Nursing care of head injury, surgery, ICO, seizures, spinal cord injury Flashcards

1
Q

What is the most common cause of traumatic brain injury

A

Falls

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2
Q

What happens to the symtoms of a hemmorage

A

They are delayed until hematoma is large enough to increase ICP

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3
Q

What are the different types of TBI?

A

open, closed, diffuse, focal.

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4
Q

What is the usual cause of an epidural hemmorage?

A

rupture of middle meningeal artery it’s a MEDICAL EMERGENCY and can cause herniation.

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5
Q

Signs of brain herniation

A

Loss of consciousness, pupil dilation, paralysis of extremity.

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6
Q

What is an epidural hemmorage?

A

above the dura under the skull, caused by rupture of middle meningeal artery.

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7
Q

What is a subdural hemmorage?

A

below dura, between dura and brain

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8
Q

What is the nature of a subdural hemmorage

A

usual venous in nature, can be acute, sub acute, and chronic

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9
Q

who is more prone to a subdural hemmorage

A

elderly on anti-coagulants.

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10
Q

Intracerebral hemmorage

A

within the brain tissue, CVA,HTN

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11
Q

traits of a contusion

A

localized, cells die, effects peak 18-36 after injury, coup-contracoup, can cause ICP increase from bleeding.

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12
Q

S&S of concussion

A

irritability, confusion, disorientation, headache

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13
Q

S&S of contusion

A

blurred vision, disorientation, unsteady gait, vomiting, slurred speech, coma.

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14
Q

Diagnosis of coup contra coup

A

CT, MRI, skull xray, angiography

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15
Q

medical care of coup contracoup - contusion

A

control ICP, intracranial bolt, mechanical ventilation, prevent hypoxemia and increase in lactic acidosis, vaso dilation, leading to an increase in ICP. reduce metabolic demands by putting into a coma, minimize secondary injury.

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16
Q

Nursing care of head injury - goal

A

address acute injuries (respirations, cerebral circulation, safety. Prevent secondary complications (infections, skin integrity, safety positing. Prevent and treat consequences with PT OT education.

17
Q

First level of nursing care following a head injury

A

assess all systems for direct impact and primary compromise, patent airway, optimal breathing pattern, optimal cerebral perfusion, appropriate fluid balance.mentation, H&H, lytes

18
Q

second level of nursing care following a head injury

A

assess for secondary compromise S&S of infection, complications, consequences.

19
Q

what should be told to the patient for eduation

A

headache, dizzyness, lethargy, irritability, emotional, fatigue, poor concentration, decreased attention span.

20
Q

following a head injury, when should a patient notify an MD?

A

Difficulty in waking and speaking, confusion, severe headache, vomiting, unilateral weakness.

21
Q

What does the frontal lobe do

A

behavior, intelligence, memory, movement

22
Q

what does the temporal lobe do

A

behavior, hearing, memory, speech, vision.

23
Q

What does the parietal lobe do

A

intelligence, lanquage, sensation, reading.

24
Q

What does the brain stem control?

A

BP, HR, breathing, consciousness, swallowing

25
Q

Why would they preform intracranial surgery

A

reduce ICP, remove tumor, ecacuate a blood clot, removal of skull to allow room for swelling, repair of skull

26
Q

What is supratentoria?

A

brain surgery above the tenrotum (eye) most of brain above cerebellum.

27
Q

What is infratentorial

A

below tentorum (eyeline) cerebellum).

28
Q

Nursing care pre brain surgery

A

document baseling neurological status, routine pre-op care and education, continuation of care. admin meds as prescribed.

29
Q

Nursing care post brain surgery

A

asses hourly, neuro with glasgow or four score, respiratory, ABG, labs, ICP monitor readings). Proper positioning depends on surgical approach. monitor OUT and IN, dressing changes.

30
Q

Glasgow

A

Eyes,verbal, motor, not good for patients on ventilators

31
Q

FOUR

A

Full Outline of Unresponsiveness - good for patients on venilator, lower score worse situation same with glasgow.

32
Q

Potential post op brain surgery complications

A

bleeding and hypovolemic shock, fluid and electrolyte disturbances, infection, increased ICP, seizures, diabetes insipidus, SIADH

33
Q

What does the monroe Kelly doctrine state?

A

increase in one component of brain, blood, and csf will lead to compensatory changes in others.

34
Q

early human responses to increase in ICP

A

Change in LOC, pupillary changes, impaired occular movement, weakness on one side, constant headache,

35
Q

Late responses to increase ICP

A

LOC, respiratory pattern alterations, loss of brainstem reflexes, cushings triad, hemiplegia, decorticate, decerebrate posturing.

36
Q

What is cushings triad?

A

Hypertension with widening pulse pressure, bradycardia, bradypnea

37
Q

Human respions to I-C-P

A

Increase in pulse pressure, Changes in LOC, Pupils, Puking, Pain, Posturing

38
Q

Decorticate

A

turn inwards damage to corticospinal tracts

39
Q

Decerebrate

A

turning outwar