NURS 444 week 3 Flashcards
Respiratory Failure and classification
when all compensatory mechanisms fail
hypoxemic- resp. failure. Insufficient O2 transferred to blood
hypercapnic- resp. failure. Inadequate CO2 removed from the lungs
Hypoxemic Respiratory Failure
Oxygenation issue
Causes
- ventilation-perfusion (v/q) mismatch
- COPD
- pneumonia
- asthma
- atelectasis
- pain
- pulmonary embolus
Hypercapnic Respiratory Failure
ventilation issues
causes:
- Airways and alveoli:
asthma, emphysema, cystic fibrosis
- CNS;
drug overdose, brainstem infarction, spinal cord injury - chest wall:
flail chest (gunshot wound), kyphoscoliosis, morbid obesity, fracture, mechanical restriction, muscle spasms - neuromuscular conditions
muscular dystrophy, guillain-barre syndrome, MS
V/Q scans
help diagnose a PE
if low –> increased circulation but low ventilation
if high –> decreased circulation but good ventilation
Consequences of hypoxemia and hypoxia
*** cells shift from aerobic to anaerobic
- lactic acid production
- metabolic acidosis and cell death
- decreased cardiac output
- impaired renal function
Sudden or Gradual onset of Respiratory Failure
Sudden: life-threatening
> greater risk if coexisting with cardiac problems or anemia
> ex. asthma exacerbation
Gradual: compensation occurs
< ex. COPD with URI. may recover faster than sudden
Resp. Failure clinical manifestations
Early SIgns: irritable/ restless
- tachycardia
- tachypnea
- mild htn
Severe morning headache
Late sign: cyanosis
- rapid, shallow breathing pattern
- tripod position
- dyspnea
- pursed lip breathing
- retractions
- change in I:E ratio
Resp. Failure Diagnostic studies
- H&P assessment
- ABG analysis
- CXR
- CBC, sputum/ blood cultures, electrolytes
- ECG
- urinalysis
- V/Q lung scan
- pulmonary artery catheter (rare and only in extreme cases)
Resp. Failure management
Oxygen Therapy
- maintain PaO2 55 to 60 mm Hg or more and SaO2 at 90% or more at the lowest O2 concentration possible
Resp. Failure meds.
- Bronchodilators
relieve of bronchospasms - Corticosteroids
reduction in airway inflammation - Diuretics, nitrates if HF present
reduction of pulm. congestion
nitrates to reduce workload of heart - IV antibiotics
Tx of pulm. infections - Benzos, narcotics
reduction of severe anxiety, pain, and agitation
Mechanical ventilation
process by which FiO2 (fraction of inspired oxygen)- 21% RA or greater, and a set amount of air volume is moved in and out
Positive pressure ventilation (PPV)- pushing pressure in
Non-invasive PPV: Bi-PAP, CPAP
BIPAP
positive pressure on inspiration
indicated for; COPD with HF or RF and sleep apnea
contraindicated for; shock, altered mental status, ^ airway secretions
indications for intubation
-apnea
-inability to breathe or protect airway
-resp. distress or muscle fatigue
-resp. failure
ET intubation prep
self-inflating bag valve mask connected to oxygen
suctioning
IV access
premedication depends on patient’s LOC and nature of procedure
Immediate actions during intubation
- Inflate cuff
- Manually ventilate patient with BVM
- Confirm placement of ET tube:
End-tidal CO2 detector
Auscultate lungs bilaterally
Ausculate epigastrium
Observe chest wall movement
Monitor Sp02
We need an x-ray to confirm placement
After ET placement
- connect to mechanical vent.
- secure ET tube (mark at lip and do before x-ray)
- suction ET tube and pharynx
- insert bite block if needed
- obtain cxr
*** 2- 6 cm above carina and observe chest wall for symmetric movement - continuously monitor pulse ox
- obtain ABGs in 30 min - 1 hr
Ventilator machine settings
AC
SIMV
FiO2
PEEP- positive end-expiratory pressure- allows pressure on exhalation. Keep alveoli open
Rate
VT