NEURO - Effects of Stroke Flashcards
Middle cerebral artery
MCA
Motor and sensory cortices—upper limb and face.
-Contralateral paralysis and sensory loss—face and upper limb.
Temporal lobe (Wernicke area); frontal lobe (Broca area). -Aphasia if in dominant (usually left) hemisphere. Hemineglect if lesion affects nondominant (usually right) side.
Anterior cerebral artery
ACA
Motor and sensory cortices—lower limb.
-Contralateral paralysis and sensory loss—lower limb.
Lenticulostriate artery
Striatum, internal capsule -Contralateral paralysis and/or sensory loss—face and body. Absence of cortical signs (eg, neglect, aphasia, visual field loss).
Common location of lacunar
infarcts, 2° to unmanaged
hypertension.
Anterior spinal artery
ASA
Lateral corticospinal tract.
-Contralateral paralysis—upper and lower limbs.
Medial lemniscus.
- DEC contralateral proprioception.
Caudal medulla—hypoglossal nerve.
-Ipsilateral hypoglossal dysfunction (tongue deviates ipsilaterally).
*Medial medullary syndrome—
caused by infarct of paramedian branches of ASA
and/or vertebral arteries.
Posterior inferior cerebellar
artery (PICA)
Lateral medulla
-Vomiting, vertigo, nystagmus;
pain and temperature sensation from ipsilateral face and contralateral body; *dysphagia, *hoarseness, gag reflex; ipsilateral Horner syndrome; ataxia, dysmetria.
*Lateral medullary (Wallenberg)
syndrome
*Nucleus ambiguus effects are
specific to PICA lesions.
“Don’t pick a (PICA) horse
(hoarseness) that can’t eat
(dysphagia) .”
Anterior inferior cerebellar
artery (AICA)
Lateral pons
-Vomiting, vertigo, nystagmus.
Paralysis of face, lacrimation,
salivation, taste from anterior 2⁄3 of tongue.
-Ipsilateral pain and temperature of the face, contralateral pain and temperature of the body.
Middle and inferior cerebellar
peduncles
-Ataxia, dysmetria
*Lateral pontine syndrome
*Facial nucleus effects are
specific to AICA lesions.
“Facial droop means AICA’s
pooped.”
Basilar artery
Pons, medulla, lower midbrain,
corticospinal and corticobulbar
tracts, ocular cranial nerve nuclei, paramedian pontine reticular formation.
-Preserved consciousness, vertical eye movement, blinking; quadriplegia, loss of voluntary facial, mouth, and tongue movements.
*“Locked-in syndrome.”
Posterior cerebral artery
PCA
Occipital cortex, visual cortex
-*Contralateral hemianopia with
macular sparing.
Anterior communicating artery (Acom)
Compression may cause bitemporal hemianopia; visual acuity deficits.
Rupture (aneurysms) may cause ischemia in ACA distribution–>contralateral lower extremity hemiparesis, sensory deficits.
Posterior communicating artery (Pcom)
Compression may cause ipsilateral CN III palsy–>mydriasis (“blown pupil”); may also see ptosis, “down and out” eye.