Neuro Flashcards
optic canal
optic nn
superior orbital fissue
III
IV
V1
V1
foramen rotundum
V2
foramen ovale
V3
foramen spinosum
middle menigeal a
foramen lacerum
internal carotid, does not go all the way thru, exits via carotid canal
internal auditory meatus
VII
VIII
jugular foramen
IX
X
XI
sigmoid sinus to inferior jugular v
hypoglossal canal
XII
what are some CIs to OMT
battle sign- basilar skull fracture
posterior fossa tumor -> herniation risk
types of cerebral palsy
spastic/pyramidal non-spastic/extrapyramidal atonic cerebellar ataxic combined
spastic/pyramidal type
most common
UMN damage
stiff rigid limbs
arms, legs, can involve tongue,, mouth, larynx, speech, eating, breathing, swallowing
associated w/hip pathology, scoliosis, limb deformities
non-spastic/extrapyramidal
2 types: ataxic and dyskinetic
decreased or fluctuating mm tone, hypotonica
impairments in involuntary movement, dyskinesias, dystonias, athetosis
mental impairment, limb deformities and seizures less likely
Dx of CP
Dx of exclusion
persistent infantile reflexes, especially moro
earliest signs of spasticity usually involve resistance to passive motion
not Dx before age 2
workup for CP
labs
imaging
clinical surveillance
joint proprioception with spasticity
postural control abnormal in kids w/spastic d/t biomechanical not neurologic factors
spastic CP and baseline tone
daily mm challenge makes mm tighter then baseline
biomechanics in spastic CP
impaired down-regulation of descending pathway of myotactic reflex
causes hypertonicity and spasticity
uncoordinated mvmt
agonists/antagonists don’t work together
each mvmt can activate reflex inhibiting mvmt
postural compenstation
related to specific mm groups
LE most common
hamstring hypertonicity
causes post innominatne decreased lumbar lordosis extended TLJ flattened thoracic kyphosis extended OA
altered hip mechanics
excessive force thru acetabulum -> increased risk of hip dislocation, fracture, or avascular necrosis
always evaluate new onset pain or changes in fnx
non-spastic CP presentation
generalized hypotonia and increased DTRs in infancy progresses to dyskinesias in childhood
hypotonic
non-spastic CP presentation
generalized hypotonia and increased DTRs in infancy progresses to dyskinesias in childhood
hypotonic and altered mvmt patterns affect proprioception input
baseline tone lower then normal
postural mechanisms impaired
other symptoms of non-spastic CP
sensorineural hearing loss
strabismus
nystagmus