neuropsych book Flashcards

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1
Q

childhood onset fluency disorder description

A

a disturbance in speech fluency that includes a number of problems with speech such as repeating syllables or words, prolonging certain sounds, making obvious pauses, or substituting words to replace ones that are difficult to articulate

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2
Q

childhood onset fluency disorder stats

A

-2x more common in boys than girls
-most often by age 6
-80% of children who stutter before enter school will no longer have stutter after they have been in school a year or so

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3
Q

childhood onset fluency disorder etiology

A

-makes people socially anxious
-multiple brain pathways
-genetic influences

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4
Q

childhood onset fluency disorder treatment

A

-regulated breathing method (stop speaking when stuttering episode starts and take deep breath)
-altered auditory feedback (electronically charging speech feedback)
-self monitoring

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5
Q

language disorder description

A

-limited speech in all situations
-expressive language (what is said) significantly lower than receptive language (what is understood)

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6
Q

language disorder stats

A

10-15% of children under 3
5x more likely in boys than girls

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7
Q

language disorder etiology

A

-parents pay not speak to them enough
-middle ear infection

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8
Q

language disorder treatment

A

may be self correcting and not require special intervention

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9
Q

social (pragmatic) communication disorder

A

difficulties with social aspects of verbal and non-verbal communication including verbosity, prosody, excessive switching of topics, dominating conversations

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10
Q

social (pragmatic) communication disorder stats

A

-cases rising

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11
Q

social (pragmatic) communication disorder treatment

A

-individualized social skills training with emphasis on teaching rules to carry on conversation and what is too much and too little info

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12
Q

tourettes description

A

-involuntary motor movements (tics) such as head twitching or vocalizations such as grunts, that often occur in rapid succession, come on suddenly , and happen in stereotypes ways, vocal tics often repetition of obscenities

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13
Q

tourette stats

A

-20% of kids show tic during growing years
-1-10 kids of 1000 have tourettes
-usually develops before 14
-high comorbidity with ADHD and OCD

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14
Q

tourettes etiollgy

A

multiple venerability genes

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15
Q

tourettes treatment

A

self monitoring, relaxation training and habit reversal

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16
Q

ADHD

A

maladaptive levels of inattention, excessive activity and impulsiveness

17
Q

inattention ADHD in DSM (ADD)

A

people may appear to not listen to others, may lose assignments or things, may not pay enough attention to details so they make careless mistakes

18
Q

hyperactivity adn impulsivity ADHD in DSM

A

fidgeting, having trouble sitting, always being on go

blurting out answers, having trouble waiting turns

19
Q

prevalence of ADHD in kids

A

5.2%
boys 2-3x more likely to have than girls
noted to be different around 3-4

20
Q

teens with ADHD more likely to have

A

pregnancy and std
driving difficulties like crashes or speeding

21
Q

ADHD in adulthood

A

Rachel Klein study following 200 boys
-employed at jobs but significantly lower positions than comparisons
-2.5 years less of education and less likely to hold higher degrees
-more likely to be divorced, have substance use problems, and antisocial personality disorder
-higher chance of head injury and ED visits

22
Q

causes of ADHD

A

-highly influenced by genetics
-mutations that create extra copies or result in the deletion of genes
-genes associated with dopamine, norepinephrine, GABA and serotonin
-look for specific ties in brain to specific behavior (endophenotypes)
-mother that smoked during pregnancy more likely to have mutation in dopamine system
-other pregnancy complications like alc use, martial problems, stress, low birth weight
-artificial food dyes and additives
-pesticides
-negative responses by teachers and parents and peers

23
Q

brain in those with ADHD

A

-brain slightly smaller in those with ADHD (3-4%)

24
Q

psychological interventions for ADHD

A

-set goals to increase time child is seated, number of path papers completed or appropriate social play with peers
-reinforcement programs reward for improvements and punish for misbehavior with loss of rewards
-structure kids day to prevent difficulties
-social skills training
-for adults cognitive behavioral intervention and improve organizational skills

25
Q

biological interventions for ADHD

A

-first type of meds used for kids are stimulants (ritalin, adderall)
-non-stimulants (atomoxetime, guanfacine, and clonidine helpful to reduce symptoms)
-stimulant meds help reinforce brain’s ability to focus attention on problem solving tasks
-drug trial may be dependent on someones genetic makeup

26
Q

Multimodal Treatment of Attention-Deficit/Hyperactivity Disorder (MTA)

A

-study by NIMH
-14 months, 579 kids
-4 groups: routine care without meds or behavior intervention (community care), medication (methylphenidate/ritalin), intensive behavioral treatment, combination of med and behavior
-found that combination and medication were better than behavioral alone and community care
-some disagreemnt/controversy

27
Q

response to intervention

A

identify child having specific learning disorder when the response to a known effective intervention is significantly inferior to the performance of peers

28
Q

statistics of learning disorders

A

-5-15% of youth lave LD
-higher frequency of diagnosis in wealthier regions (better access to services).
-1% of white children and 2.6% of black kids
-difficulties reading mort common
-students more likely to drop out of school, be unemployed and have suicidal thoughts/attempts

29
Q

communication disorders

A

-can appear deceptively benign but presence in early life can cause wide range of problems
-childhood onset fluency disorder (stuttering)
-language disorder

30
Q

genetics causes of learning disorders

A

-runs in families
-genes that affect learning and they may contribute to problems across domains
-some evidence that ability of word recognition depends on genes whereas others develop from environment (dyslexia, fluency)

31
Q

environmental causes of learning disorder

A

-home reading habits of families
-socioeconomic status, cultural expectations, child management practices, parental expectations

32
Q

biological causes of learning disorder

A

3 areas of L hemisphere for dyslexia
-Broca’s area (articulation and word analysis)
-area in left parietotemporal region (word analysis)
-and left occipototemporal area (recognizing word form)

-intrapariatal sulcus is critical for sense of numbers and implicated in math disorder

-none yet for written word

33
Q

treatment of learning disorders

A

-educational interventions :
1. specific skill instructions: vocab, main idea, facts in reading
2. strategy instruction: efforts to improve cognitive skills through decision making and critical thinking
-Direct Institution: systematic instruction (scripted lesson plans where students in small groups based on progress) and teaching for mastery (teach until understand all concepts), students tested for their understanding frequently
-MRI show that after intensive online program, those with reading disorders improved reading skills and brain started to function in similar ways to peers

34
Q

autism spectrum disorder (ASD)

A

neurodevelopmental disorder characterized by significant impairment in social interactions and communications and restricted patterns of behavior, interest and activity