Ocular Emergencies Flashcards

1
Q

List the top 10 ocular emergencies that require same day referral?

A
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2
Q

What are the 2 types of chemical ocular injuries? Which is worse? What type of necrosis do they each cause?

A

Alkali injuries cause liquefactive necrosis – can continue to penetrate over time – most household cleaning products and workplace chemicals = alkali = Plaster & cement
Acid injuries cause a coagulative necrosis.

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3
Q

Should you attempt to neutralise chemical ocular injuries? How should you manage these?

A

= NO
- DO NOT attempt to neutralise chemicals. The products of the neutralising reaction can be more toxic than the original chemical.
- If you want to know whether the chemical was acid or alkaline, litmus and urine dipstix can give a reasonable idea. Wash the eye out first as to avoid delay. Alkaline injuries remain alkaline for hours!

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4
Q

List 4 sequelae of chemical ocular injuries.

A

Sequelae of chemical ocular injuries:
- Conjunctivalisation
- Symblepharon

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5
Q

How do we classify ocular traumas?

A

Penetrating – one point of entry
Peforating – one point of entry and one point of exit at the other side
IOFB – object has entered eye and stayed there

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6
Q

What is a Hyphema?

A

Eight ball hyphema = entire anterior chamber filled with blood = poor prognosis as usually significant internal structure damage

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7
Q

How does an anteriorly dislocated lens present? Management?

A
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8
Q

What test can be performed to determine if an ocular trauma is lamellar or penetrating?

A

Seidel test positive when fluorescein dye gets washed away by aqueous fluid as the injury is full thickness  require surgery

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9
Q

How does a Uveal prolapse present?

A

Uveal prolapse
- Peaked pupil
- Corneal haze around laceration

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10
Q

Describe the FAST acronym for management ocular trauma?

A

Abx = Ciprofloxacin – penetrates the eye quite well
Antiemetics – needed as vomiting can increase intraocular pressure further and cause more damage

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11
Q

What does this indicate? How should this patient be managed?

A

Central Retinal Artery Occlusion

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12
Q

List 6 features of Giant Cell Arteritis (Temporal Arteritis)? Which 2 symptoms should you always enquire about? 3 complications?

A

CRAO – Central Renal Artery occlusion

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13
Q

How should you manage Giant Cell Arteritis?

A
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14
Q
A

= pus in the anterior chamber = Endophthalmitis

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15
Q

Features of Endophthalmitis? 2 Hx? 2 Exam?

A
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16
Q

Management of Endophthalmitis?

A
17
Q

What are the Features of Acute angle-closure glaucoma?
- 4 Hx?
- 5 Exam?

A

Acute angle-closure glaucoma (AACG): sudden obstruction of the iridocorneal angle causing a rapid, acutely symptomatic, and vision-threatening elevation of IOP, often > 30 mm Hg.

18
Q

Treatment of Acute angle-closure glaucoma? (5)

A
19
Q
A
20
Q

What is the single most important risk factor for corneal abscess?
- Describe the pneumonic PEDAL for distinguishing between infective and non-infective keratitis?

A

**Corneal Abscess **
- Contact lens wear is the single most important risk factor.
- Any contact lens wearer who presents with an acute red eye has an infection until proven otherwise.

21
Q

Treatment for a corneal abscess? (7)

A
22
Q

What is this?

A

= Orbital Cellulitis

23
Q

How can you differentiate orbital from preseptal cellulitis?

A

RAPD = Relative Afferent Pupillary Defect

24
Q

How is an orbital cellulitis managed?

A
25
Q

What is this? Work-up?

A

Third Nerve Palsy
- pupil-involving right third nerve palsy.
- The lid is ptotic and the eye position is “down and out”.