Paediatrics Flashcards

1
Q

Developmental milestones for 6 weeks old: Gross motor

A

1.) Good head control – raises head to
45o when on tummy,
2.) Stabilises head when raised to sitting
position

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2
Q

Developmental milestones for 6 weeks old: Fine motor

A

1.) Tracks face/object

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3
Q

Developmental milestones for 6 weeks old: Speech/language

A

1.) startles at loud noise

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4
Q

Developmental milestones for 6 weeks old: Social

A

Social smile

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5
Q

Developmental milestones for 6 months old: Gross motor

A

Gross Motor:
1.) Sit without support, rounded back
2.) Rolls tummy (prone) to back (supine)
. Vice versa slightly later.

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6
Q

Developmental milestones for 6 months old: Fine motor skills

A

Palmer grasp
Transfer hand to hand

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7
Q

Developmental milestones for 6 months old: Language and speech

A

1.) Turns head to loud sounds
2.) Understands “bye bye” / “no” (7m)
3.) Babbles (monosyllabic)

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8
Q

Developmental milestones for 6 months old: Social

A

1.) Puts objects to mouth (stops at 1yr)
2.) Shakes rattle
3.) Reaches for bottle / breast

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9
Q

Developmental milestones for 9 months old: Gross motor skills

A

Gross Motor:
1.) Stands holding on
2.) Sit up straight

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10
Q

Developmental milestones for 9 months old: Fine motor skills

A

1.) Inferior pincer grip
2.) Object permanence

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11
Q

Developmental milestones for 9 months old: Language and Speech

A

1.) Responds to own name
2.) Imitates adult sounds

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12
Q

Developmental milestones for 9 months old: Social

A

Holds and bites food
Stranger fears

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13
Q

Developmental milestones for 12 months old: Gross motor

A

Walks alone

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14
Q
A
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15
Q

A 12-year old girl presented with palpitation for a few week and after performing an ECG was shown to have supraventricular tachycardia. What are the 3 types of SVT?

A

1) AVRT (including WPW)
2) Atrial:
-Sinus Tachycardia- Regular
-Atrial fibrillation - Irregular
-Atrial Flutter - Regular
3) AVNRT (functional) = most common SVT

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16
Q

What are the measures to treat SVT (Both non-pharmacological in a stable patient, and pharmacological)

A

Non-pharmacological interventions:
Valsalva manoeuvre
Immersing face in ice water

Pharmacological:
Adenosine (first-line)
Amiodarone
Propranolol

17
Q

Describe the features of Wolff-Parkinson-White syndrome on an ECG

A

o Delta waves
o Short PR interval
o Wide QRS complex (>120 ms)

18
Q

Treatment for WPW (3)

A

1.) Vagal Manoeuvres
2.) IV Adenosine
3.) Synchronised DC Cardioversion

19
Q

What is contraindicated in WPW

A

Digoxin

20
Q

If SVT persists unrecognised how might infants present with?

A

1) pallor
2) poor feeding
3) vomiting
4) may progress to lethargy and shock

21
Q

What structural malformations are associated with SVT? (3)

A
  1. Ebstein anomaly (Tricuspid valve defect)
  2. Transposition of great arteries
  3. Hypertrophic cardiomyopathy (heart muscles thickened)
22
Q

3 Months Boy presents with Failure to Thrive for 1 Month. What are the possible differentials?

A

Inadequate intake:
o Organic: impaired sucking, anorexia
o Non-organic: Psychosocial deprivation

Inability to retain:
o Vomiting
o severe GERD

Malabsorption:
o lactose intolerance, milk intolerance
o IBD, Celiac disease
o short gut syndrome, post necrotising enterocolitis (NEC)

Inability to utilize nutrient
o Chromosomal: Down, IUGR
o Metabolic: congenital hypothyroidism, storage disorder, amino acid disorder

Increase energy demand
o Thyrotoxicosis, malignancy, chronic infection, CHF, CRF, VSD

23
Q

Heart failure in neonates. Likely reason and causes? (4)

A

An obstructed systemic circulation
1.) Hypoplastic left heart syndrome
2.) Aortic stenosis
3.) Severe coarctation of the aorta
4.) Interruption of the aortic arch

24
Q

Heart failure in infants. Likely reason and causes? (3)

A

High pulmonary blood flow (Left-to-right shunts) - occurs when blood leaks from the systemic circulation to the pulmonary circulation

1.) Ventricular septal defect (VSD) - Hole between R and L ventricles that leads to O2 rich blood flowing back into lungs

2.) Atrioventricular septal defect (AVSD) - Hole between atrium and ventricle

3.) Large patent ductus arteriosus (PDA) - opening between a baby’s aorta and pulmonary artery is large, causing irregular blood flow and increased pressure in the lungs

25
Q

Heart failure in older children and adolescents. Likely reason and causes?

A

Right/ left heart failure

1.) Eisenmenger syndrome - congenital heart defect causes abnormal blood circulation in the lungs and heart

2.) Rheumatic heart disease - heart valves damaged by rheumatic fever

3.) Cardiomyopathy

26
Q

A 3 month old boy presents with a large VSD with Heart Failure. What are the symptoms he may present with.

A

o Respiratory distress (e.g. during feeding), dyspnea, tachypnea
o Poor feeding and feeding difficulty
o Poor weight gain
o Sweating
o Recurrent chest infection

27
Q

What are the 2 major reason for FTT in heart failure

A

o Use a lot of energy to overcome symptoms of congenital heart disease
o Reduced milk intake due to respiratory distress

28
Q

What are the Common ddx of heart failure with heart murmur in 3 months old?

A

(heart failure in infant period usually due to high pulmonary blood flow = left to right shunt)

Large VSD, AVSD, PDA

29
Q

Management for heart failure in infants

A

Supportive & Nutrition (O2, calorie input) (no fluid and salt restriction in paediatrics unless very
severe)

Pharmacological:
o Diuretics to treat pulmonary and venous congestion: furosemide, spironolactone (K
sparing)
o Inotropic support: digoxin (less used now), dobutamine
o Afterload reduction: ACEI (captopril, only syrup form), hydralazine
o Surgical (Types & Timing)
* can be done at 3-6months old to prevent pulmonary HT
* Open surgical closure
* Interventional catheterization with occlusion device

30
Q

A boy presenting with vomiting, abdominal pain and rapid breathing and acid-base results suggesting DKA
with respiratory compensation. ABG: Na 152 / K 6.2 / pO2 normal / PCO2 low / HCO3 low / BE -20

Describe the Acid-base balance abnormality

A

Metabolic acidosis with partial respiratory compensation

Respiratory acidosis we would expect an elevated PCO2 and a near normal or elevated HCO3. A near normal HCO3 would indicate no metabolic compensation while an elevated concentration would represent some level of metabolic compensation.

If the process was a metabolic acidosis, we would expect to see a declined HCO3 level and a near normal or lower PCO3. The former indicates no respiratory compensation and the latter represents some level of respiratory compensation for metabolic acidosis.

31
Q

DKA signs

A

Kussmaul breathing
Pear drop breath
Reduced skin turgor

32
Q
A