Neuromuscular Flashcards

1
Q

Thrombus

A

Atherosclerotic plaque build up in artery causing occlusion

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2
Q

Embolus

A

Tissue (typically atherosclerotic plaque) travels through bloodstream until it reaches a vessel it cannot pass through, causing occlusion

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3
Q

L hemisphere CVA symptoms

A

R sided weakness
Frustration
Decreased processing
Aphasia
Dysphagia
Motor apraxia
Decreased R/L discrimination
R hemianopsia

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4
Q

R hemisphere CVA Symptoms

A

L sided weakness
Decreased attention span
L hemianopsia
Decreased awareness and judgment
Memory deficits
L inattention
Decreased abstract reasoning
Emotional lability
Impulsivity
Decreased spatial orientation

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5
Q

Brainstem CVA Symptoms

A

Unstable vitals
Decreased consciousness
Decreased ability to swallow
Bilateral weakness/paralysis

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6
Q

Cerebellum CVA Symptoms

A

Decreased balance
Ataxia
Decreased coordination
Nausea
Nystagmus
Decreased ability to perform postural adjustment

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7
Q

Flexor synergy pattern (UE)

A

Scapular elevation + retraction
GH abduction + ER
Elbow flexion
Wrist flexion
Forearm supination
Finger flexion and adduction
Thumb flexion and adduction

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8
Q

Extensor synergy pattern (UE)

A

Scapular depression + protraction
GH adduction + IR
Elbow extension
Wrist extension
Forearm pronation
Finger flexion and adduction
Thumb flexion and adduction

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9
Q

Flexor synergy pattern (LE)

A

Hip abduction + ER
Knee flexion
Ankle DF + supination
Toe extension

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10
Q

Extensor synergy pattern (LE)

A

Hip extension, adduction, IR
Knee extension
Ankle PF + pronation
Toe flexion + adduction

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11
Q

Motor control

A

Ability to regulate and direct movement

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12
Q

Motor learning

A

Acquisition or modification of movement

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13
Q

Motor learning principles

A

Learning vs performance
Use of feedback
Impact of practice on skill
Transfer of learning across tasks and environments

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14
Q

Adam’s closed loop theory of motor learning

A

Use of sensory feedback (compare current movement with stored information) and practice

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15
Q

Schmidt’s schema theory of motor learning

A

High emphasis on the value of feedback and variation with practice, open loop

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16
Q

Three stage model: cognitive phase

A

Controlled environment preferred
High rate of errors
Inconsistent attempts and performance
Increased cognitive work (listening, observing and processing feedback)

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17
Q

Three stage model: associative phase

A

Controlled or open environment okay
Decreased errors
More independently able to distinguish correct vs incorrect performance
Should avoid excessive external feedback (use of internal/proprioceptive feedback)
Decreased need for concentration and cognition
Skill refinement
Improved coordination

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18
Q

Three stage model: autonomous phase

A

Improved efficiency
Variable environment
Automatic response, non-cognitive
Error-free regardless of environment or distractions
Can perform more than one task simultaneously
Intrinsic feedback > extrinsic (should not be used)

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19
Q

Intrinsic feedback

A

Feedback received through sensory systems as a result of movement

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20
Q

Extrinsic feedback

A

Feedback provided during or after a movement, typically verbal or manual/tactile

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21
Q

Knowledge of results

A

Extrinsic, terminal feedback, regarding the outcome in relation to the goal

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22
Q

Knowledge of performance

A

Extrinsic, relating to the actual movement pattern

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23
Q

Massed practice

A

Practice time > rest time

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24
Q

Distributed practice

A

Practice time < (or equal to) rest time

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25
Constant practice
Uniform practice conditions
26
Variable practice
Differing practice conditions
27
Random practice
Varying practice amongst different tasks
28
Blocked practice
Consistent practice of single task
29
Whole training
Practicing entire task
30
Part training
Practicing individual component of task
31
Closed system model
Transfer of information that incorporates multiple feedback loops and larger distribution of control. Nervous system is an active participant
32
Compensation
Ability to utilize alternate strategies due to impairment that limits normal completion of task
33
Habituation
Decreased response due to consistent exposure to stimuli
34
Learning (non-associative)
Single repeated stimulus (habituation, desensitization)
35
Learning (associative)
Gaining understanding of the relationship between multiple stimuli (classic or operant conditioning)
36
Learning (procedural)
Learning of tasks that can be performed automatically without attention or concentration
37
Learning (declarative)
Learning of tasks that require attention, awareness, and reflection to attain knowledge
38
Open system model
Single transfer of information within any feedback loop, nervous system in "awaiting stimuli" to react
39
Sensitization
An increase in response that will occur as a result of a noxious stimulus
40
Closed motor skill
Skill performed under a stable and unchanging environment
41
Open motor skill
Skill performed under a consistently changing environment
42
Carr and shepherd motor relearning approach
Emphasizes that the factors involved with learning are also involved with re-learning (setting goal, inhibition of unnecessary movement, adjusting to balance/gravity, proper alignment, internal and external practice, feedback, knowledge of results)
43
Bobath NDT
-Train postural control by decreasing BOS and increasing distance between COM and BOS -Avoid compensation -Include rotation in treatment -Move in and out of midline to facilitate midline orientation/control -Facilitate and inhibit tone -Reflex inhibiting postures -Proximal control
44
Brunnstrom 7 stages of recovery (hemiplegia)
1 - no volitional movement 2 - basic synergies, spasticity beginning to form 3 - synergies performed volitionally, increasing spasticity 4 - decrease in spasticity, movement patterns exist outside of synergies 5 - decrease in spasticity, independence from synergy patterns 6 - isolated joint movements performed with coordination 7 - normal motor function
45
Associated reaction definition
Involuntary and automatic movement of a body part as a result of an intentional active or resistive movement in another body part
46
Homolateral synkinesis definition
Flexion pattern of the involved upper extremity facilitates flexion of the involved lower extremity
47
Raimiste's Phenomenon
The involved LE will abduct/adduct with applied resistance to the uninvolved LE in the same direction
48
Souques' Phenomenon
Raising the involved UE above 100 deg with elbow extension will produce abduction and extension of the fingers
49
UE PNF: D1 Flexion
Scapular elevation, abduction, upward rotation GH flexion, adduction, ER Elbow flexion or extension Radioulnar supination Wrist flexion, radial deviation Thumb adduction
50
UE PNF: D1 Extension
Scapular depression, adduction, downward rotation GH extension, abduction, IR Elbow flexion or extension Radioulnar pronation Wrist extension, ulnar deviation Thumb abduction
51
UE PNF: D2 Flexion
Scapular elevation, adduction, upward rotation GH flexion, abduction, ER Elbow flexion or extension Radioulnar supination Wrist extension, radial deviation Thumb extension
52
UE PNF: D2 Extension
Scapular depression, abduction, downward rotation GH extension, adduction, IR Elbow flexion or extension Radioulnar pronation Wrist flexion, ulnar deviation Thumb opposition
53
LE PNF: D1 Flexion
Pelvic protraction Hip flexion, adduction, ER Knee flexion or extension Ankle and toe DF, inversion
54
LE PNF: D1 Extension
Pelvic retraction Hip extension, abduction, IR Knee flexion or extension Ankle and foot PF, eversion
55
LE PNF: D2 Flexion
Pelvic elevation Hip flexion, abduction, IR Knee flexion or extension Ankle and foot DF, eversion
56
LE PNF: D2 Extension
Pelvic depression Hip extension, adduction, ER Knee flexion or extension Ankle and foot PF, inversion
57
Agonist reversals
Targets: controlled mobility, skill Isometric contraction, followed by alternating concentric and eccentric contractions with resistance
58
Alternating isometrics
Targets: stability (endurance and strength) Isometric contractions performed alternating from one side of joint to the other (without rest)
59
Contract relax
Targets: mobility End range of motion --> maximal isotonic contraction of antagonist muscle (5 s) while moving limb through ROM --> relax into range
60
Hold relax
Targets: mobility End range of motion --> maximal isometric contraction of antagonist muscle (5 s) --> relax into range
61
Hold relax active movement
Targets: mobility (used to initiate movement in muscle testing 1/5 of less) Muscle taken to shortened range --> contracted isometrically --> relaxed and taken to lengthened position + quick stretch --> isotonic contraction attempted to return limb to shortened position
62
Normal timing
Targets: skill (coordination) Performed distal to proximal, proximal components are limited until distal components activate and initiate movement - promotes appropriate muscle contraction timing (distal -- proximal)
63
Repeated contractions
Targets: mobility Used to initiate movement and sustain contraction through ROM Provide quick stretch followed by isometric/tonic contractions through ROM
64
Resisted progression
Targets: skill (coordination) Resistance applied during the gait cycle to enhance coordination, strength, endurance
65
Rhythmic initiation
Targets: mobility (initiating movement with hypertonia) passive --> active assisted --> active --> slightly resisted Slow and rhythmic through full ROM
66
Rhythmic stabilization
Targets: mobility, stability Used as a progression from alternating isometrics Isometric contractions of all muscles around a joint against progressive resistence
67
Rhythmical rotation
Target: mobility (decrease hypertonia) Passively rotating an extremity around longitudinal axis
68
Slow reversal
Target: stability, controlled mobility, skill (movement control and posture) Slow resisted concentric contractions of agonists and antagonists without rest
69
Slow reversal hold
Target: stability, controlled mobility, skill Slow reversal with the addition of isometric contraction at the end of each movement
70
NM Junction injury signs
Sensory intact, motor fatigue, normal DTR (Myesthenia gravia)
71
Chorea
Sudden, rapid, jerky, purposeless movement involving limbs, trunk, face
72
Asthenia
Weakness due to cerebellar pathology
73
Dysmetria
Inability to control the range of a movement or force of muscle activity
74
Evoke potentials purpose
Non-invasive, used to rule out MS, brain tumor, acoustic neuroma
75
Myelography purpose
Invasive, used to rule out abnormalities in subarachnoid space, spinal n injury, herniated disc, fracture, back/hip pathology, spinal tumors
76
ALS UMN signs
Incoordination Spasticity + babinski Clonus Fatigue Oral motor impairment Motor paralysis Respiratory paralysis
77
ALS LMN signs
Asymmetric muscle weakness Fasciculations Cramping Hand atrophy Distal > proximal weakness
78
Friedrich's ataxia signs
Gait unsteadiness UE ataxia Dysarthria Paresis Tremors Impaired reflexes, vibration and position sense Caused by impaired mitochondria function (autosomal recessive)
79
Huntington's disease signs
Involuntary choric movement Mild personality alteration Tongue protrusion Grimacing Ataxia
80
Multiple sclerosis signs
Visual impairment Paresthesias/sensory changes Clumsiness Weakness Ataxia Balance dysfunction Fatigue Periods of exaceration and remission
81
Myasthenia gravis signs
Extreme fatiguability Muscle weakness Ocular muscles impacted first Dysphagia CN weakness
82
Parkinson's disease signs
Resting tremor Impaired balance Bed mobility Fine motor tasks
83
PD progression
Hypokinesia Akinesia Rigidity Freezing Mask like appearance Festinating and shuffling gait Bradykinesia Poor posture Dysphagia
84
Post-polio syndrome signs
Slow and progressive weakness Fatigue Atrophy Pain Swallowing issues
85
Anti-spasticity drug names
Baclofen Valium (diazepam)
86
Cholinergic drug indications
Glaucoma Dementia Myesthenia gravis
87
Cholinergic drug names
Duroid Pilocar Aricept Tensilon
88
Dopamine replacement drug names
Levodopa Amantadine
89
Muscle relaxant drug names
Valium (diazepam) Flexeril Paraflex
90
Anti-epileptic drug side effects
Ataxia Skin changes HA Weight gain Behavior changes GI Blurred vision
91
Anti-spasticity drug side effects
Drowsiness Dizziness Confusion Weakness HA
92
Dopamine replacement drug side effects
Arrhythmias GI Orthostatic hypotension Mood Dyskinesia Tolerance
93
Muscle relaxant drug side effects
Sedation Drowsiness Dizziness N/V HA Tolerance Dependence
94
Allodynia
Pain due to non-pain provoking stimulus
95
Causalgia
Persistent, severe, burning pain due to peripheral n injury
96
Paresthesia
Abnormal sensation of numbness and tingling without objective cause
97
Peripheral n injury
Sensory loss, motor weakness, atrophy in nerve distribution Fasciculations Ex. trauma
98
Peripheral neuropathy injury
Stocking glove sensory impairments Distal > proximal weakness Fasciculations Decreased DTRs Ex. DB
99
Spinal root n injury
Dermatomal sensory loss and motor weakness Fasciculations Decreased DTRs Ex. herniated disc
100
Athetosis
Slow, writhing, involuntary movements
101
Dystonia
Abnormal muscle tone causing twisting, abnormal posture and repetitive movements Ex. PD, CP, encephalitis
102
Akinesia
Inability to initiate movement
103
Ataxia
Lack of muscle coordination
104
Dysdiadochokinesia
Inability to perform rapid alternating movements
105
Clasp knife response
Tone with greatest resistance at beginning of ROM
106
Cerebral angiography purpose
Invasive, used to diagnose CVA, brain tumor, aneurysm, vascular malformation
107
CT scan purpose
Non-invasive, used to rule out vascular malformation, tumor, cyst, herniated disc, hemorrhage, epilepsy, encephalitis, spinal stenosis, intracranial bleeding, head injury
108
Discography purpose
Invasive, CT scan + contract, used to assess integrity and pathology of spinal disc
109
EMG purpose
Invasive, used to rule out muscle pathology, nerve pathology, spinal cord disease, denervated muscle, LMN injury
110
MRI purpose
Non-invasive, used to rule out MS, brain tumor, head trauma
111
PET scan purpose
Non-invasive, used to rule out cerebral circulatory pathology, metabolism dysfunction, tumor, blood flow, brain changes
112
Spinal puncture purpose
Invasive, used to rule out hemorrhage, inflammation, infection, meningitis, tumor
113
Alzheimers signs
Changes in higher cortical functions (learning, memory, concentration) Loss of orientation Word finding difficulty Depression Poor judgement Rigidity Bradykinesia Shuffling gait
114
Bell's palsy signs
Asymmetrical facial appearance, full 1/2 face
115
Carpal tunnel syndrome signs
Sensory and motor changes to median n distribution Radiating pain Night pain Hand weakness Decreased grip strength Clumsiness Decreased wrist mobility
116
Spinocerebellar ataxia signs
Neuropathy Ataxia Restless leg syndrome Autosomal dominant
117
Acquired ataxias cause
Toxin exposure (alcohol, carbon monoxide, etc) Idiopathic (hypothydroidism)
118
Epilepsy signs
Loss of awareness Loss of consciousness Movement disturbances Sensation Mood Mental function
119
Myoclonic seizures
Muscle contractions involving one or multiple body parts
120
Atonic seizures
Loss of tone, LOC, drop attack
121
Bilateral convulsive seizure (tonic and clonic)
Sudden LOC Rigidity - tonic Jerking - clonic
122
Absence seizure
Sudden cessation of ongoing conscious activity (ie petit mal seizure)
123
Guillain barre syndrome signs
Motor weakness (distal to proximal) Sensory impairment Respiratory paralysis Peak disability @ 2-4 wk Decreased DTRs
124
Anti-epileptic drug names
Clonazepam Gabapentin Carbamezepine
125
Cholinergic drug side effects
GI Bradycardia Bronchoconstriction Parasympathetic response
126
Dorsal column (fasiculis gracilis) function
LE proprioception Vibration Two point discrimination
127
Dorsal column (fasiculis cuneatus) function
UE proprioception Vibration Two point discrimination
128
Anterior spinothalamic tract function
Crude touch Deep pressure
129
Lateral spinothalamic tract function
Pain Temperature
130
Anterior corticospinal tract function
Ipsilateral voluntary, discrete movement
131
Lateral corticospinal tract function
Contralateral voluntary, fine movement
132
Analgesia
Inability to feel pain
133
Anesthesia
Loss of sensation
134
Dysesthesia
Distortion of any sense (specifically touch)
135
Hyperesthesia
Increased sensitivity to stimulation
136
Hyperpathia
Extreme exaggerated response to pain
137
Hypesthesia
Decreased sensitivity to touch
138
Neuralgia
Nerve pain
139
Pallanesthesia
Loss of vibration sensation
140
Anterior horn cell injury
Sensory intact Motor weakness Normal or decreased DTRs Ex. polio, ALS
141
Muscle injury
Sensory intact Motor weakness Normal or decreased DTRs Ex. muscular dystrophy
142
Nerve conduction velocity purpose
Non-invasive, used to rule out peripheral neuropathy, carpal tunnel, demyelination injury, peripheral n compression
143
Rood theory to neuromuscular treatment
Motor output is the result of past and present sensory input Goal: obtain homeostasis by using key patterns to enhance motor control Sensory stimulation is used to facilitate or inhibit a response
144
Sensory facilitation techniques
Approximation Joint compression Icing Light touch Quick stretch Resistance Tapping Traction
145
Sensory inhibition techniques
Deep pressure Prolonged stretch Warmth Prolonged cold
146
Agnosia
Inability to interpret information
147
Agraphesthesia
Inability to recognize symbols, letters, or numbers traced on the skin
148
Agraphia
Inability to write due to lesion
149
Alexia
Inability to read or comprehend written language due to lesion
150
Anosognosia
Denial or unawareness of one's illness
151
Apraxia
Inability to perform purposeful learned movements (with no impairment that would hinder)
152
Astereognosis
Inability to recognize objects by touch
153
Constructional apraxia
Inability to reproduce geometric figures and designs. Unable to visually analyze how to perform a task.
154
Decerebrate rigidity
Characteristic of corticospinal lesion at the level of the brainstem Extension of trunk and all extremities
155
Decorticate rigidity
Characteristic of corticospinal lesion at the level of the diencephalone Trunk and LE in extension, UE in flexion
156
Dysprosody
Impairment in rhythm and inflection of speech
157
Ideational apraxia
Inability to formulate an initial motor plan and sequence tasks where the proprioceptive input necessary for movement is impaired
158
Ideomotor apraxia
Able to plan a movement but cannot volitionally perform it
159
Neologism
Substitution within a word that is so severe that it makes the word unrecognizable
160
Anterior cord syndrome impairments and MOI
MOI flexion Loss of motor, pain, temperature (CST and STT)
161
Brown sequard syndrome impairments and MOI
MOI stab Loss of ipsilateral motor, vibration, proprioception (DCML and CST) Loss of contralateral pain and temperature (STT)
162
Cauda equina syndrome impairments
Lesion below L1, peripheral n injury Flaccidity, areflexia, bowel and bladder dysfunction
163
Central cord syndrome impairments and MOI
MOI hyperextension STT, DCML, CST UE > LE injury Motor > sensory impairments
164
Posterior cord syndrome impairments and MOI
MOI (rare) posterior spinal a compression Loss of proprioception, two point discrimination, stereogensis Motor preserved
165
ASIA A
Complete: no sensory or motor function in S4-5
166
ASIA B
Sensory incomplete Sensory preserved below the level of lesion, including S4-5. No motor function in more than 3 levels below motor level
167
ASIA C
Motor incomplete Motor function preserved for voluntary anal contraction OR sensory incomplete + motor function of more than 3 levels below motor level Less than half of the muscle below injury have a grade of > 3
168
ASIA D
Motor incomplete At least half of muscles below injury have a grade > 3
169
ASIA E
Normal function
170
ASIA Key muscles
C5 elbow flexors C6 wrist extensors C7 elbow extensors C8 finger flexors T1 small finger abductors L2 hip flexors L3 knee extensors L4 ankle DFs L5 great toe extensors S1 ankle PFs
171
DVT symptoms
LE swelling Pain Sensitivity over clot Warmth
172
Autonomic dysreflexia
Can occur with any lesion above T6 Symptoms: high BP Severe HA Blurred vision Stuff nose Profuse sweating Goose bumps Flushing
173
Heterotopic Ossification symptoms
Edema Decreased ROM Increased temperature of involved joint
174
Myelotomy
Surgical procedure that severs certain tracts in spinal cord to decrease spasticity and improve function
175
Neurectomy
Surgical removal of a segment of a nerve to decrease spasticity and improve function
176
Neurogenic non-reflexive bladder
Flaccid bladder (underactive muscles), injuries below T12
177
Neurogenic reflexive bladder
Reflexive bladder, empties reflexively, injury above T12
178
Paradoxical breathing
Abnormal breathing where abdomen rises and chest is pulled inward during inspiration, upon expiration the abdomen falls and chest expands (common in tetraplegia)
179
Rhizotomy
Surgical resection of sensory component of spinal n to decrease spasticity and improve function
180
Tenotomy
Surgical release of a tendon in order to decrease spasticity and improve function
181
Coup lesion (TBI)
A direct lesion of the brain under the point of impact. Local brain damage sustained Primary injury
182
Contrecoup lesion (TBI)
An injury that results on the opposite side of the brain, due to rebound effect of brain after impact Primary injury
183
Epidural hematoma (TBI)
Hemorrhage that forms between skull and dura mater Secondary injury
184
Subdural hematoma (TBI)
Hemorrhage that forms between dura mater and arachnoid mater (due to venous rupture) Secondary injury
185
Secondary injury types (TBI)
Hematoma Hypoxia Ischemia Increased ICP Post traumatic epilepsy
186
Primary injury types (TBI)
Skull penetration Skull fracture Contusions
187
Coma
Unconscious and unresponsible to external and internal stimuli
188
Stupor
State of general unresponsiveness with arousal from repeated stimuli
189
Obtundity
State of sleep, reduced alertness, delayed responses to stimuli
190
Grade 1 concussion
Head injury without LOC Transient confusion, resolves in less than 15 min Removed from game and return after 1 week of no symptoms
191
Grade 2 concussion
Moderate head injury Transient confusion for > 15 min Poor concentration, retrograde/anterograde amnesia Removed from game and medical eval Return to play after 2 weeks of no symptoms
192
Grade 3 concussion
Head injury with LOC Transport to ER for full neuro evaluation Minimum of 1 month symptom free for return to play
193
Rancho level I
No response Deep sleep, completely unresponsive
194
Rancho level II
Generalized response Inconsistent, non-purposeful, non-specific response to stimuli Responses include physiological changes, gross body movements, vocalization
195
Rancho level III
Localized response Inconsistent, specific response to stimuli May follow simple commands (EC, hand squeeze) inconsistently
196
Rancho level IV
Confused agitated Heightened state of activity Bizarre, non-purposeful activity Unable to cooperate with treatment efforts Frequent incoherent, inappropriate verbalization Brief attention to environment Lacks short and long term recall
197
Rancho level V
Confused in-appropriate Responds to simple commands fairly consistently Gross attention to the environment, but is highly distractible and cannot focus on task Inappropriate vocalization Severely impaired memory May perform learned tasks with structure, unable to learn new tasks
198
Rancho level VI
Confused appropriate Goal-directed behavior, dependent on external input/direction Follows simple directions consistently, shows carryover May respond incorrectly due to memory difficulty More depth and detail to past memory than present
199
Rancho level VII
Automatic appropriate Appropriate and oriented to hospital and home setting Goes through daily routine automatically Carryover in new learning Able to initiate social or recreational activities with structure
200
Rancho level VIII
Purposeful appropriate Able to recall and integrate past and present events and is aware of and responsive to environmental stimuli Shows carryover in new learning and no longer needs supervision
201
Glasgow coma scale
< 8 severe brain injury 9-12 moderate brain injury 13-15 mild brain injury Assesses: eye opening, motor response, verbal response
202
Autonomic nervous system: sympathetic division function
Stimulating response 'fight or flight' Involved with norepinephrine
203
Somatic nervous system function
Regulates body movement through sensory and motor neurons that transmit information from the brain to muscle fibers throughout the body. Controls: voluntary movement, 5 senses, reflex arcs (DTRs)
204
Autonomic nervous system: parasympathetic division function
Conserving and restoring 'rest and digest' Involved with acetylcholine
205
Limbic system function
Mood Emotion Recent memory Olfaction Appetite Drastic behavior change with lesion
206
Frontal lobe functions
Personality Voluntary movement Orientation Executive function Brocas area (L)
207
Parietal lobe functions
Sensation, kinesthesia, vibration, temperature Memory Language interpretation Receives sensory info (hearing, vision, etc) Spatial perception
208
Temporal lobe functions
Auditory Olfactory Wernickes area (L) Interpretations of emotions and reactions
209
Occipital lobe functions
Visual processing Distance perception Color, light, shape
210
Hippocampus
L temporal lobe Forms and stores declarative memories
211
Basal ganglia
Responsible for voluntary movement, regulation of autonomic movement, posture, tone, control of motor responses Impairments may cause: PD, HD, ADHD, Tourette's OCD
212
Amygdala
Located in bilateral temporal lobes Emotion and social processing Fear, pleasure, arousal Memory processing
213
Thalamus
Relay/processing for info in cerebral cortex. Receives information from cerebellum, basal ganglia and sensory pathways and transmits to cerebrum Impairment may cause thalamic pain syndrome
214
Hypothalamus
Receives and integrate info from the ANS, regulates hormones, controls hunger, thirst, sexual behavior, sleep, body temp, glands
215
Subthalamus
Regulates skeletal muscle movement
216
Epithalamus
Pineal gland (secretes melatonin and controls circadian rhythm)
217
Midbrain function
Relay center Reflex center
218
Cerebellum function
Movement fine tuning, posture, tone, rapid alternating movements, location of extremities in space
219
Pons function
Part of hindbrain and brainstem RR regulation, head orientation in regard to visual/auditory stimuli
220
Medulla function
Part of hindbrain ANS activity, HR/RR regulation Vomiting, coughing, sneezing Relays somatic sensory info
221
Brainstem
Made up of midbrain, pons, medulla Relays info from body to cerebral cortex Primitive functions Reticular activating system
222
ACA blood supply
Anterior frontal lobe, medial frontal and parietal lobes
223
MCA blood supply
Frontal lobe, parietal lobe, outer surfaces of temporal lobe
224
PCA blood supply
Medial and inferior temporal lobes, medial occipital lobe, thalamus, hypothalamus, visual receptive area
225
Vertebrobasilar a. blood supply
Cerebellum, medulla, pons, midbrain, occipital cortex, brainstem
226
ACA lesion impairments
Personality Hemiparesis/plegia Neglect Aphasia Perseveration
227
MCA lesion impairments
Aphasia (communication & language interpretation) Spatial relations Cognition Sensation Voluntary movement (face and arm > leg)
228
PCA lesion impairments
Contralateral hemiplegia (face and arm > leg) Ataxia/tremor Homonymous hemianopsia Blindness Receptive aphasia Memory deficits
229
Vertebral basilar a lesion impairments
Unilateral or bilateral extremity weakness Loss of vibration sense, two point discrimination, and proprioception Diplopia Homonymous hemianopsia Dysphagia Dysarthria Nausea Confusion
230
L hemisphere function
Ability to understand language Producing written and spoken language Expression of positive emotions Ability to be analytical, controlled, logical Math
231
R hemisphere function
Nonverbal processing Artistic expression Spatial relationships Kinesthetic awareness Body image awareness
232