?

What causes the characteristic whooping?

A
  • URTI caused by bordetella pertussis –> g-ve bacteria
  • Causes bouts of severe paroxysmal coughing –> can’t properly take in air during bouts, results in forceful intake of air when coughing ends w a loud inspiratory whoop
  • Spread by aerosol droplets
  • Children and pregnant women vaccinated
  • Vaccine doesn’t provide lifelong protection –> can still get even though been vaccinate
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10
Q

What is the presentation of whooping cough?

What are the three phases?

A

1 week of incubation then 21 day period of being infectious

-Phase 1 –> 1/52 of coryzal symptoms w low grade fever and cough.

  • Phase 2 –> paroxysmal coughing –> coughing becomes more severe and occurs in bouts w cough free periods in between.
  • Coughing can be so severe cause –> vomiting, fainting, pneumothorax
  • Not all will have ‘whoop’ can have apnoeic episodes

-Phase 3 –> convalescent –> gradual improvement in symptoms. ‘100 day cough’. Most resolve w/i 2 months.

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11
Q

What are the investigations for whooping cough?

When should you suspect whooping cough?

A
  • Clinical diagnosis
  • Suspect when cough >14 days w no other known cause and one or more of –> post tussive vomiting, paroxysmal coughing, inspiratory whoop, apnoeic episodes (previously undiagnosed in infants)
  • Can do nasal swab w PCR/bacterial culture –> increasing as availability increases
  • Also possible test for anti-pertussis toxin immunoglobulin G when cough present >2 weeks. >5 years then oral fluid, >17 then in blood.
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12
Q

What is the management of whooping cough?

A
  • Notifiable disease –> inform PHE w/i 3 days
  • Admit hospital –> <6 months or vulnerable or acutely unwell or episodes of cyanosis/apnoea.
  • Conservative –> fluids, rest, analgesia
  • Ab –> when presenting w/i 21 days onset symptoms.
  • Ab –> macrolides –> azithro/clari/erythro. Cotriamoxaizole an alternative.
  • Prophylaxis Ab in close contacts –> ?only when vulnerable e.g pregnant
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13
Q

What is wilms tumour?

A
  • A rare, childhood tumour/cancer
  • Embryonic cancer –> undifferentiated mesodermal cells that make up blood vessels walls, kidneys, adrenal cortex
  • Presents in those <5, most commonly ages 1-3
  • <50 cases annually in UK
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14
Q

What is the typical presentation of a wilms tumour?

A
  • Consider in those <5 presenting w
  • Palpable abdominal mass, possibly w abdo pain or haematuria
  • Other possible symps –> lethargy, HTN, weightloss
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15
Q

When should you urgently refer for suspected wilms tumour?

A

VERY URGENT W/I 48 HOURS FOR SPECIALIST ASSESSMENT

  • Palpable abdo mass
  • Unexplained visible haematuria
  • Unexplained abdo organ enlargement
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16
Q

What are the investigations for wilms tumour?

A
  • Abdo US –> to visualise kidneys
  • CT and MRI to stage
  • Histology and biopsy to confirm
17
Q

What is the treatment of wilms tumour?

A
  • Surgery –> removal of tumour and nephrectomy
  • Adjuvant chemo or radio
  • Good prognosis - 90% 5 year survival rate