Nutrition Flashcards

1
Q

Vitamin __ can not be created by animals, is fat soluble, and is available in grains, vegetables, diary, and meat products.

A

A

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2
Q

Vitamin __ deficiency is seen in chronic intestinal disorders or lipid malabsorption syndromes.

Manifestations include….

Diarrhea
Bronchial Obstructions
Eye Lesions

A

A

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3
Q

Vitamin ___ or _______ is a co-factor for sugar metabolism, required for acetylcholine synthesis, and is increased during times of increased metabolism (fever, pregnancy, hyperthyroidism)

A

B1 (Thiamine)

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4
Q

________ deficiency is associated with severely malnourished states that present with fatigue, depression, decreased concentration.

You may also see…..

Decreased DTRs
Hoarseness
Ataxia
AMS

A

Thiamine (B1) Deficiency

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5
Q

Vitamin __ or ______ is commonly found in milk, eggs, organ meat, legumes, and mushrooms.

A

Vitamin B2 (Riboflavin)

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6
Q

______ deficiency is commonly caused by malasorption and can manifest with cheilosis, glossitis, keratitis, photophobia, and seborrheic dermatitis.

A

Riboflavin (B2) deficiency

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7
Q

Vitamin __ or _____ helps to form part of co-factors NAD and NADP and is commonly found in meat, fish, poultry and leafy greens.

A

Vitamin B3 (Niacin)

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8
Q

_____ deficiency is primarily seen in populations where corn is the major food source.

This will manifest clinically as anorexia, weakness, numbness, and dizziness.

A

Niacin (B3) Deficiency

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9
Q

What it the triad of prolonged Niacin (B3) deficiency?

A
  1. Dementia
  2. Dermatitis
  3. Diarrhea
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10
Q

Vitamin __ or ______ is a co-enzyme for amino acid and glycogen metabolism and neurotransmitter synthesis and is commonly found in fish, poultry, bananas, and rice.

A

Vitamin B6 (Pyridoxine)

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11
Q

_______ deficiency manifests with irritability, seizures, vomiting, and skin lesions.

A

Pyridoxine (Vitamin B6)

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12
Q

______ is a co-factor for enzymes in carboxylation reactions.

Deficiency is unlike as this is widely distributed.

A

Biotin

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13
Q

_______ is used as a co-enzyme in amino acid and nucleotide metabolism.

A

Folate

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14
Q

______ deficiency can result to to malabsorption or increased requirement (ie: sickle cell disease).

Clinical manifestations include….

Megaloblastic anemia
Neural Tube defects
Glossitis

A

Folate Deficiency

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15
Q

Vitamin __ or _____ is a co-factor for essential reaction in lipid and sugar metabolism.

It is absorbed in the ileum after binding to intrinsic factor.

A

Vitamin B12 (Cobalamin)

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16
Q

_______ deficiency is commonly seen in patients with Crohn’s disease or vegan/vegetarian diets

Manifestations include…..

Irritability
Development delay / regression
Peripheral neuritis

A

Cobalamin (B12)

17
Q

Vitamin __ or _____ ____ is important in the synthesis of collagen, involved in neurotransmitter and cholesterol metabolism,, and the biosynthesis of carnitine.

It is commonly found in citrus fruits, berries, peppers, melons, and tomatoes.

A

Vitamin C (Abscorbic Acid)

18
Q

______ ____ deficiency is associated closely with scurvy and can be seen in the defective formation of collagen in skin, bone, and vessels.

Clinical manifestations include…..

Decreased Appetite
Tenderness in legs
‘Rosary’ formation at the costochondral junctions
Anemia

A

Abscorbic Acid (Vitamin C)

19
Q

How could Vitamin C deficiency be diagnosed?

What would be shown on the above?

A

XR showing ground glass appearance on the distal end of long bones.

20
Q

Vitamin __ deficiency is a disease of growing bones in children that results in the softening of bones.

This can manifest with….

Rachitic Rosary
Craiotabes

A

Vitamin D Deficiency (Rickets)

21
Q

How is vitamin D deficiency treated?

A

Sunlight

Supplemental Nutrition

22
Q

_______ deficiency also can result in Rickets and is more common in Africa.

A

Calcium Deficiency

23
Q

________ is the most common inborn error of metabolism and involves the absence of phenylalanine hydroxylase or its co-factor tetrahydrobiopterin.

A

Phenylketonuria (PKU)

24
Q

T/F: Phenylketonuria is autosomal dominant

A

False

25
Q

How does PKU present at birth?

Later on in life?

A

Normal at birth

Gradually develop profound intellectual disability and many will have an IQ less than 35

26
Q

How does PKU present in infants?

A

Lighter skin, eyes, and hair (decreases melanin)
Eczema
Musty smell
Spasticity

27
Q

How is PKU evaluated?

What is the goal of treatment?

A

Mass screening at birth within the first 48 hours

Tx:

Monitor weekly phenylalanine levels, use formula low in phenylalanine and continue at least through childhood/early adulthood as brain is still developing.

28
Q

How is short term enteral nutrition delivered?

Long term?

A

Short: NG Tube

Long: G-Tube or J-Tube

29
Q

What are complications of enteral nutrition?

A

Diarrhea

Aspiration

30
Q

_______ nutrition is often given through a peripheral vein and primarily composed of amino acids and dextrose.

A

Parenteral Nutrition

31
Q

What are complications associated with parenteral nutrition?

A

Mechanical (regarding catheter placement)
Metabolic (Fluid overload, hyperglycemia)
Infection