Nsg: Diabetes Flashcards
Acute Complications of DM
- Hyperglycemia: HHNS, DKA.
- Hypoglycemia
Long term Complications of DM
Microvascular:
- Neuropathy,
- Retinopathy,
- Nephropathy
Macrovascular:
-Peripheral Arterial disease (from Atherosclerosis)
Erectile Dysfunction (ED): Tigh glycemic control delays onset. Tx: with viagra and referral to an ED specialist
Tx of Hyperglycemia
Inuslin
Meformin
Exercise
Diet of low CHO and sugar
Tx of Hypoglycemia
Mild: 15g CHO, 175ml OJ or soda
Wait 15min, check BG, repeat if t awake to swallow.
What is Peripheral Vascular Disease?
Narrowing and degeneration of the arteries: Neck, abdomen, and extremeties.
Tx for Peripheral Arterial Disease
Sx:
- Angiogram
- Angioplasty
- Bypassgraft
- Amputation (last resort)
Increase exercise to increase collatoral circulation, walk till point of discomfort and rest to let circulation continue then restart. Antiplatelets, pain managment when sleeping, HOB elevated when sleeping, reduce dietary cholesterol, fats, calories prn, and use meds to reduce cholesterol (all to prevent blockage of arteries). quit smoking. revascularization by sx.
S/s of Peripheal Arterial Disease
-Loss of hair on legs
-Tight and shiny skin
-Pallor of extremeties
-Slow cap refill
-Decrease arterial supply to distal tissues when pt exercises, leading to pain (buildup of Lactic Acid)
“Intermitten Claudication”
Tx: Pain is relieved by rest.
-“Resting claudication” Pain in periphery while at rest.
Tx: Pain is relieved if pt dangles legs.
Blood flow/oxygen to the periphery becomes so decreased that peripheral cells die b/c of lack of oxygen.
What is Ketoacidosis (DKA)?
For Type 1 Diabetics. Sometimes type 2.
Metabolic complication when fats are metabolised when insulin is absent. pH is very acidic b/c of the amount of BG and acid ketones (byproduct of fat breakdown)
S/s of Ketoacidosis (DKA)
Hyperglycemia BG>14mmol/L and pH <7.34
- polyuria and polydipsia
- dehydration: dec.
- skin turgor,
- tachycardia,
- orthostatic hypotension,
- lethargy,
- weakness,
- dry and loose skin,
- N/V b/c of acidosis (result=dehydration),
- KUSSMAUL RESPIRATIONS: deep and rapids breaths to rid of CO2.
- Osmotic Diuresis: to rid of excess ketone and glucose
- Low BP: D/t osmotic diuresis.
Tx of Ketoacidosis (DKA)
- 0.45% or 0.9% NaCl to restore U/O to 30-60ml/hr and incr. BP from Osmotic Diuresis
- When BG is < 14mmol/L, 5% Dextrose is added to the fluid to prevent HYPOGLYCEMIA.
- Fluid/Electrolyte Tx to replace lost electrolytes b/c of Osmotic Diuresis.
- Potassium replacement b/c of hypokalemia d/t lost electrolytes.
- Monitor K balance once Insulin Tx is started so it doesn’t decrease.
- IV insulin Tx to correct hyperglycemia and hyperketonemia. Start insulin tx once fluid replacement is restored b/c insulin also draws water into the cells along with glucose.
- Administer IV sodium bicarbonate is sever acidosis pH <7.0
What is Hyperosmolar Hyperglycemic Nonketotic Syndrome? ( HHNS)
With Type 2 Diabetics.
Life threatening syndrome.
Can produce enough insulin to prevent DKA, but not prevent hyperglycemia.
S/s of HHNS
Hyperglycemic s/s: inc. Urination, weakness, fatigue, headache, N/V, abd pain.
BG lvls can quickly elevate before it is noticed.
Tx of HHNS
Monitor:
BG lvls,
U/O (from osmotic diuresis),
IV fluid balance (to replace what was voided), Electrolytes (to replace what was lost),
Insulin therapy for the high BG lvls,
LOC,
Potassium balance from (too much insulin, and too little insulin causing and incr in urinaion and decr. potassium)
Assess renal status
Assess cardiopulmonary status r/t hydration and electrolyte levels (movement of K can alter cardiac function).
What can insulin do to potassium?
Insulin can make the cells take up potassium resulting in low potassium levels in the blood causing potential cardiac issues.
S/s hyperglycemia
BG >7mmol/L
Polyuria, Polydipsia, polyphagia
Can lead to: DKA, HHNS