Ortho part 1 Flashcards
Pathophys of bursitis
Inflammation of the bursa causes synovial cells to multiply and thereby increases collagen formation and fluid production
A more permeable capillary membrane allows entrance of high protein fluid
The bursal lining may be replaced by granulation tissue followed by fibrous tissue
Acute phase of bursitis
Local inflammation occurs and the synovial fluid is thickened
Movement becomes painful as a result
Chronic phase of bursitis
Leads to continual pain and can cause weakening of overlying ligaments and tendons and, ultimately, rupture of the tendons
Etiology of bursitis
Autoimmune disorders Crystal deposition Infectious diseases Traumatic events Hemorrhagic disorders Secondary to overuse
Hx of bursitis
Localized tenderness Decreased ROM or pain with movement Erythema or edema Hx of repetitive movement Hx of inflammatory dz Hx of trauma
PE of bursitis
Tenderness at the site of the inflamed bursa
If superficial, localized tenderness, warmth, edema, and erythema of the skin
Reduced active ROM with preserved passive ROM
With chronic bursitis, affected limb may show disuse atrophy and weakness
Tendons may also be weakened and tender
What should be part of the differential for bursitis?
Tendinitis Muscle injury Septic arthritis- ROM will not be decreased, and fever will be present Ligamentous injury Fracture OA Cellulitis Gout and Pseudogout\ RA
Common areas affected by bursitis
Subacromial Olecrenon Trochanteric Prepatellar Infrapatellar
Labs for bursitis
With septic bursitis, leukocyte count and ESR may be mildly to moderately elevated
Draw BCx if concerned about deep infection
Order ESR, ANA, RF, and anti-CCP where autoimmune dz is suspected
Procedure workup for bursitis
Joint aspiration and analysis to r/o infection or rheumatic causes
May also be therapeutic
Fluid should be analyzed for monosodium urate crystals, cell count with diff, Gram stain, and culture
Cell counts of nonseptic vs septic bursitis
Nonseptic: <2,000, with predominance of mononuclear cells
Septic: >70,000, with a predominance of PMNs
What are the most infected bursae?
Olecranon
Prepatellar
Infrapatellar
Imaging in bursitis
Plain radiography does not help with dx of bursitis but may be useful for identifying triggering pathology
Tx of bursitis
Conservative tx usually: Rest Cold and heat txs Elevation NSAIDs Bursal aspiration Intrabursal steroid injections When septic suspected, give abx -Oxacillin or first-gen cephalosporin -PCN allergy: cipro and rifampin Surgery for chronic or recurrent
When to treat bursitis surgically
Failure of needle aspiration to drain the bursa adequately
Bursa site inaccessible to repeated needle aspirations
Abscess, necrosis, or sinus formation
Need for exploration to assess the extent of infection of adjacent structures
Recurrent or refractory disease after conservative tx
What areas does tendinitis most commonly affect?
Rotator cuff
Insertion of the wrist extensors and flexors at the elbow
Patellar and popliteal tendons and iliotibial band at the knee
Insertion of the posterior tibial tendon in the leg
Achilles tendon at the heel
Etiology of tendinitis
Usually unknown RFs: -Middle aged or older -Repetitive microtrauma -Strain -Excessive or unaccustomed exercise -FQs
Workup for tendinitis
Radiographs if hx of trauma is present, but will be negative with tendinopathy
U/s is good to detect tendinitis
That with u/s is reserved for those whose dx is unclear or who fail conservative management
Nonpharmacologic tx of tendinitis
Rest or decrease activity level: restrict activities that cause pain
Ice for first 24-48 hrs
Splint or immobilize; sling for rotator cuff
Strengthening and stretching exercises once pain has subsided
Pharmacologic and surgical tx of tendinitis
NSAIDs
Consider corticosteroid injection for conservative tx failure
NEVER use injections for Achilles tendinitis
Avoid repetitive injections
Also consider surgery for conservative tx failure