Neuro part 1 Test 7 SG- DONE Flashcards

1
Q

1.How would you assess the motor response of an unconscious client? (pg.458)

A

-administering a painful stimulus to determine the client’s response.

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2
Q

2.What is Decorticate Posturing (pg.458):

A

the arms are flexed, fists are clenched, and the legs are extended

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3
Q

3.Decerebrate Posturing (pg. 458, ATI pg.17):

A

Extension of elbows & wrists, abduction of arms
Decerebrate rigidity = extremities are stiff rigid

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4
Q
  1. What is Flaccid (pg.458):
A

no motor response to stimuli

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5
Q

5.What is a Glasgow Coma Scale? (pg.459, ATI pg.17)

A

Glasgow Coma Scale: is an objective assessment tool for evaluating LOC of a client. The GCS # allows providers to immediately determine if neurologic changes have occurred.

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6
Q

6.What does the GCS scale consist of? (pp slide 3)

A

Eye- Opening response: 4 spontaneous, 3 to voice, 2 to pain, 1 none
Verbal response: 5 oriented x time, person, place ;4 confused, 3 inappropriate words, 2 incomprehensive sounds, 1 none
Motor response: 6 obeys commands, 5 moves to localized pain, 4 flex to withdraw from pain, 3 abnormal flexion, 2 abnormal extension, 1 none

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7
Q

7.At what level is the client considered to be in a coma? (pg.459, ATI pg.17)

A

Score of 8 or less.

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8
Q

8.How do you check for neck rigidity? (pg.460)

A

Move the head and chin towards the chest.

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9
Q

9.Nuchal rigidity:

A

pain & stiffness, resistance when you move the neck towards the chest or inability to place the chin on the chest

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10
Q
  1. Brudzinski? (pg.469)
A

Flexion of the neck towards the chin if the knees and hips flex up its positive.

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11
Q
  1. Kernig? (pg.469)
A

Inability to extend the leg when the thigh is flexed on the abdomen (severe stiffness in hamstring)

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12
Q

12.Manifestations of increased intracranial pressure? (PP slide 5)

A

-decreasing LOC (early manifestation) *LOC is the earliest indicator of neurologic status
-changes in pupils
-headache (more severe in the morning
-vomiting, papilledema, decorticate or decerebrate posturing
-stuporous, semi-comatose: confusion, restlessness periodic disorientation
-Cushing’s triad: pulse rate that increases initially but then decreases & respiratory rate that is irregular, Pulse pressure (difference between systolic & diastolic) 40-60 difference is normal.
-Cheyenne-Stokes respirations

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13
Q

13.Nursing considerations for intracranial pressure (pp slide 6)

A

-maintain head in midline,30-degree elevation
-maintain BP & ensure cerebral perfusion/fluids as ordered (AVOID hypotonic solutions)
-maintain airway (monitor o2 levels)
-monitor neurologic status (change in LOC)
-seizure precautions
-decrease stimuli (may need pt to be sedated)
-indwelling catheter may be placed to monitor I & O’s
-stool softener (important to avoid straining)
-avoid hypothermia

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14
Q

14.What is Cushing’s Triad? (PP slide 5)

A

pulse rate that increases initially but then decreases(bradycardia) & respiratory rate that is irregular, Pulse pressure (difference between systolic & diastolic) 40-60 difference is normal.

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15
Q

15.What is meningitis and what is it caused by? (PP slide 7)

A

Infection of the meninges which surround & protect the brain & spinal cord caused by a virus, bacteria/fungi, or parasite.

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16
Q

16.Meningitis manifestations (pp slide 8)

A

-high fever
-positive Brudzinski’s sign
-excruciating headache
-nuchal rigidity

17
Q

17.Phenytoin need to knows (pp slide 28, pg.485)

A

(seizures)
-do not abruptly stop medication
-can cause gingival hyperplasia (overgrowth of the gums, puffy & red), good dental hygiene is important
-can be toxic to the liver, monitor LFT
-check phenytoin levels (narrow therapeutic range 10-20)
-monitor calcium (can cause low calcium)
-can cause thrombocytopenia (bruising, nose bleeds, flu like symptoms) >notify PCP

-skin rash > Steven Johnson syndrome REPORT
-no oral contraceptives
-hypotension (low & slow vitals)
-suicidal thoughts

18
Q

18.Levodopa need to know(pp slide 23, flashcard, pg.480)

A

(parkinsons)
**-decrease protein (can interfere w/med) **

-do not stop abruptly
-monitor LFTS
-don’t take w/MAOI’s (risk for hypertensive crisis)
-can cause dark color in saliva, urine or sweat
-change positions slowly
-monitor for behavioral & mood changes

19
Q

19.Seizure nursing actions (PP slide 28)

A

-keep safe from injury (pillow under head)
-position client on their side, loosen restrictive clothing, keep airway patent, suction client, administer oxygen
-document of the situation that proceeded the seizure, to assist in identifying precipitating factors like an aura, duration of the seizure, parts of the body involved.

20
Q

20.Seizure classifications (PP slide 27)

A

Absence seizure: stares blankly, eyelids flutter, lack of prominent movements
Myoclonic seizure: sudden brief jerking
Tonic-Clonic seizure: muscle alternate between contraction & relaxation, jerking movements

21
Q

21.Post seizure nursing actions (slide 29, ATI pg.34)

A

Ask the patient if they know what happened?
-Ask what type of body movements? Automatisms (repetitive non purposeful actions/movements)
-did they lose consciousness?
-how long was the seizure?
-was there an aura?
-monitor postictal phase (period after the seizure, very often will be very sleepy)
-check vitals, check for injuries, perform neurological check, reorient client

22
Q

22.Parkinson’s manifestations (PP slide 22)

A

assess for progression of disease
(For progressive diseases we try to keep client as independent as possible.)
*Parkinson medications can decrease symptoms drastically at first, but overtime effects will decrease.
*Metoclopramide given for Gerd can give extrapyramidal effects
-muscle rigidity
-tremors: pill rolling
-bradykinesia (slow movements including slow speech, masklike expression & decreased blinking)
-stooped posture
-hypophonia (low volume of speech) & slow speech
-shuffling gait: difficulty redirecting forward motion
-dysphagia

23
Q

23.Guillan-Barre syndrome nursing considerations (pp slide 12)

A

-assess signs of respiratory distress
-monitor airway
-spirometer
-have emergency suction near bedside
-skin care
-change position every 2 hrs
-ROM exercises to prevent muscle atrophy

24
Q

24.Guillan-Barre syndrome manifestations (pp slide 11)

A

**disease is ascending **
-difficulty chewing, talking, swallowing
-tingling in the arms & legs
-incontinence
-weakness that progresses
-paralysis

25
Q

25.ALS manifestations (PP slide 18)

A

-wasting of the arms, legs, and trunk
-develop atrophy (experiences episodes of muscle fasciculations -twitching)
-progressive muscle weakness
-if ALS affects the brainstem > difficulty speaking & swallowing, periods of inappropriate laughter & crying, respiratory failure & total paralysis

26
Q

26.Encephalitis manifestations and diagnosis (pp slide 10, Pg.470)

A

ASK IF THEYVE HAD ANY RECENT BUG BITES
- Sudden fever
- Severe headache
- Stiff neck
- Seizures
- Spastic or flaccid paralysis
- Tremors
- Muscle weakness
- Incontinence
- Lethargy
- Irritability
- Delirium
- Visual disturbances
- Vomiting
- Drowsiness
- Coma
Dx: lumbar puncture, EEG.

27
Q

27.Baclofen need to knows(pp slide 17, pharm pg.360)

A

(MS)
-decrease & minimize the muscles spasticity & rigidity (for M.S)

-causes drowsiness, dizziness, nausea, weakness
-hypotension

28
Q

28.Brain tumor manifestations, focal (PP slide 30)

A

-headache (most common in the AM, becoming increasingly severe & occurs more frequenltly as the tumor grows)
-vomiting occurs w/out nausea or warning
-papilledema
-double vision and other sensory loss
-speech difficulty
-seizures
-muscle weakness.
-paralysis
(Focal= focused on certain part of the brain)
(Generalized= over general area> vomiting)

29
Q

29.Brain tumor priority (PP slide 31)

A

-prevent increased ICP

30
Q

30.Huntington’s Disease manifestations (PP slide 24)

A

-choreiform (jerking, riving -exaggerated movements) movements
-intellectual decline
-difficulty chewing & swallowing
-grimacing
-speech difficulty
-severe depression (can lead to suicide)
-mental apathy & emotional disturbances
-loss of bowel & bladder control

31
Q

31.Valproic acid need to knows (pp slide 24, pg.484)

A

(seizures)

-can cause thrombocytopenia (watch 4 bruising, nose bleeds, flu like symptoms) >notify PCP
-monitor LFT’s
-do not abruptly stop medication
-monitor calcium levels

  • avoid alcohol use
    -monitor platelets & bleeding time, ammonia levels
32
Q

32.Amantadine need to knows (pp slide 23, ATI pg.40, pg.480)

A

(Parkinson’s)
-can cause insomnia & lightheadedness

-monitor for discoloration of the skin that subsides when amantadine is discontinued
-may cause anxiety, confusion, anticholinergic effects (dry mouth, constipation).
-can cause hypotension (slow position changes)
-do not stop abruptly
-do not mix w/MOI’S

33
Q
  1. Selegiline need to knows (pp slide 23)
A

(Parkinson’s)
- avoid foods high in tyramine

-massive HTN crisis risk (massive headache)
-major risk for stroke

-begin diet at least 2 weeks before beginning treatment & 2 weeks after treatment.
-avoid OTC drugs (calcium, antacids, Tylenol, NSAIDS naproxen/ibuprofen)
-increase suicide risk
-2 wk wash out period (no other antidepressants)