NURS 444 week 4 heart Flashcards
Cardiac output
Heart rate (HR) x stroke volume (SV)
normal is 4-8 L/min
- preload
- afterload
- contractility
Arterial blood pressure:
BP- cardiac output (CO) x systemic vascular resistance (SVR)
**normal: SBP < 120 mm Hg, DBP < 80
Preload
volume in the ventricles at the end of diastole (end diastole pressure). Amount of blood in the ventricles right before contraction
increased in:
Hypervolemia
Regurgitation of cardiac valves
Afterload
Resistance left. Ventricle must overcome to circulate blood
Increased in:
htn
vasoconstriction
increased afterload
increased cardiac workload
Cardiomyopathy (CMP)
Primary- idiopathic and only heart involved
Secondary-
Major types: dilated, hypertrophic, restrictive
Leads to cardiomegaly and HF
leading cause of heart transplantation
Types of secondary CMP
> dilated- due to too much fluid backing up into the ventricle. think of balloon stretching; alcoholic, CAD, htn, valve disease, pregnancy
MOST COMMON mixed heart failure
> hypertrophic (enlarged vent.)- aortic stenosis, htn
more common in men. dyspnea most common symptom
> restricitve- post-radiation therapy, ventricular thrombus, scarring (not as common)
least common
classic symptoms are fatigue, exercise intolerance, and dyspnea
Management for Dilated
- control HF
- doesn’t respond well to therapy- hospitalized numerous times
- vent. assist device***
- heart resynchronization therapy
- heart tranplantation
Hypertrophic management
- beta or calcium channel blockers
- ICD
- surgical treatment
(ventriculomyotomy and myectomy) - percutaneous transluminal septal myocardial ablation
Restrictive management
- no specific tx exists
- conventional therapy for HF and dysrhythmias
Ejection Fraction
normal: 55- 65%
< 45% = HF
Heart Failure compensatory mechanism
sympathetic nervous system
- Catecholamines: epinephrine and norepinephrine
- detrimental over time. increases O2 demand, increases workload of hert
neurohormonal response
- decreases renal perfusion causes kidney to increase renin
RAAS. results in water retention
may be on ACE inhibitor or ARB
Frank Sterling Law
dilation of heart chambers, muscle fibers stretch in response to volume
hypertrophy follows dilation
Patients who have had an MI
will go on a beta blocker to prevent remodeling of the heart
also on a low dose ACE inhibitor
Monitoring HF patients
weigh: call if more than 1lb/ day
BMP stretching: check labs
Left Sided Heart Failure
Pulmonary congestion:
- cough
- crackles
- wheezes
- blood-tinged sputum
- tachypnea
restlessness
confusion
orthopnea
tachycardia
exertional dyspnea
fatigue
cyanosis
** correct fluid before med. therapy
also systolic HF
Right sided HF
“Cor Pulmonale”
- faitgue
- increased peripheral venous pressure
- Ascites
- enlarged liver/ spleen
- may be secondary to chronic pulmonary problems
- DJV
- anorexia and GI distress
- swelling in hands and fingers
- dependent edema