newborn Flashcards
apgar score
1 and 5min. 5 components: HR(absent, 100), respiratory effort, M tone, reflex irritability, color. 5min score of 7-10= normal/ doesn’t predict outcome or dev of infant
assess intrapartum hypoxia/ischemia
best is presence of metabolic acidosis in umbilical artery blood at time of birth
Vit K bleeding
bc neonates have no gut flora to make K. so present at birth or wks later with skin bruising, mucosal bleeding, bleeding at umbilicus and circumcision site or fatal intracranial hemorrhage
ophthalmia neonatorum
cause by neisseria gonorhoeae and chlamydia. Tx- erythromycin appliced to conjunctival sacs. Can use silver nitrate
dubowitz/ballard exam
done at 12-24hr of life. used when gestational age or due date unclear. Assess infant neuroM and physical maturity. NM maturity is based on infant NM tone and reflexes. Physical M - anterior posterior progression of plantar creases and progression from transparency to cracking, lanugo, extent of dev of breast tissue, eye/ear dev, maturation of genitalia.
hypoglycemia
risk group: diabetic mother, SGA, LGA, birth asphyxia.
HIV pos M
bathed at birth. Zidovudine initiated by 12hr.
withdrawal
abstinence scoring system every 4hr ck vital sign.
LGA/posterm
> 90th percentile. skin cracked (wrist ankle), assess for trauma using Moro reflex and grasp symmetry, ID clavicular fractures, brachial plexus injury, facial n palsy.
dextrose/sterile water
avoid giving bc cause hyponatremia and other electrolyte disturbances
not breastfed
incr risk obesity, asthma, diabetes, childhood leukemia
reduce severe hyperbilirubinemia
promote breastfeeding, evaluate and ID, measure totoal serum bili or transQ bili for jaundiced bay in first 24hr. Under 38wk are at higher risk.
new born discharge
doesn’t happen until pass stool and urine, safe rear facing seat
vaccination
hep B. w/in 12 hr if mother’s hep B status in unknown. Suggest- influenza, tetanus, reduced diphtheria toxoid, acellular pertusis, TdaP to postpartum mother.
Vit D
400IU per day after birth
risks for respiratory rdistress
maternal Diabetes, prematurity (lung immature), maternal GBS, c section (TTN), premature rupture of membrane (prolong PROM >18hr- RF for neonatal sepsis), meconium in amniotic fluid (meconium aspiration syndrome)
DD of respiratory distress
RDS, TTN, pxn, hypoglycemia, CHF, neonatal sepsis, congenital diaphramatic hernia, severe coarctation of aorta, mecoium aspiration, maternal drug exposure, hypothermia
RDS
Caused by a deficiency of lung surfactant and delayed lung maturation
Can occur as late as 37 weeks’ gestation
Most common cause of respiratory distress in premature infants
Remember that there may be surfactant deficiency and delayed lung maturation in infants of diabetic mothers
CXR- diffuse reticulograndular - ground glass appearance + air bronchograms
Transient tachypnea of newborn (TTN)
Result of delayed clearance of fluid from the lungs following birth. Usually fluid cleared by squeezing during uterine contraction and absorption by pul lymphatics.
Much more common in infants born to diabetic mothers and in infants born by c-section
While generally considered a disorder of term infants, TTN does occur in premature infants
CXR- wet lungs, no consolidations/ air bronchograms
Pneumothorax
Caused by a collection of gas in the pleural space with resultant collapse of lung tissue
Common risk factors are mechanical ventilation or underlying lung disease (especially meconium aspiration or severe infant respiratory distress syndrome).
While relatively uncommon, always an important consideration in an infant with respiratory distress
More likely in a premature infant with RDS
hypoglycemia
May be seen in infants of diabetic mothers due to the chronic hyperinsulinemic state that occurred during gestation
Can be more pronounced in premature infants
Tachypnea is a non-specific response to this metabolic derangement. can be asx- but even then can affect brain dev. so always ck. at birth, sep of placenta cause decr in glu lvl over first 1-2hr then stabilize by 3-4hr- 65-71. intervene for asx at
CHF
In an infant, most often caused by a congenital heart defect
May present with early cardiac failure and tachypnea
Increased risk of heart defects in IDM infants, and therefore an increased risk of CHF
neonatal sepsis
Can present initially with tachypnea and progress to more severe illness rapidly
Often due to infection with Group B Streptococcus (GBS), usually transmitted from the mother during labor
Prolonged PROM is associated with an increased incidence of neonatal sepsis
congenital diaphragmatic hernia
Occurs in 1 out of every 2,200 to 5,000 live births
Most common type (accounting for > 95% of cases) is the Bochdalek hernia, which is located posterolaterally
Absent breath sounds or presence of bowel sounds on one side of the chest are important diagnostic clues
coarctation of aorta
if there is severe LV outflow tract obstruction
hypothermia
Premature newborns are more at risk to become hypothermic because of their small body size
small for gestation age
transition from intra to extrauterine life
1) Removal of the low-resistance placental circulation by cutting the umbilical cord.
2) Initiation of air breathing by the newborn infant.
3) Reduction of the pulmonary arterial resistance.
4) Closure of the PFO and PDA.
1st hr
elevated HR (160-180) and RR (60-80)
2nd hr of life
HR (120-160), RR (40-60)
persistent pulmonary HT of newborn (PPHN)
persistence of fetal circulation
sx of respiratory distress
tachypnea, retration of inter/subcostal = incr work of breathing due to decr lung compliance. grunting- end of expiration, = air expelled from partially closed glottis
cyanosis
due to respiraotry, congenital heart defects or CNS, infectious, other
respiratory causes of cyanosis
TTN, RDS.
Congenital heart defects-cyanosis
ToF. TGA
CNS cyanosis
hypoxic-ischemic encephalopathy, intraventricular hemorrhage, sepsis/meningitis
infectious cynosis
septic shock, meningitis
cynosis other
respiratory depression 2/2 maternal med, hypothermia, polycythemia/hyperviscosity syndrome
oxygen challenge test
dd cardiac and pulmonary etiology. O2 incr PaO2 of respiratory condition but not in cardiac lesion cases
insulin
primary anabolic hormone for fetal growth especially in 3rd T- heart/liver/M are insulin sensitive. incr in fat sun and deposition. insulin insensitive organ- brain, kidney -normal size. therefore HbA1c control in T1 is predictor. >12–> 12x incr in major malformations