Neuro Flashcards
Role of APRN in HA and face pain
accurately dx rule out secondary causes recognize red flags provide acute management assist with HA prevention
HA
most common pain problem seen in family practice
Primary HA
Benign
Recurrent
NOT associated with underlying patho
The HA is the disease
Types of primary HA
Migraine with or without aura
Tention type HA
Cluster HA
Secondary HA
sudden
progressive
associated with pathology
may require immediate action
Patho of Secondary HA
Aneurysms Subarachnoid hemorrhage Thunder clap HA Meningitis Stroke Carotodynia Temporal Arteritis HTN Sinus TMJ Trigeminal Neuralgia
Worrisone Red Flags of HA “SNOOP”
S: systemic symptoms or diease N: neurological signs/symptoms O: onset sudden O: onset before 5 or after 50 P: patterns change from prior HA
Characteristics of migraine
unilateral moderate/severe intensity lasts 4-72 hours throbbing quality associated symptoms females
Characteristics of tension-type HA
bilateral mild/mod intensity lasts 30 min to 7 days pressure/tightening quality no associated symptoms females
Characteristics of cluster HA
strictly unilateral severe intensity last 15-90 minutes severe quality associated symptoms males
To Dx migraine with an aura
must have 2 attacks with the following criteria:
fully reversible visual, sensory, speech deficits
homonymous visual symptoms
aura developing over 5 mintues
each symptom lasts 5-60 minutes
not attributed to another disorder
To Dx migraine without aura
must have 5 attacks with the following: HA lasting 4-72 hours unilateral, pulsating, mod pain, aggravation by physical activity N/V Photophobia
Tension type HA Criteria
At least 10 episodes occuring < 1 day/month with: HA lasting 30min-7days Bilateral pressing/tightening quality not aggravated by physical activity No N/V only one of photophobia or phonophobia
Cluster HA Criteria
Deep pain around eyes or temporal NO throbbing Nightly occurrence 6-12 wks at a time can have facial sweating, eyelid edema, conjunctival injection, ptosis
Chronic daily HA in children
HA persists >2 h and occurs > 15 days/month
Increased intracranial pressure in children
chronic or intermittent increasing frequency progressive severity occipital neuro signs (papilledema)
Facial pain
most adults female unilateral or bilateral paroxysmal remissions or constant may be triggered by slight touch, wind, speaking
Reasons for facial pain
trigeminal neuralgia postherpetic neuralgia TMJ dental pain sinusitis glaucoma angina pectoris
Tx for Migraine
NSAIDS Triptans, Ergotamine, Dihydroergotamine rescue medication prophylaxis with amitriptyline, propranolol, timolol avoid hormone fluctuations foods alcohol environmental changes stress, lack of sleep
Tx Tension type HA
NSAIDS
don’t provide anything addicting
Tx Cluster HA acute attack
100% O2
SL ergotamine
itranasal lidocaine can be helpful
Tx Cluster HA preventive tx
verapamil 80mg QID (cardiac monitor) Lithium 300-900 mg Prednisone taper Ergotamine 2mg 2 hours before bedtime Divalproex
Refer HA and facial pain
uncertainty
tx failsure
unremitting HA
medication overuse or chronic daily HA
Episodic loss of consciousness
Seizure: temporary neuro signs from abnormal paroxysmal, hypersynchronous electrical activity in cerebral cortex
Syncope: due to a reduced supply of blood to cerebral hemispheres (vasovagal)
Symptoms present in both syncope and seizures
LOC or loss of awareness
with or without myoclonus opisthotonus
myoclonus
incontinence may occur
Events during a spell
tonic-clonic LOC with tonic stiffening then clonic jerking
hypoperfusion produces flaccid unresponsiveness, stiffening or jerking
LOC from hypoperfusion rarely lasts more than 15 second
Posture when LOC occurs
orthostatic hypotension and faints occur in the upright or sitting position
only occur in lying position suggest seizure or cardiac arrhythmia unless phelbotomy
Physical exertion with syncope
usually due to cardiac outflow obstruction
Postictal state
brief confusion, disorientation or agitation