Ortho Flashcards
What EO assessments are made for ortho?
Skeletal
- anteroposterior
- vertical
- transverse
Soft Tissue
- lip competence
- incisal display
- lip protrusion
Discuss AP ortho measurements
From side on, pt in natural head position/Frankfort plane parallel, bipalpation of deepest curve of maxilla + mandible
Skeletal
- 1: normal; chin on/up to 2mm behind zero meridian line
- 2: retrusive; chin behind line
- 3: protrusive; chin in front of line
Discuss vertical ortho measurements
Two methods Linear: Lower Ant. Facial Height - using ruler for numerical value - lower 1/3 should = middle 1/3 — glabella-> subnasale -> soft tissue gnathion
Angular: Frankfort Mandibular Plane Angle
- high/obtuse: lines meet before occiput; inc. LAFH
- av.: lines meet @ occiput
- low/acute: lines meet behind occiput; red. LAFH
Discuss transverse ortho measurements
Measured above + behind and in front of pt
Facial centreline: mid-eyebrow, tip of nose, U lip philtre, chin point
Facial asymmetry: usually mandibular
- true: X-bite + mandibular displacement
- common: molar 3, inc. LAFH
Discuss lip assessment for ortho
Morphology: thin, normal, full
Competency:
- competent: meet @ rest w/o muscular effort
- potentially: separated only by proclined UIs (Class 2/1)
- incompetent: don’t meet @ rest; Skeletal 2, inc. LAFH
Coverage: L lip cover 1/3 UIs
Length: alar base to vermillion border; 22-24mm
Discuss intro-arch dental features for ortho assessment
Crowding: dento-alveolar discrepancy
- mild: <4mm
- mod.: 4-8mm
- severe: >8mm
Spacing: dento-alveolar discrepancy
- not usually measured
- qualify where present
- maxillary median diastema: gap b/w U1s
Rotations
- describe direction from side w/ largest contact point displacement
- i.e. MB, DP
Angulation: mesio-distal tip
Inclination: bucco-lingual
- lat. ceph
— U1s: 109d to maxillary plane
— L1s: 93d to mandibular plane
What inter-arch dental features are in ortho assessment?
Incisor relationship Overjet Overbite Centrelines Molar relationship Crossbites + Displacements
Discuss incisal relationship
1: incisal edge LIs occludes on cingulum plateau UIs
2: incisal edge LIs occludes post. cingulum plateau UIs
- 1: inc. overjet; UIs proclined or normal
- 2: retroclined
3: incisal edge LIs occludes ant. cingulum plateau UIs
Discuss overjet + overbite
Overjet: horizontal distance b/w incisal edge UIs + labial aspect LIs
- av.: 2-4mm
- Class 2/1: >
- Class 3: <
Overbite: vertical overlap of LIs by UIs
- av.: 2-4mm (1/3-1/2)
- complete: contact b/w teeth + teeth/palate
- incomplete: no contact
Discuss dental midlines
U midline (dental) coincident w/ facial midline U+L midlines coincident w/ each other L midline coincident w/ chin point
Discuss molar relationship
1: MB cusp U6 occludes in midbuccal groove L6
2: U6 occludes M
3: U6 occludes D
Discuss crossbites + displacements
Crossbites
- U teeth should occlude B to L; if occlude P = X-bite
- ant. or post.
- unilateral or bilateral
Displacement: mandibular deviation b/w centric relation + centric occlusion
Discuss growth control theories for bone, cartilage and soft tissue matrix
Bone
- no innate growth potential; no growth when transplanted
- growth @ sutures responds to outside stimuli; pulled apart
Cartilage
- nasomaxillary: some innate growth
- mandibular condyle: less growth when transplanted, considered growth site
Soft Tissue Matrix: bone + cartilage react to growth of soft tissue
- cranial vault: size of brain
- nasal + oral cavities: functional need
- mandible: impaired by TMJ ankylosis
Discuss theories of growth modulation
Genetic: homeobox control generic blueprint of growth
Epigenetic
- Functional Matrix Theory
— capsular matrix: indirectly by altering vol. of capsule
— periosteal matrix: directly on skeleton
- Updated functional Matrix Theory
General features of craniofacial growth
Follows somatic growth pattern
Rate inc. pre-/@ puberty
F earlier
Red. facial convexity (class 2); mandible frowns 18+
Continues throughout life
Discuss structures of cranial vault + growth
Comprises - flat bones — frontal — occipital — parietal — squamous part of temporal - sutures + fontanelles
Intramembranous ossification
- apposition: sutures + exterior
- resorption: interior
Structures of cranial base + growth
Comprises
- basioccipital
- ethmoid
- sphenoid
- petrous part of temporal
Endochondrial ossification
Spheno-occipital synchondrosis: affects AP
Surface remodelling
5 mechanisms of maxillary growth
Sutural Surface remodelling Displacement/Myofibroblasts Nasal Septum Functional Matrix
Discuss sutural growth of maxilla
Sutures
- frontomaxillary
- zygomaticomaxillary
- pterygomaxillary
- midline
- zygomatico-frontal (directly)
- zygomatico-temporal (indirectly)
Intramembranous ossification; apposition @ sutures grows forwards + downwards
Discuss displacement growth of maxilla
From 7-15y up to 1/3 forward movement due to passive displacement
Associated w/ sutural growth
Rotational component masked by periosteal remodelling
Discuss nasal septum growth
Cartilaginous growth
1 of 1ry mechanisms of nasomaxillary complex growth
- apposition @ sutures
- surface remodelling: down + forward, widening palatal vault
Considerably affects growth of U face
Discuss growth of mandible
Intramembranous Endochondral @ condyle - elongation - ant. + downward Surface remodelling (majority)
Discuss growth rotations of mandible
Determined by
- post. face height: condyles, synchondroses
- ant. face height: tooth eruption, soft tissue growth
Backward (clockwise)
- IO: red. overbite, ant. openbite
- EO: inc. FMPA + LAFH
Forward (anti-clockwise): class 2/2
- IO: inc, overbite
- EO: red. FMPA + LAFH
Discuss predicting of facial growth
Numerous methods
- 2ry sexual characteristics
- standing height
- age
- skeletal maturity
- dental age
No strong evidence for any of these; no real way to predict max. growth
Ortho relevance of growth + Tx implications
Relevance - Tx quicker in presence of growth - favourable in XLA cases (spontaneous closure) - facilitate — overbite red. — space closure — settling — functional appliance + rapid maxillary expansion
Implications
- orthognathic: timing + stability
- timing implant placement
- stability
Reasons for early loss of teeth
Local
- caries
- trauma
- ectopic eruptions
Systemic
- gingival hyperplasia, poor OH
- cerebral palsy: red. mobility = poor OH
- syndromes/disorders
Define balancing and compensating extractions
Balancing
- tooth from opposite side of same arch; min. centreline shift
Compensating
- tooth from opposing quadrant
- min. occlusal interference by allowing teeth to maintain occlusal relationship as drift forward
- more difficult to justify
Reasons for early loss A&Bs + management options
Reasons
- retained + effecting eruption UIs
- XLA as wider Tx: mesiodens, expose + bond UE U1
- traumatised + poor prognosis + not avulsed
- carious
Management
- balancing/compensating not req.
- space maintainer: aesthetic (URA + pontic) or crowding concern
Reasons for early loss Cs
Aid eruptions 3s - provide space B 3s - improve position ectopic 3s Interceptive: unilateral loss UC causing centreline shift when other C lost Carious Trauma
Reasons for early loss D&Es
Allow eruption of 4/5s
Aid improvement position ectopic 4/5s
Utilise leeway space to relieve crowding
Carious
Management options for early loss of C&Ds
Balancing: may be req. crowded arch
Compensating: not req.
Unbalanced XLA result
- no centreline shift; don’t balance
- shift w/ complete space closure; don’t balance until sought ortho exam
- shift w/ space remaining M to XLA; monitor to see if movement occurring, if yes seek ortho exam
Management options for early loss Es
Balancing not req.: no appreciable effect on midline
May allow serious tilting + drifting of adjacent 6
Consider space maintainer
General effect of early loss of teeth
Red. arch length =
- crowding
- ectopic eruption
- impaction
- rotation
- centreline shift
- unfavourable molar relationship
Functions of space maintainers
Maintain: arch length, width of leeway space Prevent OE opposing tooth Improve aesthetic (ant.) Aid breaking habits; digit sucking
Ideal space maintainer
Simple
Durable, strong, stable
Passive
Cleansable; no inc. risk caries
When to use space maintainers + considerations
When
- presence of permanent successor
- successor covered by alveolar bone
Consider
- specific tooth
- T since tooth lost
- occlusion
- pt age; cooperation
- OH
- space; already lost? Overcrowding?
Types of space maintainers
Unilateral
- crown/band and loop
- distal shoe
Bilateral - fixed — lingual arch — transpalatal arch — nance palatal arch - removable: URA
Compare indications for lingual arch and transpalatal arch + nance palatal arch
Lingual arch
- lost several post. + L6 erupted
- maintain length + arch
- prevent loss Leeway Space
Transpalatal Arch + Nance Palatal Arch
- lost several post. + U6 erupted
- maintain length + width
- prevent Leeway Space loss by prevent M drift 6
- modify: habit breaker for digit sucking
Indications for URA
A+Bs lost: can + teeth to URA Habit breaker Maintain U6 + Leeway Space Poor OH Unable to bond enamel
Why are teeth XLA’d for ortho?
Create space for alignment
Orthodontically correct malocclusions
Aid dental development through removal 1ry/2ry teeth
Why is space making required?
Relieve crowding Incisor AP movement to obtain normal OJ Change incisor angulation/proclination Levelling occlusal curves Arch contraction Tooth enlargement or replacement
How can space be created?
Leeway Space: 2.5mm/Q L; 1.5mm/Q U D movement UMs Expansion of U teeth Incisor proclination Enamel stripping XLA
Discuss ortho XLA of incisors
U
- rare: poor aesthetics
- poor prognosis due to severe trauma
- root resorption: ectopic 3
- development malformation: dens in dente, dilaceration, fusion, macrodont
L
- easily relieve crowding where M Class 1 + I crowding
- adult: class 3 + retract LLS
- disadv: crowding reappears; red. intercanine width + inc. OB+J
- req. bonded retainer
Discuss ortho XLA 4s and 5s
4s
- provide 8-9mm space
- mod-severe crowding + aid ant./post. crowding
- impacted 3s where crowding caused shortage of space
— U4 XLA as erupt pre-3
- 40-60% XLA space available if anchorage not reinforced
5s
- mild-mod crowding: provide 3-8mm
- P/L ectopic: early loss U/LE
- fixed appliances req. contact b/w 4+6
- 25-50% XLA space available if anchorage not reinforced
Common reasons for poor prognosis 6s
Caries
Molar-incisor hypominerlisation
Enamel hypoplasia
Possible effects of early loss of 6s
Localised spacing
OE opposing
Tipping of adjacent
Factors to consider before XLA 6s
Age: dental development Crowding Malocclusion DH: hypoplasia Presence/absence of other teeth: 5s, 8s
Discuss effect of age on 6 XLA
U: spaces close faster spontaneously L - spontaneous closure if T correctly — calcification of bifurcation 7s - early: 5s drift + ectopic - late: 7s erupted + req. ortho for space closure in pt not ideal candidate
Discuss effect of crowding on XLA 6
U: potential for rapid space loss L - spaced: still spaced - aligned: min. spaces - crowded: best results
Are balancing or compensating XLAs req. for 6s?
Compensating
- U6: no
- L6: often yes
Balancing: only if severely crowded premolars
Compare XLA 6s for Class 1 w/ mild crowding to Class 1 w/ mod-severe crowding
Mild
- XLA @ optimum T for 7s
- don’t balance unilateral 6 w/ healthy 6s
- L6: compensate UNLESS L7 erupted + O contact w/ U6
- U6: don’t compensate if L6 healthy
Mod-severe
- XLA @ optimal T for 7 + relieve premolar crowding
- bilateral B crowding: balance; relief + maintain centreline
- L6: compensate to prevent OE + relieve premolar crowding
- labial crowding: little spontaneous relief w/ XLA 6
— delay until 7s erupt + use space for alignment w/ fixed
- OR XLA @ optimal T + Tx once in permanent dentition
Discuss XLA 6s for Class 2 and Class 3 malocclusions
Class 2
- req. space to correct incisor relationship
- L6: @ ideal T for 7s + control 5
- U6
— immediate XLA req.: functional/headgear to correct B segment + fixed (if pt suitable)
— temporise + wait until 7s then reassess
— consider XLA if risk OE or complication of malocclusion
Class 3
- try avoid loss 6s
Discuss ortho XLA 7s and 8s
7s
- rare: so post.
- aid D movement U buccal segment
- relieve mild L premolar crowding
- additional space prevent impacting 8s: no guarantee
8s
- no evidence for XLA to prevent LI crowding
Define class 2/1 malocclusion
Lower incisor edge lies post. to cingulum plateau of UIs
Always inc. overjet
UIs: proclined or av. inclination
Skeletal aetiology of Class 2/1
Usually skeletal 2
- skeletal 1 possible
Mainly w/ retrognathic mandible (80%)
Dental aetiology of Class 2/1
Crowding; push U1s labially
Soft tissue aetiology of class 2/1
Teeth erupt in zone of equilibrium w/ soft tissues thus important
L lip: incompetent lips w/ lip trap (push U1s labially)
Lip muscular tone
- inc. mentalis activity = retrocline L1s (normal resting lower lip line)
Habit aetiology of class 2/1
Digit sucking
- proclined UIs
- retroclined LIs
- inc. overjet
- ant. open bite; usually asymmetric
- narrow U arch w/ or w/o unilateral post. X-bite
Dental features of class 2/1
Inc. overjet M Class 2 Crowding/spacing Overbite - inc. + deep OR - red./incomplete
If Skeletal 1
- UIs proclined
- LIs retroclined
Rationale for Tx class 2/1
Aesthetics
Risk of trauma
Psychological well-being
Discuss risk of trauma associated w/ Class 2/1
Overjet
- 5mm 22% trauma incidence
- 9mm 24%
- > 9mm 44% (IOTN 5a)
No lip protection
Overbite
- pain
- gingival stripping -> PD
5 management options for class 2/1
Accept Growth modification Headgear Camouflage Orthognathic surgery
Define functional appliances
Ortho devices that utilise forces generated by stretched MoM, facial expression + periodontium to change position of teeth and/or jaw relationships in actively growing pt
Discuss accepting class 2/1
Aesthetics acceptable
Pt not concerned
OH not good enough for appliances
Must
- explain risk to pt
- provide mouth guard; red. trauma risk
Classification of functional appliances
Mode
- myotonic: passive muscle stretching
- myo-dynamic: muscular stretching during function
Retention
- tooth: passive (bionator), active (twin block)
— easy, tolerable, adjustable
- soft tissue: Functional Regulator (Frankel)
— complex, poorly tolerated
— changes zone of equilibrium
Contra/indications for growth modifications for class 2/1
Indications
- motivated
- actively growing
- mod-severe AP discrepancy
- inc. overjet
- inc. overbite
- red./av FMPA + LAFH
- lip trap
- proclined UIs, retroclined LIs
Contraindications - poor OH - non-growing - mild AP discrepancy - inc. FMPA w/ red. overbite — overbite red. in Tx - retroclined UIs, proclined LIs
Dental and skeletal effects of growth modification of class 2/1
Dental: 70%
- UIs retrocline
- LIs procline
- LMs erupt M
- UMs drift D
Skeletal: 30%
- mandibular growth @ condyles (1-2mm)
- v little restriction of maxilla (0.7mm)
- glenoid fossa remodel ant.
Discuss headgear, camouflage + orthognathic surgery in Tx of class 2/1
Headgear
- generate force to maxilla restrict AP growth
- allow catch up of mandible
- can direct force to in/extrude Ms = control VD
- compliance: wear 14h/d force 500g/side
Camouflage - fixed appliances - mild-mod. Skeletal 2 - non/XLA — U4s: easier move teeth back — L5s: preferable w/o
Orthognathic
- surgery + fixed
- adults; growth complete
- severe skeletal AP or VD discrepancy
- poor facial appearance
Define class 2/2
LIs edge lie post. to cingulum plateau UIs
UIs retroclined
Overjet usually min./dec. can be inc.
Skeletal aetiology of class 2/2
AP
- mild-mod skeletal 2
- skeletal 1 + mild 3 (rare) possible
Vertical
- red. VD
- red. FMPA
- associated w/ forward growth rotation of mandible
- progenia: prominent chin
Soft tissue aetiology of class 2/2
Influence of skeletal pattern
If LAFH red.
- lower lip line effectively higher on crowns UIs
- high resting lower lip line = retroclined UIs
Muscle activity + tone: inc. mentalis activity = high lower lip line
If lower lip line high (not v higher) U2s escape effect = av. inclination whilst U1s retroclined
- if lower lip line v higher = all UIs retroclined
Dental aetiology of class 2/2
Acute crown-root angle (crowns flatter)
Thin labial-palatal thickness
Retrusive maxillary + mandibular dento-alveolar process
Dental features of class 2/2
Retroclined U1s
U2s
- crowded
- M-L rotated
- normal or proclined (depending on lip line)
Crowding: exacerbated by retroclination U1s (red. arch length)
Inc. overbite
- retroclination UIs = inc. inter-incisal angle
- red. VD + skeletal 2 = no occlusal stop to LIs = OE
— occlude w/ UIs or P mucosa
— severe = trauma P mucosa or L gingiva LIs
Tx rationale for class 2/2
Aesthetic concerns
Dental health
- traumatic overbite (IOTN 4f)
- crowding
Tx options for class 2/2
Accept
Growth modification
Camouflage
Orthognathic surgery
Discuss accepting class 2/2
Aesthetics acceptable Pt - not concerned - not suitable (OH) Overbite: not significant clinically or aesthetically
Discuss growth modification Tx for class 2/2
Indications
- growing pt
- mild-mod skeletal 2
Functional appliance to convert to Class 2/1
- procline UIs
- inc. overjet
Finish w/ fixed to create class 1
Discuss functional appliances used for Tx class 2/2
Procline UIs by
- URA: Expansion + Labial Segment Alignment Appliance
- Twin block w/ proclining spring
- sectional fixed appliance
Discuss camouflage + orthognathic Tx of class 2/2
Camouflage
- fixed appliances
- mild-mod skeletal 2
- non/XLA: U4s, L5s (depends on tooth condition)
- none: generate space by proclining all Is
Orthognathic - surgery + fixed - suitable — adults: growth complete — severe skeletal AP or VD — poor facial appearance
What is stability of class 2/2 Tx dependent on?
Red. overbite + inter-incisal angle
If IIA not corrected OB will relapse as no occlusal stop to LIs
Define class 3
LIs edge lie ant. to cingulum plateau UIs
Skeletal 3
Molar Class 3
Skeletal aetiology of class 3
Maxillary hypoplasia (retrusion) Mandibular prognathism (protrusion) Combination
Dental aetiology of class 3
Maxillary hypodontia Narrow U arch, broad L arch Pseudo class 3: posturing forward to comfortable bite
Soft tissue aetiology of class 3
Not important part of class 3
Other etiological factors of class 3
Genetics: Hapsburg royal family
- narrow U arch
Craniofacial anomalies
- cleft lip + palate
- Binders syndrome
Indications for Tx of class 3
X-bite + displacement
Predicted pattern of future growth (mandible grows more)
Aesthetics
Functional problems
Management options for class 3
Accept: do nothing or wait Interceptive - URA - functional - face mask/reverse head gear Camouflage Orthognathic
Define early, intermediate and late in terms of ortho Tx
Early
- mixed dentition
- <10yo
Intermediate
- permanent dentition
- growing
- 10-16
Late
- permanent dentition
- non-growing
- > 16yo
Discuss early interceptive Tx of class 3
If do nothing undesirable changes to malocclusion occur
Improvable components @ this stage; not severe
- ant. X-bite
- post. X-bite + displacement
- risk of trauma/dehiscence
Problems: compliance, stability, growth
Intermediate interceptive Tx options for class 3
Growth modifications
Camouflage
Discuss growth modification for class 3
Functional appliance or reverse headgear
Indications
- cooperative, understand pt
- mild-mod
— severe; Tx not beneficial at this stage
Tx not guaranteed
Has to be worn whole T maxilla growing = long Tx T + potentially poor results
Discuss camouflage of class 3
Indications
- mild-mod skeletal 3
- pt happy w/ facial profile
- proclination UIs + retroclining LIs possible
- good overbite
Method
- fixed w/o XLA; procline U, retrocline L
- fixed w/ XLA; U5s, L4s (need more space ant.)
May req. U arch expansion to correct X-bite
- URA
- tri/quad helix
Problems associated w/ intermediate Tx of class 3
Unfavourable growth (unpredictable)
Late presentation of non-growing pt
Pt unhappy w/ facial profile
Discuss late Tx of class 3
Orthognathic surgery + fixed
Pt must have stopped growing before commencing
- growth modification not possible
If facial profile not a concern = camouflage
Why is it important pt chooses camouflage or orthognathic?
As cannot do both
First stage of orthognathic is decompensation which would reverse camouflage = extended Tx time, cost, compliance
Reasons for IOTN
Uniformity of ‘need’ Prioritise need Risk:benefit for pt Monitor standards of care Audit + research Education + teaching
Define MOCDO
Missing: 5i/h, 4t/h
- congenital absence
- traumatic loss
- ectopic/impacted
Overjet: 5a/m, 4a/b/m
- reverse: all Is lingual
Crossbite: 4c/l
- + displacement
contact-point Displacement (crowding): 4d
- measure b/w anatomical contact points w/ ruler
Overbite: 4e/f
- deep/complete/traumatic
- Openbite: 4e, 3e
IOTN Grade 5
i: impacted
h: extensive hypodontia (>1 tooth/Q) w/ restorative complication
a: overjet >9mm
m: reverse overjet >3.5mm w/ masticatory + speech difficulty
p: cleft lip/palate + craniofacial anomalies
s: submerged deciduous tooth
IOTN Grade 4
h: less extensive hypodontia
a: overjet >6<9mm
b: reverse overjet >3.5mm w/o masticatory + speech difficulty
m: reverse overjet >1<3.5mm w/ masticatory + speech difficulty
c: ant./post. X-bite w/ >2mm discrepancy
l: post. lingual X-bite w/o functional occlusal contact
d: severe crowding >4mm
e: ant./lat. open bite >6mm
f: inc. + complete overbite w/ gingival/palatal trauma
t: PE
x: supernumerary
IOTN Grade 3
a: overjet >3.5<6mm w/ incompetent lips
b: reverse overjet >1<3.5mm
c: ant./post. X-bite w/ >1<2mm discrepancy
d: crowding >2<4mm
e: ant./lat. open bite >2<4mm
f: complete overbite w/o gingival/palatal trauma
IOTN Grade 2 + 1
Grade 2
- a: overjet >3.5<6mm w/ competent lips
- b: reverse overjet >0<1mm
- c: ant./post. X-bite w/ <1mm discrepancy
- d: crowding >1<2mm
- e: ant./post. open bite >1<2mm
- f: overbite >3.5mm w/o trauma
- g: pre/post-normal occlusion w/o other anomaly
Grade 1: no Tx need
- d: crowding <1mm
Aesthetic component of IOTN
Used when 3d recorded
10 photographs of inc. unpleasant aesthetics
Subjective assessment by dentist
Limitations of IOTN
Lacks skeletal/soft tissue factors - only dental factors No account for growth potential Not index of Tx complexity Doesn’t account for pt demands/concerns Doesn’t account psychosocial effects of malocclusion Generalised spacing not recorded
What is early ortho?
Any ortho assessment or Tx during mixed dentition stage
Why is early assessment important?
Most malocclusions Tx’d in permanent dentition
Small min. req. Tx early
- red. complexity
- may eliminate need for future ortho Tx
What triggers further Ix during early ortho assessment?
Delayed eruption (i) Crowding (d) Inc. OJ (a) X-bite (c) Submergence (i) Palpation of UE U3s >10y (i)
Caries
Deep/open bite (e)
Aetiology of ectopic 3s
Tooth tissue disproportion/crowding (d) Long eruption path 2: absence/abnormality c: prolonged retention Ankylosis Pathology Clefts
Discuss clinical + radiographic diagnosis of UE 3s
Clinical
- bone contour: B/P bulge
- adjacent teeth: position, vitality, mobility, colour
- contralateral 3 erupted
Radiographic Morphology - apex - root resorption of adjacent tooth - size of follicle of 3 (cystic change) Localisation - B-P position - height of crown in relation to O plane - obliquity of long axis - proximity to midline
Tx options for UE 3
Interceptive: XLA C
- if close to line of arch
- normalisation in 78% cases
Later Tx
- no Tx
- XLA 3
- XLA + ortho
- exposure of 3 + ortho
- autotransplantation
Tx for UE 1s
Make space
- XLA: As/supernumeraries
- move 2 D (not all cases)
What is a supernumerary and why are they important?
Excess/extra tooth cf to normal
Most common cause of late/delayed eruption
Classification of supernumeraries
Supplemental: normal form Conical/peg shaped/early forming Tuberculate/barrel shaped/late forming Odontome - complex - compound
Compare conical and tuberculate supernumeraries
Conical
- most common; mesiodens
- root formation early or in time w/ permanent I
- often erupt, may be inverted
- usually don’t impact eruption
- Tx: leave unless causing malposition
Tuberculate - root formation delayed cf permanent I - usually P - rarely erupt or inverted - usually impact eruption - Tx — XLA ASAP not before 6y — not causing delayed eruption may req. XLA pre-ortho
Why should tuberculate supernumeraries not be XLA’d before 6y?
May damage developing permanent I
General Tx options for supernumeraries
Conservative
- XLA $ + 1ry, await eruption permanent
- maintain space
- review 3-6/12
Exposure of UE Open - tooth close to surface - place apically positioned flap + bracket Closed - tooth far away - bond bracket + Au chain - pull
Why should localised I X-bites be Tx’d immediately?
Eliminate displacement Prevent - PD trauma - mobility opposing tooth - excessive tooth wear
Aetiology + pathogenesis of infraoccluded teeth
Aetiology
- idiopathic
- genetic
- trauma
- absence of successor
Pathogenesis
- ankylosis during reparative phase of tooth resorption
Tx factors + options of infraoccluded teeth
Factors
- permanent successor?
- degree
- co-existing malocclusion
- long term prognosis of 1ry
Options
- conservative advocated
- pt not begun growth spurt + 1-2mm infraocclusion = XLA
- if left too late = complete coverage + req. extensive bone removal
Discuss bone resorption mechanism
Osteoclasts recruited by external stimuli
Attach to bone @ clear zone to form seal
H+ ions secreted across ruffled border to demineralise bone
Organic matrix removed by lysosomal enzymes, MMP remove osteoid
What are lacunae?
Pits of bone resorbed osteoclasts
How is bone resorption regulated?
Osteoclast differentiation and function regulated by osteoblast derived factors
RANKL stim. OC formation and function
Osteoprotegrin inhibits resorption
- acts as decoy receptor for RANKL
Discuss cellular response to light ortho force
1-2s: PDL fluid expressive
5s: PDL fibres + cells compressed
Mins: blood flow altered; prostaglandins + cytokines released
Hrs: metabolic changes; OB/OC activity
2-3d: frontal resorption, slight OTM
5-14d: lamina dura removed (no OTM w/o removing)
14-30d: OTM again; resorption + deposition
Discuss cellular changes seen with heavy ortho force
1-5s: PDL completely compressed Mins: blood flow ceased Hrs: ischaemia, necrosis, hyalinisation 2-3d: OB+OC recruited; undermining resorption begins 14d: hyalinisation zone removed 15-30d: lamina dura removed, OTM
What is the ideal force for OTM? What does this result in?
Force just > capillary pressure Dec - risk tissue + cell necrosis - pulpal damage - pt discomfort
What is the centre of resistance?
Point of tooth where single force passed through would result in translation of tooth in direction of force
What affects the centre of resistance of a tooth?
No./SA of roots
Degree of bone resorption
Degree of root resorption
How do root and bone resorption affect the centre of resistance?
Greater bone resorption, CoR more apical
Greater root resorption, CoR more coronal
What are the general optimal forces for different OTM?
Tipping: 30-60gm
Bodily: 100-150gm
Intrusion: 15-25gm
Extrusion: 30-60gm
What is ortho anchorage?
Resistance to unwanted tooth movement
Why is anchorage important?
Prevent waste of space gained from XLA not being used to align teeth
Prevent movement of Ms through fixed + removable appliances
Move teeth with higher anchorage value
Discuss headgear
EO anchorage system
Use Kloehn bow
- attaches to headgear tubes on U6 bands to prevent M movement of Ms (loss of anchorage)
- move Ms D by inc. forces to create space (up to 1/2 class 2)
Discuss temporary anchorage decides
IO device used for indirect anchorage
Titanium implant screwed into alveolus @ level of attached gingiva
No osseointegration thus easily removed when not req.
Use
- moving teeth in severe hypodontia case
- red. OJ where U6s lost
Relationship b/w anchorage value + root SA
Direct
Anchorage value proportional to SA of root
Predisposing factors for root resorption during ortho
Long Tx T Class 2 elastics Roots in cortical plate Nail biting Previous trauma Atopic pt Blunt, short pipette roots Previous ortho Previous radiotherapy FH
Mechanism of root resorption
Cementum adjacent to hyalinised PDL resorbed by cementoclasts
Progress to dentine destruction
Ortho forces removed
- repair by deposition of cementum in areas of destruction
- dentine not replaced
Types of root resorption
Slight blunting: minor blunting of apices
Moderate: <1/4 root
Severe: >1/4 root
Localised/generalised
Discuss moderate generalised root resorption
Most teeth show some resorption
Greater in pt w/ linger Tx T
Shortening of root length; UIs most severe
Clinically insignificant
Discuss severe localised and generalised resorption
Localised
- aetiology: excessive force + prolonged Tx T
- UIs greatest risk
- v high risk: roots UIs against lingual cortical plate
Generalised
- aetiology: unknown
- MH: thyroid deficiency (high risk)
- atopic pt: inc. levels inflammatory mediations
Challenges/limitations of adult orthodontics
Bone turnover: slower; tooth movement slower, more difficult, inc. risk resorption Dental + PD condition - more likely to be worse - previous Tx - resorption - XLA spaces - relapse + stability
Difference in management of overbites in adults cf adolescents
Adolescents
- post. not occluding thus extrude post. w/o intruding ant.
- acceptable as still growing; use functional appliance
- relative intrusion
Adults
- post. occluding thus intrude ant. w/o movement of post.
- true intrusion
Challenges faced w/ XLAs for ortho in adults and potential alternatives
Challenges
- slow space closure/long Tx T
- compliance/acceptance of space
- space recurrence
Alternatives
- interdental enamel reduction
- I proclination
- if XLA: 5s rather than 4s
- move Ms D
- expansion
Define impacted tooth
Tooth that has failed to erupt due to obstruction/barrier to eruption of normally positioned tooth
Incidence of UE teeth
8s: 25%
5s: 20%
U3s: 2%
6s: 0.75-6%
U1s: 0.13%
Aetiology of impaction
Delayed loss of 1ry Abnormal position of tooth germ Ectopia Supernumerary Cysts Tumours Odontomes Ankylosis Dilaceration: invulsion of 1ry Trauma Systemic Genetic
Risks associated w/ impacted teeth
Damage adjacent teeth/structures
- cyst formation
- root resorption
Poor aesthetics
Tx need for impacted teeth (IOTN)
IOTN 5i
Speech benefits
Occlusal function
Psychological benefits
Min. damage adjacent teeth
Req./steps to localisation of UE teeth
Eruption pattern Dental anatomy Visual exam Palpation X-ray
Why is knowledge of eruption pattern and anatomy important for localisation fo UE teeth?
Eruption pattern
- timing of crown + root formation; how far down should tooth be
- eruption dates; late = further Ix
- contralateral tooth erupted
Anatomy
- differentiate 2ry vs 1ry
- erupt into path of least resistance; suggests sites for palpation if ectopic
Discuss visual exam + palpation of UE teeth
Visual - obvious B/P bulges; crown under mucosa - angulation 2: UE 3 - colour changes of 1ry — previous trauma — resorption from impaction
Palpation
- carried out all 9-10yo
- palpate B/L sulcus for crown UE
- check mobility Cs + 2s
- palpate P for crown
- compare contralateral eps. if erupted
When is intervention of UE U1s req.?
Contralateral erupted >6/12
Both UE’d + L1s erupted >12/12
Deviation from normal eruption pattern; 2s before 1s
Aetiology of UE 1s
Hereditary
- supernumerary
- cleft lip + palate
- cleidocranial dystosis
- odontomes
- abnormal tooth:tissue
- generalised retarded eruption
- gingival fibromatosis
Environmental
- trauma
- early XLA/loss 1ry
- retained 1ry
- cystic formation
- endocrine abnormalities
- bone disorders
Management of UE U1s
Remove obstruction: 1ry, supernumerary Ensure sufficient space - URA - sectional fixed appliance - retained appliance Review 3-6/12; 80% erupt unaided
Management when eruption of U1 failed at 12/12
Exposure indicated
Closed if high, open if superficial
Avoid incisions in free gingiva
Palatal approach where possible
Aetiology of UE 3s
Long path of eruption Develop before 2s Small/absent 2s Guided eruption by 2s Cs resistant to resorption Polygenic inheritance
Management strategies for UE 3
No Tx Interceptive Expose + ortho Surgical XLA Transalveolar implant Surgical repositioning
Discuss no Tx for UE 3
Review 6/12 + occasional X-ray
Warn re root resorption 2s + cyst formation
Indications
- cooperative
- age
- appearance of C: colour, shape, root length
- long term restorative options
- 3 position
Interceptive Tx for UE 3
XLA both Cs
- uncrowded mouth = 78% improvement where space
- usually within 12/12
- XLA both to avoid centreline shift
Exposure + ortho for UE 3
When interceptive Tx failed Req. space for 3s - URA - distalisation or XLA 4s Expose + bone (NICE)
Surgical XLA of UE 3
Indications
- poorly positioned
- curved root
- XLA req. for ortho
In uncrowded mouth req. pros replacement
Benefit: eliminate need for X-ray monitoring
Risks associated w/ aligning impacted 3s
Root resorption adjacent teeth Loss of vitality Poor tissue contour Inc. pocket depths 3 root resorption Ankylosis
Aetiology + management of impacted 5s
Aetiology: early loss Es Management - accept - XLA/incorporate into ortho Tx - expose + bone
Aetiology + management of UE 6s
Aetiology
- bulbous E
- crowding
- M path of eruption
- 1ry failure of eruption
Management - observe - persist >8yo: interceptive — XLA E — disimpact 6
Types of ortho appliances
Removable
- Active: URA, functional
- Passive: Hawley retained, vacuum formed retainer
Fixed
- Active: pre-adjusted edgewise appliance
- Passive: fixed/bonded retainer
Difference b/w URA and functional appliance
Both active removable ortho appliances
URA: simple
Functional: postures mandible away from rest position
Design features of removable ortho appliances
Active components
Retentive complements
Anchorage
Baseplate
Discuss active components of ortho appliances
Site of force delivery
Springs: T, Z, palatal finger
- 0.5mm austenitic SS
- optimal force: 25-40g/root
- actives 2-3mm, 1mm movement/mnth
Labial Bow: retract Is
- 0.7mm SS (Hawley bow)
- acrylic must be trimmed behind Is
Screws: move 1/+ teeth
Discuss retentive components of removable ortho appliances
Maintain appliance in mouth
Adams clasps
- 0.7mm SS
- easy to adjust
- versatile; can add auxiliary fittings
Southend clasps: UIs
Fitted labial bow
Ball-ended clasps: 0.6mm SS
Discuss anchorage features of removable ortho appliances
Prevent unwanted tooth movement
IO
- Simplified: anchor teeth > moving teeth
- Reciprocal: equal no. teeth moving in opp. directions
EO: headgear
Features of removable ortho appliances baseplate
Hold components together + provide anchorage
Heat/cold cure acrylic
Incorporate
- split screw
- ant. bite plane: red. OB by discluding B segments allowing OE + compensatory development of alveolus (growing pt only)
- post. bite plane: disclude occlusion allowing correction of ant. X-bite
Uses of removable ortho appliances
Interceptive: correct ant. X-bite; space maintainer Functional Test compliance Red. OB D movement U B segments Post-Tx retainer
Dis/advantages of removable ortho appliances
Adv
- effective for tipping teeth over short distances
- easy to clean
- self-limiting
- cheap
- min. chair side T
- aesthetic
- provide anchorage
Disadv
- compliance
- limited to tipping movement
- speech + mastication difficulty
Indications for fixed appliances
When req. bodily tooth movement
- crown + root movement
- in/extrusion
- de-rotation
- space closure
Dis/advantages of fixed appliances
Adv
- 3D tooth control
- not compliance dependent
- act like bite turbos (red. OB)
- headgear
Disadv
- difficulty cleaning
- risks: perio, resorption, decalcification
- limited anchorage control; may req. adjunctive appliance
Components of fixed appliances
Brackets
Bands
Archwires
Auxiliaries
Types of archwires
Round/rectangular
NiTi/SS
Discuss difference between initial and late archwires
Initial
- flexible + elastic
- engaged in multiple displaced teeth
- round NiTi
Late
- rigid + strong
- resist distortion as teeth moved w/ auxiliaries
- rectangular SS
Types of auxiliaries
Close/open coils
Intermaxillary elastics
Springs
Hooks
What are the functional limitations of fixed appliances?
Alveolar bone has limited capacity for remodelling therefore skeletal pattern offers greatest limit to tooth movement
What is ortho relapse?
Change in
- tooth position (intra-arch)
- arch relationship (inter-arch)
From positions placed @ end of Tx
Causes and types of ortho relapse
Orthodontic relapse: predictable + avoidable
- physiological relapse: return to original malocclusion
- true relapse: poor Tx
Maturational changes: unpredictable
- unfavourable growth
Factors affecting ortho stability post-Tx
Soft tissue Occlusal Facial growth Soft tissue maturation Occlusal maturation Supporting tissue Habits
Discuss soft tissue and occlusal impact on ortho stability
Soft Tissue
- pre-Tx: malocclusion stable as in zone of equilibrium w/ soft tissues
- post-Tx: unstable unless new stability position found
- must assess soft tissues + consider during Dx + Tx planning
- Class 2/1: OJ dependent on lower L action on U labial segment
Occlusal
- ant. X-bite: req. +ve OB
- post. X-bite: req. good intercuspation
Impact of continuing facial growth on ortho stability
Facial growth continues post-Tx
Dentoalveolar adaptation usually maintains O relationships even when skeletal relationship change w/ growth
If intercuspation poor/dentoalveolar adaptation @ limit, O change - class 3, skeletal open-bite
Discuss impact of occlusal maturation + soft tissue maturation on ortho stability
Occlusal
- continues throughout life; changes from teens to adulthood
— dec.: inter-canine width, arch length
— inc.: LI crowding, OB
Soft Tissue
- lip maturation + tone -> uprighting of labial segments thus crowding
Discuss impact of supporting tissues on ortho stability
Incl. supporting bone, PDL, supracrestal fibres
Recently deposited bone esp. susceptible to resorption
- must retain tooth movements until supporting tissues fully adapted
Supporting bone + PDL: 6/12
Supracrestal fibres: >12/12
Define ortho retention
Holding teeth following ortho Tx in Tx position for period req. for maintenance of result
Rationale for ortho retention
Allow PD + gingival reorganisation
Min. changes from growth
Permit neuromuscular adaptation to correct tooth position
Maintain unstable tooth positions; if req. for compromise or aesthetics
Relapse risk factors
Pre-Tx
- rotations
- median diastema
- spacing (esp. adults)
- palatal 3s
- class 2/2
- grossly incompetent lips
- forward tongue posture
- ant. open bite
- PD
During Tx
- expansion
- I advancement/retraction
- XLA spaces (adult)
Types of ortho retention
Help by Tx occlusion
- Class 2/2 reverse OJ held by +ve OB
- lip trap: OJ red. complete, prevent L lip becoming caught
Appliances: all are passive
- removable: vacuum formed, Hawley
- fixed/bonded
Discuss duration of retention
Part time
- removable: nocturnal wear
- no evidence for FT wear removable appliance cf PT
Long term
- relapse potential ongoing
- maturational changes continue until late adulthood
Discuss the Updated Functional Matrix theory of growth
Periosteal matrix
- mechanical loading influence gene expression
- mechano-sensing enables cells to respond to extrinsic loading through mechano-reception and mechano-transduction
Periosteum
- bone loaded dynamically (muscle contraction)
- bone loaded statically (gravity)
Explain how updated functional matrix theory may apply to ortho
Ortho appliances/muscles deform bone
Mechanoreception: osteoblasts/clasts electrically activated
Mechanotransduction: osteoblasts/clasts respond
Gene expression altered
Discuss cheek and nose assessments for ortho
Cheeks: paranasal hollowing; skeletal 3, maxillary hypoplasia
Nose - nasio-labial angle; 90D — M: 90-95; F: 95-10 — av., acute, obtuse - asymmetry - flaring @ base
Define: ant. + post. X-bite and scissor bite
Ant.: 1/+ UIs in linguo-occlusion cf L
Post.: B cusp LP/M occludes B to B cusp UP/M
Scissor: B cusp LP/M occlude L to L cusp UP/M
What should be incl. in ortho Dx summary?
Short description
- Pt: name, age, CO
- I relationship
- skeletal: AP, vertical, transverse
Key dental features
- presence/absence teeth
- condition: caries, restorations
- describe L+U arch
- O features
Relevant X-ray findings + IOTN
What structures are visible on lat. ceph?
Skeletal structures - calvarium - cranial base - facial skeleton; Md, Mx - cervical spine Teeth Soft tissues
Ortho uses for lat. ceph
Pre-Tx record Dx + Tx Planning - skeletal assessment - Is: inclination, angulation, position - UE teeth - soft tissue profile assessment Assess/monitor growth During Tx - end of functional appliance Tx - monitoring anchorage req. + I inclination (before space closure) - Tx progress (surgical case) End of Tx/retention Research
Ideal features of point on ceph for tracing
Valid: represent accurately the structure concerned
Reproducible: repeatably, accurately identifiable
What are the sella and naison points on a lat. ceph?
Sella: midpoint sella turcica
Naison: most ant. point frontonasal suture
What are the orbitale, porion, menton and gonion points on lat. ceph?
Orbitale: most ant., inf. point margin of orbit
Porion: uppermost, outermost point EA
Menton: most inf. point of mandibular symphysis in midline
Gonion: most post., inf. point angle of mandible
What are ANS, PNS, A, B points on lat. ceph?
ANS: tip ant. nasal spine
PNS: tip post. nasal spine
A: most post. point profile maxilla b/w ANS + alveolar crest
B: most post. point profile mandible b/w chin point + alveolar crest
What are UIA, UIT, LIA, LIT on lat. ceph?
UIA: upper incisor apex
UIT: upper incisor tip
LIA: lower incisor apex
LIT: lower incisor tip
Lines drawn on lat. ceph
Frankfort: orbitale->porion
Maxillary plane: ANS->PNS
Mandibular: menton->gonion
McNamara: through naison perp. to Frankfort
Functional occlusal plane: through U and L P/Ms
Discuss SNA/SNB analysis of lat. ceph and what it means for ortho
SNA: sella-naison-A angle; av: 81+3
SNB: sella-naison-B angle; av 78+3
ANB: SNA-SNB
- Skeletal 1: 2-4
- Skeletal 2: >4
- Skeletal 3: <2
Discuss dental analysis of lat. ceph
Incisal inclination
UI-Mx: angle b/w Mx plane + line through UIA and UIT
- av: 109 +6
LI-Md: angle b/w Md plane + line through LIA and LIT
- av: 93 +6
Inter-incisal angle: b/w UI-Mx and LI-Md
- av: 135 +10
Importance of determining aetiology of malocclusion
Tx governed by aetiology
- accept/moody skeletal; age? Severity?
Differentiate b/w ortho Tx modalities
Orthopaedic (Growth Modification)
- growing pt w/ skeletal discrepancy
- usually headgear
Ortho Camouflage
- non/growing pt w/ mild-mod. skeletal discrepancy
- correct I, accept skeletal
Orthognathic
- non-growing pt w/ mod.-severe skeletal discrepancy
- correct I + skeletal
Aetiology of Skeletal 2 and 3
2
- Md retrognathia
- +/- Mx protrusion
3
- Mx hypoplasia
- +/- Md prognathism
Soft tissue aetiology of class 2 malocclusion
Class 2/1
- low lip line, fail to control UIs -> OJ
- stability: retract UIs
- lip trap -> exacerbate OJ
Class 2/2
- high lip line; mentalis hyperactive
— U1s: retroclined due to lip pulling back
— U2s: proclined as shorter so not impacted by high lip line
— LIs: retroclined as lack lip support
Effect of strap-like lip on arch
L arch has square shape
Effect of tongue on malocclusion
Size: large
- procline LIs
- spacing
Position: forward -> procline LIs + spacing
What is dentoalveolar disproportion? What is it’s effect?
Discrepancy b/w no and/or size teeth and size of arch/space within arch
Leads to spacing/crowding
Aetiology of crowding/spacing
Size of teeth No. of teeth Position of teeth Red. arch size Insufficient space within arch - early loss 1ry - retained 1ry
Causes of retained 1ry
Successor developmentally absent
Successor ectopic
Local abnormality in alveolar development (infra-occlusion)
Failure of permanent to resorb 1ry
Tooth size aetiology of malocclusions
Microdont: spacing in normal arch
Macrodont: crowding in normal arch
Indications for ortho space creation
Crowding relief OB red. OJ red. Arch constriction Torquing UIs
Methods and factors affecting space creation for ortho
Methods - inc. arch length — Ms D — Is forward - arch expansion - interproximal enamel red. - XLA
Factors
- dental health
- degree of crowding
- total space req.
- aetiology
Mechanism of how habits cause malocclusion
Digit sucking alters intrinsic soft tissue pressures
- > equilibrium b/w teeth + soft tissues lost
- > teeth move
What is the severity of the effect of digit sucking dependent on?
Duration; threshold 6h/d
Freq.
Intensity
Effects of digit sucking
UIs: proclined LIs: retroclined Inc. OJ Bi/unilateral X-bite +/- displacement Ant. OB; usually asymmetric - differential eruption Mx constriction - lower tongue position - inc. buccinator activity
When should digit sucking be stopped by?
5-6y
Methods for dissuading digit sucking
Explain cause + effect
Conservative: 3-6/12
- reward
- reframing
- bitter nail varnish
- plaster
- wearing sock over @ night
Persistent: 6-12/12; fixed/removable appliance
Hypnosis
Discuss management of debonded bracket and loose bands
Debonded - not urgent - usually still attached by elastic to wire — moves/spins -> irritation - Tx — wax to stop moving — ortho appt
Loose - usually no immediate harm - left 3-4/52 — swallowing — enamel demineralisation - Tx — remove if poss. — ortho appt
Management of poking ortho wire
Usually handled @ home w/ wax by pt
Cut wire flush to terminal band/bracket w/ D end cutters
Turn wire in at end
Ortho appt
Management of lost/loose ortho appliance
Urgent: see ortho
Management
- adjust URA + refit
- encourage commitment
Management of swallowed appliance/brace
Infrequent
Small + asymptotic; reassure will pass easily
Airway compromised + symptomatic
- AE chest X-ray
- provide description of inhaled object
EO risks of ortho + management
Usually headgear/protraction face mask
- skin trauma: ill fitting
- skin rash: Ni allergy
- eye damage -> blindness
Tx
- allergy: stop, refer to GP
- ill fitting: stop, don’t refit
- eye injury: AE
- urgent ortho appt
Importance of Dx UE 3s
3s cornerstone of mouth Canine guidance Last tooth to be lost IOTN5i Multidisciplinary care req. Long Tx T
What is ‘late’ eruption for U3s?
Normal: 11-12y
Late
- F: 12.3y
- M: >13.1y
When should radiographs be used for localisation of UE 3s?
> 11y + 3s not palpable or erupted
Sequelae following impacted 3
Dentingerous cyst formation Internal resorption impacted tooth External resorption impacted + neighbouring teeth Ankylosis Infection Crowding 2/4 contact + red. arch length
How to determine if correct T for XLA 6? (Ortho)
Calcification of bifurcation 7: body movement to position of 6
UE5 engaged in roots E: prevent drifting + ectopic
M angulation 7; 15-30°
Follicle 7 contact w/ 6
Additional: presence 5s, 8s?