Neurological Exam Flashcards

1
Q

How many cranial nerves are there?

A

12

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2
Q

What is cranial nerve 1?

A

Olfactory.

Unilateral olfactory loss is actually more common than bilateral and occurs with subfrontal masses.

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3
Q

What is cranial nerve 2?

A

Optic nerve.
Optic nerve projects to the lateral geniculate nucleus (LGN) and superior colliculus.

Loss of a visual HEMIFIELD on the same side in both eyes (homonymous hemianopsia) suggests a lesion involving the LGN, optic radiations, or contralateral occipital lobe. This would look like missing half of the field on left (or right) side in each eye.

Loss of a visual field QUADRANT (superior homonymous quadrantanopsia) would suggest involvement of axons in Meyer’s loop in the posterior temporal lobe opposite the field deficit. This is called pie in the sky disorder, the upper top left quadrant might be missing in each eye.

Loss of the visual temporal FIELDS (bitemporal hemianopsia) suggests a chiasmatic lesion (e.g., pituitary adenoma). This would look like missing the left half of vision on the left eye, and missing the right half of vision on the right eye, likely limiting peripheral vision significantly.

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4
Q

What is cranial nerve 3?

A

Oculomotor.
The oculomotor nerve projects from the midbrain to the iris and five of the extraocular muscles (EOMs). A lesion to this nerve may lead to fixed, dilated pupil (blown pupil). CN III controls movement of eyes downward, upward, and or mesial directions

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5
Q

What is cranial nerve 4?

A

Trochlear.

Also controls eye movements. May lead to diplopia if compressed.

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6
Q

What is cranial nerve 5?

A

Trigeminal.
Projects from the pons. Has 3 different divisions - ophthalmic, maxillary, and mandibular. Motor functioning of the trigeminal nerve may be evaluated by asking the patient to open his or her mouth. The jaw will deviate to the side of weakened muscle innervation.

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7
Q

What is cranial nerve 6?

A

Abducens.

Also controls eye movements. May lead to diplopia if compressed.

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8
Q

What is cranial nerve 7?

A

Facial.
Controls motor functions of the upper and lower face, as well as the salivary glands, tear glands, and nasal mucosa.
Assessed by asking patient to smile and watching for unequal elevation of mouth’s corners. Person with facial nerve damage will also be unable to close both eyes or wrinkle their brows symmetrically.

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9
Q

What is cranial nerve 8?

A

Vestibulocochlear.

Auditory and vestibular functions

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10
Q

What is cranial nerve 9?

A

Glossopharyngeal.

Damage to this nerve can result in loss of taste sensation in part of the tongue and impaired swallowing

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11
Q

What is cranial nerve 10?

A

Vagus.

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12
Q

What is cranial nerve 11?

A

Spinal accessory.

Tested by having the patient shrug his or her shoulders. Asymmetric shrug suggests dysfunction o the side that is lower.

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13
Q

What is cranial nerve 12?

A

Hypoglossal.

Tested by tongue protrusion. Tongue deviates toward the side of nerve weakness. Subtle dysarthria may be noted.

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14
Q

What is antalgic gait?

A

When less weight appears placed on one leg, suggesting a pain-guarding strategy.

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15
Q

When there is wide-based gait, what could that suggest?

A

Cerebellar ataxia.

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16
Q

If one leg is observed to swing around (circumduct) in an arc, this might suggest what…

A

Hemiparetic gait.

17
Q

If the lower extremities have reduced knee flexion, and gait has a “scissoring” quality (imagine one blade on surface with next blade on surface), person might walk on tiptoes, this could suggest…

A

Corticospinal dysfunction.

18
Q

Inability to do tandem gait suggests…

A

Cerebellar dysfunction or other higher motor dysfunction

19
Q

Dysmetria is?

A

When there is inaccuracy with upper extremeties in pointing from nose to doctor’s fingertip.

20
Q

What is the Romberg sign?

A

When unsteadiness occurs while standing with feet together and eyes closed. May suggest loss of propioception.

21
Q

What is sensory extinction?

A

Extinction to simultaneous sensory stimulation (ESS) is a clinical phenomenon in which a patient perceives a unilateral sensory stimulus presented in isolation but fails to perceive the same stimulus when presented simultaneously with a second stimulus. ESS is most commonly seen after right hemisphere strokes and may adversely affect prognosis. The mechanisms of ESS have not been fully elucidated. The delayed information processing model proposes that delays in the transfer of sensory information in the damaged hemisphere increase the susceptibility to interference from the intact hemisphere.

May suggest contralateral parietal lobe dysfunction

22
Q

What is the use of computed tomography (CT)?

A

Helps distinguish structures of very different tissue composition (blood vs brain vs bone). Useful in demonstrating hemorrhage, skull fracture, and mass effect. Less useful in primarily white matter conditions or in making refined differential diagnoses (tumor vs other mass).

23
Q

What is use of MRI?

A

Detects reaction of water molecules within a strong magnetic field after the introduction of a radiowave signal. T1 scans demonstrate greater anatomic detail but less tissue contrast, where as T2 scans have enhanced contrast that has greater clinical benefit in differentiating various types of brain tissue and various neuropathologies. In T2, intact white matter appears gray, but plaques (MS) will appear brighter. CSF also looks brighter on T2 scans, facilitating the identification of cavitated lesions (lacunar infarcts). FLAIR allows for the representation of even greater contrast between normal and pathological tissue.