Nursing care of head injury, surgery, ICO, seizures, spinal cord injury Flashcards

1
Q

What is the most common cause of traumatic brain injury

A

Falls

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2
Q

What happens to the symtoms of a hemmorage

A

They are delayed until hematoma is large enough to increase ICP

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3
Q

What are the different types of TBI?

A

open, closed, diffuse, focal.

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4
Q

What is the usual cause of an epidural hemmorage?

A

rupture of middle meningeal artery it’s a MEDICAL EMERGENCY and can cause herniation.

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5
Q

Signs of brain herniation

A

Loss of consciousness, pupil dilation, paralysis of extremity.

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6
Q

What is an epidural hemmorage?

A

above the dura under the skull, caused by rupture of middle meningeal artery.

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7
Q

What is a subdural hemmorage?

A

below dura, between dura and brain

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8
Q

What is the nature of a subdural hemmorage

A

usual venous in nature, can be acute, sub acute, and chronic

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9
Q

who is more prone to a subdural hemmorage

A

elderly on anti-coagulants.

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10
Q

Intracerebral hemmorage

A

within the brain tissue, CVA,HTN

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11
Q

traits of a contusion

A

localized, cells die, effects peak 18-36 after injury, coup-contracoup, can cause ICP increase from bleeding.

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12
Q

S&S of concussion

A

irritability, confusion, disorientation, headache

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13
Q

S&S of contusion

A

blurred vision, disorientation, unsteady gait, vomiting, slurred speech, coma.

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14
Q

Diagnosis of coup contra coup

A

CT, MRI, skull xray, angiography

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15
Q

medical care of coup contracoup - contusion

A

control ICP, intracranial bolt, mechanical ventilation, prevent hypoxemia and increase in lactic acidosis, vaso dilation, leading to an increase in ICP. reduce metabolic demands by putting into a coma, minimize secondary injury.

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16
Q

Nursing care of head injury - goal

A

address acute injuries (respirations, cerebral circulation, safety. Prevent secondary complications (infections, skin integrity, safety positing. Prevent and treat consequences with PT OT education.

17
Q

First level of nursing care following a head injury

A

assess all systems for direct impact and primary compromise, patent airway, optimal breathing pattern, optimal cerebral perfusion, appropriate fluid balance.mentation, H&H, lytes

18
Q

second level of nursing care following a head injury

A

assess for secondary compromise S&S of infection, complications, consequences.

19
Q

what should be told to the patient for eduation

A

headache, dizzyness, lethargy, irritability, emotional, fatigue, poor concentration, decreased attention span.

20
Q

following a head injury, when should a patient notify an MD?

A

Difficulty in waking and speaking, confusion, severe headache, vomiting, unilateral weakness.

21
Q

What does the frontal lobe do

A

behavior, intelligence, memory, movement

22
Q

what does the temporal lobe do

A

behavior, hearing, memory, speech, vision.

23
Q

What does the parietal lobe do

A

intelligence, lanquage, sensation, reading.

24
Q

What does the brain stem control?

A

BP, HR, breathing, consciousness, swallowing

25
Why would they preform intracranial surgery
reduce ICP, remove tumor, ecacuate a blood clot, removal of skull to allow room for swelling, repair of skull
26
What is supratentoria?
brain surgery above the tenrotum (eye) most of brain above cerebellum.
27
What is infratentorial
below tentorum (eyeline) cerebellum).
28
Nursing care pre brain surgery
document baseling neurological status, routine pre-op care and education, continuation of care. admin meds as prescribed.
29
Nursing care post brain surgery
asses hourly, neuro with glasgow or four score, respiratory, ABG, labs, ICP monitor readings). Proper positioning depends on surgical approach. monitor OUT and IN, dressing changes.
30
Glasgow
Eyes,verbal, motor, not good for patients on ventilators
31
FOUR
Full Outline of Unresponsiveness - good for patients on venilator, lower score worse situation same with glasgow.
32
Potential post op brain surgery complications
bleeding and hypovolemic shock, fluid and electrolyte disturbances, infection, increased ICP, seizures, diabetes insipidus, SIADH
33
What does the monroe Kelly doctrine state?
increase in one component of brain, blood, and csf will lead to compensatory changes in others.
34
early human responses to increase in ICP
Change in LOC, pupillary changes, impaired occular movement, weakness on one side, constant headache,
35
Late responses to increase ICP
LOC, respiratory pattern alterations, loss of brainstem reflexes, cushings triad, hemiplegia, decorticate, decerebrate posturing.
36
What is cushings triad?
Hypertension with widening pulse pressure, bradycardia, bradypnea
37
Human respions to I-C-P
Increase in pulse pressure, Changes in LOC, Pupils, Puking, Pain, Posturing
38
Decorticate
turn inwards damage to corticospinal tracts
39
Decerebrate
turning outwar