Neurocognitive Disorders and Grieving Flashcards

1
Q

Delirium:

A

Definition:
Delirium is a state of acute confusion and rapidly changing mental function. It is characterized by an inability to focus attention, changes in psychomotor activity, incoherent speech, disorientation, hallucinations, illusions, and an acute onset over hours or days. Importantly, it is usually temporary, and clinical features may fluctuate throughout the day, often worsening at night.
Clinical Features:

  • Inability to Focus Attention: Patients with delirium struggle to maintain attention and concentration.
  • Change in Psychomotor Activity: This can manifest as either agitation or a slowed-down, lethargic state.
  • Incoherent Speech: Speech may be disjointed, nonsensical, or difficult to follow.
  • Disorientation: Patients may be unaware of their surroundings, time, or even their own identity.
  • Hallucinations and Illusions: Perceptions may be altered, leading to seeing things that are not present.
  • Acute Onset: Delirium has a sudden and rapid onset, in contrast to some other mental health conditions.
  • Fluctuating Course: Symptoms may vary in intensity and clarity throughout the day. Worsens at night.

Etiology (Causes):
Delirium can be triggered by various underlying medical conditions. Some common causes include:

  1. Systemic Infections:
    • Example: Severe bacterial or viral infections, such as pneumonia or urinary tract infections.
  2. Fever:
    • Example: High fever associated with an infection or inflammatory condition.
  3. Endocrine Disorders:
    • Example: Pituitary disorders
  4. Seizures:
    • Example: Intense or recurrent seizures affecting the brain’s normal function.
  5. Metabolic Disorders:
    • Example: Hypo/per/glycemia ( blood sugar: poorly controlled, can lead to vascular changes that affect blood flow to the brain.) or hypoxia (inadequate oxygen supply to tissues).
  6. Trauma:
    • Example: Physical trauma, such as a head injury, or psychological trauma.
  7. Sensory or Sleep Deprivation:
    • Example: Prolonged periods of sensory isolation or chronic lack of sleep.
  8. Substance Intoxication or Withdrawal:
    • Example: Delirium tremens (DT) resulting from alcohol withdrawal.
  9. Medications:
    • Example: Adverse reactions to certain medications, particularly anticholinergic drugs or certain psychotropic medications.
  10. Electrolyte Imbalance:
    • Example: Disturbances in sodium, potassium, or calcium levels. They can fuck with the blood and the brain as well.
  11. Cerebrovascular Accidents (CVAs):
    • Example: Stroke or other cerebrovascular events affecting blood flow to the brain.
  12. Brain Abscess:
    • Example: A collection of pus within the brain tissue.
  13. Post-operative States:
    • Example: Delirium that occurs after surgery, especially in elderly individuals.

Treatment:
The primary goal in treating delirium is to identify and address the underlying cause. This may involve medical interventions such as treating infections or adjusting medications. From a psychiatric standpoint:

  • Patient Safety: Ensuring the safety of the patient is a top priority.
  • Close Observation: Regular and close monitoring of the patient’s mental status is crucial.
  • Pharmacological Intervention: If the patient becomes violent or poses a risk to themselves, medications may be used. Commonly employed drugs include haloperidol (Haldol) and lorazepam (Ativan). However, it’s essential to use these medications judiciously (with good judgment.), as polypharmacy can exacerbate delirium. Only use what is necessary for the patient’s safety.
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2
Q

Difference between dementia, depression (pseudodementia), and normal forgetting:

A

Normal Forgetting:
Normal forgetting is a part of the aging process and is considered a typical aspect of cognitive function in older individuals. Here are some key points:
- Onset and Progression: Recall processes begin to slow around age 40, and this slowing continues as individuals age.
- Memory Function: While memory remains generally intact, there is a decrease in the speed of information retrieval. This can manifest as forgetfulness in daily life.
- Other Cognitive Aspects: There may be some decrease in attention span (فترة مدى duración), abstraction, and naming ability. This is a natural consequence of aging and the decrease in the efficiency of neuronal processing.
- Neuronal Changes: Aging involves a decrease in the number of neurons and a decline in their processing ability. This contributes to the overall slowing of cognitive functions.
- Lifestyle Impact: There is increasing evidence that staying mentally and physically active can help slow the normal aging-related cognitive decline. The phrase "use it or lose it" encapsulates the idea that engaging in cognitive and physical activities can support cognitive function as people age

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3
Q

Dementia:

Latin word "dement," which means "out of one's mind" "خَرَف
Don’t confuse with delirium

A

Dementia is a broad term for a group of cognitive disorders characterized by a decline in memory and other cognitive abilities that interfere with daily life. Here are some key distinctions:
- Onset and Progression: Dementia is not a normal part of aging. It has a more insidious (خبيث) onset and a progressive course.
- Memory Impairment: Unlike normal forgetting, dementia involves significant memory impairment that interferes with daily functioning. It goes beyond occasional forgetfulness.
- Cognitive Decline: Dementia affects various cognitive functions, including memory, language, problem-solving, and executive function.
- Neuropsychiatric Symptoms: Dementia often presents with neuropsychiatric symptoms such as personality changes, mood swings, and behavioral issues.
- Irreversible: Most forms of dementia are progressive and irreversible, with Alzheimer's disease being the most common cause.
Depression (Pseudodementia):
Depression can sometimes present with symptoms that mimic dementia, a condition known as pseudodementia. Here are some distinguishing features:
- Onset and Course: Pseudodementia often has a more sudden onset, and its course is tied to the underlying depressive episode.
- Reversibility: Unlike most dementias, pseudodementia is reversible with appropriate treatment of the underlying depression.
- Memory Impairment: Memory deficits in pseudodementia are typically more related to attention and concentration difficulties, rather than the profound and widespread memory impairment seen in dementia.
- Mood Symptoms: Depression, including feelings of sadness, hopelessness, and a lack of interest or pleasure, is a prominent feature in pseudodementia.

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4
Q

Characteristics of depression in the elderly, sometimes referred to as “pseudodementia”:

A

Depression in the Elderly (“Pseudodementia”):
- Prevalence: is relatively common in the elderly population, and in some cases, it can present with symptoms that mimic dementia, leading to the term "pseudodementia."
- Global Slowing of Mental Processes: Individuals with pseudodementia, especially in the elderly, often experience a general slowing of all mental processes. This can manifest as difficulties with concentration, attention, and overall cognitive function.
- Memory Complaints: Patients may complain of forgetfulness, reflecting the cognitive impact of depression. However, it’s important to note that these memory complaints are often more related to difficulties with attention and concentration rather than the profound and widespread memory impairment seen in true dementias.
- Preservation of Certain Cognitive Functions: Despite cognitive difficulties, aspects of cognitive function such as recall, spatial orientation, and recognition tend to remain mostly intact in depression.
- Association with Stress or Loss: Depression in the elderly is often triggered or exacerbated by stressors, such as the loss of a loved one, health issues, or other life changes.
- Physical Symptoms: Physical symptoms commonly associated with depression include a loss of appetite, decrease in overall activity levels, and a flat affect (reduced emotional expression).
- Diurnal Variation: Symptoms may show diurnal (daily in Latin) variation, with some improvement as the day progresses. This contrasts with the more consistent and progressive nature of many dementias.
- Social Behavior: Despite cognitive difficulties, individuals with depression typically remain oriented to their surroundings and retain social inhibitions. They do not exhibit wandering behavior, which is sometimes seen in advanced dementia.
- Response to Treatment: One key feature that distinguishes pseudodementia from true dementia is the potential for improvement. Pseudodementia is reversible with appropriate treatment of the underlying depression, whereas most forms of dementia are progressive and irreversible.

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5
Q

Features of dementia, now categorized as Mild Neurocognitive Disorder or Major Neurocognitive Disorder in the DSM-5:
Dementia (Mild Neurocognitive Disorder or Major Neurocognitive Disorder):

A
  • Insight Diminishment: Individuals with dementia, especially as the illness progresses, may deny or underestimate their mental impairment. Insight into the cognitive decline tends to diminish over time.
  • Memory Impairment: Memory impairment, especially in the form of short-term memory loss, is a hallmark feature of dementia. As the disease advances, individuals may struggle with recall, spatial orientation, and recognition of familiar faces or places.
  • Psychotic Symptoms: Hallucinations are relatively rare in dementia, and if present, they are typically not a prominent feature. Delusions, if present, tend to be poorly structured and may have paranoid themes.
  • Appetite: Unlike depression, appetite is often intact in individuals with dementia.
  • Disorientation: Disorientation is a common feature in dementia, and individuals may become lost even in familiar environments. Wandering is a significant problem, posing safety concerns.
  • Emotional Lability and Agitation: Dementia often leads to emotional lability, (easily altered, "التقلب") with individuals experiencing rapid and unpredictable changes in mood. Agitation is a common behavioral manifestation, and it can be challenging for both individuals with dementia and their caregivers.
  • Disinhibition: Individuals with dementia may display disinhibited behaviors, such as disrobing in inappropriate settings, as a result of impaired judgment and social awareness.
  • Variability in Symptoms: The specific symptoms and their severity can vary widely depending on the type of dementia and the extent of the illness. Alzheimer's disease, vascular dementia, Lewy body dementia, and other types of dementia may present with distinct clinical features.
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6
Q

Clinical Considerations of dementia:

A

Clinical Considerations:
- Progressive Nature: Dementia is characterized by a progressive decline in cognitive function, impacting daily life activities. It is not a reversible condition.
- Challenges in Communication: As dementia progresses, communication abilities may decline, making it challenging for individuals to express themselves or understand others.
- Caregiver Burden: Dementia places a significant burden on caregivers due to the complex needs of individuals with the condition. It requires a comprehensive and compassionate approach to care.
- Multidisciplinary Approach: Management of dementia often involves a multidisciplinary team, including physicians, nurses, social workers, and therapists, to address medical, psychological, and social aspects of the condition.

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7
Q

Most common types Neurocognitive disorders:

The term Alzheimer is named after Dr. Alois Alzheimer, a German psychiatrist and neuropathologist.

A
  1. Alzheimer’s Disease:
    - Alzheimer's disease is the most prevalent cause of neurocognitive disorders. It is a progressive brain
    disorder characterized by the accumulation of abnormal protein aggregates, leading to memory loss, cognitive decline, and functional impairment.
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8
Q

Most common types Neurocognitive disorders:

Vascular Dementia

A
  1. Vascular Dementia:
    - Vascular dementia results from impaired blood flow to the brain, often due to strokes or other vascular issues. It is the second most common cause of neurocognitive disorders and can lead to a range of cognitive impairments.
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8
Q

Most common types Neurocognitive disorders:

Lewy Body Dementia

A
  1. Lewy Body Dementia:
    - Lewy body dementia involves the presence of abnormal protein deposits called Lewy bodies in the brain.
    It shares characteristics with both Alzheimer's and Parkinson's disease and is associated with cognitive fluctuations, visual hallucinations, and motor symptoms.
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9
Q

Most common types Neurocognitive disorders:

Frontotemporal dementia

A
  1. Frontotemporal Dementia:
    - Frontotemporal dementia is characterized by damage to the frontal and/or temporal lobes of the brain.
    It often presents with changes in personality, behavior, and language difficulties.
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10
Q

Most common types Neurocognitive disorders:

Parkinson’s Disease

A
  1. Parkinson’s Disease:
    - Parkinson’s disease is primarily a movement disorder, but it can also lead to cognitive decline and
    dementia in later stages. The underlying pathology involves the loss of dopamine-producing neurons.
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11
Q

Most common types Neurocognitive disorders:
Traumatic Brain Injury (TBI)

A
  1. Traumatic Brain Injury (TBI):
  • Neurocognitive disorders can result from traumatic brain injuries, which may occur due to accidents,
    falls, or other forms of head trauma. The severity and type of cognitive impairment can vary depending on the extent of the injury.
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11
Q

Most common types Neurocognitive disorders:

Huntington’s disease

A
  1. Huntington’s Disease:
    - Huntington’s disease is a genetic disorder that leads to progressive degeneration of nerve cells in the
    brain.

It is characterized by motor abnormalities, psychiatric symptoms, and cognitive decline.

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12
Q

Most common types Neurocognitive disorders:

Alcohol and Substance Abuse

A
  1. Alcohol and Substance Abuse:
    - Chronic alcohol and drug abuse can contribute to neurocognitive disorders. Prolonged substance abuse
    can lead to structural and functional brain changes, resulting in cognitive deficits.
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13
Q

Most common types Neurocognitive disorders:

Prion diseases

A
  1. Prion Diseases:
    - Prion diseases are rare neurodegenerative disorders caused by abnormal proteins called prions.
    Examples include Creutzfeldt-Jakob disease (CJD), which leads to rapidly progressive dementia.
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13
Q

Most common types Neurocognitive disorders:

Medication-Induced Neurocognitive Disorders

A
  1. Medication-Induced Neurocognitive Disorders:
    - Certain medications, especially those with anticholinergic properties or those affecting the central
    nervous system, can contribute to cognitive impairment. This includes some antipsychotics, benzodiazepines, and medications for Parkinson's disease.
14
Q

Most common types Neurocognitive disorders:
Infectious Diseases

A
  1. Infectious Diseases:
    - Certain infectious diseases can affect the brain and lead to neurocognitive disorders. Examples include
    HIV-associated neurocognitive disorders (HAND) and neurosyphilis.
15
Q

Let’s delve into the details of Neurocognitive Disorder due to Alzheimer’s Disease (AD) WITH MORE DETAILS

Neuronal Loss: Alzheimer’s disease involves the abnormal accumulation of two proteins in the brain—beta-amyloid plaques and tau tangles. These protein aggregates lead to the degeneration and death of neurons, particularly in regions critical for memory and cognitive function, such as the hippocampus and entorhinal cortex.

A

Characteristics:
- Onset and Progression:
- Alzheimer’s Disease (AD) typically has a slow onset and is characterized by a progressive deterioration
of both mental and physical functions. The entire spectrum of dementia features, including memory impairment, cognitive decline, and changes in behavior, is observed.
- Sundowning:
- Symptoms often worsen in the evening, a phenomenon known as “sundowning.” This can include
increased confusion, agitation, and restlessness during the late afternoon and evening.
- Neuro-imaging and Pathology:
- Neuroimaging studies, such as CT scans and PET scans, often reveal decreased brain volume and nutrient
uptake. Autopsy findings commonly show neurofibrillary tangles and senile plaques, which are abnormal protein deposits in the brain. There’s also a decrease in acetylcholine, a neurotransmitter important for memory and learning. Additionally, there is an increase in the neurotransmitter glutamate, which can contribute to excitotoxicity. "Excessive levels of glutamate causes the death of neurons"
- Prevalence:
- Alzheimer's Disease is the most common cause of dementia, accounting for 60-80% of all cases. If
dementia with multiple causes is considered, the percentage is even higher.
- Incurable and Progressive Nature:
- Alzheimer’s Disease is considered progressive and, thus far, incurable. Medications such as donepezil
(Aricept) and memantine (Namenda) may provide some improvement in mental function, especially in the early stages. However, it’s crucial to note that what is often termed “improvement” is more accurately a decrease in the rate of cognitive decline. These medications do not demonstrate an increase in overall survival.
- Genetic Factors:
- Genetic factors play a role, especially in early-onset Alzheimer’s Disease. The Apolipoprotein E (ApoE)
ε4 allele is the most significant known genetic risk factor. “There’s a specific gene called Apolipoprotein E (ApoE), and it comes in different forms, or “alleles.” One of these forms is called ApoE ε4. Having the ApoE ε4 allele increases the chances of getting Alzheimer’s disease, especially in cases where the disease starts at an earlier age.”
- Other Risk Factors:
- Various risk factors are associated with Alzheimer’s Disease, including head trauma, cardiovascular
disease, social isolation, diabetes, metabolic syndrome, depression, hypothyroidism, and long-term use of alcohol or benzodiazepines.
- Management:
- While there is no cure for Alzheimer’s Disease, management focuses on improving the quality of life,
supporting individuals and their families, and addressing symptoms through medications and non- pharmacological interventions.
- Nursing Home Delay:
- The use of medications such as donepezil (Aricept) and memantine (Namenda) may delay the need for
nursing home placement by about 5 months when used in combination.
Epidemiology of Dementia:
- Current Prevalence:
- Currently, there are approximately 5.7 million people in the United States living with dementia.

  • Projected Estimates by 2050:
  • By 2050, it is estimated that the number of people living with dementia in the U.S. will increase
    significantly to around 14 million.
  • Mortality and Ranking:
    - Dementia is the sixth leading cause of death in the United States. Shockingly, 1 in 3 seniors dies with
    Alzheimer’s or another form of dementia. Notably, dementia is responsible for more deaths than prostate cancer and breast cancer combined.
  • Economic Burden:
  • Dementia care comes with a substantial economic burden. Currently, around $277 billion is spent on
    dementia care each year in the U.S. The projected estimate for 2050 is a staggering $1.1 trillion. It’s important to note that only about 10% of this cost is covered by U.S. government funding, leaving the majority of the financial burden on families.
    Stages of Dementia:
  • Variable Progression:
  • The progression of dementia is variable and can differ among individuals. The stages of dementia can be
    broadly categorized, but the duration of each stage and the overall trajectory vary.
  • Average Duration:
  • On average, from the time of diagnosis to death, the duration of dementia is around 8-10 years. However,
    there is a wide range, with some individuals experiencing a more rapid decline (as short as 3 years), while others may have a longer duration (up to 20 years).
  • Table 23-3:
  • Reference is made to Table 23-3, which likely provides a breakdown of the various stages of dementia
    and associated characteristics. Understanding the stages is crucial for healthcare professionals and caregivers to provide appropriate care and support throughout the course of the illness.
    Implications and Considerations:
  • The increasing prevalence of dementia highlights the urgent need for effective interventions, including both treatment and prevention strategies.
  • The economic burden underscores the importance of public health policies and funding to support research, caregiver resources, and improved care infrastructure.
  • The significant impact on families emphasizes the need for comprehensive and compassionate care, as well as support systems for caregivers.
  • Ongoing research efforts are essential to uncover better diagnostic tools, treatment options, and potential preventive measures to mitigate the growing impact of dementia on individuals and society.
15
Q

Most common types Neurocognitive disorders:
Metabolic and Endocrine Disorders

A
  1. Metabolic and Endocrine Disorders:
    - Disorders such as hypothyroidism, vitamin B12 deficiency, and metabolic conditions can result in
    neurocognitive impairment when not properly managed.
16
Q

Neurocognitive Disorder with Lewy Bodies (DLBD):

IN DEPTH

A
  • Symptoms and Progression:
  • Symptoms of Neurocognitive Disorder with Lewy Bodies (DLBD) are similar to Alzheimer's Disease
    (AD), but the progression is often more rapid. Psychosis, especially visual hallucinations (VH), is common
    and may occur earlier in the disease course. Additionally, individuals may experience Parkinson-like symptoms. Importantly, the use of typical neuroleptics and anti-Parkinson medications can exacerbate symptoms.
  • Lewy Bodies:
  • “Lewy bodies” are eosinophilic inclusion bodies found in the cerebral cortex and the brainstem. Their
    presence is a key pathological feature of DLBD. (Abnormal clumps of protein. The term "eosinophilic" refers to the affinity of these structures for a dye called eosin, commonly used in histological staining.)
  • Relation to Parkinson’s Disease (PD):
  • Parkinson's Dementia (PD) refers to the development of dementia in individuals with Parkinson's disease.
    Lewy bodies are present in different areas of the brain. PD can eventually progress to dementia, affecting 20-60% of individuals with PD.
  • Progressive and Incurable:
  • Similar to other neurocognitive disorders, DLBD is progressive and currently considered incurable.
    Management focuses on alleviating symptoms and improving quality of life.
17
Q

Vascular Neurocognitive Disorder (Multi-infarct Dementia):

A
  • Symptoms and Etiology:
  • Vascular Neurocognitive Disorder, also known as multi-infarct dementia, is likely a result of multiple
    small strokesor transient ischemic attacks. Sudden onset of symptoms is common, and the pattern of cognitive decline is more irregular compared with AD.
  • Prevalence:
  • Vascular Neurocognitive Disorder is generally considered the second most common cause of dementia,
    accounting for about 10-20% of all cases.
  • Irreversible and Progressive:
  • Similar to other forms of dementia, Vascular Neurocognitive Disorder is irreversible and often
    progressive. The cumulative effect of multiple small strokes can result in cognitive decline.
  • Potential for Neural Pathway Compensation:
  • Unlike some other neurocognitive disorders, individuals with vascular dementia may sometimes regain
    certain functions through other neural pathways. However, this compensation is often limited.
  • Cascade of CVAs:
  • A "cascade" of cardiovascular accidents (CVAs or strokes) can exacerbate symptoms and make necessary
    life changes more challenging for individuals with vascular dementia. (it implies a series of cardiovascular accidents "CVAs or strokes" occurring over time)
18
Q

Neurocognitive Disorder Due to HIV Infection (HIV/AIDS Dementia):

A
  • Clinical Presentation:
  • Differentiating features for Neurocognitive Disorder due to HIV Infection include the individual being
    HIV positive, often for an extended period, and the fact that affected individuals are generally much younger than those with other types of dementia.
  • Preventive Measures:
  • Highly Active Antiretroviral Therapy (HAART) medications play a crucial role in preventing and managing HIV/AIDS dementia. Additionally, treatment of opportunistic infections that affect the brain can help treat or even reverse dementia symptoms in individuals with HIV.
19
Q

Diagnosis of Neurocognitive Disorder:

A
  • Mini Mental Status Exam (MMSE):
  • The MMSE is a widely used screening tool for assessing cognitive function. It is not the same as the full
    Mental Status Exam (MSE) but specifically focuses on neurocognitive function.
  • Clinical Assessment:
  • In addition to standardized tools like the MMSE, a comprehensive assessment involves exploring the
    onset of symptoms, the patient’s medical history (PMH), psychosocial assessment (PSA), and other relevant factors.
20
Q

General Nursing Care for Clients with Neurocognitive Disorder:

A
  • Communication:
  • Stay calm and keep communication simple. Focus on one thing at a time. Encourage reminiscence (recollection) about
    the past, as long-term memory is often relatively intact even in severe short-term memory impairment.
  • Dealing with Delusions/Confusion:
  • For delusions or confusion leading to agitation, validate the patient’s feelings and gently redirect or
    distract them to something else. For moderate to severe dementia, avoid insisting on correcting the patient or providing reality orientation, as it can worsen agitation.
  • Wandering and Environmental Modifications:
  • Redirect patients who wander and use large signs with pictures to assist those in the end-stage of
    dementia.
  • Promoting Independence in ADLs:
  • Encourage independence in activities of daily living (ADLs) while providing assistance as needed. Allow
    ample time for ADLs and try again later if the patient becomes agitated.
  • Consistency and Routine:
  • Maintain consistency in staff and caregivers, as well as a daily routine, to provide a structured and
    familiar environment for individuals with neurocognitive disorders.
  • Support for Caregivers:
  • Recognize the importance of supporting family members and caregivers. Caregivers often need respite
    and support, and acknowledging their feelings of despair over losing a loved one to dementia is essential.
  • Grieving Process:
  • Acknowledge the grieving process for both individuals with neurocognitive disorders and their families.
    Educate caregivers about the progressive nature of dementia and the need for ongoing support.
21
Q

Grief and Loss:
Stages of Grief - Kubler-Ross:

Swiss-American psychiatrist, a pioneer in near-death studies

A

Elisabeth Kübler-Ross proposed a model of grief that includes the following stages. It’s important to note that these stages are not necessarily experienced in a linear fashion, and individuals may move back and forth between them.

  1. Denial:
    - This stage involves a refusal to accept the reality of the situation. It’s a defense mechanism that helps
    individuals cope with the shock of the loss.
  2. Anger:
    - Anger may be directed at various targets, including God, the universe, or even innocent people. It's an
    natural emotional response to the perceived injustice of the loss.
  3. Bargaining:
    - In this stage, individuals may attempt to make deals or bargains, often with a higher power, in an effort
    to reverse or mitigate the loss. This can involve promises or commitments.
  4. Depression:
    - Depression in the grieving process is characterized by feelings of deep sadness, hopelessness, and a
    sense of emptiness. It represents a more profound acknowledgment of the reality of the loss.
  5. Acceptance:
    - Acceptance is the final stage where individuals come to terms with the reality of the loss. It does not
    necessarily mean happiness or joy but represents a state of emotional resolution and moving forward.
22
Q

Acute Grieving:

A

Normal/Healthy Grieving:
- In the acute stage of grief, which typically lasts 6-8 weeks, individuals go through a range of emotions
as part of a normal and healthy grieving process. However, complete resolution of the grief response may take years.

23
Q
  • Dysfunctional/Maladaptive Grieving:
A
  • Failure to grieve or unresolved grief can lead to dysfunctional grieving later. Various factors can
    contribute to maladaptive grieving, including:
  • Guilt over unresolved conflicts or negative feelings with the dying or deceased significant other. - Heavy emotional dependence on the deceased.
  • Young age of the deceased, challenging societal expectations.
  • Lack of social support.
  • Death from a social stigma, such as AIDS, suicide, or illegal substance use.
  • Unexpected death, such as in a motor vehicle accident, murder, or suicide.
  • A history of mental illness, including substance abuse.
24
Q

Clinical Considerations:

A
  • Individual Variability:
  • It’s important to recognize that individuals may experience grief differently, and the duration and intensity
    of grief can vary widely.
  • Supportive Interventions:
  • Healthcare professionals play a crucial role in providing support and guidance during the grieving process. Encouraging individuals to express their emotions, providing resources for grief support, and being empathetic are essential components of care.
  • Long-Term Implications:
    - Unresolved grief can have long-term implications for mental health and well-being. Identifying signs of
    maladaptive grieving and offering appropriate interventions are integral to comprehensive care.
25
Q

Nursing Care for the Grieving:

A

Providing effective nursing care for individuals experiencing grief, including those facing end-of-life situations and their significant others, requires a compassionate and supportive approach. Here are key principles for nursing care in these situations:
1. Anticipating Needs:
- Anticipate the needs of clients, especially those with cognitive disorders, and provide proactive care.
This includes addressing physical, emotional, and psychosocial needs, as well as ensuring comfort and dignity in end-of-life care (palliative care).
2. Practice the Art of Presence:
- The simple act of being present is powerful in supporting someone dealing with grief. Just being there
physically, even in silence, communicates a sense of companionship and support. Avoid trying to “fix” the situation but instead, reflect the person’s feelings to convey understanding.
3. Advocacy:
- Assertively advocate for your clients and their significant others. Nurses often have knowledge of the
healthcare system and can play a vital role in ensuring that the needs and preferences of the grieving individuals are respected and met.
4. Self-Reflection and Support:
- Regularly check in with yourself about your own feelings. Providing care in end-of-life and grief
situations can be emotionally challenging for healthcare professionals. Seek support, debrief with colleagues, and take advantage of available resources for your own well-being.
5. Reflecting on Life and Meaning:
- At the end of life, individuals often desire to reflect on their experiences and find meaning in their lives.
It’s essential to create a supportive environment for clients to express their thoughts, beliefs, and concerns. A non-judgmental and empathetic approach allows individuals to share their experiences and feelings about the meaning of life, even if they differ from your own beliefs.

**Remember to review “Care of the Older Adult” (CH 30)**
This material is indirectly addressed in many lectures, but will not be covered directly in class. Use your study guide to help you focus when studying for Exam 3.
26
Q

Medications such as donepezil
(Aricept) and memantine (Namenda) . Provide some improvement in mental function, especially in the early stages.

A

Alzheimer’s

Provide some improvement in mental function, especially in the early stages.

However, it’s crucial to note that what is often termed “improvement” is more accurately a decrease in the rate of cognitive decline.

These medications do not demonstrate an increase in overall survival.

27
Q

Infarct” is a term used in medicine to describe an area of tissue that has died or become necrotic due to a lack of blood supply (O2), typically caused by a blockage or obstruction of the blood vessels.

A