O&G Flashcards
septate, bicorniate and didelphys uterus
septate: smooth fundus. can appear to have 2 cervices
bicornuate: indented fundus. 1 cervix
didelphys: complete. indented fubdus, 2 cervices and 2 vaginas.
placenta praevia spectrum and grade
still crosses os at 28wks. can’t dx b4 20wks. repeat at 32 wks
low lying. less than 2cm from OS Partial praevia. partially covers os Complete praevia. total coverage of os
Increased risk APH and acretta. Follow up at 30wks to see if regresses
grade 1. edge within 5cm
grade 2. marginal. tissue reaches but doesn’t cover
grade 3. partially covers OS
grade 4. complete
placenta acretta
acretta. abnormal adherence to myometrium. prominent venous lakes. increased risk with previous c section or placenta praevia. worry if anterior placenta and previous section
incretta. infiltrates withing myometrium
percretta. within myometrium. no or very little plane between myometrium vessels and bladder on doppler
placental abruption
premature separation b4 20wks
antepartum haemorrhage
retro placental clot
circumvillate placenta
placental shelf at edge, lifts up
can infarct or haemorrhage
succenturiate lobe
2 lobes. put doppler between and check no vessels, esp along os. do PV
associated with praevia and retained products
abnormal cord insertion into placenta
marginal. within 2cm from edge.
velamentous. inserts direct to wall/membranes, not into placenta. vessels run between insertion and placenta
associated IUGR, vasa praevia ( vessels across os) and haemorrhage
amniotic band and shelf
shelf. thin band that runs entire length of uterine wall. has base (thick) and free edge. can have flow.
band. free floating blind end in anion with intact end to chorion. can entrap limbs and cause structures.
uterine cord
2aa, 1vv.
normal RI <0.55
normal PI <1.4
MCA artery doppler
mum sitting up or on rt side
normally has high resistance flow with no antegrade diastolic (below line)
reduced flow suggests head sparing and cerebral redistribution. IUGR progression.
IUGR
symmetric or asymmetric
symmetric. all parameters are down. EFW <10%
associated with trisomy 13 or 18, TORCH, etoh, smoking, heroin
asymmetric. placental insufficiency or pre eclampsia.
HC normal, AC reduced. increased HC/AC ratio. check MCA for sparing. look at bone density and shape.
must comment on foetal movements, liquor volume and umbilical aa SD and PI.
polyhydramnios
>25cm after 20wk idiopathic dm cns or neural tube defect Hydrops TTTS cardiovascular abnormal. svt CPAM hernia GI obstruction Microcephaly (rubella or CMV in utero, trisomy, Syndromic ie walker Warburg mm dystrophy)
most common virus causing hydrops
pavovirus
CCAM types
type 1. most common. large cysts 2-10cm size
type 2. cysts <2cm size. associated with renal agenesis, Pulm sequestration and cardiac anomalies
type 3. unlined cysts. usually only affects 1 lobe. can’t ddx from type 1
type 0. rare. global arrest of lung development. postnatal fatal.
commonest cause of hydronephrosis in utero
PUJ obstruction.
high risk renal injury with minor trauma.
bilateral 30%. L>R.
congenital is usually idiopathic. other causes: extrinsic compression (vessel, fibrosis or mass), pelvic trauma, infection with scar
TTTS in mono/di chorion, Moni/di amnionic
exclusively monochorionic pregnancies
MCDC, MCDA
Nuchal translucency thickness
> 3mm, but must correlate with maternal bloods also
- bHCG, AFP, oestriol, pregnancy associated plasma protein (PAP).
look at heart.
yolk sac present
5-6 weeks
hydrops fetalis
immune/rhesus incompatible
non inmune/rhesus compatible: cardiac> chromosomal 18, 13, 21, turners > infection (pavovirus B19, TORCH) > chest (CPAM), Urinary tract obst, TTTS, sacroccygeal tumors, anemia, skeletal dysplasia, vv galen
TTTS stages
- visible bladder. normal US
- empty bladder. normal UA
- empty bladder. UA doppler abnormal
- hydrops in recipient
- demise of either twin
eccentric gestational sac
Interstitial ectopic
high in fundus, off centre. corneal.
low near cervix - c-scar or classic c scar
*mention myometrium thickness as <3mm associated with risk perforation. Uterine anomaly. bicorniate or septate
ectopic vs miscarriage
ectopic has decidual reaction and rim of vascular it.
ectopic won’t move on probe palpation.
mx ectopic
medical: not ruptured, asymptomatic, <3cm
methotrexate wither systemic or direct injection (kcl first to kill pregnancy)
surgical: ruptured, symptoms
failed early pregnancy
MSD >25mm with no embryo
CRL >7mm with no heartbeat
*CRL out ranks MSD. repeat US in 7-14 days if borderline
suspected failed: CRL <7mm with no heartbeat MSD: 16-24mm with no embryo enlarged (>7mm), calcified or irregular yolk sac irregular gestational sac
hydatiform mole
gestational trophoblastic disease
teens, 40-50yo and Asians
complete: diploid 46XX
partial: abnormal fetus. triploid 69XXXY
bunch of grapes on US with enlarged uterus and elevated bHCG
complete can progress to invasive or choriocarcinoma
MCMA MCDA DCDA DCMA
DC. lambda sign. twin peaks. thick
DA. T sign. thin
oligohydramnios
DRIPC
Demise or drugs
Renal agenesis, posterior urethral valves, MCDA. check PUJ and bladder
IUGR
PROM or post dates
Chromosomal abnormalities 18, 21, 13, turners *Check pulm hypoplasia, limbs for club foot or deformity, facial deformity, UA for IUGR
intrauterine hydrocephalus
> 10mm diameter later ventricles at atrium
non obstructive. hemorrhage, infection
obstructive. spina bifide, aqueduct stenosis, chiari, dandy walker, encephalocele
normal intrauterine renal pelvic diammeter
7mm
cisterna magna c/w dandy walker
cisterna doesn’t have a connection to 4th ventricle
dandy walker has small posterior fossa
ovarian cysts - simple
<5cm premenopausal or <1cm postmenopausal - leave alone.
5 -7cm pre (1-7cm postmenopausal) - f/u yearly US
>7cm needs gynae rv +/- MRI for possible infiltration
ovarian cyst - haemorrhagic
<5cm no fu
>5cm - fu in 6-12 weeks to ensure resolution
in post menopausal need follow up at any size.
* hypoechoic with lace like internal echos. +/- concave solid part that has no flow.
malignant breast ca
95% adenocarcinoma
5% phylloides, lymphoma, sarcoma, scc, mets (melanoma, lymphoma).
2nd to skin ca in frequency. 2nd to lung ca in cause of death
risks. hormones (early menarche, late menopause), nulliparity, obesity, oestrogen. genetic brca 1 & 2, le fraumeni, Cowden, HTT, peutz jagher.
non invasive breast cancer vs invasive
invasive has breached the basement membrane
they all arise in terminal duct/lobule unit
non invasive breast ca
DCIS.
branching linear micro calcs that spread along the duct.
comedocarcinoma, papillary, Micropapillary, solid, cribriform.
LCIS.
usually incidental finding on bx as no calcification.
more likely to be bilateral or multilocul and can recur in either breast as ductal or lobular
more common in younger women than DCIS
- both can become invasive around 1% per year.
invasive breast ca
breached basement membrane
upper outer most common
schirrhous (dense collagen), stelate or well cx.
subtypes.
- NOS. worst prog, high grade
- Medullary. poor prog as rapid growth and local aggressive. looks like fibro adenoma. younger (50s). BRCA 1
- Lobular. poorly seen as little desmoplastic reaction and no ca+. Indian ink cells in loose clusters. multicentric or bilateral can occur
- Mucinous. older women with slow growth and good prog. cells float in mucin. soft and gelatinous.
- Tubular. spiculated mass in younger. slow growth with best prognosis.
first trimester bleeding
Normal implantation bleed, miscarriage (MSD >25mm, CRL >7mm with no FHB), ectopic, GTD, Subchorionic haemorrhage (>50% high risk)
Empty gestational sac
Blighted ovum (MSD >25mm with no embryo) Ectopic with pseudogestational sac (central cf eccentric location in uterus, no yolk sac, irregular/pointed shape)
Echogenic endometrial cavity
Early IUP, Ectopic, retained products, emdometritis
Complex intrauterine mass
Missed miscarriage with RPOC, molar, degenerative fibroid, endometrial ca (>5mm postmenopausal with bleed, >11mm without bleed)