OCD and Explanations for OCD Flashcards

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1
Q

What does OCD stand for:

A

Obsessive Compulsive Disorder

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2
Q

What is OCD:

A

It is an anxiety disorder characterised by obsessions and/or compulsions according to the DSM-5

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3
Q

State the two required symptoms of OCD:

A
  • Obsessions
  • Compulsions
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4
Q

Explain Obsessions:

A
  • recurrent and persistent thoughts which are intrusive and unwanted
  • lead to anxiety and distress
  • neutralised through another thought/ action
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5
Q

Explain Compulsions:

A
  • Repetitive behaviour (e.g. checking door) or mental action (e.g. counting)
  • done in response to obsessions - reduce anxiety
  • often excessive, can be unrealistic
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6
Q

Describe two differences between obsessions and compulsions:

A

Obsessions are recurrent and persistent thoughts WHEREAS compulsions are repetitive behaviours or mental actions
Obsessions lead to anxiety and distress WHEREAS compulsions are done in response to obsessions to reduce anxiety.

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7
Q

What are the 3 characteristics of OCD:

A
  • Cognitive
  • Behavioural
  • Emotional
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8
Q

State all 6 COGNITIVE characteristics of OCD:

A
  • Recurrent and persistent thoughts
  • Insight into excessive anxiety
  • Selective attention
  • Increased awareness of certain stimuli
  • Hypervigilance
  • Catastrophic thinking
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9
Q

Explain Recurrent and persistent thoughts:

A

Sufferers experience constantly repeated obsessive thoughts, images and ideas of an intrusive nature. They are uncontrollable and cause distress.

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10
Q

Explain Insight into excessive anxiety:

A

Sufferers are aware that their obsessions and compulsions are not rational. In fact- this is necessary for a diagnosis of OCD. If someone really believed their obsessive thoughts were based on reality - symptom of different form of mental disorder.

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11
Q

Explain Selective attention:

A

Increased awareness of source of obsession in new situations

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12
Q

Explain Increased awareness of certain stimuli

A

Sufferers have an increased awareness of certain stimuli, causing obsessions, while ignoring other equally relevant stimuli

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13
Q

Explain Hypervigilance:

A

Sufferers constantly on lookout for threats, both real and imagined. Hyperaware of surroundings, scanning for potential dangers at all times.

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14
Q

Explain Catastrophic thinking:

A

Sufferers assume that the very worst will happen in situations if they do not perform their compulsions e.g. “Someone will break in if I don’t lock my door 4 times over.”

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15
Q

State the 2 behavioural characteristics: (Compulsions)

A
  • Compulsions are repetitive
  • Compulsions reduce anxiety
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16
Q

Explain Compulsions are repetitive:

A

Sufferers feel compelled to repeat behaviours as a response to their obsessive thoughts, ideas and images. E.g. washing hands repeatedly

17
Q

Explain Compulsions reduce anxiety:

A

The vast majority of compulsive behaviours are performed in an attempt to manage the anxiety produced by obsessions. Performing these helps reduce the anxiety. E.g. checking if door is locked makes them feel better

18
Q

State the 2 emotional characteristics:

A
  • Anxiety and distress
  • Guilt and disgust
19
Q

Explain the emotional characteristic - Anxiety and distress:

A
  • obsessive thoughts are unpleasant and frightening and the anxiety that goes with these can be overwhelming. The urge to repeat a behaviour (a compulsion) creates anxiety
20
Q

Explain the emotional characteristic - Guilt and disgust:

A
  • OCD often involves irrational guilt, e.g. over a minor moral issue. Or it may cause disgust towards external things like dirt or germs.
21
Q

What is the genetic explanation for OCD: (biological approach)

A

The genetic explanation centres on OCD being inherited through genetic transmission. - we inherit OCD from our parents and OCD is acquired from specific genes being assed on. The genes that increase the likelihood of having OCD are known as candidate genes.

22
Q

What are candidate genes?

A

Specific genes associated with a disorder

23
Q

Explain how candidate genes lead to OCD + Ozaki

A
  • specific genes lead to a vulnerability for OCD called OCD candidate genes. e.g. 5-HTT (SERT gene) which is responsible for transportation of serotonin in a neuron
  • inheriting a faulty 5-HTT gene leads to disruption to transportation of serotonin - leading to low levels of serotonin in the synapse
  • Ozaki et al (2003) found mutation of 5-HTT gene in families where 6/7 ppl had OCD.
24
Q

Define Polygenic:

A

Caused by more than one gene

25
Q

Explain Polygenic:

A

Other researchers believe OCD is too complex a disorder to be caused by one gene. Believe OCD may be polygenic disorder
- Taylor (2013) did a meta-analysis and found evidence of up to 230 different genes involved in OCD

26
Q

Weakness: too simplistic + the diathesis stress model might offer a better explanation.

A

For example, according to this model certain genes leave the individual more vulnerable to OCD. To develop the symptoms of OCD - there needs also to be an environmental trigger (stressor). ** Ex - Shows - OCD cannot entirely be genetic in origin + traumatic events play a crucial role too - to truly understand the causes of OCD - need a holistic approach that considers both genetic + environmental factors
L- undermines validity of genetic explanation - incomplete
* Cromer et al (2007) - over half patients w/OCD in their sample has had a traumatic experience in their past + OCD was more severe in those with more than 1 trauma.

27
Q

P - Strength: supporting evidence - twin studies.

A

Ev - Nestadt et al - reviewed previous twin studies into OCD - found 68% MZ CR + 31% DZ CR
Ex - Shows - as the prop of shared genes increase from DZ twins (50%) to MZ twins (100%) - likelihood of both twins developing OCD also increases. - OCD develops due to genetic factors + is inherited:
L - Adds validity to genetic explanation of OCD

28
Q

State the two neural explanations for OCD:

A
  • Abnormal levels of neurotransmitters
  • Abnormal brain circuits
29
Q

Explain how Abnormal levels of neurotransmitters lead to OCD:

A
  • Serotonin is responsible for regulating mood by facilitating synaptic transmission from one neuron to another
  • OCD- serotonin is removed too quick from synapse so no chance to pass o signal to the Post synaptic neuron
  • This low level of serotonin leads to issues with mood associated w/ OCD
  • Also dopamine levels are abnormally high in OCD linked with anxiety people with OCD experinece
30
Q

Explain how Abnormal brain circuits lead to OCD:

A
  • Several areas in the frontal lobes of the brain are thought to be abnormal in people w/ OCD
  • One of roles of the ORBITO frontal cortex - send worry signals to THALAMUS e.g. when we come across a potential germ hazard
  • This is mediated by CAUDATE NUCLEUS in BASAL GANGLIA which suppresses minor worry signals and prevents us from acting on every little worry allows major signals to pass to the thalamus which initiates a reaction
  • However in ppl with OCD - CAUDATE NUCLEUS is HYPERSENSITIVE - all major and minor worry signals sent from OFC to THALAMUS which confirms and receives minor signals too- creating a worry circuit.
31
Q

Strength: practical applications in the use of SSRIs

A

Ev - Led to use of SSRI to treat depression - block the reuptake of serotonin into the pre-syn neuron -prolongs the influence of serotonin on post-syn neuron
- Soomro et al reviewed evidence into the impact of SSRI on OCD - found they were effective in 70% of cases
Ex - As SSRIs = effective treatment - infer that the underlying assumption that the treatment is based must
also be valid.
L- Increases validity of neural explanation of OCD + shows it can improve lives

32
Q

Weakness: supporting evidence is correlational

A

Ev - this means the evidence cannot be used to establish a cause and effect relationship between serotonin and OCD*
Ex 1 - It could be that low serotonin causes OCD or that having OCD in the first-place causes serotonin levels of deplete
Ex 2 - If former is true - the neural theory = validated. If latter is true, this suggests the initial cause of OCD
may be beyond scope of biological approach - e.g. OCD traits are learned through observation and imitation of compulsive behaviour and reinforcement of this behaviour
L - Therefore this raises questions about the validity of the supporting evidence and neural explanation itself