OB Exam 3, High Risk Care, FHR, Labor Flashcards

1
Q

what does blood loss lead to in pregnancy and for the fetus?

A

In preg: hypovolemia, anemia, infection

Fetus: premature birth, hypoxemia, death

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2
Q

Placenta previa, s/sx

A

placenta overlying os
1/200 pregs
s/sx = painless vaginal bleeding, 3rd trim, hypovolemia, decreased FHR

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3
Q

risk factors for placenta previa

A

Risk factors = c/s prior, endometrial scarring, abortion with dilation/curretage (D/C), short preg interval, AMA, DM, HTN, smoking, multipara

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4
Q

contraindication for placenta previa?

A

do not do vaginal exam

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5
Q

Placental abruption, s/sx

A

placental detachment before delivery

s/sx = PAINFUL bleeding, fetal hypoxemia bad FHR (late decelerations), hypertonic uterine ctx, tenderness

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6
Q

Risk factors for placental abruption

A

Risks = previous abruption, HTN, AMA, c/s prior, cocaine, meth, smoking, multipara, PPROM, abd trauma, thrombophilia

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7
Q

Placenta accreta

A

Placenta accreta = partial/complete placental invasion of uterine wall

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8
Q

what can placenta accreta lead to?

A

PPH→ DIC → hysterectomy due to risk of bleeding out

Blood loss 3000-5000mL, may need blood transfusion

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9
Q

abortion

A

Spontaneous (miscarriage) = before 20 weeks

non viable fetus

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10
Q

risk factors for abortion

A

Risk factors = increased parity, AMA, diabetes, drug use, autoimmune disease, infection, genetics, uterine/cervical abnormalities

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11
Q

what to give for abortion

A

Give rhogam w/in 72 hr

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12
Q

what are surgical treatments for an abortion?

A
D/C = dilation & curettage 
D/E = dilation & evacuation
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13
Q

when can medical abortions take place? What 2 meds are given

A

Medical = 1st trimester only (13-14 weeks), later surgical

Mifepristone, misoprostol

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14
Q

what is the main reason medical abortions take place

A

cancer in mother or fetal abnormalities

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15
Q

ectopic pregnancy, s/sx

A

Egg growing outside uterus, 95% in fallopian tube, Non viable–can result in hemorrhage

s/sx = sudden, sharp pain, one sided, referred shoulder pain, light bleeding, hypovolemia

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16
Q

risks for ectopic preg

A

Risks = prior EP, fallopian tube abnorm, pelvic inflammatory disease PID, infertility, pelvic abdominal surgery, endometriosis, STIs, tubal surgery

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17
Q

what is surgery for ectopic preg

A

Surgery = laparoscopic surgery

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18
Q

meds for ectopic preg

A

Meds = methotrexate (chemotherapy agent) dissolves ectopic mass

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19
Q

Gestational trophoblastic disease

A

Hydatidiform mole/molar pregnancy = abnormal fertilized egg by multiple sperm, splits and makes neoplasms, rare, trophoblast cell growth

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20
Q

risks factors for GT disease

A

Risks = <20 >35, previous hx, anemia, uterine enlargement, u/s no fetus, VERY elevated hCG

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21
Q

management for GT disease

A

Management = D/C, chemo drug, monitor for increased cancer risk (from incr hCG)

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22
Q

what is the leading cause of pregnancy death?

A

Trauma = leading cause of preg death, hemorrhagic shock

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23
Q

how are outcomes of trauma in pregnancy defined?

A

Outcome defined by injury and when in preg–750-1000mL/min, 8-10min, FHR changes indicate maternal deterioration

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24
Q

to which side do you displace the uterus for CPR?

A

L side

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25
Q

what do drugs/alcohol do to fetus?

A

Low birth weight, develop disabilities, preterm birth, infant death, teratogenic, addiction

Growth defects, facial dysmorphia, CNS impair, behavior disorders, imparied intellectual

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26
Q

why does alcohol affect fetus?

A

Baby has inadequate liver, can’t metabolize alcohol

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27
Q

What do illicit drugs do to mother and fetus?

A

Drugs (cocaine, amphetamines, heroin, ecstasy)

- Cocaine causes vasoconstriction of placenta/uterus→ placental abruption or PTB

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28
Q

what do cigarettes do to fetus/mother?

A

2x LBW, IUGR, increased preterm birth, miscarriage, stillbirth, placenta previa or abruption

3x likely SIDS, asthma, infantile colic, obesity in childhood

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29
Q

what is the 2nd leading cause of infant death? 1st?

A

2nd leading cause of infant death

1st is congenital malform & chromosome issues

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30
Q

what are the ages of very premature, premature, and late premature?

A

Very Premature: Neonates born at <32 weeks
Premature: Neonates born 32-34 weeks
Late Premature: Neonates born 32-37 weeks

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31
Q

What are 2 survival predictors of a premie?

A

Survival predictors = period of gestation & birth weight

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32
Q

What are some issues of premie babies?

A
Respiratory Distress Syndrome (RDS)
Retinopathy of Prematurity (ROP)
Bronchopulmonary Dysplasia (BPD)
Patent Ductus Arteriosus (PDA)
Periventricular-intraventricular hemorrhage
NEC: Necrotizing Enterocolitis
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33
Q

an infant with inadequate muscle tone has:

A

hypotonia

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34
Q

when do eyelids open in fetus?

A

Eyelids fused in premie, (open 26-30 weeks)

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35
Q

What are some characteristics of premies?

A
Hypotonia
Skin – translucent, transparent, red
Decreased subcutaneous fat
Lanugo
Eyelids fused (open 26-30 weeks)
Pinna (thin, soft, flat)
Undescended testes
Tremors/Jittery
Weak Cry
Diminished/Absent Reflexes
Immature Suck/Swallow --> Unable to tolerate oral feedings
Apnea (20 sec +)
Heart Murmur
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36
Q

how to help premie with thermoregulation

A

To help = keep head covered, polyethylene barriers for <29 weeks, warming mattress

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37
Q

how to measure tube for gavage premie feedings

A

Measure tube from mouth to ear, ear to lower end of sternum

OG/NG route, 5-8 French, gravity or pump

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38
Q

What is RDS? s/sx?

A

decreased alveoli surface tension→ atelectasis (moms given betamethasone IM if preterm labor is happening)

s/sx = Tachypnea, Retractions (Seesaw Breathing), Nasal Flaring
→ Hypoxemia, hypercarbia → metabolic/resp acidosis

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39
Q

what are complications of RDS for the baby?

A

Complications = PDA, pneumothorax, BPD, hypotension, pulm edema, hypoglycemia, ROP, intraventricular hemorrhage

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40
Q

What med is administered for RDS?

A

admin exogenous surfactant

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41
Q

What is a CPAP used for in baby? What is used if CPAP doesn’t work

A

for neonates who are at risk for RDS or who have RDS. It can be administered by nasal cannula, nasal mask, nasal prongs, endotracheal tube, or nasopharyngeal route.

mechanical ventilation used when CPAP is not effective

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42
Q

what is HFOV?

A

High frequency oscillatory ventilation

used when mechanical ventilation has proven unsuccessful.
Delivers small volumes of gas at a high rate (greater than 300 breaths/minute). Less traumatic on fragile lung tissue

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43
Q

what are benefits of surfactant therapy?

A

Benefits of Surfactant Therapy = Prophylactic therapy decreases the occurrence of RDS and mortality in preterm neonates.

  • Decreased risk of pneumothorax
  • Decreased risk of intraventricular hemorrhage
  • Decreased risk of bronchopulmonary dysplasia
  • Decreased risk of pulmonary interstitial emphysema
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44
Q

What is necrotizing enterocolitis? s/sx?

A

Necrotizing enterocolitis–neonate GI disease from decreased blood flow, typically 3-10 days after birth

s/sx =
Abdominal Distension
Feeding intolerance
Emesis
Residuals
Bloody stools
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45
Q

what causes necrotizing enterocolitis?

A
  • Altered blood flow regulation, particularly to the intestines.
  • Impaired gastrointestinal host defense when faced with stress/injury to the intestinal tissue.
  • Alterations in the inflammatory response
  • formula feeding increases risk
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46
Q

what meds given for necrotizing enterocolitis? what labs done?
what other interventions?

A

Meds = Antibiotics, Analgesia, Antihypertensive

Labs/tests = abd XR to see gas/lack of, labs (blood culture, CBC, CRP, Stool culture, CMP, ABG)

Gastric decompression
Surgery

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47
Q

what needs to be stopped for enterocolitis?

A

no oral feedings

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48
Q

Retinopathy of prematurity (ROP)

A

incidence of ROP increases as gestational age and birth weight decrease
Occurs primarily in neonates <29 weeks with LBW (<1500g)

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49
Q

what is prevention for ROP?

A

Administer Oxygen (87-94% SPO2)

Oxygen blenders & Oxygen calibrating systems for exact oxygen concentration

Avoid bright lights – cover isolettes with blankets

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50
Q

Meconium aspiration syndrome, s/sx, management

A

Fetal asphyxia in utero, Post term (42 weeks)

Aspiration of meconium fluid → Respiratory failure; inhibits surfactant

Main finding = respiratory distress (flaring, grunting, chest retractions), mec stained amniotic fluid

Management = resp support

51
Q

Hyperbilirubinemia

A

Increased bilirubin in the blood

52
Q

difference between physiological hyperbili and pathological jaundice?

A
  • Physiological
    Occurs after 24 hours (anything before is considered pathological); peak 3 day, progressive head to lower extremities
  • Pathological Jaundice
    Before 24 hours
    Can lead to Kernicterus = Abnormal accumulation unconjugated bilirubin in the brain → neurological disorders
53
Q

what are risk factors for pathological jaundice?

A

Risk factors = asian, native, greek mom; ABO incompatibility (M: O, NB: A or B), Rh incompatibility, low breastfeeding intake (pooping less, clearing bili out less); for newborn = delayed cord clamping, hypoxia, acidosis, hypothermia, hypoglycemia bruising, delayed feedings, cephalohematoma (pieces of broken RBC = bili), prematurity, sibling with hyperbili

54
Q

how does phototherapy for hyperbili work? How much should total serum bili drop after 4-6 hr?

A

Phototherapy results in photoconverting bilirubin molecules to water-soluble isomers that can be excreted in the urine and stool without conjugation in the liver

Total serum bilirubin levels should drop 1 to 2 mg/dL within 4 to 6 hours after the initiation of phototherapy.

55
Q

what are s/sx of neonate withdrawal

A

s/sx of neonate withdrawal = diarrhea, crying, high pitched, excessive sucking, fever, irritability, hyperreflexia, hypertonia, tremors, nasal congestion, poor feeding, emesis

56
Q

what meds for opioid and alcohol withdrawal?

A

For Opioid: Methadone, morphine, clonidine, and phenobarbital
For Alcohol: Benzodiazepines

57
Q

what substances pass through breastmilk?

A

Cocaine, Meth, Alcohol, Heroin, THC

58
Q

what is intrapartum

A

Intrapartum = labor = onset of regular contractions → placenta expulsion

59
Q

what is lightening?

A

Lightening = fetus descending into true pelvis

–urinary frequency at this stage from increased bladder pressure. In subsequent pregnancies, this may not occur until labor begins.

60
Q

braxton hicks vs real

A

Real = Contractions regular and do not change with position, cervix effacement and dilation

Cervix dilates (enlargement, widening) 0-10cm

Cervix effaces (thinning, shortening) 0-100%

61
Q

possible triggers of labor?

A

Not well known

Stretching uterine muscles, estrogen/progesterone changes, oxytocin release, prostaglandin release during contractions, fetal cortisol changes, placenta ages

62
Q

5 Ps

A
Power
Passageway 
Passenger
Psyche
Position
63
Q

For Power, what are primary and secondary

A

Power =

primary = frequency & duration, contractions mild, moderate, strong

secondary = pushing effort, ferguson reflex)

64
Q

what 2 hormones soften and increase elasticity of pelvic ligaments for passageway/cervix

A

Estrogen & relaxin

65
Q

what is cephalopelvic disproportion

A

Cephalopelvic disproportion (CPD) = head shape does not fit proportionally in pelvis, C/S indicated

66
Q

what is station?

what is engagement?

A

Station = -3-3+ head in relation to ischial spines

Engagement = station 0 is narrowest part fetus must pass through

67
Q

what is Passenger?

A

Passenger = fetus and relationship to passageway

Attitude, lie, presentation (vertex/cephalic-95%, breech, shoulder/transverse), position (ROA & LOA most common), size

Presenting part = O
Woman’s pelvis side = L or R
Relationship of presenting part = A,P,T

68
Q

what is the proper term for sunny side up baby?

A

Occiput posterior = OP, sunny side up, malpresentation, causes back pain

69
Q

what is Psyche?

A

Psyche = response of woman

mental/physical prep, influenced by culture, expectations, support

70
Q

what is Position?

A

Position = maternal posture and position

Freedom of movement, upright position (gravity) lithotomy position

71
Q

what indicates true or false labor?

A

True labor = contractions @ regular intervals, increasing in frequency, intensity and duration

72
Q

what is ROM? when should delivery happen after? what are tests to tell that ROM has occurred?

A

Rupture of membranes

  • Deliver within 24 hr, loss of protective barrier
  • How to tell = ferning (amnio fluid on slide), amnisure (rapid monoclonal assay detects PAMG 1 protein), nitrazine paper (turns blue with amnio fluid)

Assess for cord prolapse

73
Q

what are the 4 stages of labor

A

Stage 1 = longest, latent (beginning to 6cm), active (6-8cm), transition (8-10cm) contractions 1-2 min, bloody show

Stage 2 = pushing (ferguson)/delivery, beings @ 10cm until baby born

Stage 3 = delivery to placenta, begins w baby birth

Stage 4 = immediate PP recovery, placental delivery–2 hr post

74
Q

what is a Sterile vaginal exam

A

Cervical dilation, effacement, check position of cervix (posterior, mid, anterior), station, presentation of baby (cephalic, breech)

75
Q

what is leopolds maneuver

A

Leopold’s maneuver = determine fetal position and size

1- The first maneuver is to determine what part of the fetus is located in the fundus of the uterus
2 - The second maneuver is to determine location of the fetal back
3 - The third maneuver is to determine the presenting part
4 - The fourth maneuver is to determine the location of the cephalic prominence

76
Q

what pain meds are used in labor? Other treatments?

A

Analgesia = IV fentanyl, dilaudid, stradol, nitrous oxide

Anesthesia = regional pudendal (pelvic block), regional epidural, regional spinal, regional general, local anesthesia

Relaxation, breathing, cutaneous stim, counter pressure, thermal stimulation, hydrotherapy, mental stim, support

Complementary = aromatherapy, massage, birthing ball, hydrotherapy, music

77
Q

epidural anesthesia site of admin and infusion type

A

Regional, continuous infusion

4-5 vertebrae

78
Q

what are s/sx of epidural intravascular injection (bad)?

A

Maternal bradycardia, hypertension, tinnitus, metallic taste

79
Q

what are post delivery priorities

A

Assess uterus position, tone, location

Lochia color, amount, clots

Vitals Q 15 min

Ice pack to perineum

Monitor for bladder distension

Assess for return of motor function

Promote skin/skin

80
Q

what is the EFM?

A

Electronic Fetal Monitoring (EFM) = EFM is a technique for fetal assessment based on the fact that the FHR reflects fetal oxygenation

  • detects FHR baseline, variability, accelerations, decelerations
81
Q

what are the goals of FHR monitoring?

A
  • Interpret and Assess Fetal Oxygenation
  • Prevent Significant Fetal Acidemia
  • Support maternal coping and labor progress
  • Maximize uterine blood flow
  • Maximize umbilical blood flow
  • Maximize oxygenation
  • Maintain appropriate uterine activity
82
Q

what does a toco transducer monitor vs an u/s trandsducer?

A

Toco transducer = uterine contractions

Ultrasound transducer = assesses FHR

83
Q

what are internal devices to assess FHR

A

Fetal scalp electrode = assess FHR

Intrauterine pressure catheter = UCs and IUPC (measures strength of contraction in mmHg)

84
Q

what does the baseline FHR be?

A

Baseline FHR 110-160

  • Tachycardia >160
  • Bradycardia <100
85
Q

what causes FHR tachycardia vs bradycardia?

A

Tachycardia >160

  • Maternal Fever/Infection
  • Chorioamnionitis
  • Dehydration
  • Medications: Ephedrine, Terbutaline
  • Drugs: Cocaine

Bradycardia <100

  • Maternal Position (Supine)
  • Hypotension
  • Uterine Rupture
  • Placental Abruption
  • Medications: Anesthetics
86
Q

what are the types of FHR variability?

A

Fluctuations in the baseline FHR – irregularity in amplitude & frequency

  • Absent: Amplitude range is undetectable
  • Minimal: Amplitude range is <=5 bpm
  • Moderate: Amplitude 6-25 bpm
  • Marked: Amplitude >25 bpm
87
Q

what are accelerations in FHR?

A

Accelerations = abrupt increase in FHR above the baseline

Peak (15x15): >=15 bpm for >=15 seconds

<32 weeks (10x10)

88
Q

what are FHR decelerations types?

A

Nadir = Lowest point of a FHR Deceleration

Periodic = Changes in baseline of FHR that occur in relation to Contractions

Episodic = Changes in baseline of FHR that occur independent of Contractions

89
Q

what are prolonged, early, variable, late for FHR decrease from baseline?

A

Prolonged = abrupt or gradual decrease >15bpm lasts 2-10min

Early = gradual decrease from baseline to nadir >30sec, periodic

Variable = abrupt decrease to nadir <30sec, periodic, episodic, U W V shape

Late = gradual decrease to nadir >30sec, always periodic, delayed from UC

90
Q

what are causes of FHR decelerations? VEAL CHOP

A
VEAL CHOP = 
Variable    -- Cord compression
Early          -- Head compression
Accelerations -- Ok
Late           -- Placenta perfusion
91
Q

what are intrauterine resuscitation interventions

A
  • Change maternal position → oxygenation
  • IVF bolus
  • decrease/d/c oxytocin
  • Administer O2 10L LNRB
92
Q

how to treat tachysystole FHR

A

To treat = maternal position change, IVF bolus, ½ or d/c oxytocin, administer tocolytic (terbutaline)

93
Q

what are the 4 causes of dystocia (difficult birth)?

A
  • Hypotonic Uterine Dysfunction = Pressure of uterine contraction is not strong enough to dilate/efface cervix
  • Hypertonic Uterine Dysfunction = Frequent, painful but ineffective in dilation/effacement
  • Failure to Progress (First Stage Arrest) = Failure to continue dilation with confirmed adequate uterine contractions (IUPC)
  • Failure to Descend (Second Stage Arrest) = No descent/rotatio or cessation of descent; 4 hs for nulliparous women with an epidural
94
Q

What are causes for induction of labor (IOL)?

A
  • Deliberate stimulation of uterine contractions before onset of spontaneous labor
  • Bishop Score of >8 for nulliparous women
  • Vertex presentation
  • Informed Consent
  • Elective inductions–no medical indication > = 39 weeks
- Medical Inductions:
Fetal demise
Gestational Hypertension
Pre-Eclampsia
PROM (34 weeks)
Post-Term Pregnancy
AMA (39 weeks)
GDM (39 weeks)
IUGR
95
Q

what med is given for induction of labor? contraindications?

A

Oxytocin (Pitocin) – Most common agent

  1. 5 mU/min and increasing the dose by 1 to 2 mU/min every 30 to 60 minutes until adequate labor progress is achieved (effacement & dilation)
    - Titration Infusion (IV Pump): mu/min
    - contraindications: placenta previa, uterine surgery, non vertex position

GOAL = stimulate labor, UCs every 2-3 min

96
Q

what are risks of IOL?

A

Risks of IOL = Failure, uterine tachysystole fetal intolerance

97
Q

what is cervical ripening?

what is cervical status?

A

Cervical Ripening = process of physical softening, thinning, dilation of cervix
Cervical status = most important predictor of successful IOL

98
Q

For IOL, a bishop score of less than what is unfavorable?

A

Bishop score of <6 = unfavorable

99
Q

what medications used for IOL for cervical ripening? Contraindication?

A

Prostaglandins
Cytotec, Cervidil

Contraindication = previous uterine surgery

100
Q

what is mechanical dilation for IOL?

A
  • Pressure applied via balloon to lower segment of uterus and cervix
  • Balloon Catheter – “Foley Bulb”
  • Filled with saline: 30-50mL
  • Induces cervical ripening and dilation, causes prostaglandin release
101
Q

what is amniotomy? what is contraindication?

A

Artificial Rupture of Membranes (AROM)
- to induce/augment labor

Contraindication = fetal head not yet engaged (impacts fetal station)

102
Q

what is labor augmentation?

A
  • When spontaneous uterine contractions fail to produce progressive cervical change or fetal descent (labor dystocia)
  • GOAL = Strengthen and regulate UC’s, shorten length of labor
  • AROM (Amniotomy) or Pitocin Infusion
  • Contraindication = previous uterine surgery
103
Q

Chorioamnionitis, s/sx?

A

Intra-amniotic infection

- s/sx = 
Maternal Fever (Oral Temp >102F)
Maternal and/or Fetal Tachycardia
Elevated WBC
Uterine Tenderness
Purulent discharge
104
Q

risk factors for Chorioamnionitis

A
Prolonged ROM
 Multiple SVE
 Internal monitors 
 Meconium Stained Amniotic Fluid
 GBS, STIs
105
Q

cord prolapse, risk factors

A

Prolapsed cord or cord prolapse (0.1-0.6%) = when cord comes out before head
- leads to fetal hypoxia

Risk Factors

  • Malpresentation (Breech)
  • ROM before engagement (higher station)
106
Q

nursing actions for cord prolapse

A
Elevation of presenting part--hands/knees positioning, or physically push finger against presenting part
Notify provider
Education to pt/significant other
O2 10L NRB Mask
IV Fluid bolus
Knee-Chest Position
D/C Oxytocin – administer tocolytic
107
Q

what is operative vaginal delivery with Vacuum assisted delivery? risks and 3 pop off rule

A

Vacuum Assisted Delivery (3%) Indications

  • Shorten 2nd stage of labor
  • Maternal exhaustion, fetal compromise
  • Prolonged 2nd stage
  • Maternal cardiac disease
  • Risks – same as forceps = Greater incidence of cephalohematoma
  • 3 pop-off rule = if suction pops off, stop after 3x
108
Q

what are maternal and fetal risks of forceps assisted delivery?

A

Maternal risks

  • ​​Vaginal/Cervical lacerations
  • Hemorrhage
  • Hematoma

Fetal risks

  • Cephalohematoma
  • Skin lacerations
  • Nerve injuries
  • Skull fractures
  • Intracranial hemorrhage
109
Q

difference between unscheduled 3 types of c/s birth

A
  1. Planned – Scheduled
  2. Unscheduled – Unplanned
    - Non-urgent
    - Urgent–asap
    - Emergent–general anesthesia used, problems like cord prolapse, rupture of uterus etc
110
Q

c/s pre op tasks of RN

A

PRE OP

FHR monitoring 
 Pre-Op Checklist
 Initiate IV
 Informed Consent
 Incision prep
shave, chlorhexidine wipes 
Administer medications
Antibiotics, Bicitra, IVF
111
Q

c/s intra op tasks of RN

A

INTRA OP

 Positioning for Spinal
 FHR Doppler 
 Grounding Pad/Suction
 Insert Foley
 Positioning for Surgery (hip tilt)
 Leg strap
 Skin prep – sterile
 Instrument/Needle/Sponge Counts
 Education/Support
112
Q

c/s post op tasks of RN

A

POST OP

PACU – 1 hour minimum
 2 hours immediate post-op (1:1)
 Pain Control
 Initiate skin-skin, breastfeeding
 NB assessments/meds
 VS/motor/strength Q 15 Min
113
Q

What is VBAC

A

VBAC: Vaginal Birth After Cesarean

114
Q

what is TOLAC

A

TOLAC: Trial of Labor After Cesarean (called this until after vaginal birth has been completed, when previous birth was c/s)

115
Q

what are benefits of TOLAC

A

shorter recovery time and overall lower morbidity and mortality, less blood loss, fewer infections, fewer thromboembolic problems

116
Q

what kind of labor is preferred?

A

Spontaneous preferred

IOL – no cytotec, limited Pitocin

117
Q

contraindications for TOLAC

A
Classical, T, or unknown uterine incision
Previous uterine rupture
Pelvic abnormalities (CPD)
Placenta Previa
Inability to perform Emergency C/S
118
Q

shoulder dystocia, what is 1st sign?

A
Obstetric Emergency (0.2-3%)
Difficulty with delivery of the shoulders
1st sign – retraction of fetal head (turtle sign)
119
Q

what are neonate complications from shoulder dystocia?

A

Brachial plexus injuries
Clavicle fracture
Neurological injury (r/t asphyxia)
Death

120
Q

what are maternal complications of shoulder dystocia?

A

Maternal complications = Perineal laceration (4th degree), infection, bladder injury, PPH

121
Q

what are risk factors for shoulder dystocia?

A
Fetal Macrosomia
Maternal Diabetes
Hx Shoulder Dystocia
Prolonged 2nd stage
Excessive weight gain
122
Q

what is management for shoulder dystocia?

A
McRoberts Maneuver
Suprapubic Pressure
Woods corkscrew maneuver (provider)
Document series of interventions & time intervals
Prepare for neonatal resuscitation
123
Q

what is uterine rupture? s/sx? what kind of delivery needed?*

A

Partial/complete tear in uterine muscle
Rare Obstetric Emergency (0.07%)
Most common w/ VBAC

  • s/sx:
  • Tearing sensation, burning/stabbing
  • Internal hemorrhage → Maternal hypovolemia (shock: hypotension, tachypnea, tachycardia, and pallor)
  • Fetal compromise = Uteroplacental insufficiency

emergency C/S needed