Orthopaedics Flashcards
Bone purpose and make up
ORGAN
Functions - stability for locomotion, haematopoeisis, calcium homeostasis
Organic - cells and type I collagen (to resist tension like fibrocartilage - type II is hyaline cartilage)
Inorganic - hydroxyapatite and calcium phosphate to resist compression
Structure of bone
Metaphysis - proximal to growth plate (good blood supply, heals well)
Epiphysis - distal to growth plate
Diaphysis - rest of the shaft
Types of bone
Lamellar - mature, regular arrangement of collagen, strong, few cells - cortical and cancellous bone
Woven - immature, disorganised, in children or adults pathologically/fracture repair
Describing a fracture
- mechanism of injury (trauma, stress, pathological)
- which bone, what part
- closed/compound
- simple/comminuted
- displaced (angulated/shortened - according to distal fragment, no. of cortical widths by if lateral) or not displaced
- intra/extra-articular
- fracture pattern (transverse/spiral/oblique/segmental/greenstick/avulsion)
4 needs in bone healing
CASS Contiguity Alignment Stability Stimulation
Primary bone healing
Abnormal in real life, needs intervention for absolute stability
Cutting cones formed by osteoclasts for intramembranous ossification
Without callus - haematoma, inflammation and proliferation, cutting cones, consolidation
Secondary bone healing
With callus - haematoma, inflammation and proliferation, soft callus, hard callus, remodelling
Endochrondral ossification
Needs micromotion
Fracture time to heal
Perkin’s principles
Upper limb
- if under 12yo, 3-4 weeks
- in adult, 6-8 weeks
Lower limb (double)
- in child, 6-8 weeks
- in adult, 12-16 weeks
Axial skeleton
- 8-12 weeks (rare in children)
Factors affecting bone healing
Biological
- energy transfer
- neurovascular status
- infection
- the patient - age, smoking, diabetes, alcohol
Mechanical
- degree of displacement/reduction
- degree of movement
- mode of infection
Non-union of fracture
If not healed in double the time expected, will never heal naturally
- host factors
- injury factors
- treatment factors
Hypertrophic - excessive strain/movement at site, callus forms but fracture line persists
Atrophic - host + injury factors, absence of callus
Wolff’s law
Form follows function
Bent bone will reform straight according to where the pressure is
Secondary signs of fracture
Lipohaemarthosis
- if intra-articular, commonly in knee
- fat and blood from bone marrow into joint
- leg must be elevated to show fluid level on imaging
Haemarthrosis
- less serious
- haemorrhage into joint space
Principles of treatment of fractures
Aim to restore normal function
REDUCE
IMMOBILISE
REHABILITATE
Reduction of fractures
Only necessary if
- pressure on surrounding tissues
- intra-articular (synovial fluid washes away haematoma so can’t heal)
- multiple fractures (to get patient mobilising sooner)
To restore length, alignment, rotation in extra-articular fracture
Immobilisation of fractures
Non-operative
- plaster of paris cast
- splint
- traction
Operative
- K-wires (still callus formation, just to stabilise slightly, secondary healing)
- screws/plates (holds in absolute stability, primary healing)
- intramedullary nail (callus so secondary healing - need weight bearing 24h after surgery to get micromotion necessary)
- external fixator (secondary healing, for compound fractures, bridging plate so allows micromotion)
Rehabilitation of fractures
The earlier to move the better
MDT - physio, OT, GP, district nurse Analgesia Social worker (adapting housing) Work assessment Repeat risk management - falls, malignancy, epilepsy, osteoporosis
Fractures in children
Commonest in the areas of rapid growth – distal humerus, radius and around the growth plate
Greenstick - break on tension side, buckling on compression side (single cortical #, eg FOOSH)
Torus - 360degree buckling, bulge ‘donut’ around
Growth plate - Salter-Harris
Salter-Harris classification
For fractures of the growth plate in children, or commonly in adolescents (lots of growth, lots of cartilage)
- 90% type II
I - Straight across II - Above III - Lower IV - Two V - ERasure (crush)
Blood supply to NOF
Abdominal aorta -> common iliac
-> internal iliac
- obturator artery to head
- superior gluteal artery to greater trochanteric head
- inferior gluteal artery to lesser trochanter
All not enough to keep head of femur alive
-> external iliac -> femoral -> profunda femoris
-> LCFA + MCFA -> extracapsular anastomotic ring
Blood supply to femoral head
So if in fracture, cut the retinacular/ascending arteries, all blood supply cut off and avascular necrosis
Intracapsular NOF fractures
Shortened, internally rotates (ileopsoas to lesser trochanter pulls up)
Rare than extracapsular
Displaced or undisplaced
Garden’s classification - 1/2 give it a screw, 3/4 hip no more
NEED SURGERY within 36h (avascular necrosis will not heal alone)
- if under 55/undisplaced - cannulated screws
- if 55-75 - THR
- if 75+ - hemiarthroplasty
Extracapsular NOF fractures
More common than intracapsular, and better prognosis and blood supply not compromised
NEED SURGERY (for secondary healing, micromotion)
- intertrochanteric - DHS
- subtrochanteric (needs more support, closer to moment) - long intermedullary nail
Hip problems in children by age
Newborn - developmental dysplasia, dislocatable hips
First year - Tom-Smiths arthritis (septic)
Toddler - irritable hip, spontaneous improvement
Age 5-9 - Legg-Calve-Perthes
Age 10-14 - SUFE (limp, external fixation deformity)
Age 15-30 - early arthritis secondary to childhood problems
Age 50+ - impingement (CAM if cup too shallow, pincer if cup too big)
Old age - osteoarthritis