Oxygenation Key Terms Flashcards

0
Q

Arterial Blood Gas (ABG)

A

Provides a direct indication of oxygen and carbon dioxide exchange and the acid-base balance within the blood.

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1
Q

Apnea

A

The absence of breathing -manifested by lack of respiratory effort can lead to respiratory arrest. -NIC: identify/treat underlying cause, & administer respiratory stimulants, as appropriate.

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2
Q

Atelectasis

A

The collapse of lung tissue affecting affecting all or part of a lung, impacting the exchange exchange of oxygen and carbon dioxide.

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3
Q

Bronchovesicular

A

The movement of air within the bronchial tree creates a mixture of sounds of air flowing through a tube and the breeziness of the open alveolar lung fields.

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4
Q

Crackles

A

High-pitched popping sounds. Crackles are heard on inspiration and are caused by fluid associated with or resulting from inflammation or exudates, within the lung fields or localized atelectasis.

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5
Q

Dyspnea

A

Labored breathing or SOB that is uncomfortable or painful, also occurs when breathing is insufficient to meet oxygen demand -manifested by: clearly audible, labored breathing; anxiety, distressed facial expression, & nasal flaring. -NIC: Identify/Treat Cause & administer oxygen if O2 falls below 90%

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6
Q

Eupnea

A

The process manifested by oxygenation

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7
Q

Hypercarbia

A

An increased level of carbon dioxide in the blood. *The drive to breathe

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8
Q

Hypoxemia

A

Decreased level of oxygen. -Chest wall in-drawing is an early indicator. -Cyanosis is a late indicator that it’s occurring.

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9
Q

Orthopnea

A

Difficulty breathing when an individual is laying supine -Manifested by: Dyspnea while laying down -NIC: I/T cause, elevate head,neck, & chest while sleeping.

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10
Q

Oxygenation

A

The mechanism that facilitates or impairs the body’s ability to supply oxygen to all cells of the body.

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11
Q

Palpation

A

To feel the areas related to the body system being assessed.

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12
Q

Peak expiratory flow rate (PEFR)

A

Used to monitor the ability of an individual to exhale a specific volume of air related to the individuals age,gender, height, & weight.

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13
Q

Percussion

A

A method of tapping the chest or back to assess underlying structures; tones heard during percussion determine solid-filled or air-filled spaces at the area percussed.

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14
Q

Pneumothorax

A

A partial lung collapse resulting from air or gas collecting in the lung or pleural space that surrounds the lungs. -Respiratory Emergency -manifested by: chest pain, SOB -NIC: I/T cause, observation, needle or chest tube insertion, surgery.

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15
Q

Pulmonary function test (PFTs)

A

Provide information about ventilation airflow, lung volume, and capacity and the diffusion of gas, and they incorporate spirometry, peak flow meters, and the body plethysmograph.

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16
Q

Rhonchi

A

Is a long, low-pitched sound that continues throughout inspiration. Rhonchi suggests blockage of large airway passages which can sometimes be cleared with coughing.

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17
Q

Stridor

A

A high-pitched sound within the trachea and larynx that suggests a narrowing of the tracheal passage.

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18
Q

Surfactant

A

Controls surface tension and keeps the alveoli from collapsing and sticking to themselves.

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19
Q

Thoracentesis

A

Both an intervention and a test. Performed to drain excessive pleural fluid from between the pleural linings. -fluid drained is often analyzed for blood, fiber, & microbe content.

20
Q

Ventilation-perfusion

A

The movement of oxygen across alveolar-capillary membrane into a well-perfusing capillary. -the concentration levels of O2 & CO2 dictate the movement of each gas across the alveolar-capillary membrane.

21
Q

Vesicular

A

The sounds of air moving into & out of the lobes at the alveolar the alveolar level. -they are soft & breezy sounds

22
Q

Wheezing

A

A high-pitched whistling sound most often heard on expiration and caused by the narrowing of bronchi, but wheezes can also be heard on inspiration. -when inflamed can be low-pitched, grating sound caused by pleural surfaces rubbing together. Occurs more during inspiration, but can also be heard during expiration.

23
Q

Respiratory Rate of 30-60 per minute (normal)

A

Newborn

24
Q

RR of 20-40 breaths/min

A

Infants

25
Q

RR of 10-20 breaths/min

A

Adult

26
Q

Respiratory Acidosis

A

⬆️CO2➡️vasodilation➡️⬆️ICP & PR Client c/o headache, irritability, ⬇️ LOC, flushed skin. Important in chest trauma, aspiration, pneumonia, & OD Be alert in pt with problems r/t airway clearance, limited ambulation, anxiety or signs/symptoms of ⬇️ O2

27
Q

Pain from ischemic events including: -Cerebral -Cardiac -Shock States -Pulmonary -Pediatric congenital issues such as heart defects -Adult infarcts, CHF, valvular issues, cardiomyopathy

A

⬇️O2 to tissues manifests as pain -Assess related symptoms such as ⬆️PR, RR, BP, restlessness, anxiety, diaphoresis, reports of discomfort.

28
Q

⬇️Tissue perfusion creates oxygen deficit to organs.

A

Perfusion -Assess perfusion including pulses, nail beds, color, body position for comfort, orientation. -Administer Oxygen -Pharmacotherapy to improve CO, surgery to correct defect -Monitor ABGs

29
Q

Thoracic Wall Symmetry Assessment

A

Normal findings: trachea is midline, hand placement on chest is symmetric. Abnormal: asymmetry of movement occurs, decreased expansion, trachea shifts from midline.

30
Q

Muscles of Breathing Inspection

A

NF: Chest gently rises and falls, neck muscles are relaxed, trachea is midline, intercostals raise the chest up & out with inhalation and calmly relax with exhalation. Abnormal: retraction of intercostals, sternocleidomastoid muscles in neck contract, posturing occurs.

31
Q

Skin Assessment related to Respiratory System Findings

A

NF: pink skin=adequate oxygenation of the cells thru out body. Abnormal: cyanosis (blue coloring) in fair or. or gray coloring in darker pt.

32
Q

RS: Nail bed Assessment Findings

A

NF: Nail beds are an extension of the finger & are normally curved with a 160* angle of the nail bed to the finger. Abnormal: Clubbed nail beds have an angle of 180* or greater, depending on the duration of time am individual has had hypoxemia.

33
Q

Arterial Blood Gas Values (ABGs) Normal ranges

A

pH=7.35-7.45 PaCO2= 35-45mm Hg PaO2= 75-100mm Hg HCO3= 24-28 mEq/I

34
Q

Independent Nursing Oxygenation Interventions

A

-Deep Breathing Exercises -Positioning -Encouraging Smoking Cessation -Monitor Activity Intolerance -Promoting Secretion Clearance -Suctioning -Assisting pt. with ADLs

35
Q

Collaborative Oxygenation Nursing Interventions

A

-Improving Nutrition (Diatary, MD) -Pharmacological Therapies (MD, RX) -Non-Pharmacological Therapies i.e. Oxygen admin. & thoracic catheter insertion. (MD) (TCI is used when fluid enters the pleural cavity, causing lung collapse. Emergency conditions treated as surgery.)

36
Q

Nasal Cannula -Flow Rate Setting? -O2 Concentration (FiO2)?

A

FR: 1-6 L/min FiO2: 24%-44%

37
Q

Oxymizer Flow Rate? FiO2?

A

FR: 1-6 L/min FiO2: 24%-88%

38
Q

Vapotherm Flow Rate? FiO2?

A

FR: 1-40 L/min FiO2: 24%-100%

39
Q

Face Mask Flow Rate? FiO2?

A

FR: 5-10 L/min FiO2: 30%-50%

40
Q

Nonrebreather Flow Rate? FiO2?

A

FR: 10-15 L/min FiO2: >60%

41
Q

Venturi Mask Flow Rate? FiO2

A

Venturi is set with jet adapter for FR & FiO2

42
Q

Characteristics of Acute Respiratory Distress Syndrome (ARDS)

A
  1. Initiation of ARDS-alveoli & capillary damage 2. Onset of Pulmonary Edema- alveoli & capillary walls are more permeable, allowing plasma, proteins, & erythrocytes to enter the interstitial space. 3. Alveolar Collapse- protein rich fluid accumulates, inactivating surfactant and damaging alveolar type II cells, as surfactant is lost alveoli stiffen & collapse, increasing breathing effort. -All this interferes with gas exchange across a/c membrane, blood O2 (PaO2) levels fall as well as PaCO2 initially due to rapid expirations. 4. End Stage ARDS- fibrin & cell debris from necrotic cells combine forming hyaline membranes, CO2 can’t diffuse across hyaline mem. PaCO2 levels rise while O2 continues to fall, risk of Respiratory Acidosis r/t ⬆️CO2. If patient doesn’t receive respiratory support, respiratory failure will set in. Even with aggressive treatments almost 50% of ARDS pt. die.
43
Q

ARDS- Initial Clinical Manifestations

A

Initial manifestations develop within 24-48 hours -Dyspnea -Tachonea -ABGs may be in range -Lab findings are consistant with presenting illness -Chest X-Ray to be clear of infiltrates, with the exception of direct Pulmonary illness. Baseline labs & diagnostic tests aid in identifying change in pulmonary status.

44
Q

ARDS-Progressive Manifestations

A

-Progressive respiratory distress -⬆️ RR, Intercostal Retractions, Use of muscles of respiration -Tachycardia occurs as the demand for O2 in cells⬇️ -CXR shows interstitial changes w/ patchy infiltrates -Pulse Oximetry & ABG levels reflect Hypoxemia refractory to O2 admin. -Breathe sounds clear initially, with crackles (rales) & rhonchi developing later. -Respiratory failure progresses causes mental status changes: agitation, confusion & lethargy

45
Q

ARDS Diagnostic Testing

A

-ABGs analysis of O2 levels in blood -Chest Radiography (X-Ray or CT) assessing fluid vol. in lungs. -Blood Tests: CBC, Blood Chemistries, & cultures (help determine cause of ARDS, such as infection.) -Sputum Culture: exact cause of infection

46
Q

ARDS Treatments

A

-Surfactant Therapies -Mechanical Ventilation -Artificial Airways -Nutrition & Fluids -Treatment of infections or other underlying condition

47
Q

Caring for ARDS patients require constant monitoring of ?

A

Airway, breathing, & circulation Any changes in perfusion, LOC, or oxygenation require rapid Nursing interventions to maintain life. Focusing on meeting essential needs of the pt.