Ophthalmic Procedures Flashcards
A characteristic feature of postoperative visual loss due to posterior ischaemic optic
neuropathy is:
a) Painful
b) Normal light reflexes
c) Normal fundoscopy
d) Visual inattention
c) Normal fundoscopy
21.1 The most common cause of postoperative visual loss after spinal surgery is
a. Central retinal artery occlusion
b. Central retinal vein occlusion
c. Ischemic optic neuropathy
d. Haemorrhage
e. corneal abrasion
c. Ischemic optic neuropathy
Cardiac: Anterior
Spinal: Posterior
ION
19.1, 20.1 Soon after a peribulbar block, the patient’s eye rapidly becomes proptosed and tense, and the visual acuity is markedly decreased. A lateral canthotomy is indicated to:
a) Allow globe to continue to swell
b) Drain blood from behind eyeball
c) Allow the eye to proptose
d) Reduce pressure on the optic nerve
c) Allow the eye to proptose
Orbital Compartment Syndrome
The orbital compartment is a fixed space with limited capacity for expansion. If something like blood fills part of that space the pressure increases and may result in ischaemia of the optic nerve or the retina. A lateral canthotomy is a way of releasing this pressure.
You have up to approximately 2 hours before irreversible visual loss occurs. It may occur in less than 2 hours however, so speed is of the essence.
use local anesthetic but warn the patient that they may feel pain
Perform the canthotomy:
place the scissors across the lateral canthus and incise the canthus full thickness
Perform cantholysis:
Grasp the lateral lower eyelid with toothed forcepsPull the lower eyelid anteriorlyPoint the scissors toward the patient’s nose, place the blades either side of the lateral canthal tendon, and cut.
By cutting the canthal tendon,the counter pressure of the eyelid on the is relieved and the eye is allowed to proptose and pressure is relieved.
21.2 Stellate ganglion block is NOT contraindicated in patients with
a) Contralateral phrenic nerve palsy
b) Glaucoma
c) Recent MI
d) Arrhythmia
d) Arrhythmia
- caution if conduction disease however
Contraindications are current coagulopathy (or anticoagulated), recent myocardial infarction, pathologic bradycardia, and glaucoma.
Source Radiopaedia
Contralateral stellate ganglion/phrenic nerve block/neuropathy
20.1, 22.2 Your patient underwent a stellate ganglion block 2 hours ago. Prior to discharge you are asked to review the patient in recovery because of a droopy upper eyelid. The patient would also be expected to have ipsilateral
a) Pupillary constriction and reaction to light
b) Pupillary constriction and no response to light
c) Pupillary dilation and response to light
d) Pupillary dilation and no response to light
a) Pupillary constriction and reaction to light
Stellate ganglion block causes ipsilateral Horner’s Syndrome:
Ptosis (eyelid droop)
Miosis (constricted pupils)
Anhydrosis (loss of sweating)
Enophthalmos (sinking of eyeball into the bony cavity that protects the eye)
*Pupillary constriction in response to light is controlled by the Edinger-Westphal nucleus of CN3, which will remain intact.
21.2 Suxamethonium causes a sustained contraction of the extraocular muscles for up to
a) 2 minutes
b) 3 minutes
c) 5 minutes
d) 10 minutes
e) 20 minutes
d) 10 minutes
- best answer; one of those shit questions that depends on your source.
Morgan & Mikhail’s (chapter 36: anaesthesia for ophthalmic surgery):
“ Succinylcholine increases IOP by 5-10mmHg for 5-10 minutes”.
- due to prolonged contracture of the EOM
BARASH:
Succinylcholine increases IOP 7 to 10 mmHg reaching a peak pressure 1 to 2 minutes after IV administration and returns to the baseline in 5 to 7 minutes. This increase may be attenuated by pretreatment with anesthetics, although none completely eliminates the increase in IOP. In the presence of a lacerated globe, this increase in IOP may increase the extrusion of intraocular contents although greater increases in IOP may occur during crying and coughing.
Yao & Artusio’s:
- also quotes same information: increases IOP 7 to 10mmHg, returning to baseline in 5 - 7 minutes.
Stoelting’s:
Intraoccular pressure peaks at 2-4 minutes after administration and returns to normal by 6 minutes
22.1 A patient has undergone a multilevel cervical spine fusion and upon emergence from anaesthesia reports complete visual loss. Fundoscopic examination shows a pale optic disc with haemorrhages. This supports a diagnosis of
a. CRAO
b. AION
c. PION
Ischaemic optic neuropathy (anterior)
https://www.researchgate.net/figure/Top-Funduscopic-examination-revealed-pale-and-swollen-discs-with-small-hemorrhages-on_fig2_6759964
21.1 Sensory innervation of the cornea is by the
A. Ophthalmic division of the Trigeminal nerve
B. Nasocilliary Nerve
C. Frontal Nerve
D. Oculomotor
B. Nasocilliary Nerve
a branch of Ophthalmic division of trigeminal
20.2 The most common cause of post operative visual loss after spinal surgery is
a) Corneal abrasion
b) Retinal artery occlusion
c) Central retinal vein occlusion
d) Ischaemic optic neuropathy
e) Occipital infarct
a) Ischaemic optic neuropathy
Postoperative visual loss (POVL) occurs in 1/60 000–1/125 000 operations. Spinal surgery has the highest incidence of POVL.
American Society of Anesthesiologists (ASA) Post Operative Visual Loss Registry, spinal surgery accounted for 93/131 (70%) of all cases of visual loss after non-ophthalmic surgery.
Of these:
> 83 were attributable to ischaemic optic atrophy (ION)
> 10 were caused by central retinal artery occlusion (CRAO).
CRAO
- caused by direct pressure on the globe causing raised intraocular pressure and compromising retinal perfusion.
- visual loss is usually unilateral and associated with other signs of pressure (e.g. ophthalmoplegia, ptosis, or altered sensation in the territory of the supraorbital nerve).
- Initial careful positioning of the head and regular checks throughout the procedure in case of movement minimizes the risk
- documentation of eye checks should occur every 30mins and horseshoe shaped head rests should be avoided in prone patients
ION
> associated with:
- male gender
- obesity
- increasing blood loss
- operative procedures >6 hrs in length.
- The use of the Wilson frame has also been implicated.
> final common pathway is thought to be hypoperfusion of the optic nerve, there is no clear association with either intraoperative systemic hypotension or with the presence of peripheral vascular disease or diabetes.
> recently updated ASA practice advisory for POVL associated with spinal surgery recommends regular intraoperative testing of haemoglobin concentration. However, it was unable to suggest a transfusion threshold that would prevent POVL.
Other possible causes of POVL:
1. Cortical ischaemia
2. Haemorrhage into a cerebral tumour.
In high-risk cases, assessment of vision should be performed as soon as possible in PACU and an early ophthalmic opinion sought if there is a suggestion of visual compromise.
Initial management
1. optimization of arterial pressure
2. oxygenation
3. correction of anaemia.
Treatment with agents such as acetazolamide has not been beneficial and there is rarely any useful improvement in vision with either injury, so attention should be focused on preventative measures:
1. Careful positioning with the head at the same level as the heart
2. Meticulous haemostasis,
3. Possibly staging prolonged procedures should be considered.
Because of the devastating nature of this complication, patients should be informed of an increased incidence of visual loss after spinal operations that are expected to be of prolonged duration and associated with significant blood loss.
22.1 You are planning to perform a peribulbar block and wish to check the axial length of the eye prior to proceeding. The average axial length of the globe in adults as measured by ultrasound is
a. 20mm
b. 23mm
c. 26mm
d. 29mm
e. 32mm
B 23mm
22.2 The normal axial length of the globe of an adult eye is
a. 20mm
b. 23mm
c. 26mm
d. 29mm
e. 32mm
23mm
21.2 Techniques to improve the speed of onset and spread of a peribulbar block include all of the
following EXCEPT
a) Honan balloon
b) Digital pressure
c) Ocular massage
d) Hyalase
c) Ocular massage
Hyalase
Mixing with lignocaine
Higher concentration
Higher volume
Occular pressure (spread and IOP reduction)
Source: 2x BJA Ed articles
21.2 The medical laser LEAST likely to cause eye injury is
a) CO2
b) Nd:YAG
c) Argon
d) Green light
CO2
Laser danger is proportional to penetration.
Penetration inversely proportional to the laser wavelength.
CO2 laser has very little penetration (~ 10micrometres), as it has a wavelength of 10 600nm.
Helium-Neon laser also has very little penetration.
Nd:YAG is the most powerful, with a penetration of 2-6mm, as it has a wavelength of 1064nm.
21.2 The oculocardiac reflex results in
a) Hypertension
b) Apnoea
c) Junctional rhythm
d) Torsades
c) Junctional rhythm
Up to date: Anaesthesia for elective eye surgery
Oculocardiac reflex manifestations —
Manifestations of the oculocardiac reflex commonly occur when pressure is applied to extraocular muscles.
These include bradycardia (a decrease of 10 to 20 percent in the basal heart rate), junctional rhythms, hypotension, and, rarely, asystole.
This reflex can occur during injection of local anesthesia or during the surgical procedure itself.
Management includes stopping the stimulus (eg, release of traction or manipulation of the extraocular muscles).
If this is ineffective, an anticholinergic medication (eg, atropine or glycopyrrolate) is administered.
The risk of inducing this reflex may be reduced by an effective regional anesthetic block or general anesthesia with adequate depth.