Opthalmological emergencies Flashcards

1
Q

Why are why marks on the cornea a concern

A

Corneal infiltrate
Corneal ulcer
Scar->herpetic
Severe allergy

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2
Q

Concern about metal on metal

A

Sharp object, metal penetrate through the eye

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3
Q

What are the concerns with a painful photophobic eye

A

Iritis
Keratitis
Acute angle glaucoma

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4
Q

Should topical steroids be used

A

Not if unsure of diagnosis especially if herpes not ruled out

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5
Q

Components of functional eye examination

A

Pupils->afferent/efferent, direct/consensual
Optic nerve->snellen, pinhole, confrontation, VF/red colour
Motility 3, 4, 6->posture, ptosis, cover/uncover, movement

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6
Q

Anatomical eye examination

A
General
Lids, lacrimal, position, movement
Conjunctiva, sclrea
Cornea->clairty, fluroscein stain
Subtarsal lid
Anterior chamber
Iris 
Pupil
Opthalmoscope->red reflex, disc, vessels, periphery, red/white spots, masses
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7
Q

What are sight threatening conditions which require urgent consultation to an opthalmologist

A
Lid/globe lacerations
Chemical burns
Corneal ulcer
Gonoccocal conjunctivities
Acute iritis
Acute angle-closure glaucoma
CRAO
Intraocular foreign body
Retinal detachment
Endophthalmitis
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8
Q

What is CRAO

A

Central retinal artery occlusion

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9
Q

What are life threatening ocular emergencies

A
Proptosis
CN3 palsy w/ dilated palsy
Papilloedema
Orbital cellulitis
Temporal arteritis
Leukocoria
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10
Q

When there is proptosis, what are they at risk of

A

Cavernous sinus fistula or thrombosis

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11
Q

What does a CN3 palsy with dilated pupils suggest

A

IC aneurysm
Herniation
Neoplastic lesion

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12
Q

Which is worse, acid or alkali burns, and why

A
Alkali worse-->
lime, cement, dishwashing, 
caustic soda-->
Even with clear cornea,
can burn for weeks
Acids coagulate tissue
and stop further corneal
penetration
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13
Q

Management of chemical burn

A
\+++Irrigate w/
Saline water-->continuous drip
Swab upper and lower
lids to remove
possible particulate matter
Do not neutralise-->
heat produced= further
damage
Refer urgently!
\+/- dilate
Antibiotics, patch
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14
Q

What is a dendritic ulcer

A

Herpes simplex keratitis

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15
Q

What is hutchinson’s sign

A

If tip of nose if involved with herpes, 75% will have globe involved

Xnose involved, 1/3 eye involved

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16
Q

Management of dendritic ulcer

A

Refer
Aciclovir 5X daily
+/- minimal wipe debridement

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17
Q

Complications of herpes zoster occular involvement

A
Corneal keratitis
Ulceration
Perforation
Scarring
Secondary- iritis, glaucoma, cataracts
Muscle palsies
Severe post herpetic neuralgia
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18
Q

Signs requiring referral

A
Decreased VA
Shallow anterior chamber
Hyphema
Abnormal pupil
Ocular misalignment
Retinal damage
19
Q

What does penetrating trauma inclue

A

Ruptured globe, prolapsed iris, IO foreign body

20
Q

Initial management of penetrating trauma

A
REFER
ABCs
Do not press on eye globe
Dont check IOP 
Check vision, diplopia
Apply rigid eye sheild
Keep head elevated
Keep NPO
Tetanus status
Give IV antibiotics
CT orbits
21
Q

Management of suspected globe rupture

A
CT orbits
Cefazolin + aminoglycoside
NPO
Tetanus
Pethidine
Metoclopramide
22
Q

Management of central retinal artery occlusion

A

Massage globe to dislodge thrombus
Decrease IOP->B blocker, IV mannitol, IV acetazolamide, rebreathing CO2, CCB, ant chamber paracentesis
Treat underlying cause

23
Q

Causes of CRAO

A

Emboli->arrythmia, endocarditis, valvular disease
Thrombus
Temporal arteritis

24
Q

Presentation of CRAO

A
Sudden, painless, severe monocular LOV
RAPD
May have had episodes of amaurosis fugax
Fundoscopy->
Cherry red spot
Retinal pallor
Narrowed arterioles
Cotton wool spots->infarct
After 6 weeks cherry red spot recedes and optic disc pallor becomes evident
25
What is the timeframe to initiate treatment for CRAO before vision loss
2 hours
26
What is the hallmark for central venous occlusion
Dilated arteries and veins
27
What are the associated conditions with CRVO
Hypertension Diabetes Hyperviscosity syndromes Glaucoma
28
How does CRVO present
Rapid monocular vision loss RAPD Fundoscopy->blood and thunder
29
Distinct groups of CRVO
1. ƒƒvenous stasis/non-ischemic retinopathy ŠŠno RAPD, VA approximately 20/80 ŠŠmild hemorrhage, few cotton wool spots ŠŠresolves spontaneously over weeks to months ŠŠmay regain normal vision if macula intact 2. ƒƒhemorrhagic/ischemic retinopathy ŠŠusually older patient with deficient arterial supply ŠŠRAPD, VA approximately 20/200, reduced peripheral vision ŠŠmore hemorrhages, cotton wool spots, congestion ŠŠpoor visual prognosis
30
Etiology of painless sudden LOV
``` CRVO CRAO IO hemorrhage Retinal detachment Optic neuropathy Optic neuritis Migraines ```
31
If suspect temporal arteritis/GCA- investigations and management
``` Refer ESR->elevated C-reactive protein (CRP)->elevated FBC->normochromic, normocytic anemia LFTs->mild elevation temporal artery biopsy temporal artery ultrasound ``` no visual or neurological symptoms or signs 1st line: prednisolone visual or neurological symptoms or signs 1st line: methylprednisolone pulse therapy confirmed GCA ``` 1st line: prednisolone adjunct: aspirin adjunct: osteoporosis prevention recurrent or relapsing disease or severe corticosteroid adverse effects plus: methotrexate ```
32
Purpose of acetazolamide in acute glaucoma
Diamox= carbonic anhydrase inhibitor= | reduction in aqueous humor->reduction in IOP
33
Purpose of pilocarpine 4% in acute glaucoma
Cholinergic= iris sphincter miosis->ciliary muscle +outflow through trabecular
34
Presentation of acute glaucoma
``` presence of risk factors halos around lights aching eye or brow pain headache nausea, vomiting reduced visual acuity eye redness elevated intraocular pressure (IOP) corneal oedema fixed dilated pupil ```
35
Investigations in acute glaucoma
Refer gonioscopy, examination of anterior chamber angle slit-lamp examination automatic static perimetry
36
Initial management acute glaucoma
initial presentation: acute angle-closure glaucoma 1st line: carbonic anhydrase inhibitors and/or topical beta-blocker and/or topical alpha-2 agonist adjunct: topical ophthalmic cholinergic agonists adjunct: hyperosmotic agents->glycerol plus: laser peripheral iridotomy after acute attack resolved (after corneal oedema resolves)
37
Ongoing management of acute glaucoma
residual angle closure after laser peripheral iridotomy with elevated intra-ocular pressure 1st line: topical prostaglandin analogues and/or topical beta-blocker and/or topical alpha-2 agonist->latanaprost + timolol + brimonidine adjunct: carbonic anhydrase inhibitors adjunct: argon laser peripheral iridoplasty (when there is a component of plateau iris) adjunct: lens extraction surgery ± goniosynechialysis adjunct: topical cholinergic agonists adjunct: trabeculectomy or tube shunt implantation
38
Red eye differential
``` Lids/orbit Conjunctival/sclera Cornea Anterior chamber Other ```
39
Lids/orbit/lacrimal causes of red eye
``` Chalazion Blephritis Entropion/ectropion Foreign body Laceration Dacryocystitis ```
40
Conjunctival/scleral causes of red eye
``` SC hemorrhage Conjunctivitis Dry eye Pterygium Epi/scleritis Orbital cellulitis ```
41
Corneal causes of red eye
Foreign body Keratitis Abrasion/laceration Ulcer
42
Anterior chamber causes of red eye
Anterior uveitis AA glaucoma Hyphema Hypopyon
43
Other causes of red eye
Trauma Post op Endophthalmitis
44
What is arc eye/snow blindness and how to manage
``` Arc eye/snow blindness--> Radiation keratitis, welding, skiing without eye protection, treat as for abrasion--> Topical antibiotic, topical NSAID, patch, dilate ```