Neurogenic Dysphonias Flashcards
Neurogenic Dysphonias
Any dysphonia which occurs as a result of an impairment in the innervation, function, or motor planning aspects of the muscles involved in phonation, and resonance/articulation resulting in a change in voice production
Motor Speech Disorders
Disorders of speech resulting from neurologic impairment affecting the motor programming or neuromuscular execution of speech
Dysarthria, Apraxia
Dysarthrias
Group of MSD resulting from disturbances in muscular control over the speech mechanism
Due to damage of the central or peripheral nervous system
Results from paralysis, paresis, or incoordination of the speech musculature
Vocal fold paralysis
When one or both vocal folds are unable to move either towards or away from midline
Etiology: damage to CN X
90% due to LMN lesions
Vocal fold paralysis characteristics
Flaccid dysphonias due to a reduction in muscle tone resulting from a loss of motor supply to the muscle
Bilateral vocal fold paralysis
Usually result of lesions high in trunk of vagus nerve or at the nuclei of origin in the medulla
Etiology: tumors, carcinoma, trauma
Adductor vocal fold paralysis
Neither vocal fold is capable of moving toward the midline
Phonation = impossible
At risk for aspiration
Abductor vocal fold paralysis
Vocal folds remain at midline
Serious respiratory problems
Unilateral vocal fold paralysis
Most common cause = disruption of recurrent laryngeal nerve on one side
Etiology: damage or trauma to the RLN
Laryngeal adductor muscles cannot adduct the vocal folds
Paralyzed vocal fold remains fixed in paramedian position during both inspiration and expiration
Unilateral vocal fold paralysis Characteristics
Dysphonic or aphonic Breathy, hoarse Phonation time reduced Reduced or monoloud Diplophonia, pitch breaks
Myasthenia Gravis
Autoimmune disease in which the neuromuscular junction becomes impaired as the patient uses that particular muscle or muscle group resulting in extreme muscle fatigue
Muscles innervated by head and neck are very vulnerable to this disease
Possible difficulty swallowing
Results in flaccid dysarthria/dysphonia
Etiology: damage to the receptors at the neuromuscular junction, prevents the normal transfer of impulse from the nerve in the particular muscle
Myasthenia Gravis Characteristics
Typically normal voice changes to weak, breathy, barely audible voice; after a few minutes of rest voice is restored
Guillain-Barre
Focal demyelination of spinal and cranial nerves
Flaccid dysarthria/dysphonia
Possible dysphagia
Etiology: unknown cause but frequently preceded by a viral infection
Affects extremities as well as facial, oro-pharyngeal and occular muscles
Spastic Dysphonia
Muscular hypertonia: significantly increased muscle tone wherein the limb cannot be moved passively or volitionally with ease
UMN dysphonia
Spasticity of the laryngeal muscles
Etiology: small bilateral lesions that occur just above the nucleus of CN X
Frequently co-occurs with spastic dysarthria
Ataxic Dysphonia
Lesion in cerebellum
Loss of regulatory movements
Spastic Dysphonia Characteristics
Strained, strangled, harsh vocal quality, low pitch, monopitch, reduced/variable loudness, possible hypernasality
Ataxic Dysphonia Characteristics
Normal vocal quality, hoarse/harsh vocal quality, low pitch or monopitch, monoloudness, inappropriate/imprecise prosody, excessive loudness, vocal tremor
Dysphonias related to Extrapyramidal System
Depletion/functional reduction of Acetylcholine or dopamine on the activities of the basal ganglia
Imbalance of the two neurotransmitters causes movement disorders with related dysarthrias/dysphonias