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OMED > OMM 1 > Flashcards

OMM 1 Flashcards

1
Q
A
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2
Q

TMJ dysfunction symptoms

A

pain, clicking when mouth is open or closed, HA, sinus congestion, dizziness, facial pain, tinnitus, hoarseness, ear pain

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3
Q

Digastric and suprahyoid

A

depresses mandible

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4
Q

Depression/opening of the jaw ->

A

depresses mandible

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5
Q

Unilateral contraction of pterygoids

A

contralateral deviation and anterior

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6
Q

Contralateral pterygoids

A

draw articular disc anteriorly to facilitate opening

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7
Q

Direct blow to a closed mouth or WHIPLASH

A

posterior capsule injury

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8
Q

Malocclusion Class 2

A

overbite

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9
Q

Malocclusion Class 3

A

underbite

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10
Q

Upper molar extraction

A

temporal bone compressed into occiput

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11
Q

Lower molar extraction

A

temporal bone compressed into TMJ -> strained sphenomandibular joint -> sphenoid pulled inferiorly and contralaterally

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12
Q

Internal rotation of temporal bone

A

anterior lateral movement of mandible

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13
Q

External rotation of temporal bone

A

posterior and medial movement of mandible

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14
Q

Mandible deviates away from _______ rotated mandible

A

internally

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15
Q

Mandible deviates toward ______ rotated mandible

A

externally

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16
Q

Stylomandibular ligament

A

attaches temporal to mandible

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17
Q

Short leg ->

A

unleveling of occiput -> temporal and TMJ dysfunction

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18
Q

Flexion head

A

external rotation of paired bones (wide, flat)

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19
Q

Extension head

A

internal rotation of paired bones (long, narrow)

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20
Q

CNV1 may be affected by dysfunction of

A

Dysfunction of temporal bone

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21
Q

CNV2 may be affected by dysfunction of

A

temporals, sphenoid, maxillae and mandible

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22
Q

CNV3 may be affected by dysfunction of

A

sphenoid bone

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23
Q

CNV1 dysfunction may affect what

A

ethmoid sinus

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24
Q

Tic doulourex

A

CNV2 dysfunction

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25
Q

Trigeminal neuralgia

A

CNV3 dysfunction

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26
Q

External carotid may be affected by dysfunction of

A

temporal, occipital, sphenoid

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27
Q

Internal carotid may be affected by dysfunction of

A

cervical dysfunction C6-C2

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28
Q

Internal jugular may be affected by dysfunction of

A

temporal

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29
Q

Lymphatics to head may be affected by dysfunction of

A

upper thoracic spine, upper ribs (1-4), and clavicle

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30
Q

External carotid dysfunction may cause

A

weakness and altered sensation on opposite side

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31
Q

Internal carotid dysfunction may cause

A

vision abnormalities, and dizziness

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32
Q

Internal jugular dysfunction may cause

A

head congestion

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33
Q

Which n passes through the pterygopalatine fossa

A

CNV2

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34
Q

Abducens n lies under the

A

petrosphenoidal ligament

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35
Q

Entrapment of CN VI can lead to

A

strabismus and diplopia or sixth nerve palsy

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36
Q

Abducens n may be affected by dysfunction of

A

Sphenoid or temporal dysfunction

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37
Q

Bell’s Palsy

A

dysfunction of temporal bone impinging on facial n

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38
Q

parasympathetics to the eye are carried by

A

CNIII

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39
Q

parasympathetics to the lacrimal gland and nasopharyngeal mucosa are carried by

A

CNVII

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40
Q

CNVII parasympathetic hyperactivity ->

A

thin, watery secretions, excessive tear production

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41
Q

Somatic dysfunction at T1-T4 may cause

A

photophobia, tinnitus and unsteadiness, vasoconstriction

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42
Q

Hearing decrease and vertigo may result from dysfunction of __________ impinging on CN8

A

sphenoid, occiput and temporal bones

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43
Q

HEENT Chapman’s Reflex Points - anterior

A

clavicle to 2nd rib

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44
Q

HEENT Chapman’s Reflex Points - posterior

A

suboccipital musculature, intertransverse spaces C1-C2

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45
Q

All facial bones except mandible are driven by

A

sphenoid

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46
Q

Referred sinus pain follows

A

trigeminal

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47
Q

CNV1 innervates all sinuses except

A

Maxillary (CNV2)

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48
Q

Otitis Media may be caused by ______________ of the temporal bone

A

internal rotation

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49
Q

secretory to submandibular, sublingual and lacrimals

A

CNVII

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50
Q

Parasympathetic n carried by CNVII

A

Greater Petrosal

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51
Q

Parasympathetics carried by glossopharyngeal synapse ____________ and innervate

A

otic ganglion; parotid gland

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52
Q

Sympathetics to the head are carried via

A

T1-T4 -> sphenopalatine ganglion

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53
Q

Vertigo may result from dysfunction of

A

temporal bone

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54
Q

Internal rotation of the temporal bone

A

closes off the ET; high pitched ringing

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55
Q

Treating sinusitis with OMT

A

inhibitory trigeminal stimulation over CNV1 and CNV2

56
Q

relaxing medial pterygoid muscle, allows

A

tensor veli palatine to open ET

57
Q

out pouching of respiratory diverticulum

58
Q

bifurcation into R/L bronchial buds

59
Q

branching into lobes, 3 on R, 2 on L

60
Q

branching into tertiary buds, 16w (terminal bronchial buds) -> 300-700 million sacs in mature lung, 20-70 million sacs

61
Q

Rib lesions are common with

A

extended Type II dysfunctions

62
Q

Diaphragm and quadratus lumborum attach to which ribs

63
Q

Inhalation ribs

A

up in front, down in back

64
Q

Structural Rib Dysfunction

A

Not related to breathing, rather a disturbance due to rib motion and biomechanical restrictions of the thoracic spine

65
Q

key ribs are often

A

structural ribs

66
Q

Functional Thoracic Inlet

A

vertebral units of T1-4, ribs 1 and 2 plus their costo-cartilage, and the manubrium

67
Q

Anatomic Thoracic Inlet

A

manubrium, rib 1, body of T1

68
Q

Chronic Obstructive Lung Disease

A

chronically contracted diaphragm, overuse syndrome of accessory muscles

69
Q

Accessory Muscles: Forced inhalation

A

scalenes, SCM, serratus anterior, external intercostals

70
Q

Accessory Muscles: Forced exhalation

A

rectus abdominus and internal intercostals

71
Q

asthma reflex

72
Q

linking of airway

A

Normal epithelium is ciliated columnar with mucus secreting goblet cells

73
Q

___________ influence is dominant in normal functioning lung

A

parasympathetic (watery mucus)

74
Q

While asthma is “obstructive”, there may be a component of

A

restrictive lung disease

75
Q

Restrictive lung disease limits

A

the amount of air that can get in and out of the lung

76
Q

Obstructive lung disease limits

A

the rate of airflow out of the lung

77
Q

COPD

A

chronic bronchitis AND/OR emphysema + airway hyperreactivity

78
Q

antitrypsin

A

inhibits lung elastase and prevents lung destruction and emphysema/COPD

79
Q

COPD findings

A

hyperinflation/barrel chest, hyperresonance, sd in right upper thoracics

80
Q

FEV1 >80% FEV1 50-80% FEV1 30-50% FEV1

A

stage 1 stage 2 stage 3 stage4

81
Q

Chronic bronchitis

A

long-term exposure to irritant (smoking), productive cough

82
Q

Emphysema

A

abnormal permanent entrapment of air - enlarged alveolar sacs

83
Q

Obstructive Lung Disease

A

air is trapped in lung, maximum inhalation (barrel chest), forced expiration (no IRV, FVC

84
Q

blue bloater

A

COPD - Chronic hypoxemia leads to erythrocytosis, pulmonary hypertension, and eventually right ventricular failure

85
Q

restrictive lung disease

A

A stiff thoracic cage results, increased respiratory effort

86
Q

Order of treatment of thoracic cage

A

Treat the spinal segment first, then any structural rib, then any respiratory dysfunction.

87
Q

key area of lymphatic obstruction

88
Q

Anterior and middle scalenes attach to

89
Q

Posterior scalene attaches to

90
Q

Diaphragm: 3 apertures levels

A

vena cava (T8), esophagus (T10), aorta (T 12)

91
Q

lRT Parasympathetics

A

OA, AA, C2, suboccipital

92
Q

Earl Miller

A

1923 - developed lymphatic pump technique

93
Q

Thoracic pump is more effective if the chest cage is

94
Q

A.T. Still Research Institute established

95
Q

Louisa Burns D.O., a pioneer in osteopathic research, joined AT Still research institute

96
Q

research grants from the AOA to Osteopathic institutions

97
Q

Frank Chapman

A

Neurolymphatic reflexes in 1920

98
Q

“Lymphatic Reflexes: A Specific Method of Osteopathic Diagnosis and Treatment” published in

A

1929 by Chapman

99
Q

Charles Owens, D.O.

A

pelvic-thyroid-adrenal syndrome” (PTA

100
Q

Paul Kimberly, D.O.

A

FAAO re-introduced Chapman’s reflexes in the KCOM curriculum in the late 70’s

101
Q

Ward E. Perrin

A

1943 grad from CCOM, CCOM faculty

102
Q

first recipient of the AOA’s Bureau of Research Gutensohn/Denslow Award in 1984

103
Q

William Garner Sutherland, D.O

A

1939 Cranial

104
Q

Floyd Peckham, DO

A

1921 grad CCOM - helped keep CCOM afloat financially

105
Q

Denslow

A

1941-1943: spinal reflex research

106
Q

HEENT parasympathetics (VS)

A

occiput, C1, C2

107
Q

HEENT sympathetics (VS)

108
Q

The input from both visceral and somatic structures end on common

A

interneurons

109
Q

Lawrence Jones

A

1955 tender points

110
Q

Upper cervical left side dysfunction may cause

A

AV node effects (PNS) –> impaired conduction and dropped ventricular contractions

111
Q

Upper cervical right side dysfunction may cause

A

SA node effects (PNS) –> Bradycardia

112
Q

Upper thoracic right side dysfunction may cause

A

SA node effects (SNS) –> tachycardia

113
Q

Upper thoracic left side dysfunction may cause

A

AV node effects (SNS) –> premature ventricular contractions

114
Q

Cardiac Rhythm

115
Q

Myocardium

A

T1-T5 (L > R)

116
Q

Posterior wall MI

117
Q

Bronchomotor

A

T1-T3 (b/l or ipsilateral)

118
Q

Lung

A

T1-T4 (b/l or ipsilateral)

119
Q

Esophagus

120
Q

Gastric

121
Q

Upper RT

122
Q

Oropharynx

123
Q

Thyroid

124
Q

Ventricular involvement

125
Q

Atrial involvement

126
Q

Anterior infarct

127
Q

Inferior infarct

A

T3-T5L, C2

128
Q

HTN Linkage Pattern

A

C6, T2ESR-R & Left Inhalation Rib 2 T6FSR-L & Right Exhalation Rib 6

129
Q

Underlying CAD

A

Type I curve convex Right

130
Q

Anterior Adrenal CP

A

2-2.5” above & 1” lateral to umbilicus

131
Q

Posterior Adrenal CP

A

b/w T11-T12 b/w SP and TP

132
Q

Anterior Kidney CP

A

1” above & 1” lateral to umbilicus

133
Q

Posterior Kidney CP

A

b/w T12-L1 b/w SP and TP

134
Q

MI Anterior CP

A

2nd ICS near sternum

135
Q

MI Posterior CP

A

b/w T2-T3 b/w SP and TP

OMED (4 decks)

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